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Journal Article
Nature Immunology, ISSN 1529-2908, 11/2001, Volume 2, Issue 11, pp. 1032 - 1039
Journal Article
Journal Article
European Heart Journal, ISSN 0195-668X, 08/2014, Volume 35, Issue 29, pp. 1917 - 1924
Epidemiological research over the last 50 years has discovered a plethora of biomarkers (including molecules, traits or other diseases) that associate with... 
Coronary artery disease | Mendelian randomization | C-REACTIVE PROTEIN | FUTURE MYOCARDIAL-INFARCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | ESTER TRANSFER PROTEIN | CARDIOVASCULAR EVENTS | SUBTILISIN/KEXIN TYPE 9 | ISCHEMIC-HEART-DISEASE | PHOSPHOLIPASE A | DENSITY-LIPOPROTEIN CHOLESTEROL | LAPLACE-TIMI 57 | GENOME-WIDE ASSOCIATION | Triglycerides - physiology | Cholesterol Ester Transfer Proteins - physiology | Humans | Obesity - genetics | Cholesterol, HDL - physiology | C-Reactive Protein - metabolism | Cholesterol Ester Transfer Proteins - genetics | Mendelian Randomization Analysis | Diabetes Complications - genetics | Genetic Pleiotropy - genetics | Hypertension - genetics | Phospholipases A2 - physiology | Telomere - genetics | Fibrinogen - physiology | Obesity - complications | Receptors, Interleukin-6 - genetics | Risk Factors | Linkage Disequilibrium - genetics | Lipoprotein(a) - physiology | C-Reactive Protein - genetics | Cholesterol, LDL - physiology | Cholesterol, HDL - genetics | Randomized Controlled Trials as Topic | Cholesterol, LDL - genetics | Serum Amyloid P-Component - genetics | Genetic Markers - genetics | Coronary Artery Disease - genetics | Hypertension - complications | Polymorphism, Single Nucleotide - genetics | Triglycerides - genetics | Lipoprotein(a) - genetics | Phospholipases A2 - genetics
Journal Article
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 01/2014, Volume 370, Issue 5, pp. 421 - 432
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0159294
Lysozyme amyloidosis is a hereditary disease in which mutations in the gene coding for lysozyme leads to misfolding and consequently accumulation of amyloid... 
APOPTOSIS | UNFOLDED PROTEIN RESPONSE | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | NEURODEGENERATION | ENDOPLASMIC-RETICULUM STRESS | MODEL | FAMILY | BETA | Neurons - pathology | Central Nervous System - metabolism | Humans | Central Nervous System - pathology | Drosophila melanogaster - genetics | Protective Factors | Drosophila melanogaster - metabolism | Protein Aggregation, Pathological - pathology | Protein Aggregation, Pathological - prevention & control | Amyloidosis - genetics | Neurons - metabolism | Transgenes | Protein Aggregation, Pathological - genetics | Disease Models, Animal | Gene Expression | Amyloidosis - pathology | Plaque, Amyloid - pathology | Animals, Genetically Modified | Muramidase - genetics | Muramidase - metabolism | Longevity - genetics | Plaque, Amyloid - genetics | Serum Amyloid P-Component - genetics | Animals | Plaque, Amyloid - metabolism | Plaque, Amyloid - prevention & control | Serum Amyloid P-Component - metabolism | Mutation | Amyloidosis - metabolism | Protein Aggregation, Pathological - metabolism | Apoptosis | Complications and side effects | Drosophila | Central nervous system | Physiological aspects | Development and progression | Influence | Amyloidosis | Genetic aspects | Research | Lysozyme | Amyloidogenesis | Biotechnology | Cytotoxicity | Nervous system | Biology | SAP protein | Accumulation | Proteins | Neurotoxicity | Neurodegeneration | Coding | Cell cycle | Amyloid | Pathogens | Fibrils | Gene expression | Physics | Life span | Insects | Cell death | Protein expression | Flies | Endoplasmic reticulum | Amyloid P component | Biological Sciences | Naturvetenskap | Natural Sciences | Developmental Biology | Utvecklingsbiologi | Biologiska vetenskaper
Journal Article
Journal Article