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Journal of Immunology, ISSN 0022-1767, 06/2011, Volume 186, Issue 11, pp. 6119 - 6128
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2004, Volume 10, Issue 7, pp. S2 - S9
The molecular bases underlying the pathogenesis of neurodegenerative diseases are gradually being disclosed. One problem that investigators face is... 
MEDICINE, RESEARCH & EXPERIMENTAL | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ONSET PARKINSONS-DISEASE | AXONAL-TRANSPORT | AMYLOID PRECURSOR PROTEIN | ALPHA-SYNUCLEIN | CELL BIOLOGY | A-BETA | MITOCHONDRIAL DYSFUNCTION | HUNTINGTONS-DISEASE | SUPEROXIDE-DISMUTASE | TRANSGENIC MICE | Protein Kinases - metabolism | Oncogene Proteins - genetics | Protein Kinases - genetics | Membrane Glycoproteins - metabolism | Humans | Huntington Disease - pathology | Membrane Glycoproteins - chemistry | Ubiquitin - metabolism | Nerve Degeneration - genetics | Protein Kinases - chemistry | Serum Amyloid A Protein - genetics | Serotonin Plasma Membrane Transport Proteins | Nerve Degeneration - metabolism | Alzheimer Disease - pathology | Nerve Tissue Proteins - chemistry | Peptides - metabolism | Protein Denaturation | Carrier Proteins - chemistry | Parkinson Disease - metabolism | Parkinson Disease - pathology | Neurodegenerative Diseases - pathology | Serum Amyloid A Protein - metabolism | Oncogene Proteins - chemistry | Amyotrophic Lateral Sclerosis - genetics | Oncogene Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | Intracellular Signaling Peptides and Proteins | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - metabolism | Parkinson Disease - genetics | Ubiquitin-Protein Ligases - chemistry | Huntington Disease - metabolism | Nerve Tissue Proteins - genetics | Protein Folding | Membrane Glycoproteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Protein Deglycase DJ-1 | Amyotrophic Lateral Sclerosis - pathology | Animals | Carrier Proteins - metabolism | Serum Amyloid A Protein - chemistry | Membrane Transport Proteins | Alzheimer Disease - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Huntington Disease - genetics | Alzheimer Disease - genetics | Ubiquitin-Protein Ligases - genetics
Journal Article
Oncogene, ISSN 0950-9232, 01/2015, Volume 34, Issue 4, pp. 424 - 435
Journal Article
Cell Metabolism, ISSN 1550-4131, 07/2014, Volume 20, Issue 1, pp. 103 - 118
Chronic inflammation constitutes an important link between obesity and its pathophysiological sequelae. In contrast to the belief that inflammatory signals... 
OBESITY | HOMEOSTASIS | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | FAT | NECROSIS-FACTOR-ALPHA | DIFFERENTIATION | IDENTIFICATION | ADIPOGENESIS | EXPRESSION | PROGRESSION | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Adipogenesis - drug effects | Tumor Necrosis Factor-alpha - genetics | Adipose Tissue, White - metabolism | Body Weight - drug effects | Adrenergic Agonists - pharmacology | Male | Serum Amyloid A Protein - genetics | Fatty Acid-Binding Proteins - metabolism | Recombinant Proteins - biosynthesis | Inflammation - metabolism | Leptin - blood | Diet, High-Fat | Adiponectin - metabolism | Female | Models, Animal | Glucose Tolerance Test | Lipopolysaccharides - toxicity | Serum Amyloid A Protein - metabolism | Fatty Liver - physiopathology | Mice, Inbred C57BL | Mice, Transgenic | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Intra-Abdominal Fat - metabolism | Fatty Acid-Binding Proteins - genetics | Tumor Necrosis Factor-alpha - pharmacology | Animals | Mice | Adipose Tissue, White - drug effects | Adipose tissues | Glucose metabolism | Analysis | Physiological aspects | Inflammation | Glucose | Cells | Dextrose | Index Medicus | Medicinska grundvetenskaper | Basic Medicine | Physiology | Immunologi inom det medicinska området | Immunology in the medical area | Fysiologi
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2015, Volume 19, Issue 6, pp. 1418 - 1425
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2010, Volume 207, Issue 7, pp. 1453 - 1464
Acute-phase proteins (APPs) are an evolutionarily conserved family of proteins produced mainly in the liver in response to infection and inflammation. Despite... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | LIVER-INJURY | PATHWAY | INFLAMMATION | SERUM-AMYLOID-A | GP130 | MICE | MECHANISMS | IMMUNOLOGY | SEVERE SEPSIS | SIGNAL TRANSDUCER | Myeloid Cells - cytology | Liver - pathology | Spleen - immunology | Cytokine Receptor gp130 - genetics | Liver - microbiology | Apoptosis - genetics | Hepatocytes - pathology | Male | Sepsis - complications | Serum Amyloid A Protein - genetics | Chemokine CXCL1 - genetics | Gene Expression Profiling | Hepatocytes - metabolism | Cell Movement - genetics | Spleen - microbiology | Cell Movement - immunology | Inflammation - complications | Signal Transduction - immunology | Bacteria - immunology | Liver - immunology | Myeloid Cells - immunology | Chemokine CXCL1 - metabolism | Spleen - pathology | STAT3 Transcription Factor - metabolism | Sepsis - immunology | Serum Amyloid A Protein - metabolism | Cytokine Receptor gp130 - metabolism | Signal Transduction - genetics | Hepatocytes - immunology | Sepsis - genetics | Immunity, Innate - immunology | Animals | Apoptosis - immunology | Inflammation - genetics | Myeloid Cells - metabolism | Inflammation - prevention & control | Mice | CD11b Antigen - metabolism | Hepatocytes - microbiology | Sepsis - microbiology | Acute-Phase Proteins - immunology | severe sepsis | signal transducer | inflammation | serum-amyloid-a | pathway | liver-injury | gp130 | activation | mice | mechanisms
Journal Article
eLife, ISSN 2050-084X, 07/2014, Volume 3, Issue 2014, pp. 1 - 18
Journal Article
Journal Article