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Diabetes, ISSN 0012-1797, 03/2018, Volume 67, Issue 3, pp. 423 - 436
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 11/2017, Volume 21, Issue 11, pp. 2732 - 2747
Metastasis associated lung adenocarcinoma transcript 1(MALAT1) is a long non‐coding RNA, broadly expressed in mammalian tissues including kidney and... 
MALAT1 | β‐catenin | high glucose | diabetic nephropathy | SRSF1 | podocyte | β-catenin | beta-catenin | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | RETAINED NONCODING RNA | PROLIFERATION | MESENCHYMAL TRANSITION | CELL BIOLOGY | GLOMERULAR SLIT DIAPHRAGM | ROLES | DYSFUNCTION | WNT | SPLICING FACTOR | RNA, Small Interfering - genetics | Diabetes Mellitus, Experimental - genetics | Male | Serine-Arginine Splicing Factors - metabolism | Serine-Arginine Splicing Factors - genetics | Glucose - toxicity | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Wnt Signaling Pathway | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Diabetic Nephropathies - chemically induced | Gene Expression Regulation | Diabetic Nephropathies - genetics | RNA, Long Noncoding - genetics | Podocytes - pathology | beta Catenin - metabolism | beta Catenin - genetics | Protein Transport | Serine-Arginine Splicing Factors - antagonists & inhibitors | Feedback, Physiological | Animals | Podocytes - drug effects | beta Catenin - antagonists & inhibitors | Diabetes Mellitus, Experimental - pathology | Protein Binding | Mice | Streptozocin - toxicity | RNA, Long Noncoding - antagonists & inhibitors | RNA, Long Noncoding - metabolism | Cell Line, Transformed | RNA, Small Interfering - metabolism | Original
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 09/2016, Volume 23, Issue 9, pp. 1515 - 1528
Bidirectional interactions between astrocytes and neurons have physiological roles in the central nervous system and an altered state or dysfunction of such... 
MOTOR-NEURONS | STEM-CELLS | DELTA-133P53-ALPHA | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | P53-BETA | GROWTH | AUTOPHAGY | CELLULAR SENESCENCE | EXPRESSION | CELL BIOLOGY | Alternative Splicing | Tumor Suppressor Protein p53 - antagonists & inhibitors | Coculture Techniques | Humans | Neurons - cytology | Serine-Arginine Splicing Factors - metabolism | Serine-Arginine Splicing Factors - genetics | Autophagy - drug effects | Tumor Suppressor Protein p53 - genetics | Alzheimer Disease - pathology | Brain - metabolism | Protein Isoforms - metabolism | RNA Interference | Cellular Senescence | Neurons - metabolism | Interleukin-6 - metabolism | Sequestosome-1 Protein - metabolism | Astrocytes - cytology | Astrocytes - drug effects | Interleukin-6 - genetics | Sequestosome-1 Protein - antagonists & inhibitors | Cells, Cultured | Neuroprotection - physiology | Tumor Suppressor Protein p53 - metabolism | Genetic Vectors - metabolism | Genetic Vectors - genetics | Serine-Arginine Splicing Factors - antagonists & inhibitors | Leupeptins - pharmacology | Sequestosome-1 Protein - genetics | Amyotrophic Lateral Sclerosis - pathology | Alzheimer Disease - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Brain - pathology | Protein Isoforms - antagonists & inhibitors | Astrocytes - metabolism | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | Index Medicus | Original Paper
Journal Article
Advances in Experimental Medicine and Biology, ISSN 0065-2598, 2016, Volume 907, pp. 215 - 228
Journal Article
Advances in Biological Regulation, ISSN 2212-4926, 01/2017, Volume 63, pp. 59 - 70
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0165453
Hereditary myopathy with lactic acidosis (HML) is an autosomal recessive disease caused by an intronic one-base mutation in the iron-sulfur cluster assembly... 
SUCCINATE-DEHYDROGENASE | GENE | MULTIDISCIPLINARY SCIENCES | SR PROTEINS | LACTIC-ACIDOSIS | MYOPATHY | EXPRESSION | INTRON MUTATION | FACTOR SRP20 | DEFICIENCY | PRE-MESSENGER-RNA | Acidosis, Lactic - genetics | Humans | Iron-Sulfur Proteins - genetics | Muscle, Skeletal - metabolism | Serine-Arginine Splicing Factors - metabolism | Serine-Arginine Splicing Factors - genetics | Myoblasts - metabolism | RNA - isolation & purification | RNA Splicing | RNA Interference | Myoblasts - cytology | Mice, Inbred CBA | Muscular Diseases - congenital | Real-Time Polymerase Chain Reaction | RNA - metabolism | Mutagenesis, Site-Directed | Cells, Cultured | Mice, Transgenic | Muscular Diseases - pathology | Acidosis, Lactic - pathology | Serine-Arginine Splicing Factors - antagonists & inhibitors | Animals | Acidosis, Lactic - congenital | Iron-Sulfur Proteins - metabolism | Mice | Muscular Diseases - genetics | RNA, Small Interfering - metabolism | Proteins | Muscles | Sulfur compounds | Genetic aspects | Comparative analysis | Tricarboxylic acid cycle | IscU protein | Alternative splicing | Brain | Neurosciences | Senescence | Transcription | Liver | Serine | Iron | Genomes | Soleus muscle | Arginine | Transgenic animals | Rodents | Cell cycle | Sulfur | Lactic acidosis | Kidneys | Splicing | Transgenic mice | Patients | Skeletal muscle | Myoblasts | Musculoskeletal system | Quadriceps muscle | Mutation | Electron transport | Acidosis | Splicing factors | Myopathy | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, pp. e0131073 - e0131073
T lymphocytes from many patients with systemic lupus erythematosus (SLE) express decreased levels of the T cell receptor (TCR)-associated CD3 zeta (ζ)... 
RNA, Small Interfering - genetics | Luciferases - metabolism | Alternative Splicing | Humans | Transcriptional Activation | Lupus Erythematosus, Systemic - metabolism | Serine-Arginine Splicing Factors - metabolism | Serine-Arginine Splicing Factors - genetics | Case-Control Studies | Luciferases - genetics | Dose-Response Relationship, Drug | CD3 Complex - genetics | Chromatin Immunoprecipitation | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | T-Lymphocytes - pathology | 3' Untranslated Regions | Genes, Reporter | Promoter Regions, Genetic | Receptors, Antigen, T-Cell - metabolism | Signal Transduction | Jurkat Cells | CD3 Complex - metabolism | Serine-Arginine Splicing Factors - antagonists & inhibitors | Lupus Erythematosus, Systemic - genetics | Protein Binding | Receptors, Antigen, T-Cell - genetics | Primary Cell Culture | Lupus Erythematosus, Systemic - pathology | RNA, Small Interfering - metabolism | Alternative splicing | Chromatin | Phosphorylation | Immunoprecipitation | Serine | Chains | Activation | Lymphocytes T | Arthritis | Kinases | Medical schools | Defects | Proteins | T-cell receptor | Cell activation | Systemic lupus erythematosus | Arginine | Lymphocytes | Transcription activation | Rheumatism | Lupus | Antigens | Cytokines | Splicing | Blood & organ donations | Rheumatology | Mass spectroscopy | CD3 antigen | T cell receptors | Gene expression | Medicine | Signaling | Chronic conditions | 3' Untranslated regions | Autoimmune diseases | Mass spectrometry | Splicing factors | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2010, Volume 285, Issue 8, pp. 5532 - 5540
Journal Article