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Developmental Biology, ISSN 0012-1606, 12/2018, Volume 444, pp. S356 - S365
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 05/2018, Volume 314, Issue 5, pp. H954 - H966
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 6, p. e39586
...), pro-apoptotic BH3 domain-only, BCL2 family members, have previously been shown to regulate ER stress-induced cell death, but the upstream signaling pathways that regulate this response in neuronal... 
MITOCHONDRIAL APOPTOSIS | TRANSCRIPTION FACTOR CHOP | UNFOLDED PROTEIN RESPONSE | INDUCED APOPTOSIS | KINASE | BIOLOGY | GENE-EXPRESSION | SYMPATHETIC NEURONS | DEATH | ENDOPLASMIC-RETICULUM STRESS | BH3-ONLY PROTEINS | Transcription Factor CHOP - genetics | Apoptosis - drug effects | Neurons - cytology | Bcl-2-Like Protein 11 | Forkhead Transcription Factors - metabolism | Telencephalon - drug effects | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | Neurons - metabolism | Neurons - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - physiology | Proto-Oncogene Proteins - metabolism | Cell Survival - drug effects | Tumor Suppressor Proteins - metabolism | Telencephalon - metabolism | Telencephalon - cytology | Membrane Proteins - genetics | Proto-Oncogene Proteins - genetics | Forkhead Transcription Factors - genetics | Apoptosis Regulatory Proteins - metabolism | Animals | Signal Transduction - drug effects | Tunicamycin - pharmacology | Signal Transduction - physiology | Mice | Apoptosis - physiology | Forkhead Box Protein O3 | Transcription Factor CHOP - metabolism | Endoplasmic Reticulum Stress - physiology | Nervous system diseases | Parkinson's disease | Huntington's chorea | Neurons | Tumor proteins | Protein kinases | Apoptosis | Phosphorylation | Transcription factors | Transcription | p53 Protein | Tunicamycin | Homology | AKT protein | CCAAT/enhancer-binding protein | Kinases | Proteins | Signal transduction | Mitochondria | Cell activation | Cell growth | Cascades | Rodents | Forkhead protein | Alzheimer's disease | Movement disorders | Deoxyribonucleic acid--DNA | FOXO3 protein | Stresses | Neurodegenerative diseases | Mortality | Gene expression | Stress | Neurological diseases | Pathology | Signaling | Cell death | Endoplasmic reticulum | CHOP protein | BIM protein | Deoxyribonucleic acid | DNA
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 2010, Volume 299, Issue 2, pp. H422 - H430
An increase in plasma ANG II causes neuronal activation in hypothalamic nuclei and a slow pressor response, presumably by increasing sympathetic drive... 
Eplerenone | Angiotensin II | Fra-like immunoreactivity | Telemetry | Aldosterone synthase inhibitor | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MYOCARDIAL-INFARCTION | POSTMYOCARDIAL INFARCTION | eplerenone | SUBFORNICAL ORGAN | CENTRAL INFUSION | MINERALOCORTICOID RECEPTOR | INDUCED HYPERTENSION | RENIN-ANGIOTENSIN SYSTEM | angiotensin II | CARDIAC-HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | telemetry | PREVENTS SYMPATHETIC HYPERACTIVITY | aldosterone synthase inhibitor | Immunohistochemistry | Fadrozole | Angiotensin II - administration & dosage | Rats, Wistar | Spironolactone - administration & dosage | Cardenolides - metabolism | Angiotensin II - blood | Imidazoles - administration & dosage | Male | Brain - metabolism | Enzyme Inhibitors - administration & dosage | Aldosterone - metabolism | Time Factors | Blood Pressure - drug effects | Neurons - metabolism | Neurons - drug effects | Cytochrome P-450 CYP11B2 - metabolism | Subfornical Organ - metabolism | Heart Rate | Mineralocorticoid Receptor Antagonists - administration & dosage | Brain - cytology | Infusion Pumps, Implantable | Pyridines - administration & dosage | Proto-Oncogene Proteins c-fos - metabolism | Infusions, Parenteral | Rats | Infusions, Subcutaneous | Brain - drug effects | Immunoglobulin Fab Fragments - administration & dosage | Animals | Hypothalamus - metabolism | Cytochrome P-450 CYP11B2 - antagonists & inhibitors | Neural Pathways - metabolism | Renin-Angiotensin System - drug effects | Saponins - metabolism | Blood Pressure Monitoring, Ambulatory | Neurons | Angiotensin | Physiological aspects | Genetic aspects | Research | Aldosterone | Health aspects
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