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Nature communications, ISSN 2041-1723, 2016, Volume 7, Issue 1, p. 13280
Sepsis, severe sepsis and septic shock are the main cause of mortality in non-cardiac intensive care units. Immunometabolism has been linked to sepsis;... 
IL-1-BETA PRODUCTION | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | PYRUVATE-KINASE M2 | LACTATE | HMGB1 RELEASE | SEPTIC SHOCK | INTRACELLULAR LPS | SEPSIS | PKR ACTIVATION | INNATE IMMUNITY | Interleukin-18 - immunology | Phosphorylation | eIF-2 Kinase - metabolism | Humans | Male | HMGB1 Protein - immunology | HMGB1 Protein - secretion | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - secretion | Female | Membrane Proteins - metabolism | Carrier Proteins - immunology | Macrophages - immunology | Disease Models, Animal | eIF-2 Kinase - genetics | Interleukin-1beta - secretion | Sepsis - immunology | DNA-Binding Proteins - immunology | Pyruvate Kinase - metabolism | Membrane Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Coinfection - immunology | Carrier Proteins - antagonists & inhibitors | Interleukin-1beta - immunology | Membrane Proteins - immunology | Naphthoquinones - pharmacology | Thyroid Hormones - genetics | eIF-2 Kinase - antagonists & inhibitors | Mice, Knockout | Thyroid Hormones - immunology | Carrier Proteins - genetics | Coinfection - microbiology | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Membrane Proteins - antagonists & inhibitors | Thyroid Hormones - metabolism | Inflammasomes - immunology | Myeloid Cells - metabolism | Interleukin-18 - secretion | Mice | Mice, Inbred BALB C | Pyruvate Kinase - genetics | Glycolysis - immunology | Sepsis - microbiology
Journal Article
Nature (London), ISSN 1476-4687, 2010, Volume 464, Issue 7285, pp. 104 - 107
Injury causes a systemic inflammatory response syndrome (SIRS) that is clinically much like sepsis. Microbial pathogen-associated molecular patterns (PAMPs)... 
SHOCK | SIRS | OPERATED CALCIUM INFLUX | HUMAN NEUTROPHILS | PEPTIDES | MODELS | MOBILIZATION | CHEMOTAXIS | MULTIDISCIPLINARY SCIENCES | RECEPTORS | TRAUMA | Liver - pathology | Phosphorylation | Humans | Male | Mitochondria - immunology | Liver - injuries | N-Formylmethionine Leucyl-Phenylalanine - metabolism | Liver - immunology | Systemic Inflammatory Response Syndrome - blood | Muscle, Skeletal - immunology | Sepsis - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Neutrophils - metabolism | Calcium Signaling | Femur - injuries | Sepsis - immunology | CpG Islands - immunology | Neutrophils - enzymology | Wounds and Injuries - pathology | Fractures, Bone - immunology | Wounds and Injuries - blood | Cells, Cultured | Neutrophils - immunology | Wounds and Injuries - complications | Rats | Wounds and Injuries - immunology | Acute Lung Injury - pathology | DNA, Mitochondrial - blood | Rats, Sprague-Dawley | DNA, Mitochondrial - immunology | Immunity, Innate - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Mitochondria - secretion | Acute Lung Injury - immunology | Fractures, Bone - pathology | Systemic Inflammatory Response Syndrome - complications | Muscle, Skeletal - pathology | Toll-Like Receptor 9 - metabolism | Sepsis - microbiology | Mitogen-Activated Protein Kinases - metabolism | N-Formylmethionine Leucyl-Phenylalanine - immunology | Infection | Peptides | Physiological aspects | Mitochondrial DNA | Inflammation | Research | Risk factors | Fractures | Bacterial proteins | Rheumatoid arthritis | Trauma | Deoxyribonucleic acid--DNA
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2013, Volume 288, Issue 23, pp. 16225 - 16234
Sepsis-associated immunosuppression (SAIS) is regarded as one of main causes for the death of septic patients at the late stage because of the decreased innate... 
IFN-BETA | CELLS | ACTIVATION | PROTEIN | NEGATIVE REGULATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | DEGRADATION | SEPSIS | TRIGGERED INFLAMMATORY RESPONSES | INNATE IMMUNITY | Interferon-beta - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | Intracellular Signaling Peptides and Proteins - immunology | Male | Monocytes - immunology | Lipopolysaccharides - immunology | Gene Knockdown Techniques | Ubiquitin-Protein Ligases - immunology | Sepsis - pathology | Reactive Oxygen Species - immunology | Monocytes - pathology | Tumor Necrosis Factor-alpha - immunology | Interferon-beta - genetics | Female | Tumor Necrosis Factor alpha-Induced Protein 3 | Hydro-Lyases - genetics | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Macrophages - immunology | Immune Tolerance - drug effects | Cysteine Endopeptidases | Sepsis - immunology | DNA-Binding Proteins - immunology | Lipopolysaccharides - toxicity | Histones - immunology | Immunity, Innate - drug effects | Interleukin-6 - genetics | Macrophages - pathology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | DNA-Binding Proteins - genetics | Nuclear Proteins - immunology | Proteins - immunology | Sepsis - genetics | Gene Expression Regulation - drug effects | Proteins - genetics | Animals | Histones - genetics | Interleukin-6 - immunology | Mice | Ubiquitin-Protein Ligases - genetics | Hydro-Lyases - immunology | Toll-like Receptors (TLR) | Immunology | Reactive Oxygen Species (ROS) | Innate Immunity | Endotoxin Tolerance | ROS | Inflammation | Macrophages | A20 | Immune Responsive Gene 1
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 135, Issue 6, pp. 1424 - 1432
Anaphylaxis is an acute, severe, life-threatening multisystem allergic reaction resulting from the sudden systemic release of biochemical mediators and... 
Allergy and Immunology | Allergy | signaling | mediators | receptor antagonists | anaphylaxis | platelet-activating factor | mast cells | acetylhydrolase | platelets | sepsis | PHOSPHOLIPASE A INHIBITOR | CLINICAL-TRIAL | IMMUNOLOGY | FACTOR ACETYLHYDROLASE ACTIVITY | PHASE-II TRIAL | FACTOR PAF | FACTOR RECEPTOR ANTAGONIST | DOUBLE-BLIND | HUMAN NASAL AIRWAY | CORONARY-ARTERY-DISEASE | SEVERE SEPSIS | Blood Platelets - pathology | Blood Platelets - immunology | Humans | Platelet Activation - immunology | Platelet Activating Factor - immunology | Eosinophils - immunology | 1-Alkyl-2-acetylglycerophosphocholine Esterase - immunology | Receptors, G-Protein-Coupled - immunology | 1-Alkyl-2-acetylglycerophosphocholine Esterase - genetics | Sepsis - pathology | Asthma - immunology | Anaphylaxis - immunology | Eosinophils - pathology | Platelet Aggregation - genetics | Neutrophils - pathology | Mast Cells - immunology | Platelet Membrane Glycoproteins - immunology | Sepsis - immunology | Platelet Aggregation - immunology | Signal Transduction | Anaphylaxis - pathology | Gene Expression Regulation | Neutrophils - immunology | Platelet Activating Factor - genetics | Platelet Membrane Glycoproteins - genetics | Asthma - genetics | Sepsis - genetics | Anaphylaxis - genetics | Platelet Activation - genetics | Mast Cells - pathology | Receptors, G-Protein-Coupled - genetics | Asthma - pathology | Enzymes | Immune response | Anaphylaxis | Permeability | Metabolites | Analysis | Proteins | Antigens | Blood platelets | E coli | Rodents | Preeclampsia | Lipids | Smooth muscle | Carbon | Biological activity
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2009, Volume 284, Issue 36, pp. 24035 - 24048
The role of endogenous inducers of inflammation is poorly understood. To produce the proinflammatory master cytokine interleukin (IL)-1β, macrophages need... 
GENE | LEUCINE-RICH PROTEOGLYCANS | CASPASE-1 | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE IMMUNE-RESPONSE | HYALURONAN | IL-1-BETA | SECRETION | ATP | NALP3 INFLAMMASOME | Toll-Like Receptor 2 - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Immunity, Innate - genetics | Extracellular Matrix - metabolism | Caspase 1 - metabolism | Male | Ureteral Obstruction - metabolism | Interleukin-1beta - genetics | Inflammation - metabolism | Kidney - metabolism | Carrier Proteins - immunology | Signal Transduction | Proteoglycans - immunology | Extracellular Matrix Proteins - genetics | Proteoglycans - metabolism | Toll-Like Receptor 4 - genetics | Biglycan | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Ureteral Obstruction - immunology | Macrophages - metabolism | Receptors, Purinergic P2 - metabolism | Toll-Like Receptor 2 - immunology | Mice | Proteoglycans - genetics | Extracellular Matrix - immunology | Kidney - immunology | Caspase 1 - immunology | Extracellular Matrix - genetics | Receptors, Purinergic P2 - genetics | Ureteral Obstruction - genetics | Interleukin-1beta - biosynthesis | Lung - metabolism | Receptors, Purinergic P2 - immunology | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Carrier Proteins - biosynthesis | Interleukin-1beta - immunology | Inflammation - immunology | Protein Structure, Tertiary - genetics | Receptors, Purinergic P2X7 | Carrier Proteins - genetics | Immunity, Innate - immunology | Animals | Caspase 1 - genetics | Extracellular Matrix Proteins - immunology | Receptors, Purinergic P2X4 | Inflammation - genetics | Lung - immunology | Animal models | Reactive oxygen species | Oligomerization | Proteoglycans | Transcription | Lung | Inflammation | Macrophages | Kidney | Stress | Caspase-1 | Purine P2X receptors | Signal transduction | Cell activation | TLR2 protein | Interleukin 1 | Toll-like receptors | Extracellular matrix | Sepsis | Cooperativity | Injuries | Glycobiology and Extracellular Matrices
Journal Article
Molecular immunology, ISSN 0161-5890, 2009, Volume 46, Issue 14, pp. 2753 - 2766
Journal Article
Clinical Immunology, ISSN 1521-6616, 2016, Volume 163, pp. 60 - 65
Abstract Immune dysregulation, polyendocrinopathy, enteropathy, X-linked (IPEX) syndrome is a monogenic autoimmune disease characterized by early-onset... 
Allergy and Immunology | Autoimmunity | IPEX | Mutation | FOXP3 | Primary immunodeficiency | X-LINKED SYNDROME | ENTEROPATHY | STABILITY | IMMUNOLOGY | IMMUNE DYSREGULATION | DIMER | SEQUENCE | DISEASE | REGULATORY T-CELLS | POLYENDOCRINOPATHY | IMMUNODYSREGULATION | Eczema - genetics | Eczema - immunology | Immune System Diseases - genetics | Humans | Diabetes Mellitus, Type 1 - congenital | Infant | Male | Meningoencephalitis - immunology | Thrombocytopenia - genetics | Immunoglobulin E - immunology | Diarrhea - immunology | Eosinophilia - genetics | Fatal Outcome | Hepatomegaly - immunology | Genetic Diseases, X-Linked - genetics | Diabetes Mellitus, Type 1 - immunology | Dimerization | Hemorrhage - genetics | Sepsis - immunology | Splenomegaly - genetics | Growth Disorders - immunology | Hemorrhage - immunology | Splenomegaly - immunology | Thrombocytopenia - immunology | Immune System Diseases - immunology | Leukocytosis - immunology | Meningoencephalitis - genetics | Diabetes Mellitus, Type 1 - genetics | Models, Molecular | Klebsiella Infections - genetics | Hepatomegaly - genetics | Forkhead Transcription Factors - genetics | Sepsis - genetics | Lung Diseases - immunology | Genetic Diseases, X-Linked - immunology | Immune System Diseases - congenital | Thymus Gland - abnormalities | Lung Diseases - genetics | Eosinophilia - immunology | Diarrhea - genetics | Klebsiella Infections - immunology | Phenylalanine - genetics | Hydrophobic and Hydrophilic Interactions | Growth Disorders - genetics | Leukocytosis - genetics
Journal Article