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Experimental Neurology, ISSN 0014-4886, 12/2012, Volume 238, Issue 2, pp. 225 - 234
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2013, Volume 110, Issue 2, pp. 690 - 695
Diabetic peripheral neuropathy (DPN) is the most common complication in both type 1 and type 2 diabetes. Here we studied some phenotypic features of a... 
Antioxidants | Diabetic neuropathies | Animal models | Diabetes complications | Neurons | Spinal ganglia | Nerves | Type 2 diabetes mellitus | Peripheral nervous system diseases | Type 1 diabetes mellitus | Electrophysiology | Neurodegeneration | Neuropeptide | neuropeptide | CONDUCTION-VELOCITY | MULTIDISCIPLINARY SCIENCES | neurodegeneration | SPINAL-CORD | DORSAL-ROOT GANGLION | FACTOR NGF | NERVE GROWTH-FACTOR | electrophysiology | C-PEPTIDE REPLACEMENT | MITOCHONDRIAL DYSFUNCTION | BB-WISTAR RAT | PRIMARY SENSORY NEURONS | CUTANEOUS INNERVATION | Immunohistochemistry | Sciatic Nerve - injuries | Electric Stimulation | Age Factors | Receptors, Leptin - genetics | Hyperalgesia - pathology | Ubiquinone - pharmacology | In Situ Hybridization | Peripheral Nervous System Diseases - prevention & control | Mice, Mutant Strains | Statistics, Nonparametric | Neural Conduction - drug effects | Neurons - drug effects | Diabetes Mellitus, Type 2 - complications | Peripheral Nervous System Diseases - etiology | Phospholipase C beta - metabolism | Sciatic Nerve - pathology | Mice, Inbred C57BL | Ubiquinone - analogs & derivatives | Blotting, Western | Receptors, Leptin - deficiency | Animals | Mice | Diabetes Mellitus, Type 2 - pathology | Ganglia, Spinal - drug effects | Ganglia, Spinal - metabolism | Biological Sciences | Neurologi | Endokrinologi och diabetes | Klinisk medicin | Medicin och hälsovetenskap
Journal Article
Journal Article
Neuron, ISSN 0896-6273, 08/2012, Volume 75, Issue 4, pp. 633 - 647
The radical response of peripheral nerves to injury (Wallerian degeneration) is the cornerstone of nerve repair. We show that activation of the transcription... 
SCIATIC-NERVE | MYELINATION | RAT | SIGNALING PATHWAY | PERIPHERAL NERVOUS-SYSTEM | WALLERIAN DEGENERATION | IN-VIVO | AXONAL REGENERATION | DORSAL-ROOT GANGLION | NEUROSCIENCES | NEGATIVE REGULATOR | Nerve Regeneration - physiology | Benzofurans | Cell Movement - genetics | Motor Neurons - pathology | Motor Neurons - ultrastructure | Spinal Cord - pathology | Sciatic Neuropathy - therapy | Adenoviridae - genetics | Disease Models, Animal | Microfluidic Analytical Techniques | Macrophages - ultrastructure | Microscopy, Electron, Transmission | Macrophages - pathology | Myelin Sheath - pathology | Gene Expression Regulation - genetics | Proto-Oncogene Proteins c-jun - genetics | Sciatic Neuropathy - pathology | Mice, Transgenic | Schwann Cells - pathology | Schwann Cells - metabolism | Motor Neurons - metabolism | Macrophages - metabolism | Animals | Schwann Cells - ultrastructure | Analysis of Variance | Proto-Oncogene Proteins c-jun - metabolism | Sciatic Neuropathy - physiopathology | Genetic Vectors - physiology | Mice | Myelin Sheath - ultrastructure | Sciatic Neuropathy - metabolism | Neurons | Neurosciences | Medical research | Genotype & phenotype | Transcription factors | Rodents | Leprosy | Grants | Mammals | Gene expression | Regeneration | Myelin | Neurodegeneration | Peripheral nerves | Schwann cells | Recovery of function | c-Jun protein | Trophic factors | Injuries | Radicals | Cell adhesion molecules | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Journal Article
Experimental Neurology, ISSN 0014-4886, 2010, Volume 223, Issue 2, pp. 599 - 608
Bortezomib, a proteasome inhibitor, is an antineoplastic drug to treat multiple myeloma and mantle cell lymphoma. Its most clinically significant adverse event... 
Mice | Bortezomib | Neuropathy | Chemotherapy-induced peripheral neuropathy | Proteasome inhibitor | MULTIPLE-MYELOMA | PLUS MELPHALAN | GENE-RELATED PEPTIDE | SUDOMOTOR FUNCTION | PROTEASOME INHIBITION | DORSAL-ROOT GANGLIA | COMBINATION | CANCER | NEUROSCIENCES | PROGRAMMED CELL-DEATH | PERIPHERAL NEUROPATHY | Boronic Acids - toxicity | Immunohistochemistry | Peripheral Nervous System Diseases - chemically induced | Tibial Nerve - pathology | Electrophysiology | Myelin Sheath - drug effects | Nerve Fibers, Unmyelinated - ultrastructure | Pain - chemically induced | Motor Neurons - pathology | Antineoplastic Agents - toxicity | Pyrazines - toxicity | Protease Inhibitors - toxicity | Nerve Fibers, Unmyelinated - drug effects | Female | Neural Conduction - drug effects | Peripheral Nervous System Diseases - pathology | Sciatic Nerve - drug effects | Action Potentials - drug effects | Motor Neurons - drug effects | Sensory Receptor Cells - pathology | Myelin Sheath - pathology | Sciatic Nerve - pathology | Microscopy, Electron | Pain - pathology | Nerve Fibers, Unmyelinated - pathology | Animals | Sensory Receptor Cells - drug effects | Ganglia, Spinal - pathology | Skin - innervation | Myelin Sheath - ultrastructure | Tibial Nerve - drug effects | Ganglia, Spinal - drug effects | Chemotherapy | Neurosciences | Multiple myeloma | Physiological aspects | Lymphomas | Chemical properties | Cancer
Journal Article
Journal Article
Journal of Neurology, Neurosurgery and Psychiatry, ISSN 0022-3050, 2018, Volume 90, Issue 4, pp. 424 - 427
Journal Article
Cancer Research, ISSN 0008-5472, 02/2018, Volume 78, Issue 3, pp. 817 - 829
Journal Article