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1. Full Text Enrichment of endoplasmic reticulum with cholesterol inhibits sarcoplasmic-endoplasmic reticulum calcium ATPase-2b activity in parallel with increased order of membrane lipids: Implications for depletion of endoplasmic reticulum calcium stores and apoptosis in cholesterol-loaded macrophages
by Li, Yankun and Ge, Mingtao and Ciani, Laura and Kuriakose, George and Westover, Emily J and Dura, Miroslav and Covey, Douglas F and Freed, Jack H and Maxfield, Frederick R and Lytton, Jonathan and Tabas, Ira
Journal of Biological Chemistry, ISSN 0021-9258, 08/2004, Volume 279, Issue 35, pp. 37030 - 37039
Macrophages in advanced atherosclerotic lesions accumulate large amounts of unesterified, or "free," cholesterol (FC). FC accumulation induces macrophage... 
ACYLTRANSFERASE | ELECTRON-SPIN-RESONANCE | ACYL-COENZYME-A | PROTEIN | HUMAN ATHEROSCLEROTIC PLAQUES | BIOCHEMISTRY & MOLECULAR BIOLOGY | CA-ATPASE | FLUIDITY | STRESS | PHOSPHATIDYLCHOLINE | MICROSOMAL MEMBRANE | Microsomes - metabolism | Cyclodextrins - metabolism | Humans | Endoplasmic Reticulum - metabolism | Magnetics | Lipids - chemistry | Time Factors | Calcium-Transporting ATPases - metabolism | Cell Membrane - metabolism | Phospholipids - metabolism | Cell Line | Electron Spin Resonance Spectroscopy | Phosphatidylcholines - chemistry | Cholesterol - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases | Protein Folding | Macrophages - metabolism | Animals | beta-Cyclodextrins | Protein Isoforms | Protein Conformation | Mice | Subcellular Fractions | DNA, Complementary - metabolism | Apoptosis
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2. Full Text Sarco(endo)plasmic reticulum Ca²⁺-ATPase 2b is a major regulator of endoplasmic reticulum stress and glucose homeostasis in obesity
by Sang Won Park and Yingjiang Zhou and Jaemin Lee and Justin Lee and Umut Ozcan and Michael Karin
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 45, pp. 19320 - 19325
Increased endoplasmic reticulum (ER) stress is one of the central mechanisms that lead to dysregulated metabolic homeostasis in obesity. It is thus crucial to... 
Obesity | Messenger RNA | Calcium | Refeeding | Liver | Adenoviruses | Homeostasis | Mice | Type 2 diabetes mellitus | Insulin | Type 2 diabetes | Insulin resistance | Unfolded protein response | unfolded protein response | type 2 diabetes | calcium | INSULIN-RESISTANCE | ER STRESS | MULTIDISCIPLINARY SCIENCES | insulin resistance | obesity | Liver - metabolism | Stress, Physiological | Diabetes Mellitus - metabolism | Endoplasmic Reticulum - metabolism | Lipid Metabolism | Glucose Intolerance | Obesity - metabolism | Fatty Liver | Sarcoplasmic Reticulum Calcium-Transporting ATPases - physiology | Animals | Blood Glucose - metabolism | Ca super(2+)-transporting ATPase | Diabetes mellitus | Lipids | Triglycerides | Drug development | Gene expression | Blood | Stress | Glucose tolerance | Endoplasmic reticulum | Biological Sciences
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3. Full Text Cytokines Downregulate the Sarcoendoplasmic Reticulum Pump Ca2+ ATPase 2b and Deplete Endoplasmic Reticulum Ca2+, Leading to Induction of Endoplasmic Reticulum Stress in Pancreatic β-Cells
by Alessandra K. Cardozo and Fernanda Ortis and Joachim Storling and Ying-Mei Feng and Joanne Rasschaert and Morten Tonnesen and Françoise Van Eylen and Thomas Mandrup-Poulsen and André Herchuelz and Décio L. Eizirik
Diabetes, ISSN 0012-1797, 02/2005, Volume 54, Issue 2, pp. 452 - 461
Cytokines Downregulate the Sarcoendoplasmic Reticulum Pump Ca 2+ ATPase 2b and Deplete Endoplasmic Reticulum Ca 2+ , Leading to Induction of Endoplasmic... 
SKELETAL-MUSCLE | ACTIVATION | UNFOLDED PROTEIN RESPONSE | MESSENGER-RNA | INDUCED APOPTOSIS | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | MAMMALIAN-CELLS | ISLET CELLS | EXPRESSION | INDUCED GENES | Endoplasmic Reticulum - enzymology | Gene Expression Regulation, Enzymologic - drug effects | Islets of Langerhans - drug effects | Islets of Langerhans - physiology | Rats, Wistar | Islets of Langerhans - enzymology | Sarcoplasmic Reticulum - drug effects | Oxidative Stress - physiology | Rats | Calcium-Transporting ATPases - genetics | DNA Primers | Reverse Transcriptase Polymerase Chain Reaction | Sarcoplasmic Reticulum - enzymology | Animals | Calcium - physiology | Base Sequence | Endoplasmic Reticulum - drug effects | Thapsigargin - pharmacology | Cytokines - pharmacology
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4. The development of sarcoplasmic reticulum
by Martonosi, Anthony N
2000, ISBN 9057026023, xi, 618 p., [4] p. of plates
Sarcoplasmic reticulum is a form of endoplasmic reticulum found in large quantities in mature muscle cells. Anthony Martonosi presents general information... 
Heart | Sarcoplasmic reticulum | Contraction | Biology, life sciences
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5. Full Text Enhanced Sarcoplasmic Reticulum Ca2+ Leak and Increased Na+-Ca2+ Exchanger Function Underlie Delayed Afterdepolarizations in Patients With Chronic Atrial Fibrillation
by Voigt, N and Li, N and Wang, QL and Wang, W and Trafford, AW and Abu-Taha, I and Sun, Q and Wieland, T and Ravens, U and Nattel, S and Wehrens, XHT and Dobrev, D
CIRCULATION, ISSN 0009-7322, 05/2012, Volume 125, Issue 17, pp. 2059 - U98
Background-Delayed afterdepolarizations (DADs) carried by Na+-Ca2+-exchange current (I-NCX) in response to sarcoplasmic reticulum (SR) Ca2+ leak can promote... 
ACTION-POTENTIALS | CARDIAC & CARDIOVASCULAR SYSTEMS | CONTRACTILE DYSFUNCTION | HEART-FAILURE | DOWN-REGULATION | CARDIAC RYANODINE RECEPTOR | FKBP12.6 DEFICIENCY | sarcoplasmic reticulum | ryanodine receptor calcium release channel | CALCIUM-HANDLING ABNORMALITIES | atrial fibrillation | sodium-calcium exchanger | ADENOSINE A(2A) RECEPTORS | RECTIFIER POTASSIUM CURRENT | delayed afterdepolarizations | PERIPHERAL VASCULAR DISEASE | RAT VENTRICULAR MYOCYTES | Phosphorylation | Calcium - metabolism | Humans | Male | Ryanodine Receptor Calcium Release Channel - metabolism | Atrial Fibrillation - physiopathology | Recombinant Fusion Proteins - metabolism | Action Potentials | Biological Transport, Active | Lipid Bilayers | Caffeine - pharmacology | Female | Sarcoplasmic Reticulum - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Calmodulin - metabolism | Sodium-Calcium Exchanger - physiology | Sulfonamides - pharmacology | Gene Knock-In Techniques | Atrial Fibrillation - metabolism | Patch-Clamp Techniques | Animals | Membrane Potentials | Ryanodine Receptor Calcium Release Channel - genetics | Myocytes, Cardiac - physiology | Aged | Mice | Protein Processing, Post-Translational | Benzylamines - pharmacology | Chronic Disease | Measurement | Research | Sarcoplasmic reticulum | Atrial fibrillation | Tetracaine | ryanodine receptor | Na+-Ca2+-exchanger | sarcoplasmic reticulum Ca2+-leak
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6. Full Text Altered sarcoplasmic reticulum calcium cycling - Targets for heart failure therapy
by Kho, Changwon and Lee, Ahyoung and Hajjar, Roger J
Nature Reviews Cardiology, ISSN 1759-5002, 12/2012, Volume 9, Issue 12, pp. 717 - 733
Cardiac myocyte function is dependent on the synchronized movements of Ca2+ into and out of the cell, as well as between the cytosol and sarcoplasmic... 
PROTEIN-KINASE-A | POLYMORPHIC VENTRICULAR-TACHYCARDIA | OPERATED CA2+ ENTRY | BETA-ADRENERGIC STIMULATION | CARDIAC CONTRACTILE DYSFUNCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | STROMAL INTERACTION MOLECULE-1 | PRESSURE-OVERLOAD HYPERTROPHY | HUMAN DILATED CARDIOMYOPATHY | CHANNEL RYANODINE RECEPTOR | FAILING HUMAN MYOCARDIUM | Heart Failure - therapy | Animals | Excitation Contraction Coupling | Calcium - metabolism | Humans | Myocytes, Cardiac - metabolism | Heart Failure - metabolism | Sarcoplasmic Reticulum - metabolism | Heart failure | Care and treatment | Gene therapy | Properties | Sarcoplasmic reticulum | Methods
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7. Full Text Interactions between sarco-endoplasmic reticulum and mitochondria in cardiac and skeletal muscle-pivotal roles in Ca2+ and reactive oxygen species signaling
by Eisner, Verónica and Csordás, György and Hajnóczky, György
Journal of Cell Science, ISSN 0021-9533, 07/2013, Volume 126, Issue 14, pp. 2965 - 2978
Mitochondria are strategically and dynamically positioned in the cell to spatially coordinate ATP production with energy needs and to allow the local exchange... 
Mitochondria | Reactive oxygen species | Sarcoplasmic reticulum | Uniporter | ROS | Ryanodine receptor | MAM | Mitochondria-associated membranes | signalling | RAT-LIVER | MALIGNANT HYPERTHERMIA | ER-MITOCHONDRIAL | SUBCELLULAR COMPARTMENTALIZATION | NADPH OXIDASE ACTIVITY | SARCOPLASMIC-RETICULUM | PERMEABILITY TRANSITION | CELL BIOLOGY | CALCIUM-RELEASE CHANNEL | reactive oxygen species | IN-VIVO | Ca2+ signalling | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Mitochondrial Membranes - physiology | Humans | Endoplasmic Reticulum - physiology | Muscle, Skeletal - ultrastructure | Sarcoplasmic Reticulum - physiology | Muscle, Skeletal - physiology | Biological Transport, Active | Animals | Mitochondria, Heart - physiology | Myocytes, Cardiac - physiology | Mitochondria, Muscle - physiology | Myocytes, Cardiac - ultrastructure | Calcium Signaling
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8. The sarcoplasmic reticulum
: transport and energy transduction
by Meis, Leopoldo de
1981, Transport in the life sciences, ISBN 0471050253, Volume 2, xv, 163
Bioenergetics | Calcium | Biological transport | Calcium metabolism | Sarcoplasmic Reticulum | Metabolism | Energy transfer
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9. Full Text Role of sarco/endoplasmic reticulum calcium content and calcium ATPase activity in the control of cell growth and proliferation
by Lipskaia, Larissa and Hulot, Jean-Sébastien and Lompré, Anne-Marie
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 1/2009, Volume 457, Issue 3, pp. 673 - 685
Ca2+, the main second messenger, is central to the regulation of cellular growth. There is increasing evidence that cellular growth and proliferation are... 
Calcium signaling | Biomedicine | Human Physiology | Sarcoplasmic reticulum calcium transporting ATPases | Cell growth processes | PHYSIOLOGY | SARCOPLASMIC-RETICULUM | CAPACITATIVE CA2+ ENTRY | PULMONARY-ARTERY MYOCYTES | ACTIVATED T-CELLS | STAGE HEART-FAILURE | SMOOTH-MUSCLE-CELLS | ENDOPLASMIC-RETICULUM | NUCLEAR FACTOR | INTERACTION MOLECULE-1 STIM1 | RECEPTOR TYROSINE KINASE | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Animals | Cell Proliferation | Calcium - metabolism | Isoenzymes - metabolism | Calcium Signaling - physiology | Humans | Endoplasmic Reticulum - metabolism | Sarcoplasmic Reticulum - metabolism | Growth | Adenosine triphosphatase | Cell growth | Cells | Life Sciences | Human health and pathology | Calcium | Isoenzymes | Endoplasmic Reticulum | Sarcoplasmic Reticulum | Sarcoplasmic Reticulum Calcium-Transporting ATPases | Calcium Signaling | Cardiology and cardiovascular system
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10. Full Text Enhanced activity of multiple TRIC‐B channels: an endoplasmic reticulum/sarcoplasmic reticulum mechanism to boost counterion currents
by O'Brien, Fiona and Eberhardt, David and Witschas, Katja and El‐Ajouz, Sam and Iida, Tsunaki and Nishi, Miyuki and Takeshima, Hiroshi and Sitsapesan, Rebecca and Venturi, Elisa
The Journal of Physiology, ISSN 0022-3751, 05/2019, Volume 597, Issue 10, pp. 2691 - 2705
There are two subtypes of trimeric intracellular cation (TRIC) channels but their distinct single‐channel properties and physiological regulation have not been... 
release | TRIC channels | sarcoplasmic reticulum | Ryanodine receptor | CONDUCTANCE | VOLTAGE | PHYSIOLOGY | MUSCLE | BLOCK | SELECTIVE CHANNEL | NEUROSCIENCES | CALCIUM-RELEASE CHANNELS | CA2 | FRAGMENTED SARCOPLASMIC-RETICULUM | DELETION MUTATION | Ca2+ release | K+ CHANNELS | Muscles | Physiological aspects | Musculoskeletal system | Channel opening | Channel gating | Sarcoplasmic reticulum | Receptor channels | ISDN | Rodents | Potassium | Endoplasmic reticulum | Contraction | Skeletal muscle | Research Paper | Muscle
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11. Full Text Ca2+ spark‐dependent and ‐independent sarcoplasmic reticulum Ca2+ leak in normal and failing rabbit ventricular myocytes
by Zima, Aleksey V and Bovo, Elisa and Bers, Donald M and Blatter, Lothar A
The Journal of Physiology, ISSN 0022-3751, 12/2010, Volume 588, Issue 23, pp. 4743 - 4757
Sarcoplasmic reticulum (SR) Ca 2+ leak is an important component of cardiac Ca 2+ signalling. Together with the SR Ca 2+ ‐ATPase (SERCA)‐mediated Ca 2+ uptake,... 
CHRONIC HEART-FAILURE | CELLS | PHYSIOLOGY | EVENTS | LOAD | SR CA2 | CARDIAC MYOCYTES | MUSCLE | LUMINAL CA2 | CHANNEL RYANODINE RECEPTOR | NEUROSCIENCES | SPONTANEOUS CALCIUM-RELEASE | Rabbits | Animals | Calcium - metabolism | Calcium Signaling - physiology | Myocytes, Cardiac - metabolism | Sarcoplasmic Reticulum - physiology | Ryanodine Receptor Calcium Release Channel - metabolism | Heart Failure - metabolism | Cardiovascular
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12. Full Text Calmodulin kinase II-mediated sarcoplasmic reticulum Ca2+ leak promotes atrial fibrillation in mice
by Chelu, Mihail G and Sarma, Satyam and Sood, Subeena and Wang, Sufen and van Oort, Ralph J and Skapura, Darlene G and Li, Na and Santonastasi, Marco and Müller, Frank Ulrich and Schmitz, Wilhelm and Schotten, Ulrich and Anderson, Mark E and Valderrábano, Miguel and Dobrev, Dobromir and Wehrens, Xander H. T
Journal of clinical investigation, ISSN 0021-9738, 2009, Volume 119, Issue 7, pp. 1940 - 1951
A trial fibrillation (AF), the most common human cardiac arrhythmia, is associated with abnormal intracellular Ca2+ handling. Diastolic Ca2+ release from the... 
CANINE PULMONARY VEINS | MEDICINE, RESEARCH & EXPERIMENTAL | POLYMORPHIC VENTRICULAR-TACHYCARDIA | RYANODINE RECEPTOR | SUDDEN-DEATH | HEART-FAILURE | CALCIUM-RELEASE | CARDIAC-HYPERTROPHY | DOWN-REGULATION | FKBP12.6 DEFICIENCY | TRANSGENIC MICE | Heart Atria - pathology | Phosphorylation | Cyclic AMP Response Element Modulator - physiology | Goats | Calcium - metabolism | Peptides - physiology | Humans | Mice, Transgenic | Cardiac Pacing, Artificial | Heart Atria - metabolism | Atrial Fibrillation - metabolism | Animals | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - physiology | Atrial Fibrillation - etiology | Electrocardiography | Mice | Sarcoplasmic Reticulum - metabolism | Atrial Fibrillation - prevention & control | Ryanodine Receptor Calcium Release Channel - physiology
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