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1. Full Text Diagnostic Testing for Severe Acute Respiratory Syndrome-Related Coronavirus 2: A Narrative Review
by Cheng, Matthew P and Papenburg, Jesse and Desjardins, Michaël and Kanjilal, Sanjat and Quach, Caroline and Libman, Michael and Dittrich, Sabine and Yansouni, Cedric P
Annals of internal medicine, ISSN 0003-4819, 06/2020, Volume 172, Issue 11, pp. 726 - 734
COVID-19 | Antigens | Pulmonary diseases | Upper respiratory tract infections | Research laboratories | Antibodies | Nucleic acids | Prevention, policy, and public health | Cells | Infectious disease immunology | Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Pandemics | COVID-19 Testing | Clinical Laboratory Techniques | Coronavirus Infections - diagnosis | Point-of-Care Testing | Humans | Specimen Handling - methods | Biomarkers - blood | SARS-CoV-2 | COVID-19 Vaccines | Betacoronavirus | Pneumonia, Viral - diagnosis | Radiography, Thoracic | Real-Time Polymerase Chain Reaction | Serologic Tests | Index Medicus | Abridged Index Medicus
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2. Severe Acute Respiratory Syndrome Historical, Epidemiologic, and Clinical Features
by Hui, David S. C and Zumla, Alimuddin
Infectious disease clinics of North America, ISSN 0891-5520, 12/2019, Volume 33, Issue 4, p. 869
Infectious Diseases | Immunology | Life Sciences & Biomedicine | Science & Technology | Severe Acute Respiratory Syndrome - history | Severe Acute Respiratory Syndrome - epidemiology | Global Health | History, 21st Century | Severe Acute Respiratory Syndrome - virology | Humans | SARS Virus - physiology
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3. Full Text Severe Acute Respiratory Syndrome Coronavirus 2-Specific Antibody Responses in Coronavirus Disease Patients
by Okba, Nisreen M. A and Mueller, Marcel A and Li, Wentao and Wang, Chunyan and GeurtsvanKessel, Corine H and Corman, Victor M and Lamers, Mart M and Sikkema, Reina S and de Bruin, Erwin and Chandler, Felicity D and Yazdanpanah, Yazdan and Le Hingrat, Quentin and Descamps, Diane and Houhou-Fidouh, Nadhira and Reusken, Chantal B. E. M and Bosch, Berend-Jan and Drosten, Christian and Koopmans, Marion P. G and Haagmans, Bart L
Emerging infectious diseases, ISSN 1080-6040, 07/2020, Volume 26, Issue 7, pp. 1478 - 1488
Infectious Diseases | Immunology | Life Sciences & Biomedicine | Science & Technology | Pandemics | Enzyme-Linked Immunosorbent Assay | COVID-19 Testing | Coronavirus Infections - diagnosis | Humans | Neutralization Tests | Antibodies, Viral - blood | Betacoronavirus - immunology | COVID-19 | Clinical Laboratory Techniques - methods | SARS-CoV-2 | Sensitivity and Specificity | Pneumonia, Viral - diagnosis | Serologic Tests | Medical research | Severe acute respiratory syndrome | Immunoglobulin G | Medicine, Experimental | Coronaviruses | Immunoglobulin A | Health aspects | Enzyme-linked immunosorbent assay | Pathogens | Antigens | Blood & organ donations | Laboratories | Antibodies | Viruses | Infections | Blood banks | Patients | Middle East respiratory syndrome | Severe acute respiratory syndrome coronavirus 2 | Proteins | Respiratory diseases | Index Medicus | viruses | human coronavirus | Expedited | HCoV | Severe Acute Respiratory Syndrome Coronavirus 2−Specific Antibody Responses in Coronavirus Disease 2019 Patients | neutralization | spike protein | receptor-binding domain | coronavirus | zoonoses | Research | respiratory infections | RBD | coronavirus disease 2019 | nucleocapsid protein | antibodies | serologic analysis | ELISA | 2019 novel coronavirus disease
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4. Full Text Complement Activation Contributes to Severe Acute Respiratory Syndrome Coronavirus Pathogenesis
by Gralinski, Lisa E and Sheahan, Timothy P and Morrison, Thomas E and Menachery, Vineet D and Menachery, Vineet D and Jensen, Kara and Jensen, Kara and Leist, Sarah R and Whitmore, Alan and Heise, Mark T and Baric, Ralph S and Baric, Ralph S
mBio, ISSN 2161-2129, 09/2018, Volume 9, Issue 5, p. e01753-18
coronavirus | animal models | complement | respiratory viruses | SARS-CoV | Life Sciences & Biomedicine | Microbiology | Science & Technology | SARS Virus - immunology | Chemokines - blood | Lung - pathology | Complement Activation | Severe Acute Respiratory Syndrome - virology | Mice, Inbred C57BL | Severe Acute Respiratory Syndrome - immunology | Immunity, Innate | SARS Virus - pathogenicity | Viral Load | Host-Pathogen Interactions - immunology | Lung - virology | Animals | Complement C3 - deficiency | Virus Replication | Female | Mice | Complement C3 - genetics | Lung - immunology | Cytokines - blood | Disease Models, Animal | Index Medicus
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5. Full Text SARS-CoV-2 infection and overactivation of Nlrp3 inflammasome as a trigger of cytokine "storm" and risk factor for damage of hematopoietic stem cells
by Ratajczak, Mariusz Z and Kucia, Magda
Leukemia, ISSN 0887-6924, 07/2020, Volume 34, Issue 7, pp. 1726 - 1729
Oncology | Life Sciences & Biomedicine | Hematology | Science & Technology | Betacoronavirus - pathogenicity | Severe Acute Respiratory Syndrome - epidemiology | Coronavirus Infections - drug therapy | Furans - pharmacology | Humans | Pneumonia, Viral - drug therapy | Severe Acute Respiratory Syndrome - immunology | T-Lymphocytes - virology | Betacoronavirus - immunology | COVID-19 | SARS-CoV-2 | Inflammasomes - antagonists & inhibitors | Myocarditis - epidemiology | T-Lymphocytes - drug effects | Myocarditis - prevention & control | Acute Kidney Injury - immunology | Hematopoietic Stem Cells - virology | Cytokines - genetics | Sulfones | Spike Glycoprotein, Coronavirus - genetics | Risk Factors | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Sulfonamides - pharmacology | Coronavirus Infections - immunology | Pyroptosis - genetics | Angiotensin-Converting Enzyme 2 | Coronavirus Infections - virology | T-Lymphocytes - immunology | Immunologic Factors - pharmacology | Pandemics | Pyroptosis - drug effects | Severe Acute Respiratory Syndrome - virology | Heterocyclic Compounds, 4 or More Rings | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Peptidyl-Dipeptidase A - genetics | Acute Kidney Injury - virology | Hematopoietic Stem Cells - immunology | Coronavirus Infections - epidemiology | Pneumonia, Viral - epidemiology | Spike Glycoprotein, Coronavirus - immunology | Myocarditis - immunology | Cytokines - immunology | Hematopoietic Stem Cells - drug effects | Cytokine Release Syndrome - immunology | Cytokine Release Syndrome - virology | Immunity, Innate - drug effects | Cytokine Release Syndrome - prevention & control | Betacoronavirus - drug effects | Gene Expression Regulation | Cytokine Release Syndrome - epidemiology | Pneumonia, Viral - immunology | Acute Kidney Injury - prevention & control | Inflammasomes - genetics | Myocarditis - virology | Severe Acute Respiratory Syndrome - prevention & control | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Inflammasomes - immunology | Acute Kidney Injury - epidemiology | Cytokines - antagonists & inhibitors | Peptidyl-Dipeptidase A - immunology | Pneumonia, Viral - virology | Pyroptosis - immunology | Medical colleges | Cytokines | Health aspects | Hematopoietic stem cells | Severe acute respiratory syndrome | Viral diseases | Storm damage | Viruses | Risk analysis | Progenitor cells | Cytokine storm | Risk factors | Hemopoiesis | Severe acute respiratory syndrome coronavirus 2 | Myocarditis | Stem cells | Heart diseases | Index Medicus
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6. Full Text Anti-spike IgG causes severe acute lung injury by skewing macrophage responses during acute SARS-CoV infection
by Liu, Li and Wei, Qiang and Lin, Qingqing and Fang, Jun and Wang, Haibo and Kwok, Hauyee and Tang, Hangying and Nishiura, Kenji and Peng, Jie and Tan, Zhiwu and Wu, Tongjin and Cheung, Ka-Wai and Chan, Kwok-Hung and Alvarez, Xavier and Qin, Chuan and Lackner, Andrew and Perlman, Stanley and Yuen, Kwok-Yung and Chen, Zhiwei
JCI insight, ISSN 2379-3708, 02/2019, Volume 4, Issue 4
Pulmonology | Infectious disease | Immunoglobulins | Cytokines | Macrophages | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | SARS Virus - immunology | Antibodies, Neutralizing - administration & dosage | Immunoglobulin G - blood | Severe Acute Respiratory Syndrome - virology | Humans | Middle Aged | Severe Acute Respiratory Syndrome - immunology | Male | Macaca mulatta | Antibodies, Viral - blood | Vaccinia virus - genetics | Acute Lung Injury - blood | Vaccinia virus - immunology | Young Adult | Antibodies, Neutralizing - immunology | Lung - virology | Immunoglobulin G - immunology | Aged, 80 and over | Adult | Female | Spike Glycoprotein, Coronavirus - immunology | Antibodies, Viral - administration & dosage | Macrophages, Alveolar - immunology | Severe Acute Respiratory Syndrome - blood | Disease Models, Animal | Cell Line | Immunization, Passive - methods | Lung - pathology | Spike Glycoprotein, Coronavirus - genetics | Immunoglobulin G - administration & dosage | SARS Virus - pathogenicity | Viral Vaccines - administration & dosage | Animals | Acute Lung Injury - immunology | Antibodies, Viral - immunology | Aged | Acute Lung Injury - virology | Macrophages, Alveolar - metabolism | Viral Vaccines - immunology | Antibodies, Neutralizing - blood | Lung - immunology | Index Medicus
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7. Full Text Inhibition of NF-κB-Mediated Inflammation in Severe Acute Respiratory Syndrome Coronavirus-Infected Mice Increases Survival
by Marta L. DeDiego and Jose L. Nieto-Torres and Jose A. Regla-Nava and Jose M. Jimenez-Guardeño and Raul Fernandez-Delgado and Craig Fett and Carlos Castaño-Rodriguez and Stanley Perlman and Luis Enjuanes
Journal of Virology, ISSN 0022-538X, 01/2014, Volume 88, Issue 2, pp. 913 - 924
Life Sciences & Biomedicine | Science & Technology | Virology | Viral Envelope Proteins - genetics | Severe Acute Respiratory Syndrome - virology | Down-Regulation | Humans | NF-kappa B - immunology | Severe Acute Respiratory Syndrome - immunology | Severe Acute Respiratory Syndrome - mortality | Animals | SARS Virus - genetics | NF-kappa B - genetics | Viral Envelope Proteins - metabolism | Severe Acute Respiratory Syndrome - genetics | Female | Mice | Mice, Inbred BALB C | SARS Virus - physiology | Cytokines - genetics | Cytokines - immunology | Index Medicus | Pathogenesis and Immunity
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8. Full Text Association of COVID-19 inflammation with activation of the C5a–C5aR1 axis
by Carvelli, Julien and Demaria, Olivier and Vély, Frédéric and Batista, Luciana and Chouaki Benmansour, Nassima and Fares, Joanna and Carpentier, Sabrina and Thibult, Marie-Laure and Morel, Ariane and Remark, Romain and André, Pascale and Represa, Agnès and Piperoglou, Christelle and Assante  Miranda, Laura and Baron, William and Belaid, Nourhène and Caillet, Clarisse and Caraguel, Flavien and Carrette, Barbara and Carrette, Florent and Chanuc, Fabien and Courtois, Rachel and Fenis, Aurore and Giordano, Marilyn and Girard-Madoux, Mathilde and Giraudon-Paoli, Marc and Gourdin, Nicolas and Grondin, Gwendoline and Guillot, Franceline and Habif, Guillaume and Jaubert, Solène and Lopez, Julie and Le Van, Mélanie and Lovera, Naouel and Mansuy, Marine and Bonnet, Elodie and Sansaloni, Audrey and Reboul, Annick and Mitry, Emmanuel and Nekkar-Constant, Camille and Péri, Valentine and Ricaut, Paul and Simon, Léa and Vallier, Jean-Baptiste and Vétizou, Marie and Zerbib, Robert and Ugolini, Sophie and Etiennot, Marion and Galluso, Justine and Lyonnet, Luc and Forel, Jean-Marie and Papazian, Laurent and Velly, Lionel and André, Baptiste and Briantais, Antoine and Faucher, Benoit and Jean, Estelle and Seguier, Julie and Veit, Veronique and Harlé, Jean-Robert and Pastorino, Boris and Delteil, Clémence and Daniel, Laurent and Boudsocq, Jean-Paul and Clerc, Axelle and Delmond, Emmanuel and Vidal, Pierre-Olivier and Savini, Hélène and Coutard, Bruno and Cordier, Pierre Yves and Le Dault, Erwan and Guervilly, Christophe and Simeone, Pierre and Gainnier, Marc and Morel, Yannis and Ebbo, Mikael and Schleinitz, Nicolas and Vivier, Eric and Explore COVID-19 Marseille Immunop and Explore COVID-19 IPH Grp and Explore COVID-19 Marseille Immunopole group and Explore COVID-19 IPH group and the Explore COVID-19 IPH group and the Explore COVID-19 Marseille Immunopole group
Nature (London), ISSN 0028-0836, 12/2020, Volume 588, Issue 7836, pp. 146 - 150
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | COVID-19 - immunology | Inflammation - pathology | Humans | Bronchoalveolar Lavage Fluid - immunology | Respiratory Distress Syndrome - immunology | Complement C5a - biosynthesis | Inflammation - complications | Respiratory Distress Syndrome - prevention & control | Acute Lung Injury - prevention & control | COVID-19 - complications | Inflammation - drug therapy | Myeloid Cells - immunology | COVID-19 - blood | Receptor, Anaphylatoxin C5a - immunology | SARS-CoV-2 - drug effects | Female | Myeloid Cells - drug effects | Respiratory Distress Syndrome - drug therapy | SARS-CoV-2 - immunology | Acute Lung Injury - drug therapy | Cytokine Release Syndrome - drug therapy | Disease Models, Animal | Cytokine Release Syndrome - immunology | Lung - pathology | CD11b Antigen - immunology | Cytokine Release Syndrome - prevention & control | Mice, Inbred C57BL | Inflammation - immunology | Complement C5a - antagonists & inhibitors | Animals | SARS-CoV-2 - pathogenicity | Receptor, Anaphylatoxin C5a - blood | Lung - drug effects | COVID-19 - pathology | Acute Lung Injury - immunology | Mice | Bronchoalveolar Lavage Fluid - chemistry | CD11b Antigen - metabolism | Complement C5a - immunology | Myeloid Cells - pathology | Receptor, Anaphylatoxin C5a - antagonists & inhibitors | Lung - immunology | Myeloid cells | Immune response | Complement component C5a | Severe acute respiratory syndrome | Viral diseases | Organs | Bronchus | Leukocytes (neutrophilic) | Inflammation | Coronaviruses | Blood | Recruitment | Severe acute respiratory syndrome coronavirus 2 | COVID-19 | Smoke inhalation | Receptors | Cell activation | Respiratory distress syndrome | Lungs | Bronchoalveolar lavage | Respiratory diseases | Monoclonal antibodies | Alveoli | Immune system | Index Medicus
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9. Full Text Contribution of monocytes and macrophages to the local tissue inflammation and cytokine storm in COVID-19: Lessons from SARS and MERS, and potential therapeutic interventions
by Jafarzadeh, Abdollah and Chauhan, Prashant and Saha, Bhaskar and Jafarzadeh, Sara and Nemati, Maryam
Life sciences (1973), ISSN 0024-3205, 09/2020, Volume 257, pp. 118102 - 118102
COVID-19 | SARS-CoV-2 | Monocytes | Macrophages | Pathogenesis | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Betacoronavirus - pathogenicity | Pandemics | Cytokines - metabolism | Severe Acute Respiratory Syndrome - virology | Humans | Pneumonia, Viral - pathology | Severe Acute Respiratory Syndrome - immunology | Monocytes - metabolism | Respiratory Distress Syndrome - immunology | Inflammation - immunology | Coronavirus Infections - immunology | Pneumonia, Viral - immunology | Betacoronavirus - immunology | Respiratory Distress Syndrome - pathology | Macrophages - metabolism | Respiratory Distress Syndrome - virology | Coronavirus Infections - virology | Coronavirus Infections - pathology | Severe Acute Respiratory Syndrome - pathology | Pneumonia, Viral - virology | Chemokines | Acute respiratory distress syndrome | Coronavirus infections | Dendritic cells | Lymphocytes | Inflammation | Disease susceptibility | Interferon | Coronaviruses | Health aspects | Index Medicus
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