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Nature, ISSN 0028-0836, 11/2010, Volume 468, Issue 7321, pp. 263 - 269
Mutations in the X-linked MECP2 gene, which encodes the transcriptional regulator methyl-CpG-binding protein 2 (MeCP2), cause Rett syndrome and several... 
GLUTAMIC-ACID DECARBOXYLASE | GENE | 67 KDA | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | MUTATION | NEURONAL MECP2 | SCHIZOPHRENIA | MICE | CPG-BINDING PROTEIN-2 | YOUNG GIRLS | Autistic Disorder - physiopathology | gamma-Aminobutyric Acid - metabolism | Methyl-CpG-Binding Protein 2 - metabolism | Male | Psychomotor Disorders - complications | Methyl-CpG-Binding Protein 2 - deficiency | Compulsive Behavior - physiopathology | Neurons - metabolism | Disease Models, Animal | Autistic Disorder - genetics | Signal Transduction | Compulsive Behavior - genetics | Genotype | Mice, Transgenic | Survival Rate | Hippocampus - pathology | Rett Syndrome - physiopathology | Self-Injurious Behavior - genetics | Phenotype | Neuronal Plasticity | Long-Term Potentiation | Stereotypic Movement Disorder - genetics | Compulsive Behavior - complications | Glutamate Decarboxylase - metabolism | Mice | Hippocampus - physiopathology | Autistic Disorder - pathology | Rett Syndrome - complications | Neural Inhibition | Stereotypic Movement Disorder - pathology | Reflex, Startle - genetics | Electroencephalography | Methyl-CpG-Binding Protein 2 - genetics | Synaptic Transmission | Psychomotor Disorders - genetics | Self-Injurious Behavior - complications | Psychomotor Disorders - physiopathology | Inhibitory Postsynaptic Potentials | Stereotypic Movement Disorder - physiopathology | Brain - cytology | Homeodomain Proteins - genetics | Stereotypic Movement Disorder - complications | Self-Injurious Behavior - physiopathology | Animals | Rett Syndrome - pathology | Vesicular Inhibitory Amino Acid Transport Proteins - genetics | Presynaptic Terminals - metabolism | Autistic Disorder - complications | Respiration | Rett Syndrome - genetics | Autism | Care and treatment | Rett syndrome | Schizophrenia | GABA | Genetic aspects | Diagnosis | Cognition disorders | Seizures (Medicine) | Health aspects | Studies | Anxieties | Social interaction | Neurons | Rodents | Bipolar disorder | Behavior | Gene expression
Journal Article
Journal Article
Nature, ISSN 0028-0836, 08/2007, Volume 448, Issue 7156, pp. 894 - 900
Journal Article
Journal of Autism and Developmental Disorders, ISSN 0162-3257, 4/2009, Volume 39, Issue 4, pp. 572 - 588
Journal Article
Journal of Intellectual Disability Research, ISSN 0964-2633, 02/2011, Volume 55, Issue 2, pp. 109 - 120
Background  Self‐injurious and aggressive behaviours are reported as components of some behavioural phenotypes but there are few studies comparing across... 
behavioural phenotype | impulsivity | challenging behaviour | hyperactivity | autism spectrum disorder | affect | Behavioural phenotype | Impulsivity | Challenging behaviour | Hyperactivity | Affect | Autism spectrum disorder | POPULATION | MENTAL-RETARDATION | INTELLECTUAL DISABILITIES PREVALENCE | ADULTS | MALADAPTIVE BEHAVIOR | EDUCATION, SPECIAL | CHILDREN | SMITH-MAGENIS-SYNDROME | FRAGILE-X-SYNDROME | REHABILITATION | DE-LANGE-SYNDROME | PRADER-WILLI-SYNDROME | Intervention | At Risk Persons | Emotional Response | Self Destructive Behavior | Genetic Disorders | Incidence | Phenomenology | Comparative Analysis | Behavior Problems | Pervasive Developmental Disorders | Questionnaires | Genetics | Injuries | Prader-Willi Syndrome - epidemiology | Chromosome Disorders - epidemiology | Angelman Syndrome - psychology | Prevalence | Humans | Middle Aged | Child, Preschool | Male | Genetic Diseases, Inborn - epidemiology | De Lange Syndrome - epidemiology | Case-Control Studies | Smith-Magenis Syndrome - epidemiology | Chromosome Disorders - psychology | Young Adult | Prader-Willi Syndrome - psychology | Oculocerebrorenal Syndrome - psychology | Self-Injurious Behavior - epidemiology | Abnormalities, Multiple - epidemiology | Adult | Female | Child | Abnormalities, Multiple - genetics | Fragile X Syndrome - epidemiology | Behavioral Symptoms - psychology | Child Development Disorders, Pervasive - epidemiology | Aggression | Comorbidity | Self-Injurious Behavior - psychology | Angelman Syndrome - epidemiology | Behavioral Symptoms - epidemiology | Cri-du-Chat Syndrome - epidemiology | Abnormalities, Multiple - psychology | Genetic Diseases, Inborn - psychology | Syndrome | Behavioral Symptoms - genetics | Child Development Disorders, Pervasive - psychology | Smith-Magenis Syndrome - psychology | Fragile X Syndrome - psychology | Adolescent | Cri-du-Chat Syndrome - psychology | Intellectual Disability - psychology | De Lange Syndrome - psychology | Oculocerebrorenal Syndrome - epidemiology | Prader-Willi syndrome | Genetic research | Prevalence studies (Epidemiology) | Genotype & phenotype | Autism | Comparative analysis | Self destructive behavior | Risk factors | Aggressiveness
Journal Article
Journal Article
European Neuropsychopharmacology, ISSN 0924-977X, 2013, Volume 23, Issue 10, pp. 1125 - 1142
Abstract Suicide is one of the leading causes of death in the world. Its aetiology is complex and diverse, however, epidemiological studies show that suicidal... 
Internal Medicine | Psychiatry | Gene–environment interaction | Suicidal behavior | Serotonin genes | Endophenotypes | Gene-environment interaction | PSYCHIATRY | BIPOLAR AFFECTIVE-DISORDER | MAJOR DEPRESSION | TRYPTOPHAN-HYDROXYLASE GENE | NEUROSCIENCES | CLINICAL NEUROLOGY | ALCOHOL-RELATED SUICIDE | BORDERLINE PERSONALITY-DISORDER | 1A RECEPTOR GENE | TRANSPORTER GENE | MONOAMINE-OXIDASE-A | PHARMACOLOGY & PHARMACY | GENOME-WIDE ASSOCIATION | TPH GENE | Humans | Suicide - prevention & control | Brain - enzymology | Monoamine Oxidase - genetics | Receptors, Serotonin - genetics | Gene-Environment Interaction | Case-Control Studies | Self-Injurious Behavior - metabolism | Brain - metabolism | Genetic Variation | Serotonin Plasma Membrane Transport Proteins - genetics | Protein Isoforms - metabolism | Neurons - metabolism | Tryptophan Hydroxylase - metabolism | Genetic Predisposition to Disease | Genetic Association Studies | Self-Injurious Behavior - prevention & control | Evidence-Based Medicine | Nerve Tissue Proteins - genetics | Receptors, Serotonin - metabolism | Tryptophan Hydroxylase - genetics | Nerve Tissue Proteins - metabolism | Serotonin Plasma Membrane Transport Proteins - metabolism | Self-Injurious Behavior - genetics | Animals | Neurons - enzymology | Alleles | Self-Injurious Behavior - etiology | Monoamine Oxidase - metabolism | Protein Isoforms - genetics | Monoamine oxidase | Serotonin | Analysis | Genes | Genetic research | Tryptophan | Suicide
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0149646
Journal Article