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International Journal of Cancer, ISSN 0020-7136, 03/2014, Volume 134, Issue 6, pp. 1346 - 1358
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2012, Volume 287, Issue 30, pp. 25530 - 25540
Fas is a member of the death receptor family. Stimulation of Fas leads to induction of apoptotic signals, such as caspase 8 activation, as well as... 
CD95 | AUTOIMMUNE LYMPHOPROLIFERATIVE SYNDROME | CANCER DEVELOPMENT | IFN-GAMMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE MODEL | IN-VIVO | ALPHA-DEPENDENT APOPTOSIS | IKK-BETA | RECEPTOR | CELL-DEATH | Colonic Neoplasms - genetics | Fas Ligand Protein - metabolism | Humans | Caspase 8 - metabolism | Colonic Neoplasms - chemically induced | Neoplasms, Experimental - chemically induced | fas Receptor - metabolism | Promoter Regions, Genetic - genetics | Transcription Factor RelA - genetics | Colonic Neoplasms - metabolism | Embryo, Mammalian - metabolism | Caspase 8 - genetics | Sarcoma - chemically induced | NF-kappa B p52 Subunit - genetics | Tumor Suppressor Proteins - genetics | Neoplasms, Experimental - genetics | fas Receptor - genetics | Gene Expression Regulation, Neoplastic - drug effects | Sarcoma - genetics | Gene Expression Regulation, Neoplastic - genetics | Fibroblasts - metabolism | Tumor Suppressor Proteins - metabolism | Embryo, Mammalian - pathology | Fibroblasts - pathology | Sarcoma - pathology | Methylcholanthrene - toxicity | NF-kappa B p52 Subunit - metabolism | Mice, Knockout | Sarcoma - metabolism | Animals | Transcription Factor RelA - metabolism | Mice | Neoplasms, Experimental - metabolism | Fas Ligand Protein - genetics | Apoptosis | Index Medicus | Molecular Bases of Disease | Cancer Tumor Promoter | Fas | NF-kappaB Transcription Factor | NF-kappaB | Cancer
Journal Article
Cancer Cell, ISSN 1535-6108, 2007, Volume 11, Issue 2, pp. 175 - 189
In response to DNA damage, eukaryotic cells activate ATM-Chk2 and/or ATR-Chk1 to arrest the cell cycle and initiate DNA repair. We show that, in the absence of... 
SIGNALING | CANCER-CELLS | ONCOLOGY | 7-HYDROXYSTAUROSPORINE UCN-01 | MITOTIC CATASTROPHE | SECKEL-SYNDROME | MAPKAP KINASE-2 | NUCLEAR EXPORT | INDUCED CYTOTOXICITY | 14-3-3 PROTEIN-BINDING | RADIOSENSITIZING AGENT | PHOSPHORYLATION SITES | Protein Kinases - genetics | Antibiotics, Antineoplastic - pharmacology | Humans | DNA Repair - radiation effects | Staurosporine - analogs & derivatives | Bone Neoplasms - pathology | G2 Phase - drug effects | Bone Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Neoplasms, Experimental - pathology | Tumor Suppressor Protein p53 - physiology | Ultraviolet Rays | Antineoplastic Agents - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | cdc25 Phosphatases - metabolism | Osteosarcoma - metabolism | Protein-Serine-Threonine Kinases - metabolism | DNA Damage - drug effects | DNA Repair - drug effects | Tumor Suppressor Proteins - metabolism | Mitosis - radiation effects | G2 Phase - radiation effects | Signal Transduction | Cell Survival | Cell Cycle Proteins - metabolism | Cells, Cultured | Cell Division - radiation effects | Intracellular Signaling Peptides and Proteins | Protein Kinase C - antagonists & inhibitors | Ataxia Telangiectasia Mutated Proteins | Cisplatin - pharmacology | Cell Division - drug effects | Mice, Knockout | Phosphorylation - radiation effects | S Phase - radiation effects | Animals | Mitosis - drug effects | Mice, Nude | Protein Kinases - physiology | Mice | S Phase - drug effects | Neoplasms, Experimental - metabolism | DNA Damage - radiation effects | Doxorubicin - pharmacology | Osteosarcoma - pathology | Staurosporine - pharmacology | DNA | Index Medicus
Journal Article
Developmental Cell, ISSN 1534-5807, 03/2014, Volume 28, Issue 6, pp. 633 - 646
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 10, pp. e26631 - e26631
Journal Article
Blood, ISSN 0006-4971, 2011, Volume 118, Issue 10, pp. 2889 - 2895
Journal Article
Cancer Research, ISSN 0008-5472, 05/2011, Volume 71, Issue 9, pp. 3306 - 3316
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, pp. e13943 - e13943
Background: Cancer stem cells are a chemotherapy-resistant population capable of self-renewal and of regenerating the bulk tumor, thereby causing relapse and... 
PRIMITIVE NEUROECTODERMAL TUMOR | BREAST-CANCER CELLS | SIDE POPULATION | IN-VITRO | RNA HELICASE | MARKER | PANCREATIC-CANCER | BIOLOGY | ALDEHYDE DEHYDROGENASE 1 | PROLIFERATION | BONE | Immunohistochemistry | Oncogene Proteins, Fusion - metabolism | Sarcoma, Experimental - metabolism | Neoplastic Stem Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Sarcoma, Ewing - pathology | Spheroids, Cellular - pathology | Transplantation, Heterologous | Interleukin Receptor Common gamma Subunit - metabolism | Neoplastic Stem Cells - metabolism | Proto-Oncogene Protein c-fli-1 - metabolism | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Spheroids, Cellular - drug effects | Aldehyde Dehydrogenase - metabolism | Cell Survival - drug effects | Sarcoma, Ewing - metabolism | Proto-Oncogene Protein c-fli-1 - genetics | Spheroids, Cellular - metabolism | Calcium Channel Blockers - pharmacology | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Sarcoma, Experimental - pathology | Interleukin Receptor Common gamma Subunit - genetics | Verapamil - pharmacology | Sarcoma, Experimental - drug therapy | Mice, Knockout | Sarcoma, Ewing - drug therapy | Animals | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Mice, Inbred NOD | Cell Proliferation - drug effects | Mice | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | RNA-Binding Protein EWS | Chemotherapy | Sarcoma | Analysis | Stem cells | Metastasis | Cancer | Protein binding | EWS protein | Adolescence | Pediatrics | Dehydrogenases | Brain cancer | Genes | Stem cell transplantation | Cytotoxicity | Oncology | Bone tumors | Drug resistance | Aldehyde dehydrogenase | Cancer therapies | Ewing's sarcoma | Metastases | Allografts | Xenografts | Bone marrow | Remission | Biocompatibility | Population | Inhibition | Adolescents | Protein transport | Young adults | Bioavailability | Sensitivity | Inhibitors | Pancreatic cancer | Cell lines | Verapamil | Adults | Tumors | Apoptosis | Index Medicus
Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 2008, Volume 13, Issue 3, pp. 261 - 271
Malignant mesothelioma is a devastating disease that has been associated with loss of Neurofibromatosis type 2 ( ) and genetic lesions affecting and pathways.... 
CELLCYCLE | SITE-SPECIFIC RECOMBINATION | ONCOLOGY | DEFICIENCY COOPERATE | CRE RECOMBINASE | NEUROFIBROMATOSIS TYPE-2 GENE | TUMOR-SUPPRESSOR PROTEIN | LARGE-T-ANTIGEN | CROCIDOLITE ASBESTOS | SARCOMATOID MESOTHELIOMA | SPINDLE-CELL NEOPLASMS | MUTANT NF2 PROTEIN | CELL BIOLOGY | Immunohistochemistry | Thoracic Cavity - pathology | Epithelial Cells - metabolism | Mesothelioma - pathology | Luminescent Measurements | Loss of Heterozygosity | Tumor Suppressor Protein p53 - genetics | Neoplasms, Experimental - pathology | Mixed Tumor, Malignant - pathology | Time Factors | Cell Transformation, Neoplastic - genetics | Recombination, Genetic | Neoplasms, Experimental - genetics | Adenoviridae - genetics | Integrases - metabolism | Neurofibromatosis 2 - metabolism | Mixed Tumor, Malignant - metabolism | Epithelioid Cells - pathology | Neoplasm Invasiveness | Thoracic Neoplasms - pathology | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Epithelial Cells - pathology | Genotype | Mesothelioma - genetics | Neurofibromatosis 2 - genetics | Sarcoma - pathology | Cell Transformation, Neoplastic - metabolism | Thoracic Neoplasms - metabolism | Epithelioid Cells - metabolism | Mice, Knockout | Sarcoma - metabolism | Phenotype | Animals | Mesothelioma - metabolism | Thoracic Neoplasms - genetics | Cell Line, Tumor | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Mice | Neoplasms, Experimental - metabolism | Cell Transformation, Neoplastic - pathology | Genetic Vectors | Integrases - genetics | Thoracic Cavity - metabolism | Tumor proteins | Mesothelioma | Index Medicus
Journal Article
Neuroscience, ISSN 0306-4522, 2000, Volume 98, Issue 3, pp. 585 - 598
The aim of this investigation was to determine whether murine models of inflammatory, neuropathic and cancer pain are each characterized by a unique set of... 
dynorphin | bone | astrocyte hypertrophy | c-Fos protein | osteolysis | persistent pain | Astrocyte hypertrophy | Bone | Osteolysis | Persistent pain | Dynorphin | INBRED MOUSE STRAINS | GENE-RELATED PEPTIDE | ROOT GANGLION-CELLS | PRIMARY AFFERENT NEURONS | NITRIC-OXIDE SYNTHASE | NEUROSCIENCES | GROWTH-ASSOCIATED PROTEIN | PERIPHERAL TISSUE INFLAMMATION | MORPHINE-INDUCED ANALGESIA | SUBSTANCE-P RECEPTOR | C-FOS EXPRESSION | Neuritis - metabolism | Neoplasm Transplantation | Palpation | Sciatic Nerve - injuries | Spinal Cord - metabolism | Sarcoma, Experimental - metabolism | Astrocytes - pathology | Neuralgia - metabolism | Tumor Cells, Cultured - transplantation | Male | Neuritis - pathology | Neuralgia - pathology | Pain - metabolism | Neurons, Afferent - chemistry | Sarcoma, Experimental - complications | Axotomy | Sciatic Nerve - metabolism | Neuralgia - chemically induced | Pain - etiology | Receptors, Neurokinin-1 - metabolism | Spinal Cord - cytology | Dynorphins - metabolism | Osteolysis - pathology | Spinal Nerves - injuries | Disease Models, Animal | Physical Stimulation | Proto-Oncogene Proteins c-fos - analysis | Proto-Oncogene Proteins c-fos - metabolism | Receptors, Neurokinin-1 - analysis | Behavior, Animal - physiology | Spinal Nerves - metabolism | Dynorphins - analysis | Sarcoma, Experimental - pathology | Osteolysis - metabolism | Pain - pathology | Mice, Inbred C3H | Freund's Adjuvant | Animals | Fluorescent Antibody Technique | Ganglia, Spinal - pathology | Mice | Neurons, Afferent - metabolism | Ganglia, Spinal - metabolism | Index Medicus
Journal Article