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Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 1976 - 1985.e10
Background & Aims MUTYH -associated polyposis (MAP) is characterized by a lifetime risk of colorectal cancer of up to 100%. However, no systematic evaluation... 
Gastroenterology and Hepatology | BASE-EXCISION-REPAIR | URINARY-TRACT CANCER | APC GENE MUTATION | MUIR-TORRE-SYNDROME | NONPOLYPOSIS COLORECTAL-CANCER | DUODENAL ADENOMATOSIS | FAMILIAL ADENOMATOUS POLYPOSIS | MYH-ASSOCIATED POLYPOSIS | LYNCH SYNDROME | GASTROENTEROLOGY & HEPATOLOGY | MSH2 MUTATION CARRIERS | Gastrointestinal Neoplasms - genetics | Humans | Middle Aged | Adenomatous Polyposis Coli - epidemiology | Ovarian Neoplasms - pathology | Male | Adenomatous Polyposis Coli - pathology | Incidence | Ovarian Neoplasms - genetics | Young Adult | Gastrointestinal Neoplasms - pathology | Gastrointestinal Neoplasms - epidemiology | Urinary Bladder Neoplasms - genetics | Urinary Bladder Neoplasms - pathology | Adult | Female | Retrospective Studies | Endoscopy | Adenomatous Polyposis Coli - genetics | Child | Urinary Bladder Neoplasms - epidemiology | Breast Neoplasms - epidemiology | Skin Neoplasms - pathology | Genetic Predisposition to Disease | Sebaceous Gland Neoplasms - epidemiology | Risk Assessment | Risk Factors | Sebaceous Gland Neoplasms - genetics | DNA Glycosylases - genetics | Kaplan-Meier Estimate | Europe - epidemiology | Skin Neoplasms - epidemiology | Ovarian Neoplasms - epidemiology | Phenotype | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Sebaceous Gland Neoplasms - pathology | Skin Neoplasms - genetics | Adolescent | Age of Onset | Aged | Medical colleges | Animal pigments | Genetic disorders | Oncology, Experimental | Colorectal cancer | Research | Cancer | Tumors | Medical informatics
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1894 - 9
Journal Article
The EMBO Journal, ISSN 0261-4189, 05/2019, Volume 38, Issue 9, p. n/a
Mutations in Lef1 occur in human and mouse sebaceous gland (SG) tumors, but their contribution to carcinogenesis remains unclear. Since Gata6 controls lineage... 
Lef1 | Gata6 | sebaceous gland | DNA mismatch repair | sebaceous tumors | STEM-CELLS | MOUSE EPIDERMIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR | BETA-CATENIN | CELL BIOLOGY | IN-VITRO | REPAIR | GENE | HAIR FOLLICLE MORPHOGENESIS | DIFFERENTIATION | MUTATIONS | Cell Proliferation | Humans | Lymphoid Enhancer-Binding Factor 1 - genetics | Male | MutS Homolog 2 Protein - metabolism | Stem Cells - metabolism | Hair Follicle - pathology | Lymphoid Enhancer-Binding Factor 1 - physiology | Female | Skin Neoplasms - pathology | Sebaceous Gland Neoplasms - genetics | MutS Homolog 2 Protein - genetics | Lymphoid Enhancer-Binding Factor 1 - metabolism | beta Catenin - metabolism | GATA6 Transcription Factor - metabolism | beta Catenin - genetics | Mice, Knockout | Skin Neoplasms - metabolism | Animals | Sebaceous Gland Neoplasms - pathology | Hair Follicle - metabolism | MutL Protein Homolog 1 - genetics | Skin Neoplasms - genetics | Stem Cells - pathology | Mice | DNA Damage | MutL Protein Homolog 1 - metabolism | Mutation | Sebaceous Gland Neoplasms - metabolism | Transcription factors | Carcinoma | Sox9 protein | Carcinogenesis | DNA repair | Sebaceous gland | Morphogenesis | Carcinogens | Mismatch repair | Tumorigenesis | Repair | Catenin | Deoxyribonucleic acid--DNA | Hair | Sebaceous glands | MLH1 protein | MSH2 protein | Gene expression | Ablation | Tumor suppressor genes | Skin | Cancer | Tumors | Index Medicus | Transcription | Development & Differentiation
Journal Article
British Journal of Cancer, ISSN 0007-0920, 06/2017, Volume 117, Issue 1, pp. 41 - 50
Background: Some highly penetrant familial cancer syndromes exhibit elevated leukaemia risk, and there is evidence for familial clustering of lung cancer and... 
familial clustering | lung cancer | high-energy beam | ionising radiation | myeloid leukaemia | space radiation exposure | mice | BRCA2 MUTATION CARRIERS | CATALYTIC SUBUNIT | EFFECTIVENESS RBE VALUES | BIOLOGICAL EFFECTIVENESS | ATOMIC-BOMB SURVIVORS | RETROSPECTIVE COHORT | BREAST-CANCER | LUNG-CANCER | SPACE RADIATION | ONCOLOGY | MYELOID-LEUKEMIA | Carcinoma, Hepatocellular - mortality | Harderian Gland | Neoplasms - etiology | Gamma Rays - adverse effects | Adenoma - genetics | Adenoma - mortality | Lung Neoplasms - mortality | Adenocarcinoma - etiology | Leukemia, Myeloid - genetics | Sebaceous Gland Neoplasms - etiology | Liver Neoplasms, Experimental - mortality | Neoplastic Syndromes, Hereditary - etiology | Neoplastic Syndromes, Hereditary - mortality | Neoplasms, Radiation-Induced - mortality | Neoplasms, Radiation-Induced - genetics | Lung Neoplasms - etiology | Neoplasms - genetics | Carcinoma, Hepatocellular - genetics | Adenocarcinoma - genetics | Carcinoma, Hepatocellular - etiology | Liver Neoplasms, Experimental - genetics | Neoplastic Syndromes, Hereditary - genetics | Leukemia, Myeloid - mortality | Radiation, Ionizing | Lung Neoplasms - genetics | Sebaceous Gland Neoplasms - genetics | Proportional Hazards Models | Neoplasms - mortality | Adenoma - etiology | Liver Neoplasms, Experimental - etiology | Leukemia, Myeloid - etiology | Animals | Sebaceous Gland Neoplasms - mortality | Mice | Adenocarcinoma - mortality | Adenocarcinoma | Beams (radiation) | Correlation | Leukemia | Lung cancer | Disorders | Hepatocellular carcinoma | Risk factors | Risk assessment | Circularity | Background radiation | Radiation effects | Particle beams | γ Radiation | Breeding | Periodicity | Health risks | Ions | Exposure | Clustering | Radiation dosage | Gamma rays | Gamma radiation | Irradiation | Charged particles | Health risk assessment | Cancer | Tumors | Translational Therapeutics
Journal Article
The Journal of Pathology, ISSN 0022-3417, 09/2016, Volume 240, Issue 1, pp. 84 - 95
Journal Article
Human Pathology, ISSN 0046-8177, 2014, Volume 45, Issue 3, pp. 533 - 539
Summary Periocular sebaceous carcinoma is an aggressive neoplasm with significant morbidity and mortality. Its pathogenesis is poorly understood. It is only... 
Pathology | Periocular sebaceous carcinoma | Human papillomavirus | Promoter hypermethylation | p16INK4a | p16 | PROTEIN | TUMOR-SUPPRESSOR GENE | PATHOLOGY | HUMAN-PAPILLOMAVIRUS DNA | IDENTIFICATION | P53 | INACTIVATION | MUIR-TORRE-SYNDROME | GLAND CARCINOMA | SQUAMOUS-CELL CARCINOMA | p16(INK4a) | EXPRESSION | Microsatellite Instability | Age Factors | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Carcinoma, Squamous Cell - virology | Epigenesis, Genetic | Humans | Middle Aged | Carcinoma - virology | Male | Uterine Cervical Neoplasms - pathology | Eyelid Neoplasms - virology | Papillomavirus Infections - pathology | DNA Methylation | Aged, 80 and over | Biomarkers, Tumor - metabolism | Female | Eyelid Neoplasms - genetics | Sebaceous Gland Neoplasms - virology | Carcinoma - pathology | Promoter Regions, Genetic | Sebaceous Gland Neoplasms - genetics | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Eyelid Neoplasms - pathology | Uterine Cervical Neoplasms - genetics | Papillomavirus Infections - genetics | Eyelid Neoplasms - metabolism | Sebaceous Gland Neoplasms - pathology | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Uterine Cervical Neoplasms - virology | Carcinoma - genetics | Papillomaviridae | Aged | Biomarkers, Tumor - genetics | Carcinoma - metabolism | Sebaceous Gland Neoplasms - metabolism | Immunohistochemistry | Proteins | Carcinoma | Analysis | Genetic research | Development and progression | Genetic aspects | Methylation | Papillomavirus infections | Cancer | Pathogenesis | Genes | Cloning | Mortality | Infections | Patients | Polymerase chain reaction | Studies | Medical prognosis | DNA methylation | Epigenetics | Cervical cancer | Age | Deoxyribonucleic acid--DNA | Tumors
Journal Article
Journal Article
Journal of Cancer Research and Clinical Oncology, ISSN 0171-5216, 1/2016, Volume 142, Issue 1, pp. 125 - 133
To identify crucial molecular alterations of receptor tyrosine kinases that can be used as potential therapeutic targets for eyelid sebaceous gland carcinoma... 
Eyelid | Sebaceous gland carcinoma | Medicine & Public Health | Hematology | C-MET | Oncology | Cancer Research | Internal Medicine | HER2 | EGFR | FGFR1 | HEAD | ADENOCARCINOMA | IN-SITU HYBRIDIZATION | CELL LUNG-CANCER | BREAST-CANCER | GROWTH-FACTOR RECEPTOR | AMPLIFICATION | ONCOLOGY | NECK | EXPRESSION | AMERICAN SOCIETY | Proto-Oncogene Proteins c-met - metabolism | Receptor, Epidermal Growth Factor - genetics | Prognosis | Follow-Up Studies | Receptor, ErbB-2 - genetics | Adenocarcinoma, Sebaceous - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Male | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Chromosomes, Human, Pair 17 - genetics | Immunoenzyme Techniques | Neoplasm Recurrence, Local - pathology | Receptor, Epidermal Growth Factor - metabolism | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Eyelid Neoplasms - genetics | Adenocarcinoma, Sebaceous - pathology | Gene Dosage - genetics | Neoplasm Recurrence, Local - metabolism | Neoplasm Invasiveness | Sebaceous Gland Neoplasms - genetics | In Situ Hybridization, Fluorescence | Lymphatic Metastasis | Eyelid Neoplasms - pathology | Proto-Oncogene Proteins c-met - genetics | Gene Amplification | Eyelid Neoplasms - metabolism | Sebaceous Gland Neoplasms - pathology | Neoplasm Recurrence, Local - genetics | Aged | Biomarkers, Tumor - genetics | Neoplasm Staging | Adenocarcinoma, Sebaceous - genetics | Sebaceous Gland Neoplasms - metabolism | Immunohistochemistry | Carcinoma | Genes | Genetic research | Statistics | Health aspects | Phosphotransferases | Cancer
Journal Article
Archives of Pathology and Laboratory Medicine, ISSN 0003-9985, 2014, Volume 138, Issue 6, pp. 759 - 787
Context.-Skin neoplasms include the most common malignancies affecting humans. Many show an ultraviolet (UV)-induced pathogenesis and often affect the head and... 
DESMOPLASTIC NEUROTROPIC MELANOMA | MEDICINE, RESEARCH & EXPERIMENTAL | METASTASIZING ATYPICAL FIBROXANTHOMA | TUMOR-SUPPRESSOR GENE | IN-SITU HYBRIDIZATION | MERKEL-CELL-CARCINOMA | PATHOLOGY | DNA MISMATCH REPAIR | GERM-LINE MUTATIONS | MUIR-TORRE-SYNDROME | MULTIPLE FAMILIAL TRICHOEPITHELIOMA | MEDICAL LABORATORY TECHNOLOGY | BROOKE-SPIEGLER-SYNDROME | Melanoma - etiology | Carcinoma, Basal Cell - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Male | Hair Follicle - pathology | Basal Cell Nevus Syndrome - pathology | MAP Kinase Signaling System - genetics | Hedgehog Proteins - genetics | Melanoma - genetics | Carcinoma, Basal Cell - etiology | Histiocytoma, Benign Fibrous - pathology | Pilomatrixoma - genetics | Female | Skin Neoplasms - pathology | Histiocytoma, Benign Fibrous - genetics | Carcinoma, Adenoid Cystic - pathology | Risk Factors | Sebaceous Gland Neoplasms - genetics | Carcinoma, Merkel Cell - genetics | Carcinoma, Basal Cell - pathology | Pilomatrixoma - pathology | Signal Transduction - genetics | Melanoma - pathology | Ultraviolet Rays - adverse effects | Head and Neck Neoplasms - pathology | Carcinoma, Squamous Cell - etiology | Carcinoma, Adenoid Cystic - genetics | Head and Neck Neoplasms - etiology | Sebaceous Gland Neoplasms - pathology | Skin Neoplasms - genetics | Head and Neck Neoplasms - genetics | Skin Neoplasms - etiology | Basal Cell Nevus Syndrome - genetics | Mutation | Carcinoma, Merkel Cell - pathology | Squamous cell carcinoma | Ionizing radiation | Genetic aspects | Skin | Melanoma | Skin cancer
Journal Article