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Nature Cell Biology, ISSN 1465-7392, 03/2018, Volume 20, Issue 3, pp. 307 - 319
Reactive oxygen species (ROS) contribute to tissue damage and remodelling mediated by the inflammatory response after injury. Here we show that ROS, which... 
STEM-CELLS | PHAGOCYTOSIS | ACTIVATION | SUPEROXIDE-PRODUCTION | ROS | KINASE | PTEN | NEURONS | MECHANISMS | OUTGROWTH | CELL BIOLOGY | Ganglia, Spinal - injuries | Sciatic Nerve - injuries | Axons - enzymology | Reactive Oxygen Species - metabolism | Exosomes - pathology | Sciatic Nerve - physiopathology | Spinal Cord Injuries - genetics | Spinal Cord Injuries - pathology | Endocytosis | CX3C Chemokine Receptor 1 - metabolism | Ganglia, Spinal - enzymology | Peripheral Nerve Injuries - physiopathology | Spinal Cord Injuries - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Dyneins - metabolism | Cell Line | NADPH Oxidase 2 - deficiency | Macrophages - pathology | Signal Transduction | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Endosomes - pathology | Nerve Regeneration | Macrophages - enzymology | Mice, Knockout | Animals | Peripheral Nerve Injuries - enzymology | NADPH Oxidase 2 - metabolism | Axons - pathology | Ganglia, Spinal - pathology | Exosomes - enzymology | Endosomes - enzymology | Spinal Cord Injuries - physiopathology | NADPH Oxidase 2 - genetics | Nerve Degeneration | Peripheral Nerve Injuries - genetics | Sciatic Nerve - enzymology | Axons | Regeneration | Reactive oxygen species | NADP (Coenzyme) | Nervous system | Cellular signal transduction | Research | Gene expression | Oxidase | Recovery of function | Spinal injuries | CX3CR1 protein | Inflammatory response | AKT protein | Exosomes | Macrophages | Inactivation | NAD(P)H oxidase | Signal transduction | Dorsal root ganglia | Neurodegeneration | Dieback | CYBB protein | Degeneration | Cell body | Oxygen | Deactivation | Inflammation | Ganglia | 1-Phosphatidylinositol 3-kinase | Signaling | Dynein | PTEN protein | Sciatic nerve | Endosomes
Journal Article
Glia, ISSN 0894-1491, 09/2018, Volume 66, Issue 9, pp. 1960 - 1971
Myelinating glial cells (MGCs), oligodendrocytes (OLs) in the central nervous system (CNS) and Schwann cells (SCs) in the peripheral nervous system (PNS),... 
NG2 | neurofilament | PLP | axon integrity | sciatic nerve | RANVIER | MECHANISMS | AMYOTROPHIC-LATERAL-SCLEROSIS | MICRORNAS | MATURATION | NEUROSCIENCES | OLIGODENDROCYTE DIFFERENTIATION | SUPPORT | METABOLISM | INTEGRITY | ABLATION | Motor Activity - physiology | Optic Nerve - pathology | Axons - enzymology | Male | Paralysis - pathology | Atrophy | Spinal Cord - pathology | DEAD-box RNA Helicases - deficiency | Female | Myelin Sheath - enzymology | Paralysis - enzymology | Ataxia - pathology | Nerve Degeneration - enzymology | Optic Nerve - enzymology | Ribonuclease III - genetics | Myelin Sheath - pathology | Sciatic Nerve - pathology | White Matter - enzymology | Mice, Inbred C57BL | Spinal Cord - enzymology | Mice, Transgenic | Ribonuclease III - deficiency | Disease Progression | Nerve Degeneration - pathology | White Matter - pathology | DEAD-box RNA Helicases - genetics | Animals | Ataxia - enzymology | Axons - pathology | Sciatic Nerve - enzymology | Physiological aspects | Enzymes | Recombination | Peripheral nerves | Central nervous system | Motors | Nervous system | Glial cells | Clonal deletion | Sheaths | Myelin proteolipid protein | Demyelination | Neurodegeneration | Rodents | Oligodendrocytes | Nerves | Deletion | Ataxia | Degeneration | Paralysis | Peripheral nervous system | Cell survival | Myelin | Schwann cells | Tamoxifen | Survival | Axons | Myelination | Mice
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2013, Volume 123, Issue 7, pp. 3002 - 3013
Peripheral neuropathic pain is a disabling condition resulting from nerve injury. It is characterized by the dysregulation of voltage-gated sodium channels... 
ALPHA-SUBUNITS | MEDICINE, RESEARCH & EXPERIMENTAL | NA(V)1.7 | RECORDINGS | NOCICEPTIVE NEURONS | NA+ CHANNEL | GENE DELETION | MODEL | DORSAL-ROOT GANGLION | SPARED NERVE INJURY | EXPRESSION | Ubiquitin | Research | Ligases | Health aspects | Peripheral nerve diseases | Proteins | Neurosciences | Behavior | Experiments | Pain management | Colleges & universities
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0152750
Modulation of extracellular matrix (ECM) remodeling after peripheral nerve injury (PNI) could represent a valid therapeutic strategy to prevent maladaptive... 
NEUROPATHIC PAIN | ASTROGLIOSIS | REGENERATION | MULTIDISCIPLINARY SCIENCES | DISEASE | EXTRACELLULAR-MATRIX | AMYOTROPHIC-LATERAL-SCLEROSIS | GROWTH-FACTOR | MODEL | EXPRESSION | MALADAPTIVE PLASTICITY | Glutamate Plasma Membrane Transport Proteins - metabolism | Glycine Plasma Membrane Transport Proteins - metabolism | Sciatic Nerve - injuries | gamma-Aminobutyric Acid - metabolism | Astrocytes - pathology | Male | Astrocytes - enzymology | Synapses - pathology | Anterior Horn Cells - enzymology | Spinal Cord - pathology | Gelatinases - metabolism | Microglia - pathology | Gelatinases - antagonists & inhibitors | Sciatic Nerve - pathology | Dipeptides - pharmacology | Anterior Horn Cells - pathology | Peripheral Nerve Injuries - pathology | Spinal Cord - enzymology | Microglia - enzymology | Rats | Synapses - enzymology | Rats, Sprague-Dawley | Animals | Peripheral Nerve Injuries - enzymology | Glutamic Acid - metabolism | Sciatic Nerve - enzymology | Care and treatment | Proteases | Physiological aspects | Extracellular matrix | Genetic aspects | Research | Peripheral nerve diseases | Ventral horn | Spinal cord | Matrix metalloproteinases | Central nervous system | Homeostasis | Nerve growth factor | Spinal cord injury | Glycine | Neuronal-glial interactions | Infusion | γ-Aminobutyric acid | Pain | Motor neuron diseases | Rodents | Modulation | Inhibition | Paralysis | Enzymes | Cytokines | Neurons | Glial fibrillary acidic protein | Amyotrophic lateral sclerosis | Liquid chromatography | High-performance liquid chromatography | Microglia | Injury prevention | Gliosis | Plasticity (synaptic) | Glycine transporter | Correlation analysis | Spinal cord injuries | Glutamic acid transporter | Transporter | Sodium-glutamic acid cotransporter | Sciatic nerve
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 2013, Volume 216, Issue 1, pp. 1 - 11
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 11/2017, Volume 216, Issue 11, pp. 3655 - 3675
Axon degeneration is an early event and pathological in neurodegenerative conditions and nerve injuries. To discover agents that suppress neuronal death and... 
NEURONAL APOPTOSIS | AXON DEGENERATION | WALLERIAN DEGENERATION | SYMPATHETIC NEURONS | INHIBITOR FORETINIB | CYTOCHROME-C RELEASE | RECEPTOR TYROSINE-KINASE | NERVE GROWTH-FACTOR | CELL-DEATH | STATISTICAL-MODEL | CELL BIOLOGY
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 10/2017, Volume 106, pp. 147 - 157
Journal Article
Nature Neuroscience, ISSN 1097-6256, 04/2011, Volume 14, Issue 4, pp. 429 - 436
Journal Article
The Journal of cell biology, 11/2017, Volume 216, Issue 11, p. 3655
Axon degeneration is an early event and pathological in neurodegenerative conditions and nerve injuries. To discover agents that suppress neuronal death and... 
Axons - enzymology | Apoptosis - drug effects | Crush Injuries - genetics | Motor Neurons - pathology | Wallerian Degeneration | Neuroprotective Agents - pharmacology | Time Factors | Superoxide Dismutase-1 - metabolism | Transcription, Genetic | Cytoprotection | Disease Models, Animal | Signal Transduction | Adrenergic Fibers - pathology | Sciatic Neuropathy - pathology | Genotype | Mice, Transgenic | Mitochondria - pathology | Motor Neurons - enzymology | Rats, Sprague-Dawley | Sciatic Neuropathy - enzymology | Apoptosis Regulatory Proteins - metabolism | Phenotype | Axons - pathology | Neurons - enzymology | Anilides - pharmacology | Sensory Receptor Cells - enzymology | Mutation | Mitochondria - enzymology | Neurons - pathology | Phosphorylation | Sciatic Nerve - injuries | Receptor, trkA - antagonists & inhibitors | Adrenergic Fibers - drug effects | Adrenergic Fibers - enzymology | Quinolines - pharmacology | Dose-Response Relationship, Drug | Sciatic Neuropathy - genetics | Apoptosis Regulatory Proteins - genetics | Crush Injuries - pathology | Sciatic Nerve - drug effects | Neurons - drug effects | Sciatic Neuropathy - drug therapy | Motor Neurons - drug effects | Sensory Receptor Cells - pathology | Sciatic Nerve - pathology | Mice, Inbred C57BL | Cells, Cultured | Axons - drug effects | Crush Injuries - enzymology | Mitochondria - drug effects | Animals | Sensory Receptor Cells - drug effects | Receptor, trkA - metabolism | Superoxide Dismutase-1 - genetics | Receptor, trkA - genetics | Protein Kinase Inhibitors - pharmacology | Crush Injuries - drug therapy | Sciatic Nerve - enzymology
Journal Article
Journal Article