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1991, ISBN 9780195069211, 438
This book examines neuroplasticity. Chapters represent work on neuroplasticity at all levels: behavioural, neural, and molecular. They describe work on memory... 
Neuroplasticity | Physiological aspects | Memory | Psychology | Neural plasticity
eBook
Journal Article
Science, ISSN 0036-8075, 08/2018, Volume 361, Issue 6402, pp. 548 - 549
Clusters of synaptic vesicles (SVs) are distinctive features of axon terminals in neurons. SVs contain neurotransmitters that are released upon electrical... 
MULTIDISCIPLINARY SCIENCES | SYNAPSIN-I | Presynapse | Phase transformations | Neurons | Phase transitions | Proteins | Vesicles | Neurotransmitters | Synaptic transmission | Synapsin | Phase separation | Synaptic vesicles | Clusters | Neurotransmission | Index Medicus
Journal Article
Molecular Psychiatry, ISSN 1359-4184, 12/2012, Volume 17, Issue 12, pp. 1254 - 1260
Journal Article
2016, ISBN 1848722818, xv, 198 pages
Neural Plasticity Across the Lifespan reviews the recent scientific developments which are transforming our understanding of the human brain. For many years it... 
Neuroplasticity | Cerebral cortex | Sensory Deprivation | Age Factors | Neuronal Plasticity | Growth | physiology | Biology, life sciences | Neuropsychology | Developmental Neuroscience | Biological Development | Sensory Deprivation - physiology | Brain
Book
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2009, Volume 106, Issue 42, pp. 17998 - 18003
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e92765 - e92765
Introduction: Metabotropic glutamate receptor 5 (mGluR5) that regulates glutamatergic neurotransmission contributes to pathophysiology of epilepsy. In this... 
DEPRESSION | SUBTYPE-5 | REDUCTION | ROLES | MULTIDISCIPLINARY SCIENCES | METABOTROPIC GLUTAMATE RECEPTORS | INDUCED STATUS-EPILEPTICUS | C-11-ABP688 | UP-REGULATION | EXPRESSION | BINDING | Amygdala - diagnostic imaging | Amygdala - physiopathology | Muscarinic Agonists - pharmacology | Amygdala - metabolism | Epilepsy - diagnostic imaging | Rats | Epilepsy - metabolism | Male | Positron-Emission Tomography | Epilepsy - physiopathology | Receptor, Metabotropic Glutamate 5 - metabolism | Rats, Sprague-Dawley | Hippocampus - diagnostic imaging | Radiography | Hippocampus - metabolism | Animals | Epilepsy - chemically induced | Muscarinic Agonists - adverse effects | Pilocarpine - pharmacology | Pilocarpine - adverse effects | Oximes - pharmacology | Synaptic Transmission - drug effects | Pyridines - pharmacology | Hippocampus - physiopathology | Brain | Seizures (Medicine) | Glutamate | Epilepsy | Neuroimaging | Nuclear medicine | Animal models | Nuclear magnetic resonance--NMR | Amygdala | Physicians | Spatial discrimination | Science | Pilocarpine | Glutamic acid receptors | Caudate-putamen | Image acquisition | Convulsions & seizures | Computed tomography | Rodents | Tomography | Drug dosages | Seizures | Binding | Medical imaging | Positron emission | Putamen | Studies | Video recorders | In vivo methods and tests | Glutamatergic transmission | Positron emission tomography | Glutamic acid receptors (metabotropic) | Hippocampus | Neurotransmission | Index Medicus | Nuclear magnetic resonance | NMR
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, pp. e73846 - e73846
Amyotrophic lateral sclerosis is characterized by a progressive degeneration of the corticospinal tract motor neurons. Growing evidence suggests that... 
SKELETAL-MUSCLE | WILD-TYPE | NERVE-TERMINALS | NEUROTRANSMITTER RELEASE | UNIT LOSS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | ACETYLCHOLINE-RELEASE | MUTANT SOD1 | AMYOTROPHIC-LATERAL-SCLEROSIS | BOTULINUM TOXIN | Motor Activity - physiology | Superoxide Dismutase - genetics | Synaptic Transmission - physiology | Amyotrophic Lateral Sclerosis - physiopathology | Synaptic Transmission - genetics | Neuromuscular Junction - metabolism | Humans | Male | Muscle, Skeletal - innervation | Muscle, Skeletal - metabolism | Time Factors | Female | Diaphragm - innervation | Diaphragm - metabolism | Superoxide Dismutase - metabolism | Disease Models, Animal | Synaptic Potentials - physiology | Synaptic Potentials - genetics | Amyotrophic Lateral Sclerosis - genetics | Mice, Transgenic | Motor Activity - genetics | Amyotrophic Lateral Sclerosis - pathology | Animals | Mice | Neuromuscular Junction - physiology | Amyotrophic lateral sclerosis | Deregulation | Genetic engineering | Neurosciences | Laboratories | Innervation | Superoxide dismutase | Males | Sclerosis | Synaptic transmission | Transgenic animals | Rodents | Diaphragm (anatomy) | Degeneration | Age | Nerve endings | Motor neurons | Calcium (intracellular) | Transgenic mice | Muscles | Pharmacology | Pyramidal tracts | Neuromuscular transmission | Medicine | Musculoskeletal system | Hypotheses | Neuromuscular junctions | Acetylcholine | Mutation | Diaphragm | Females | Synaptic cleft | Denervation | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2012, Volume 32, Issue 47, pp. 16857 - 16871
Amid controversy, the cellular form of the prion protein PrPc has been proposed to mediate oligomeric amyloid-beta (A beta)-induced deficits. In contrast,... 
OLDER PERSONS | TYROSINE PHOSPHORYLATION | ENDOGENOUS TAU | AMYLOID-BETA | PLAQUE-FORMATION | MILD COGNITIVE IMPAIRMENT | TRANSGENIC MOUSE MODEL | SYNAPTIC PLASTICITY | DEPENDENT MECHANISM | NEUROSCIENCES | CELLULAR PRION PROTEIN
Journal Article