X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (1215) 1215
Publication (114) 114
Newsletter (46) 46
Newspaper Article (19) 19
Book / eBook (14) 14
Book Review (11) 11
Book Chapter (10) 10
Conference Proceeding (9) 9
Government Document (1) 1
Magazine Article (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (867) 867
scoliosis (745) 745
female (597) 597
index medicus (528) 528
male (480) 480
adolescent (471) 471
child (373) 373
orthopedics (283) 283
adult (261) 261
animals (230) 230
scoliosis - metabolism (215) 215
clinical neurology (187) 187
adolescent idiopathic scoliosis (164) 164
surgery (151) 151
scoliosis - genetics (148) 148
endocrinology & metabolism (140) 140
scoliosis - etiology (140) 140
scoliosis - pathology (139) 139
research (136) 136
middle aged (133) 133
scoliosis - surgery (133) 133
mutation (132) 132
analysis (131) 131
idiopathic scoliosis (128) 128
spine (127) 127
scoliosis - physiopathology (117) 117
children (112) 112
pediatrics (108) 108
medicine & public health (105) 105
child, preschool (101) 101
osteoporosis (101) 101
radiography (96) 96
bones (88) 88
genetics & heredity (88) 88
bone density (87) 87
genetic aspects (86) 86
medicine (86) 86
care and treatment (85) 85
patients (84) 84
physiological aspects (81) 81
scoliosis - complications (81) 81
aged (79) 79
young adult (79) 79
risk factors (78) 78
scoliosis - diagnostic imaging (78) 78
pathogenesis (77) 77
metabolism (76) 76
growth (73) 73
mice (73) 73
proteins (73) 73
age (72) 72
density (70) 70
phenotype (70) 70
case-control studies (69) 69
diagnosis (68) 68
genes (67) 67
melatonin (67) 67
health aspects (64) 64
neurosciences (64) 64
article (63) 63
collagen (62) 62
disease models, animal (61) 61
medical research (61) 61
expression (60) 60
gene expression (60) 60
bone (59) 59
genetics (57) 57
research article (57) 57
fractures (56) 56
bone-mineral density (54) 54
scoliosis - blood (54) 54
studies (54) 54
development and progression (53) 53
disease (53) 53
medicine, research & experimental (53) 53
multidisciplinary sciences (53) 53
bone mineral density (52) 52
rats (52) 52
infant (51) 51
medicine, experimental (51) 51
neurosurgery (51) 51
gene (50) 50
pain (49) 49
biochemistry & molecular biology (48) 48
vertebrae (48) 48
prospective studies (47) 47
genetic disorders (46) 46
cells, cultured (45) 45
intervertebral disc - metabolism (45) 45
osteopenia (45) 45
surgical orthopedics (45) 45
treatment outcome (45) 45
cell biology (44) 44
spinal fusion (44) 44
retrospective studies (43) 43
rheumatology (43) 43
scoliosis - diagnosis (43) 43
original (42) 42
pedigree (41) 41
abnormalities (40) 40
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (1212) 1212
German (24) 24
Chinese (19) 19
Russian (19) 19
French (7) 7
Italian (7) 7
Polish (5) 5
Czech (2) 2
Japanese (2) 2
Spanish (2) 2
Turkish (2) 2
Hungarian (1) 1
Portuguese (1) 1
Slovenian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Trends in Molecular Medicine, ISSN 1471-4914, 2016, Volume 22, Issue 7, pp. 615 - 628
A key aspect of cellular function is the proper assembly and utilization of ribonucleoproteins (RNPs). Recent studies have shown that hyper- or hypo-assembly... 
Pathology | stress granules | dyskeratosis congenita | spinal muscular atrophy | RNA quality control | RNP assembly | therapy | MEDICINE, RESEARCH & EXPERIMENTAL | MYOTONIC-DYSTROPHY TYPE-1 | PRECURSOR RIBOSOMAL-RNA | C9ORF72 REPEAT EXPANSIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | POLY(A)-SPECIFIC RIBONUCLEASE PARN | CARTILAGE-HAIR HYPOPLASIA | AMYOTROPHIC-LATERAL-SCLEROSIS | DISORDER MOPD I | CELL BIOLOGY | EUKARYOTIC STRESS GRANULES | SPINAL MUSCULAR-ATROPHY | DYSKERATOSIS-CONGENITA | Immunologic Deficiency Syndromes - pathology | Microcephaly - genetics | Scoliosis - genetics | Hair - abnormalities | Humans | Walker-Warburg Syndrome - genetics | Muscular Dystrophies - genetics | Ribonucleoproteins - analysis | Dwarfism - metabolism | Osteochondrodysplasias - genetics | Walker-Warburg Syndrome - metabolism | Hair - pathology | Muscular Atrophy, Spinal - metabolism | Parkinson Disease - pathology | Muscular Dystrophies - metabolism | Hirschsprung Disease - pathology | Fetal Growth Retardation - metabolism | Ribonucleoproteins - metabolism | RNA Stability | Muscular Atrophy, Spinal - pathology | Fetal Growth Retardation - pathology | Osteochondrodysplasias - congenital | Mutation | Osteochondrodysplasias - metabolism | Osteochondrodysplasias - pathology | Hirschsprung Disease - genetics | Hair - metabolism | Fetal Growth Retardation - genetics | Dyskeratosis Congenita - genetics | Scoliosis - pathology | Muscular Atrophy, Spinal - genetics | Microcephaly - pathology | Ribonucleoproteins - genetics | Dyskeratosis Congenita - metabolism | Parkinson Disease - metabolism | Dwarfism - pathology | Walker-Warburg Syndrome - pathology | Mallory Bodies - genetics | Mallory Bodies - pathology | Microcephaly - metabolism | Scoliosis - metabolism | Parkinson Disease - genetics | Muscular Dystrophies - pathology | Dwarfism - genetics | Mallory Bodies - metabolism | Animals | Dyskeratosis Congenita - pathology | Immunologic Deficiency Syndromes - genetics | Immunologic Deficiency Syndromes - metabolism | Hirschsprung Disease - metabolism | RNA | Therapy | Spinal Muscular Atrophy | Stress granules | Dyskeratosis Congenita
Journal Article
Scientific Reports, ISSN 2045-2322, 03/2017, Volume 7, Issue 1, p. 44260
The primary cilium is an outward projecting antenna-like organelle with an important role in bone mechanotransduction. The capacity to sense mechanical stimuli... 
LOCALIZATION | FLUID SHEAR-STRESS | MELATONIN SIGNALING DYSFUNCTION | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY LOCI | OSTEOGENIC DIFFERENTIATION | BONE-CELLS | WNT | ASSOCIATION | MECHANOSENSORS | FLOW | Paired Box Transcription Factors - genetics | Bone and Bones - pathology | Cell Adhesion Molecules - genetics | Scoliosis - genetics | Receptors, G-Protein-Coupled - metabolism | Homeodomain Proteins - metabolism | Humans | Gene Expression Profiling | Scoliosis - pathology | Exome | Bone and Bones - metabolism | Kinesin - genetics | Female | Fibroblast Growth Factor 3 - metabolism | Mechanotransduction, Cellular - genetics | Osteogenesis - genetics | Cilia - pathology | Fibroblast Growth Factor 3 - genetics | Gene Expression Regulation | Scoliosis - metabolism | Transcription Factors - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Cell Adhesion Molecules - metabolism | T-Box Domain Proteins - genetics | Cilia - metabolism | Homeodomain Proteins - genetics | T-Box Domain Proteins - metabolism | Cilia - genetics | Kinesin - metabolism | Transcription Factors - metabolism | Carrier Proteins - genetics | Osteoblasts - pathology | Carrier Proteins - metabolism | Receptor Protein-Tyrosine Kinases - genetics | Adolescent | Receptors, G-Protein-Coupled - genetics | Primary Cell Culture | Osteoblasts - metabolism | Paired Box Transcription Factors - metabolism | Animal models | Mechanotransduction | Mechanical stimuli | Scoliosis | Elongation | Cilia
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 23, Issue 12, pp. 2242 - 2251
Summary Objective Lumbar facet joint degeneration (FJD) may be an important cause of low back pain (LBP) and sciatica. The goal of this study was to... 
Rheumatology | Human facet joint capsular tissue | Angiogenesis | Inflammatory cytokines | Nerve ingrowth | Low back pain | Inflammatory pain mediators | GENE-RELATED PEPTIDE | LOW-BACK-PAIN | OSTEOARTHRITIS PAIN | CLINICAL-FEATURES | RHEUMATOLOGY | Angiogenesis Nerve ingrowth | NERVE GROWTH-FACTOR | DISC DEGENERATION | LUMBAR SPINAL-DISORDERS | SUBSTANCE-P | ORTHOPEDICS | UP-REGULATION | Immunohistochemistry | Up-Regulation | Low Back Pain - immunology | Scoliosis - genetics | Zygapophyseal Joint - metabolism | Coculture Techniques | Humans | Middle Aged | Intervertebral Disc Degeneration - immunology | Male | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Intervertebral Disc Degeneration - metabolism | Young Adult | Osteoarthritis, Spine - genetics | Aged, 80 and over | Low Back Pain - genetics | Adult | Female | Cytokines - genetics | Real-Time Polymerase Chain Reaction | Spondylolisthesis - genetics | Ganglia, Spinal | Spondylolisthesis - immunology | Cytokines - immunology | Intervertebral Disc Degeneration - genetics | Joint Capsule - metabolism | Osteoarthritis, Spine - metabolism | Spondylolisthesis - metabolism | Nerve Growth Factor - metabolism | Cytokines - metabolism | Rats | Scoliosis - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Blotting, Western | Osteoarthritis, Spine - immunology | Scoliosis - immunology | Animals | Zygapophyseal Joint - immunology | Receptor, trkA - metabolism | Joint Capsule - immunology | Aged | Lumbar Vertebrae | Low Back Pain - metabolism | Cadaver | Sects | Backache | Neovascularization | Enzymes | Endothelial growth factors | Analysis | nerve ingrowth | inflammatory pain mediators | human facet joint capsular tissue | inflammatory cytokines | low back pain | angiogenesis
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e83080
Aberrant proliferation of nucleus pulposus cell is implicated in the pathogenesis of intervertebral disc degeneration. Recent findings revealed that microRNAs,... 
OVARIAN-CANCER CELLS | BREAST-CANCER | TRANSFORMATION | INVASION | METASTASIS | SIGNALING PATHWAY | MIR-B-10 | MULTIDISCIPLINARY SCIENCES | GROWTH | UP-REGULATION | EXPRESSION | RNA, Small Interfering - genetics | Phosphorylation | Cell Proliferation | rho GTP-Binding Proteins - antagonists & inhibitors | Homeodomain Proteins - metabolism | Humans | Middle Aged | Spine - metabolism | Male | MicroRNAs - metabolism | Proto-Oncogene Proteins c-akt - genetics | Intervertebral Disc Degeneration - metabolism | Intervertebral Disc Degeneration - pathology | Adult | Female | Proto-Oncogene Proteins c-akt - metabolism | Spine - pathology | Chondrocytes - metabolism | Intervertebral Disc Degeneration - genetics | Chondrocytes - pathology | Signal Transduction | rho GTP-Binding Proteins - genetics | Gene Expression Regulation | rhoC GTP-Binding Protein | Transcription Factors - genetics | Homeodomain Proteins - genetics | Transcription Factors - metabolism | rho GTP-Binding Proteins - metabolism | MicroRNAs - genetics | Primary Cell Culture | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | RNA, Small Interfering - metabolism | Physiological aspects | Development and progression | MicroRNA | Comparative analysis | Scoliosis | Cell proliferation | Disease | Laboratories | Pathogenesis | AKT protein | Nucleus pulposus | Intervertebral discs | Tissues | Nuclei | Stomach cancer | Ovarian cancer | Cell growth | Rodents | Surgery | Degeneration | Inhibition | MiRNA | Hospitals | Ribonucleic acids | MicroRNAs | Medical prognosis | Nuclei (cytology) | Aberration | Apoptosis
Journal Article
Developmental Biology, ISSN 0012-1606, 10/2017, Volume 430, Issue 1, pp. 69 - 79
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2017, Volume 7, Issue 1, p. 41803
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 07/2017, Volume 21, Issue 7, pp. 1373 - 1387
Intervertebral disc degeneration is widely recognized as a cause of lower back pain, neurological dysfunction and other musculoskeletal disorders. The major... 
intervertebral disc degeneration | IL‐1β | calcium | human nucleus pulposus cells | mitochondrial damage | NLRP3 inflammasome | advanced glycation end products | IL-1β | MEDICINE, RESEARCH & EXPERIMENTAL | PERMEABILITY TRANSITION PORE | APOPTOSIS | SIGNALING PATHWAYS | IL-1 | NLRP3 INFLAMMASOME ACTIVATION | MITOCHONDRIA | RECEPTOR | CELL BIOLOGY | TOLL-LIKE | PATTERN | EXPRESSION | Inflammation - pathology | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Scoliosis - genetics | Humans | Interleukin-1beta - genetics | Scoliosis - pathology | Intervertebral Disc Degeneration - metabolism | Intervertebral Disc Degeneration - pathology | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Interleukin-1beta - metabolism | Antigens, Neoplasm - metabolism | Low Back Pain - genetics | Low Back Pain - pathology | Glycation End Products, Advanced - genetics | Intervertebral Disc Degeneration - genetics | Antigens, Neoplasm - genetics | Nucleus Pulposus - metabolism | Oxidative Stress - genetics | Nucleus Pulposus - pathology | Mitochondria - metabolism | Mitochondria - pathology | Inflammasomes - genetics | Glycation End Products, Advanced - metabolism | Inflammation - genetics | Mitogen-Activated Protein Kinases - genetics | Mitogen-Activated Protein Kinases - metabolism | Oxidative stress | Back pain | Reactive oxygen species | Calcium | Mitochondrial permeability transition pore | Membrane permeability | Disorders | Inflammatory response | Activation | Nucleus pulposus | Intervertebral discs | Tissues | Calcium signalling | Pain | Scoliosis | Bioaccumulation | Interleukin 1 | Back | Aging | Degeneration | Damage | Assembly | Age | Activation analysis | Advanced glycosylation end products | Secretion | Diabetes mellitus | Glycosylation | Permeability | Patients | Calcium permeability | Molecular modelling | Neurological complications | Nuclei (cytology) | MPTP | Immunofluorescence | In vitro methods and tests | Original
Journal Article
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 08/2018, Volume 48, Issue 3, pp. 880 - 890
Journal Article