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Immunity, ISSN 1074-7613, 09/2012, Volume 37, Issue 3, pp. 511 - 523
Interferon-γ (IFN-γ) promotes a population of T-bet CXCR3 regulatory T (Treg) cells that limit T helper 1 (Th1) cell-mediated pathology. Our studies... 
BET | RESPONSES | TOXOPLASMA-GONDII INFECTION | DENDRITIC CELLS | IFN-GAMMA | IN-VIVO | REGULATORY T-CELLS | IL-27 | IMMUNOLOGY | EXPRESSION | VIRAL-INFECTION | Forkhead Transcription Factors - immunology | Toxoplasma - immunology | T-Lymphocytes, Regulatory - metabolism | Oligonucleotide Array Sequence Analysis | Interleukin-17 - immunology | Male | Gene Expression Profiling | T-Box Domain Proteins - immunology | Interleukin-17 - pharmacology | T-Lymphocytes, Regulatory - immunology | Salmonella Infections, Animal - immunology | STAT1 Transcription Factor - metabolism | Flow Cytometry | Toxoplasmosis, Animal - parasitology | Forkhead Transcription Factors - metabolism | Mice, Inbred CBA | Female | Interferon-gamma - genetics | Receptors, CXCR3 - metabolism | Salmonella Infections, Animal - pathology | Salmonella typhimurium - immunology | Mice, Inbred C57BL | Cells, Cultured | Interleukin-17 - genetics | Mice, Transgenic | Forkhead Transcription Factors - genetics | STAT1 Transcription Factor - genetics | T-Box Domain Proteins - genetics | STAT1 Transcription Factor - immunology | T-Box Domain Proteins - metabolism | Mice, Knockout | Cell Differentiation - immunology | T-Lymphocytes, Regulatory - drug effects | Animals | Cell Differentiation - drug effects | Interferon-gamma - immunology | Receptors, CXCR3 - genetics | Receptors, CXCR3 - immunology | Salmonella Infections, Animal - microbiology | Toxoplasmosis, Animal - immunology | Mice | Interferon-gamma - pharmacology | Toxoplasmosis, Animal - pathology
Journal Article
The Journal of Infectious Diseases, ISSN 0022-1899, 2/2000, Volume 181, Issue 2, pp. 671 - 680
Journal Article
Science, ISSN 0036-8075, 2/2013, Volume 339, Issue 6122, pp. 975 - 978
Caspases are either apoptotic or inflammatory. Among inflammatory caspases, caspase-1 and -11 trigger pyroptosis, a form of programmed cell death. Whereas both... 
Statistical significance | Secretion | REPORTS | Bacteria | Cytotoxicity | Infections | Signals | Macrophages | Cytosol | T tests | Vacuoles | FLAGELLIN | III SECRETION APPARATUS | INFLAMMASOME ACTIVATION | INTERLEUKIN-1-BETA | MULTIDISCIPLINARY SCIENCES | NLRC4 INFLAMMASOME | TRAFFICKING | MAINTAINS | SALMONELLA INFECTION | MELIOIDOSIS | CELL-DEATH | Phagosomes - microbiology | Inflammasomes - metabolism | Gram-Negative Bacterial Infections - enzymology | Gram-Negative Bacterial Infections - immunology | Burkholderia - pathogenicity | Gram-Negative Bacterial Infections - microbiology | Salmonella Infections, Animal - immunology | Burkholderia Infections - enzymology | Caspases - metabolism | Burkholderia pseudomallei - pathogenicity | Cell Death | Salmonella Infections, Animal - enzymology | Macrophages - immunology | Macrophages - microbiology | Salmonella typhimurium - physiology | Vacuoles - microbiology | Burkholderia Infections - immunology | Salmonella typhimurium - pathogenicity | Mice, Inbred C57BL | Immunity, Innate | Animals | Salmonella Infections, Animal - microbiology | Burkholderia Infections - metabolism | Burkholderia - physiology | Burkholderia pseudomallei - physiology | Cytosol - microbiology | Mice | Physiological aspects | Cellular immunity | Research | Apoptosis | Enzymes | Cellular biology | Cytoplasm | Immune system
Journal Article
PLoS Pathogens, ISSN 1553-7366, 10/2012, Volume 8, Issue 10, p. e1002933
Salmonella enterica infections result in diverse clinical manifestations. Typhoid fever, caused by S. enterica serovar Typhi (S. Typhi) and S. Paratyphi A, is... 
DENDRITIC CELLS | GAMMA-INTERFERON | MICROBIOLOGY | NATURAL-RESISTANCE | COMPLETE GENOME SEQUENCE | PATTERN-RECOGNITION | VIROLOGY | VIRULENCE FACTORS | MEMBRANE-VESICLES | ENTERICA SEROVAR TYPHI | PARASITOLOGY | III SECRETION SYSTEM | MOLECULAR-MECHANISMS | Humans | Toll-Like Receptors - immunology | Neutrophils - immunology | Salmonella Infections - immunology | Salmonella enterica - pathogenicity | Salmonella enterica - genetics | Typhoid Fever - transmission | Inflammasomes - biosynthesis | Intestines - immunology | Host-Pathogen Interactions | Salmonella Infections, Animal - immunology | Animals | Typhoid Fever - microbiology | Typhoid Fever - pathology | Inflammasomes - immunology | Salmonella Infections, Animal - microbiology | Salmonella enterica - immunology | Salmonella Infections - microbiology | Mice | Typhoid Fever - immunology | Macrophages - immunology | Cytokines - immunology | Receptors, Cytoplasmic and Nuclear - metabolism | Host-parasite relationships | Physiological aspects | Development and progression | Typhoid fever | Salmonellosis | Virulence (Microbiology) | Research | Health aspects | Salmonella | Medical research | Infections | Typhoid | Animal models | Disseminated infection | Solitary tract nucleus | Genomes | Infants | Macrophages | Lipopolysaccharides | Life cycle | Cell activation | Polysaccharides | Interleukin 1 | Toll-like receptors | Digestive tract | Pathogens | Immune response | Diarrhea | Inflammation | Leukocytes (neutrophilic) | Bacteremia | virulence factors | Host-pathogen interactions | Infection | Plasmids | Tumor necrosis factor | pathogenicity islands | gamma -Interferon | Immunocompromised hosts | Flagella
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2011, Volume 17, Issue 7, pp. 837 - 844
Journal Article
Nature Immunology, ISSN 1529-2908, 10/2012, Volume 13, Issue 10, pp. 954 - 962
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2013, Volume 110, Issue 24, pp. 9897 - 9902
Organisms adapt to day-night cycles through highly specialized circadian machinery, whose molecular components anticipate and drive changes in organism... 
Datasets | Salmonella | Cytokines | Secretion | Antimicrobials | Genes | Neutrophils | Infections | Gene expression regulation | Inflammation | Clock genes | Intestine | Gastroenteritis | Microbes | intestine | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | INNATE | TYPHIMURIUM | LIPOCALIN-2 | clock genes | microbes | NK CELLS | inflammation | gastroenteritis | MEDIATED TRANSCRIPTION | IMMUNE-RESPONSES | GENES | TNF-ALPHA | EXPRESSION | Oligonucleotide Array Sequence Analysis | CLOCK Proteins - genetics | Circadian Clocks - genetics | Gene Expression Profiling | Cecum - microbiology | Lipopolysaccharides - immunology | CLOCK Proteins - deficiency | Host-Pathogen Interactions - immunology | Salmonella Infections, Animal - immunology | Time Factors | Cecum - metabolism | Inflammation Mediators - metabolism | Gene Regulatory Networks - immunology | Cytokines - genetics | Macrophages - immunology | Macrophages - microbiology | Cytokines - immunology | Inflammation Mediators - immunology | Salmonella typhimurium - physiology | Gene Regulatory Networks - genetics | CLOCK Proteins - immunology | Cytokines - metabolism | Salmonella typhimurium - immunology | Mice, Inbred C57BL | Cells, Cultured | Circadian Clocks - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Macrophages - metabolism | Animals | Salmonella Infections, Animal - genetics | Salmonella Infections, Animal - microbiology | Mice | Cecum - immunology | Cluster Analysis | Circadian rhythms | Immune response | Physiological aspects | Host-parasite relationships | Research | Health aspects | Biological Sciences
Journal Article
Science, ISSN 0036-8075, 7/2012, Volume 337, Issue 6093, pp. 477 - 481
Defensins are antimicrobial peptides that contribute broadly to innate immunity, including protection of mucosal tissues. Human α-defensin (HD) 6 is highly... 
Molecules | Epithelial cells | Transgenic animals | Biosensing techniques | Humans | REPORTS | Cell lines | Bacteria | Mice | Monomers | Vehicles | HD5 | MICE | PANETH CELLS | MULTIDISCIPLINARY SCIENCES | Intestine, Small - ultrastructure | Protein Multimerization | Immunity, Mucosal | env Gene Products, Human Immunodeficiency Virus - metabolism | Nanostructures | Salmonella Infections, Animal - immunology | Adhesins, Bacterial - metabolism | Salmonella typhimurium - ultrastructure | Macromolecular Substances - chemistry | Peptides - metabolism | Paneth Cells - immunology | Intestinal Mucosa - immunology | Protein Structure, Quaternary | Macromolecular Substances - immunology | alpha-Defensins - metabolism | Yersinia enterocolitica - immunology | Intestine, Small - immunology | Paneth Cells - metabolism | Cell Line | Microscopy, Electron, Scanning | alpha-Defensins - chemistry | Intestinal Mucosa - ultrastructure | Peptides - chemistry | Salmonella typhimurium - immunology | Salmonella typhimurium - pathogenicity | Yersinia enterocolitica - pathogenicity | Intestine, Small - microbiology | Models, Molecular | Mice, Transgenic | Intestinal Mucosa - microbiology | Immunity, Innate | Animals | alpha-Defensins - immunology | Salmonella Infections, Animal - microbiology | Protein Binding | Bacterial Proteins - metabolism | Macromolecular Substances - metabolism
Journal Article
Journal Article