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Journal Article
Journal Article
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Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1271 - 1278
Temporal lobe epilepsy (TLE) is accompanied by an abnormal location of granule cells in the dentate gyrus. Using a rat model of complex febrile seizures, which... 
NEURONAL MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | TEMPORAL-LOBE EPILEPSY | GABA(A) RECEPTOR ACTIVATION | HIGH-FREQUENCY OSCILLATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | POSTNATAL SUBVENTRICULAR ZONE | CELL BIOLOGY | RAT DENTATE GYRUS | PYRAMIDAL CELLS | ANIMAL-MODEL | INDUCED STATUS EPILEPTICUS | CORTICAL MALFORMATIONS | Neurons - pathology | Up-Regulation | Hyperthermia, Induced - adverse effects | Epilepsy, Temporal Lobe - physiopathology | Male | GABA Agonists - therapeutic use | Brain Diseases - physiopathology | Seizures, Febrile - pathology | Choristoma - etiology | gamma-Aminobutyric Acid - physiology | Bumetanide - therapeutic use | Dentate Gyrus | Seizures, Febrile - physiopathology | RNA Interference | Animals, Suckling | Nerve Tissue Proteins - biosynthesis | Neurons - metabolism | Brain Diseases - etiology | Receptors, GABA-A - biosynthesis | Epilepsy, Temporal Lobe - prevention & control | Genes, Reporter | Nerve Tissue Proteins - antagonists & inhibitors | Nerve Tissue Proteins - physiology | Disease Susceptibility | GABA Antagonists - toxicity | Rats | Sodium-Potassium-Chloride Symporters - genetics | Epilepsy, Temporal Lobe - etiology | Hippocampus - pathology | Organ Specificity | Choristoma - physiopathology | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Bumetanide - pharmacology | Cell Lineage | Animals | Brain Diseases - prevention & control | Solute Carrier Family 12, Member 2 | Receptors, GABA-A - genetics | Sodium-Potassium-Chloride Symporters - physiology | Receptors, GABA-A - physiology | Choristoma - prevention & control | Hippocampus - physiopathology | Seizures, Febrile - complications | Cell Movement | Granulation tissue | Complications and side effects | Care and treatment | Epilepsy | GABA | Physiological aspects | Febrile convulsions | Diagnosis | Health aspects | Signal transduction | Convulsions & seizures | Pathogenesis | Neurons | Cell adhesion & migration
Journal Article
Epilepsia, ISSN 0013-9580, 05/2017, Volume 58, Issue 5, pp. 772 - 780
Summary Objective Diffusion magnetic resonance imaging (MRI) studies have demonstrated acute white matter changes following prolonged febrile seizures (PFS),... 
Neuroplasticity | Axonal injury | Status epilepticus | White matter | Diffusion tensor imaging | NERVOUS-SYSTEM | LOBE RESECTION | ABNORMALITIES | MESIAL TEMPORAL SCLEROSIS | MATURATION | CLINICAL NEUROLOGY | TRACTOGRAPHY | DIFFUSION MRI | INFANTS | BRAIN | White Matter - physiopathology | Follow-Up Studies | Humans | Child, Preschool | Image Interpretation, Computer-Assisted | Infant | Male | Reference Values | Neural Pathways - pathology | Sclerosis | Seizures, Febrile - pathology | Seizures, Febrile - physiopathology | Neuronal Plasticity - physiology | Female | Neural Pathways - physiopathology | Imaging, Three-Dimensional | Temporal Lobe - pathology | Child | Seizures, Febrile - diagnosis | Brain - physiopathology | Organ Size - physiology | Hippocampus - pathology | Echo-Planar Imaging | White Matter - pathology | Magnetic Resonance Imaging | Temporal Lobe - physiopathology | Diffusion Magnetic Resonance Imaging | Brain - pathology | Hippocampus - physiopathology | Anisotropy | Epilepsy | Febrile convulsions | Attention deficit disorder | Neuroimaging | Nuclear magnetic resonance--NMR | Statistical analysis | Image processing | Spatial discrimination | Plasticity (hippocampal) | Substantia alba | Plasticity (axonal) | Handedness | Magnetic resonance imaging | Children | Hippocampus | Seizures | Full‐Length Original Research
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