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Journal of Applied Physiology, ISSN 8750-7587, 01/2013, Volume 114, Issue 1, pp. 131 - 147
We have previously reported chronic low-intensity interval exercise training attenuates fibrosis, impaired cardiac mitochondrial function, and coronary... 
Diastolic heart failure | Exercise | Coronary flow | Fibrosis | MVO | Speckle tracking | Hypertrophic signaling | Protein Kinases - genetics | Connectin | Heart - physiology | Natriuretic Peptide, Brain - metabolism | Ventricular Function, Left - genetics | Heart Failure - physiopathology | Extracellular Matrix - metabolism | Male | Oxygen Consumption - physiology | Tissue Inhibitor of Metalloproteinases - genetics | Hypertrophy, Left Ventricular - genetics | Swine | Myocardium - metabolism | Ventricular Remodeling - genetics | Sarcomeres - physiology | Proto-Oncogene Proteins c-akt - metabolism | Natriuretic Peptide, Brain - genetics | Fibrosis - physiopathology | Matrix Metalloproteinase 2 - metabolism | Sarcomeres - metabolism | Diastole - genetics | Heart Failure - genetics | Heart Failure - metabolism | Arterial Pressure - physiology | Diastole - physiology | Muscle Proteins - genetics | Matrix Metalloproteinase 2 - genetics | MAP Kinase Kinase 4 - genetics | Hypertrophy, Left Ventricular - physiopathology | Regional Blood Flow - physiology | Protein Kinases - metabolism | Regional Blood Flow - genetics | Myocardial Contraction - physiology | Collagen Type III - metabolism | Fibrosis - metabolism | Oxygen - metabolism | Proto-Oncogene Proteins c-akt - genetics | Extracellular Matrix - genetics | Myocardial Contraction - genetics | Arterial Pressure - genetics | MAP Kinase Kinase 4 - metabolism | Sarcomeres - genetics | Muscle Proteins - metabolism | Ventricular Function, Left - physiology | Citrate (si)-Synthase - metabolism | Heart Failure - rehabilitation | Fibrosis - genetics | Hypertrophy, Left Ventricular - metabolism | RNA, Messenger - genetics | Collagen Type III - genetics | Ventricular Remodeling - physiology | Physical Conditioning, Animal - physiology | Tissue Inhibitor of Metalloproteinases - metabolism | Animals | Oxygen Consumption - genetics | Citrate (si)-Synthase - genetics | Index Medicus | speckle tracking | coronary flow | hypertrophic signaling | exercise | fibrosis | diastolic heart failure | MV̇o2
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 19 - 26
Genetic mutations account for a significant percentage of cardiomyopathies, which are a leading cause of congestive heart failure... 
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Cardiomyopathy, Dilated - pathology | Humans | Heart Failure - physiopathology | Myosin Heavy Chains - genetics | Myosin Heavy Chains - metabolism | Arrhythmias, Cardiac - physiopathology | Sarcomeres - pathology | Arrhythmias, Cardiac - pathology | Mitochondria - genetics | Sarcomeres - genetics | Myocardium - metabolism | Heart Failure - etiology | Arrhythmias, Cardiac - genetics | Cardiac Myosins - metabolism | Arrhythmias, Cardiac - metabolism | Calcium-Binding Proteins - metabolism | Cardiomyopathy, Dilated - genetics | Cytoskeleton - pathology | Cardiomyopathy, Dilated - complications | Sarcomeres - metabolism | Cardiac Myosins - genetics | Cytoskeleton - genetics | Heart Failure - genetics | Myocardium - pathology | Arrhythmias, Cardiac - etiology | Heart Failure - metabolism | Mitochondria - metabolism | Heart Failure - pathology | Mitochondria - pathology | Cardiomyopathy, Dilated - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cardiomyopathy, Dilated - physiopathology | Cytoskeleton - metabolism | Mutation | Calcium-Binding Proteins - genetics | Gene mutations | Cardiomyopathy | Genetic aspects | Congestive heart failure | Diagnosis | Heart diseases | Genetic screening | Methods | Index Medicus | Abridged Index Medicus | Review Series
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 3730 - 3741
The clinical utility of anthracycline anticancer agents, especially doxorubicin, is limited by a progressive toxic cardiomyopathy linked to mitochondrial... 
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Antibiotics, Antineoplastic - pharmacology | Apoptosis - drug effects | Mitochondria, Heart - pathology | Antibiotics, Antineoplastic - adverse effects | Apoptosis - genetics | Male | Cardiomyopathies - enzymology | Necrosis - pathology | Proto-Oncogene Proteins c-akt - genetics | Sarcomeres - pathology | Cardiomyopathies - genetics | DNA, Mitochondrial - genetics | Carbon Monoxide - pharmacology | Sarcomeres - genetics | Caspase 3 - genetics | Caspase 3 - biosynthesis | Heme Oxygenase (Decyclizing) - metabolism | Mitochondria, Heart - genetics | Proto-Oncogene Proteins c-akt - metabolism | Heme Oxygenase (Decyclizing) - genetics | 3-Phosphoinositide-Dependent Protein Kinases | Protein-Serine-Threonine Kinases - metabolism | Antimetabolites - pharmacology | GATA4 Transcription Factor - metabolism | DNA, Mitochondrial - metabolism | Cells, Cultured | Gene Silencing | Mitochondria, Heart - enzymology | Protein-Serine-Threonine Kinases - genetics | Rats | Cardiomyopathies - pathology | GATA4 Transcription Factor - genetics | Guanylate Cyclase - metabolism | Myocardium - pathology | Necrosis - chemically induced | Myocardium - enzymology | Animals | Fibrosis | Guanylate Cyclase - genetics | Mice | Necrosis - genetics | Sarcomeres - enzymology | Doxorubicin - adverse effects | Doxorubicin - pharmacology | Necrosis - enzymology | Cardiomyopathies - chemically induced | Cardiomyopathy | Dosage and administration | Research | Drug therapy | Doxorubicin | Heart diseases | Risk factors | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, pp. e0181465 - e0181465
Journal Article