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1. Full Text Sympathetic Nervous System Activation in Human Heart Failure. Clinical Implications of an Updated Model
by Floras, John S
Journal of the American College of Cardiology, ISSN 0735-1097, 07/2009, Volume 54, Issue 5, pp. 375 - 385
Disturbances in cardiovascular neural regulation, influencing both disease course and survival, progress as heart failure worsens. Heart failure due to left... 
norepinephrine | heart failure | baroreceptor reflex | sympathetic nervous system | exercise | human | sleep apnea | OBSTRUCTIVE SLEEP-APNEA | CARDIAC & CARDIOVASCULAR SYSTEMS | RECEPTOR BLOCKADE | IDIOPATHIC DILATED CARDIOMYOPATHY | ATRIAL-NATRIURETIC-PEPTIDE | PEAK OXYGEN-UPTAKE | BAROREFLEX SENSITIVITY | FAILING HUMAN HEART | AFFERENT REFLEX | POSITIVE AIRWAY PRESSURE | NORMAL VENTRICULAR-FUNCTION | Heart Rate | Norepinephrine - blood | Sleep Apnea Syndromes - physiopathology | Baroreflex | Humans | Heart Failure - physiopathology | Sympathetic Nervous System - physiopathology | Systole | Ventricular Dysfunction, Left - physiopathology | Blood pressure | Heart attacks | Drug therapy | Rodents | Reflexes
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2. Full Text Sympathetic nervous activation in obesity and the metabolic syndrome—Causes, consequences and therapeutic implications
by Lambert, Elisabeth A and Lambert, Gavin W and Straznicky, Nora E and Dixon, John B and Schlaich, Markus P
Pharmacology and Therapeutics, ISSN 0163-7258, 2010, Volume 126, Issue 2, pp. 159 - 172
The world wide prevalence of obesity and the metabolic syndrome is escalating. Contrary to earlier experimental evidence, human obesity is characterised by... 
Target organ damage | Microneurography | Metabolic syndrome | Sympathetic nervous system | Overweight | OBSTRUCTIVE SLEEP-APNEA | BLOOD-PRESSURE ELEVATION | RANDOMIZED CONTROLLED-TRIAL | BODY-MASS-INDEX | CONVERTING ENZYME-INHIBITION | NUTRITION EXAMINATION SURVEY | PHARMACOLOGY & PHARMACY | POSITIVE AIRWAY PRESSURE | LEFT-VENTRICULAR MASS | CHRONIC-RENAL-FAILURE | CARDIOVASCULAR RISK-FACTORS | Metabolic Syndrome - etiology | Body Weight | Obesity - complications | Sympathetic Nervous System - drug effects | Humans | Metabolic Syndrome - metabolism | Metabolic Syndrome - therapy | Obesity - physiopathology | Adipokines - metabolism | Sympathetic Nervous System - physiopathology | Obesity - genetics | Sympathetic Nervous System - metabolism | Exercise Therapy | Metabolic Syndrome - complications | Obesity - metabolism | Animals | Receptors, Adrenergic, beta - genetics | Metabolic Syndrome - genetics | Adipokines - genetics | Metabolic Syndrome - physiopathology | Obesity - therapy | Obesity - etiology | Diet Therapy | Receptors, Adrenergic, beta - metabolism
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3. Full Text Renal Denervation Reduces Monocyte Activation and Monocyte–Platelet Aggregate Formation: An Anti-Inflammatory Effect Relevant for Cardiovascular Risk
by Zaldivia, Maria T.K and Rivera, Jennifer and Hering, Dagmara and Marusic, Petra and Sata, Yusuke and Lim, Bock and Eikelis, Nina and Lee, Rebecca and Lambert, Gavin W and Esler, Murray D and Htun, Nay M and Duval, Jacqueline and Hammond, Louise and Eisenhardt, Steffen U and Flierl, Ulrike and Schlaich, Markus P and Peter, Karlheinz
Hypertension, ISSN 0194-911X, 02/2017, Volume 69, Issue 2, pp. 323 - 331
Overactivation of renal sympathetic nervous system and low-grade systemic inflammation are common features of hypertension. Renal denervation (RDN) reduces... 
monocyte activation | renal denervation | inflammation | hypertension | sympathetic nervous system | CELLS | SUBSETS | ENDOTHELIAL FUNCTION | BLOOD-PRESSURE | TRIAL | PERIPHERAL VASCULAR DISEASE | MICE | TREATMENT-RESISTANT HYPERTENSION | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Catheter Ablation | Male | Monocytes - metabolism | Platelet Aggregation - physiology | Hypertension - surgery | Hypertension - blood | Young Adult | Flow Cytometry | Time Factors | Sympathectomy - methods | Adolescent | Aged, 80 and over | Adult | Female | Kidney - innervation | Aged | Blood Pressure - physiology | Blood Pressure Monitoring, Ambulatory
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4. Full Text Adrenergic receptor-mediated activation of FGF-21-adiponectin axis exerts atheroprotective effects in brown adipose tissue-transplanted apoE−/− mice
by Kikai, Masakazu and Yamada, Hiroyuki and Wakana, Noriyuki and Terada, Kensuke and Yamamoto, Keita and Wada, Naotoshi and Motoyama, Shinichiro and Saburi, Makoto and Sugimoto, Takeshi and Irie, Daisuke and Kato, Taku and Kawahito, Hiroyuki and Ogata, Takehiro and Matoba, Satoaki
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 03/2018, Volume 497, Issue 4, pp. 1097 - 1103
Brown adipose tissue (BAT) has been found as an endocrine organ that maintains metabolic homeostasis; however, the effects on atherosclerosis remain undefined.... 
Fibroblast growth factor-21 | Brown adipose tissue | Sympathetic nervous system | Atherosclerosis | Adiponectin | GAMMA | FIBROBLAST-GROWTH-FACTOR-21 | BIOCHEMISTRY & MOLECULAR BIOLOGY | INSULIN SENSITIVITY | GLUCOSE-HOMEOSTASIS | ENERGY-EXPENDITURE | BIOPHYSICS | METABOLISM | GROWTH | FAT | Adipose Tissue, Brown - transplantation | Fibroblast Growth Factors - metabolism | Animals | Apolipoproteins E - deficiency | Atherosclerosis - therapy | Apolipoproteins E - genetics | Adiponectin - metabolism | Mice | Receptors, Adrenergic - physiology | Atherosclerosis - prevention & control | Mice, Knockout
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5. Full Text Sustained Reduction of Blood Pressure With Baroreceptor Activation Therapy Results of the 6-Year Open Follow-Up
by de Leeuw, Peter W and Bisognano, John D and Bakris, George L and Nadim, Mitra K and Haller, Hermann and Kroon, Abraham A and DEBuT- HT Rheos Trial Investig and DEBuT-HT and Rheos Trial Investigators
Hypertension, ISSN 0194-911X, 05/2017, Volume 69, Issue 5, pp. 836 - 843
Baroreflex activation therapy is a novel technique for treating patients with resistant hypertension. Although short-term studies have demonstrated that it... 
blood pressure | heart failure | pressoreceptors | SURGICAL TECHNIQUE | baroreflex | SYMPATHETIC-NERVOUS-SYSTEM | MULTICENTER FEASIBILITY | RESISTANT HYPERTENSION | SAFETY | HEART-FAILURE | RENAL DENERVATION | TRIAL | BAROREFLEX FUNCTION | hypertension | PERIPHERAL VASCULAR DISEASE | Antihypertensive Agents - pharmacology | Double-Blind Method | Follow-Up Studies | Pressoreceptors - physiopathology | Humans | Middle Aged | Hypertension - drug therapy | Male | Treatment Outcome | Combined Modality Therapy | Antihypertensive Agents - therapeutic use | Hypertension - physiopathology | Heart Rate - drug effects | Electric Stimulation Therapy - methods | Heart Rate - physiology | Adult | Female | Aged | Baroreflex - physiology | Blood Pressure - drug effects | Blood Pressure - physiology | Hypertension - therapy | Blood Pressure Determination
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6. Full Text Sympathetic activation in chronic renal failure
by Schlaich, Markus P and Socratous, Flora and Hennebry, Sarah and Eikelis, Nina and Lambert, Elisabeth A and Straznicky, Nora and Esler, Murray D and Lambert, Gavin W
Journal of the American Society of Nephrology, ISSN 1046-6673, 05/2009, Volume 20, Issue 5, pp. 933 - 939
The potential involvement of sympathetic overactivity has been neglected in this population despite accumulating experimental and clinical evidence suggesting... 
NEURAL-CONTROL | RENOVASCULAR HYPERTENSION | SPONTANEOUSLY HYPERTENSIVE-RATS | AFFERENT DENERVATION PREVENTS | POLYCYSTIC KIDNEY-DISEASE | NITRIC-OXIDE SYNTHESIS | UROLOGY & NEPHROLOGY | PLASMA-CATECHOLAMINES | LEFT-VENTRICULAR HYPERTROPHY | BLOOD-PRESSURE | AUTONOMIC NERVOUS-SYSTEM | Kidney Failure, Chronic - physiopathology | Norepinephrine - therapeutic use | Monoamine Oxidase - secretion | Humans | Hypertension - drug therapy | Nitric Oxide - physiology | Catecholamines - blood | Clonidine - therapeutic use | Sympathetic Nervous System - physiopathology | Efferent Pathways - physiopathology | Monoamine Oxidase - deficiency | Animals | Nerve Fibers - physiology | Kidney Failure, Chronic - epidemiology | Kidney Failure, Chronic - complications | Cardiovascular Diseases - epidemiology | Cardiovascular Diseases - mortality | Debrisoquin - therapeutic use | Kidney - innervation | Hypertension - mortality | Hypertension - epidemiology | Disease Models, Animal
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7. Full Text Chronic lowering of blood pressure by carotid baroreflex activation: Mechanisms and potential for hypertension therapy
by Lohmeier, Thomas E and Iliescu, Radu
Hypertension, ISSN 0194-911X, 05/2011, Volume 57, Issue 5, pp. 880 - 886
Recent technical advances have renewed interest in device-based therapy for the treatment of drug-resistant hypertension. Findings from recent clinical trials... 
SYMPATHETIC ACTIVATION | RESISTANT HYPERTENSION | ARTERIAL-PRESSURE | NERVES | INCREASES | DENERVATION | PERIPHERAL VASCULAR DISEASE | COMMITTEE | PROLONGED ACTIVATION | BARORECEPTOR STIMULATION | Humans | Pressoreceptors - physiology | Baroreflex - physiology | Blood Pressure - physiology | Carotid Sinus - physiopathology | Hypertension - physiopathology | Kidney - physiopathology
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8. Full Text Sympathetic Nerve Traffic Activation in Essential Hypertension and Its Correlates: Systematic Reviews and Meta-Analyses
by Grassi, Guido and Pisano, Anna and Bolignano, Davide and Seravalle, Gino and D’Arrigo, Graziella and Quarti-Trevano, Fosca and Mallamaci, Francesca and Zoccali, Carmine and Mancia, Giuseppe
Hypertension, ISSN 0194-911X, 06/2018, Volume 72, Issue 2, pp. 483 - 491
Muscle sympathetic nerve activity (MSNA) has shown that sympathetic activation may occur in essential hypertension (EHT). However, the small sample size of the... 
Heart rate | Essential hypertension | Sample size | Humans | Meta-analysis | heart rate | essential hypertension | meta-analysis | LEFT-VENTRICULAR HYPERTROPHY | OBESITY-RELATED HYPERTENSION | BLOOD-PRESSURE | PRIMARY ALDOSTERONISM | BORDERLINE HYPERTENSION | PERIPHERAL VASCULAR DISEASE | NEURAL MECHANISMS | REFLEX ABNORMALITIES | POSTMENOPAUSAL WOMEN | humans | sample size | MILD ESSENTIAL-HYPERTENSION | BAROREFLEX CONTROL | Vasoconstriction - physiology | Muscle Contraction - physiology | Muscle, Skeletal - physiopathology | Muscle, Skeletal - innervation | Blood Pressure - physiology | Autonomic Pathways - physiopathology | Essential Hypertension - physiopathology | Hypertension | Nervous system, Sympathetic | Research
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9. Full Text Renal Denervation Prevents Immune Cell Activation and Renal Inflammation in Angiotensin II–Induced Hypertension
by Xiao, Liang and Kirabo, Annet and Wu, Jing and Saleh, Mohamed A and Zhu, Linjue and Wang, Feng and Takahashi, Takamune and Loperena, Roxana and Foss, Jason D and Mernaugh, Raymond L and Chen, Wei and Roberts, Jackson and Osborn, John W and Itani, Hana A and Harrison, David G
Circulation Research, ISSN 0009-7330, 08/2015, Volume 117, Issue 6, pp. 547 - 557
RATIONALE:Inflammation and adaptive immunity play a crucial role in the development of hypertension. Angiotensin II and probably other hypertensive stimuli... 
norepinephrine | dendritic cells | inflammation | sympathetic nerve block | lymphocyte | CARDIAC & CARDIOVASCULAR SYSTEMS | RATS | PERIPHERAL VASCULAR DISEASE | MECHANISMS | HEMATOLOGY | EXPRESSION | T-LYMPHOCYTE | Kidney - drug effects | Sympathectomy | Mice, Inbred C57BL | Kidney - immunology | Hypertension - immunology | Random Allocation | Hypertension - pathology | Mice, Knockout | Animals | Immunity, Cellular - drug effects | Immunity, Cellular - physiology | Kidney - innervation | Mice | Angiotensin II - toxicity | isoketals | T cells | sympathetic nerves | immunology
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10. Full Text Role of JNK activation in apoptosis： A double-edged sword
by JingLIU AnningLIN
细胞研究：英文版, ISSN 1001-0602, 2005, Volume 15, Issue 1, pp. 36 - 42
JNK is a key regulator of many cellular events, including programmed cell death (apoptosis). In the absence of NF-κB activation, prolonged JNK activation... 
细胞凋亡 | TNF-β | NF-KB | JNK激酶 | 细胞信号转导途径 | IL-3 | 活化作用 | NF-κB | TNF-α | JNK | Apoptosis | TNF-alpha | STRESS-INDUCED APOPTOSIS | SYMPATHETIC NEURONS | apoptosis | JUN NH2-TERMINAL KINASE | PROTEIN-KINASES | CELL BIOLOGY | PROGRAMMED CELL-DEATH | SIGNAL-TRANSDUCTION | ALPHA-INDUCED APOPTOSIS | NF-kappa B | C-JUN | NF-KAPPA-B | TERMINAL KINASE ACTIVATION | Cell Line | Phosphorylation | Interleukin-3 - metabolism | Signal Transduction | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Animals | Carrier Proteins - metabolism | MAP Kinase Kinase 4 | Ultraviolet Rays | Models, Biological | Hematopoietic Stem Cells - cytology | Mice | Enzyme Activation | bcl-Associated Death Protein
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11. Full Text Activation of Central PPAR-γ Attenuates Angiotensin II–Induced Hypertension
by Yu, Yang and Xue, Bao-Jian and Wei, Shun-Guang and Zhang, Zhi-Hua and Beltz, Terry G and Guo, Fang and Johnson, Alan Kim and Felder, Robert B
Hypertension, ISSN 0194-911X, 08/2015, Volume 66, Issue 2, pp. 403 - 411
Inflammation and renin–angiotensin system activity in the brain contribute to hypertension through effects on fluid intake, vasopressin release, and... 
Brain | Inflammation | Peroxisome proliferator-activated receptors | Angiotensin II | Renin-angiotensin system | OXIDATIVE STRESS | peroxisome proliferator-activated receptors | INTERLEUKIN-1 RECEPTOR ANTAGONIST | PROTEIN-KINASE | FACTOR-KAPPA-B | brain | SUBFORNICAL ORGAN | AGONISTS | HYPOTHALAMIC PARAVENTRICULAR NUCLEUS | BLOOD-PRESSURE | PIOGLITAZONE | inflammation | ROSTRAL VENTROLATERAL MEDULLA | angiotensin II | PERIPHERAL VASCULAR DISEASE | renin-angiotensin system | Angiotensin II - administration & dosage | Sympathetic Nervous System - drug effects | Male | PPAR gamma - metabolism | Thiazolidinediones - administration & dosage | Brain - metabolism | Sympathetic Nervous System - physiology | Hypertension - chemically induced | Hypertension - prevention & control | Blood Pressure - drug effects | Blood Pressure - physiology | Angiotensin II - adverse effects | Thiazolidinediones - pharmacology | Disease Models, Animal | Angiotensin II - pharmacology | Rats | Renin-Angiotensin System - physiology | Ventricular Remodeling - physiology | Rats, Sprague-Dawley | Hypertension - metabolism | Infusions, Subcutaneous | Animals | PPAR gamma - antagonists & inhibitors | Anilides - administration & dosage | Anilides - pharmacology | PPAR gamma - agonists | Renin-Angiotensin System - drug effects | Infusions, Intraventricular | Ventricular Remodeling - drug effects | peroxisome proliferator-activated receptor-γ | autonomic regulation
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12. Full Text Endogenous activation of spinal 5-hydroxytryptamine (5-HT) receptors contributes to the thermoregulatory activation of brown adipose tissue
by Madden, Christopher J and Morrison, Shaun F
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 03/2010, Volume 298, Issue 3, pp. R776 - R783
Madden CJ, Morrison SF. Endogenous activation of spinal 5-hydroxytryptamine (5-HT) receptors contributes to the thermoregulatory activation of brown adipose... 
Thermoregulation | Sympathetic premotor neurons | Serotonin | Metabolism | Raphe | CORD | PHYSIOLOGY | RAT | sympathetic premotor neurons | raphe | ADULT HUMANS | SYMPATHETIC OUTFLOW | IDENTIFICATION | thermoregulation | THERMOGENESIS | serotonin | MEDULLARY | COLD-EXPOSURE | metabolism | RAPHE NEURONS | SEROTONERGIC NEURONS | Receptors, Serotonin - physiology | Spinal Cord - drug effects | Evoked Potentials - physiology | Male | Methysergide - pharmacology | N-Methylaspartate - pharmacology | Adipose Tissue, Brown - innervation | Sympathetic Nervous System - physiology | Energy Metabolism - physiology | Phenols - pharmacology | Body Temperature Regulation - physiology | Microinjections | Adipose Tissue, Brown - physiology | Rats | Receptor, Serotonin, 5-HT1A - physiology | Bacteriocin Plasmids | Excitatory Amino Acid Agonists - pharmacology | Bicuculline - pharmacology | Serotonin Antagonists - pharmacology | Sulfonamides - pharmacology | Piperazines - pharmacology | Rats, Sprague-Dawley | GABA Antagonists - pharmacology | Evoked Potentials - drug effects | Animals | Raphe Nuclei - physiology | Thoracic Vertebrae | Spinal Cord - physiology | Pyridines - pharmacology | Adipose tissues | Physiological aspects | Neural receptors | Regulation | Research | Body temperature
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13. Full Text Sympatho-adrenal activation by chronic intermittent hypoxia
by Nanduri R. Prabhakar and Ganesh K. Kumar and Ying-Jie Peng
Journal of Applied Physiology, ISSN 8750-7587, 10/2012, Volume 113, Issue 8, pp. 1304 - 1310
Recurrent apnea with chronic intermittent hypoxia (CIH) is a major clinical problem in adult humans and infants born preterm. Patients with recurrent apnea... 
Hypoxia-inducible factors | Reactive oxygen species | Catecholamine secretion | Carotid chemoreceptor | Sleep apneas | Carotid baroreceptor | OBSTRUCTIVE SLEEP-APNEA | NADPH OXIDASE | SPORT SCIENCES | OXIDATIVE STRESS | PHYSIOLOGY | RAT CHROMAFFIN CELLS | SYNAPTIC-TRANSMISSION | hypoxia-inducible factors | BLOOD-PRESSURE | catecholamine secretion | sleep apneas | carotid baroreceptor | carotid chemoreceptor | LONG-TERM INTERMITTENT | ADULT-RAT | reactive oxygen species | SYMPATHETIC-NERVE ACTIVITY | CAROTID-BODY | Adrenal Medulla - physiopathology | Hypoxia - physiopathology | Apnea - physiopathology | Animals | Sympathetic Nervous System - physiopathology | Humans | Highlighted Topic
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14. Full Text Sympathetic Nervous System Regulates Bone Marrow–Derived Cell Egress Through Endothelial Nitric Oxide Synthase Activation: Role in Postischemic Tissue Remodeling
by Récalde, Alice and Richart, Adèle and Guérin, Coralie and Cochain, Clément and Zouggari, Yasmine and Yin, Kiave Ho Wang and Vilar, José and Drouet, Isabelle and Lévy, Bernard and Varoquaux, Odile and Silvestre, Jean-Sébastien
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2012, Volume 32, Issue 3, pp. 643 - 653
OBJECTIVE—Catecholamines have been shown to control bone marrow (BM)–derived cell egress, yet the cellular and molecular mechanisms involved in this effect and... 
catecholamines | nitric oxide | nitric oxide synthase | angiogenesis | ischemia | PROGENITOR CELLS | RECRUITMENT | MOBILIZATION | STEM | OVEREXPRESSION | PERIPHERAL VASCULAR DISEASE |