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Clinical Immunology, ISSN 1521-6616, 2010, Volume 137, Issue 1, pp. 89 - 101
Abstract Activated macrophages have been characterized as M1 and M2 according to their inflammatory response pattern. Here we analyzed the M2 marker expression... 
Allergy and Immunology | M2 phenotype | Th2 cytokines | Pulmonary fibrosis | IL-1RA | CCL22 | Sarcoidosis | JAK/STAT pathway | CCL18 | Alveolar macrophages | CCL17 | INTERLEUKIN-1 RECEPTOR ANTAGONIST | CYTOKINE | IMMUNOLOGY | SYSTEMIC-SCLEROSIS | STAT3 ACTIVATION | INTERSTITIAL LUNG-DISEASES | MANNOSE RECEPTOR | GENE-EXPRESSION | TNF-ALPHA RELEASE | GROWTH-FACTOR | UP-REGULATION | Mannose-Binding Lectins - metabolism | Tumor Necrosis Factor-alpha - metabolism | Gene Expression - drug effects | Humans | Middle Aged | STAT Transcription Factors - metabolism | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Monocytes - metabolism | Monocytes - immunology | Sarcoidosis, Pulmonary - diagnosis | Idiopathic Pulmonary Fibrosis - metabolism | Young Adult | Lectins, C-Type - metabolism | Signal Transduction - immunology | Interleukin-4 - pharmacology | Interleukin 1 Receptor Antagonist Protein - metabolism | Scleroderma, Systemic - complications | Aged, 80 and over | Adult | Female | Bronchoalveolar Lavage Fluid - cytology | Pulmonary Fibrosis - metabolism | Interleukin-8 - metabolism | Macrophages, Alveolar - immunology | Pulmonary Fibrosis - etiology | Sarcoidosis, Pulmonary - complications | Cytokines - metabolism | Macrophage Activation - immunology | Scleroderma, Systemic - metabolism | Pulmonary Fibrosis - immunology | Receptors, Cell Surface - metabolism | Idiopathic Pulmonary Fibrosis - immunology | Monocytes - drug effects | Signal Transduction - drug effects | Macrophages, Alveolar - drug effects | Sarcoidosis, Pulmonary - metabolism | Chemokines, CC - genetics | Aged | Macrophages, Alveolar - metabolism | Macrophage Activation - drug effects | Interleukin-10 - pharmacology | Chemokines, CC - metabolism | Respiratory tract diseases | Systemic scleroderma | Transforming growth factors | Macrophages | Scleroderma (Disease) | Alveoli
Journal Article
Science, ISSN 0036-8075, 3/2013, Volume 339, Issue 6126, pp. 1448 - 1453
Type I interferons (IFN-α and IFN-β) are important for protection against many viral infections, whereas type II interferon (IFN-γ) is essential for host... 
Monocytes | Messenger RNA | Tuberculosis | Mycobacterium tuberculosis | Antimicrobials | REPORTS | Leprosy | Infections | Skin | Lesions | Gene expression | LISTERIA-MONOCYTOGENES | ACTIVATION | PROFILES | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | HUMAN TUBERCULOSIS | IL-1-BETA | INDUCTION | DEPENDENT ANTIMICROBIAL PATHWAY | SARCOIDOSIS | LEPROSY | Up-Regulation | Interferon-beta - immunology | Humans | Transcriptome | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - genetics | Monocytes - metabolism | Monocytes - immunology | Interferon-gamma - metabolism | Antimicrobial Cationic Peptides - metabolism | RNA, Messenger - metabolism | Receptors, Calcitriol - genetics | Mycobacterium leprae - immunology | Interferon-beta - genetics | Interleukin-10 - metabolism | beta-Defensins - metabolism | Interferon-gamma - genetics | Antimicrobial Cationic Peptides - genetics | Leprosy, Lepromatous - genetics | Mycobacterium leprae - physiology | Leprosy, Tuberculoid - immunology | beta-Defensins - genetics | Leprosy, Lepromatous - immunology | RNA, Messenger - genetics | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - metabolism | Microbial Viability | Receptors, Calcitriol - metabolism | Leprosy, Tuberculoid - genetics | Tuberculosis - immunology | Leprosy, Lepromatous - metabolism | Interferon-beta - metabolism | Tuberculosis - genetics | Leprosy, Tuberculoid - metabolism | Interferon-gamma - immunology | Interleukin-10 - genetics | Interleukins | Physiological aspects | Host-parasite relationships | Anti-infective agents | Interferon | Genetic aspects | Health aspects | Antimicrobial agents | Pathogenesis | Genes | Bacteria | Human behavior | Antiinfectives and antibacterials | Digital signatures
Journal Article
Journal of Breath Research, ISSN 1752-7155, 06/2015, Volume 9, Issue 2, p. 027107
Journal Article
Advances in Experimental Medicine and Biology, ISSN 0065-2598, 06/2016, Volume 910, pp. 23 - 30
Journal Article
Journal of Histochemistry and Cytochemistry, ISSN 0022-1554, 07/2006, Volume 54, Issue 7, pp. 763 - 771
CD163 is a member of the scavenger receptor cysteine-rich superfamily restricted to the monocyte/macrophage lineage and is thought to be a useful marker for... 
Macrophage heterogeneity | Macrophage markers | CD163 | Atherosclerotic lesions | Granulomatous diseases | NITRIC-OXIDE SYNTHASE | GIANT-CELL TUMOR | atherosclerotic lesions | MONOCLONAL-ANTIBODY | GLUCOCORTICOID-INDUCIBLE MEMBER | HUMAN MONOCYTES | CYSTEINE-RICH FAMILY | CELL BIOLOGY | macrophage heterogeneity | TUMOR-NECROSIS-FACTOR | DIFFERENTIATION ANTIGEN | MEMBRANE-PROTEIN RM3/1 | macrophage markers | granulomatous diseases | HEMOGLOBIN SCAVENGER RECEPTOR | Immunohistochemistry | Antigens, CD - immunology | Tuberculosis, Pulmonary - metabolism | Histiocytosis, Non-Langerhans-Cell - metabolism | Giant Cell Tumors - immunology | Antigens, CD - biosynthesis | Humans | Synovial Membrane - pathology | Histiocytosis, Sinus - metabolism | Giant Cell Tumors - metabolism | Antigens, CD - metabolism | Bone Neoplasms - metabolism | Paraffin Embedding | Soft Tissue Neoplasms - immunology | Histiocytosis, Sinus - immunology | Foreign-Body Reaction - immunology | Mass Spectrometry | Sarcoidosis - immunology | Cell Differentiation | Receptors, Cell Surface - biosynthesis | Antigens, Differentiation, Myelomonocytic - immunology | Histiocytosis, Non-Langerhans-Cell - immunology | Macrophages - immunology | Antigens, Differentiation, Myelomonocytic - biosynthesis | Atherosclerosis - immunology | Gaucher Disease - metabolism | Electrophoresis, Polyacrylamide Gel | Soft Tissue Neoplasms - metabolism | Foreign-Body Reaction - metabolism | Gaucher Disease - immunology | Receptors, Cell Surface - metabolism | Bone Neoplasms - immunology | Macrophages - cytology | Receptors, Cell Surface - immunology | Atherosclerosis - metabolism | Macrophage Activation | Macrophages - metabolism | Phenotype | Tuberculosis, Pulmonary - immunology | Antigens, Differentiation, Myelomonocytic - metabolism | Sarcoidosis - metabolism
Journal Article
European Journal of Clinical Investigation, ISSN 0014-2972, 05/2016, Volume 46, Issue 5, pp. 418 - 424
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0137358
Macrophages are involved in low-grade inflammation in diabetes, and play pathogenic roles in proliferative diabetic retinopathy (PDR) by producing... 
VEGF | MULTIDISCIPLINARY SCIENCES | IL-17 | INTERLEUKIN-31 | EXPRESSION | T-CELLS | ANGIOGENIC FACTORS | IL-31 | Tumor Necrosis Factor-alpha - metabolism | Eye - pathology | Inflammation - pathology | Humans | Middle Aged | Male | Diabetic Retinopathy - pathology | Inflammation - metabolism | Retinal Perforations - metabolism | Th17 Cells - metabolism | Vitreous Body - metabolism | Sarcoidosis - immunology | Tumor Necrosis Factor-alpha - immunology | Aged, 80 and over | Adult | Female | Vitrectomy | Macrophages - immunology | Th2 Cells - pathology | Cytokines - blood | Cytokines - immunology | Uveitis - metabolism | Eye - metabolism | Diabetic Retinopathy - immunology | Cytokines - metabolism | Macrophages - pathology | Inflammation - immunology | Diabetic Retinopathy - surgery | Immunity, Innate | Th2 Cells - metabolism | Diabetic Retinopathy - blood | Macrophages - metabolism | Th17 Cells - immunology | Vitreous Body - immunology | Vitreous Body - pathology | Aged | Retinal Perforations - pathology | Inflammation - surgery | Sarcoidosis - metabolism | Uveitis - pathology | Sarcoidosis - pathology | Diabetic retinopathy | Cytokines | Physiological aspects | Development and progression | Genetic aspects | Research | T cells | Retinopathy | Pathogenesis | Helper cells | Homeostasis | Interleukin 22 | Retina | Interleukin 23 | Lymphocytes T | Macrophages | Dermatitis | Hemorrhage | Inflammatory diseases | Interleukin 6 | Interleukin 4 | Lymphocytes | Quality | Surgery | Uveitis | Interleukin 21 | Immune response | Sarcoidosis | Review boards | Diabetes mellitus | Inflammation | Tumor necrosis factor-α | Patients | Allergies | γ-Interferon | Interleukin 10 | Ligands | Infiltration | Interferon | Diabetes
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, pp. e0161669 - e0161669
Background Sarcoidosis is characterised by up-regulation of cytokines and chemokine ligands/receptors and proteolytic enzymes. This pro-inflammatory profile is... 
AIRWAY EPITHELIAL-CELLS | MATRIX METALLOPROTEINASES | IDIOPATHIC INTERSTITIAL PNEUMONIAS | MESSENGER-RNA | HUR | MULTIDISCIPLINARY SCIENCES | DISEASE | NUCLEOLIN | NITRIC-OXIDE | INDUCED MATRIX-METALLOPROTEINASE-9 EXPRESSION | CD4(+) T-CELLS | RNA-Binding Proteins - genetics | Asthma - metabolism | Idiopathic Interstitial Pneumonias - metabolism | Humans | Middle Aged | Male | Phosphoproteins - metabolism | Heterogeneous-Nuclear Ribonucleoprotein D - metabolism | Young Adult | ELAV-Like Protein 1 - metabolism | T-Lymphocytes - metabolism | Poly(A)-Binding Proteins - metabolism | Sarcoidosis, Pulmonary - genetics | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | T-Cell Intracellular Antigen-1 | Pulmonary Disease, Chronic Obstructive - genetics | Heterogeneous-Nuclear Ribonucleoprotein D - genetics | PTEN Phosphohydrolase - genetics | Poly(A)-Binding Proteins - genetics | ELAV-Like Protein 1 - genetics | Gene Expression Regulation | PTEN Phosphohydrolase - metabolism | Gene Expression Profiling - methods | Phosphoproteins - genetics | Asthma - genetics | GPI-Linked Proteins - metabolism | Sarcoidosis, Pulmonary - metabolism | Idiopathic Interstitial Pneumonias - genetics | Aged | RNA-Binding Proteins - metabolism | GPI-Linked Proteins - genetics | Sarcoidosis | Causes of | Physiological aspects | Genetic aspects | Research | Binding proteins | Gene expression | Post-transcription | Flow cytometry | Pneumonia | Disease | Laboratories | CD8 antigen | Fluorescence | Lymphocytes T | Genomes | Proteins | Airway management | Receptors | Lymphocytes | Proteolysis | Peripheral blood | Tumor necrosis factor-TNF | Physiology | Chronic obstructive pulmonary disease | Alveoli | HuR protein | Enzymes | Cytokines | Lung diseases | Proteolytic enzymes | Bronchus | CD3 antigen | Inflammation | Dentistry | Patients | Asthma | CD4 antigen | Medicine | Polymerase chain reaction | Cytometry | RNA-binding protein | Ribonucleic acids | Respiratory diseases | Obstructive lung disease | In vivo methods and tests | Binding sites | PTEN protein | Smoking | Index Medicus
Journal Article
European Respiratory Journal, ISSN 0903-1936, 11/2011, Volume 38, Issue 5, pp. 1136 - 1144
Journal Article