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animals (576) 576
shal potassium channels - metabolism (320) 320
rats (266) 266
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shal potassium channels (226) 226
potassium channels (217) 217
shal potassium channels - genetics (202) 202
neurosciences (185) 185
patch-clamp techniques (185) 185
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potassium channels, voltage-gated (168) 168
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potassium channels - metabolism (152) 152
potassium channels - genetics (131) 131
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physiology (117) 117
cardiac & cardiovascular systems (115) 115
female (115) 115
rats, sprague-dawley (114) 114
expression (107) 107
biochemistry & molecular biology (100) 100
cardiovascular system (94) 94
kv channel-interacting proteins - metabolism (93) 93
potassium - metabolism (92) 92
transient outward current (92) 92
potassium channel (88) 88
k+ channel (84) 84
proteins (84) 84
electrophysiology (83) 83
membrane potentials - physiology (83) 83
myocardium - metabolism (83) 83
kinetics (81) 81
article (79) 79
neurons (79) 79
neurons - metabolism (78) 78
cell biology (73) 73
k+ channels (73) 73
myocytes, cardiac - metabolism (73) 73
rna, messenger - metabolism (72) 72
kv4.2 (71) 71
molecular sequence data (71) 71
kv channel-interacting proteins - genetics (69) 69
modulation (67) 67
gene expression (65) 65
potassium (65) 65
amino acid sequence (64) 64
transfection (64) 64
ion channels (62) 62
action potentials - physiology (61) 61
cell line (61) 61
action potentials (59) 59
membrane potentials - drug effects (58) 58
calcium - metabolism (57) 57
i-to (57) 57
ion channel gating - physiology (57) 57
heart (55) 55
neurons - physiology (55) 55
peripheral vascular disease (55) 55
down-regulation (53) 53
shal potassium channels - physiology (53) 53
kv channel-interacting proteins (51) 51
potassium channel blockers - pharmacology (51) 51
xenopus laevis (51) 51
currents (50) 50
immunohistochemistry (50) 50
kv4.3 (50) 50
mice, inbred c57bl (50) 50
potassium channels - physiology (50) 50
rats, wistar (50) 50
long-term potentiation (49) 49
cho cells (48) 48
rodents (48) 48
cricetinae (47) 47
heart ventricles - metabolism (47) 47
potassium channels, voltage-gated - metabolism (47) 47
pyramidal neurons (47) 47
time factors (47) 47
brain (46) 46
biophysics (45) 45
differential expression (45) 45
ion channel gating (45) 45
phosphorylation (45) 45
dose-response relationship, drug (43) 43
hippocampus - metabolism (43) 43
reverse transcriptase polymerase chain reaction (43) 43
blotting, western (42) 42
cells (42) 42
gene-expression (42) 42
inactivation (42) 42
calcium-binding proteins - metabolism (41) 41
mice, knockout (41) 41
physiological aspects (41) 41
protein binding (41) 41
shal potassium channels - chemistry (41) 41
ventricular myocytes (41) 41
action potentials - drug effects (40) 40
channels (40) 40
heart-failure (40) 40
in vitro techniques (40) 40
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1. Full Text Transient outward current (Ito ) gain-of-function mutations in the KCND3 -encoded Kv4.3 potassium channel and Brugada syndrome
by Giudicessi, John R., BA and Ye, Dan, MD and Tester, David J., BS and Crotti, Lia, MD, PhD and Mugione, Alessandra, PhD and Nesterenko, Vladislav V., PhD and Albertson, Richard M., BS and Antzelevitch, Charles, PhD and Schwartz, Peter J., MD and Ackerman, Michael J., MD, PhD
Heart Rhythm, ISSN 1547-5271, 2011, Volume 8, Issue 7, pp. 1024 - 1032
Background Brugada syndrome (BrS) is a sudden death–predisposing genetic condition characterized electrocardiographically by ST segment elevation in the leads... 
Cardiovascular | Kv4.3 channels | J-wave syndromes | Brugada syndrome | I to current | Ion channels | Genetic diseases | Sudden cardiac death | Ito current | CARDIAC & CARDIOVASCULAR SYSTEMS | PHENOTYPE | CHILDREN | CELLULAR BASIS | YOUNG | I-to current | LONG QT SYNDROME | IONIC MECHANISMS | ST-SEGMENT ELEVATION | Brugada Syndrome - genetics | Action Potentials - genetics | Brugada Syndrome - physiopathology | Gene Frequency | Humans | Middle Aged | Cells, Cultured | Genotype | Male | Myocardium - pathology | Mutation, Missense | DNA - genetics | Young Adult | Patch-Clamp Techniques | Shal Potassium Channels - genetics | Shal Potassium Channels - metabolism | Brugada Syndrome - metabolism | Myocardium - metabolism | Alleles | Polymerase Chain Reaction | Electrocardiography | Adult | Female | Genetic disorders | Electrocardiogram | Genetic aspects | Liquid chromatography | Potassium channels | Cardiology | Index Medicus | genetic diseases | ion channels, Ito current | J wave syndromes | sudden cardiac death
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2. Full Text Encephalitis and antibodies to dipeptidyl‐peptidase–like protein‐6, a subunit of Kv4.2 potassium channels
by Boronat, Anna and Gelfand, Jeffrey M and Gresa‐Arribas, Nuria and Jeong, Hyo‐Young and Walsh, Michael and Roberts, Kirk and Martinez‐Hernandez, Eugenia and Rosenfeld, Myrna R and Balice‐Gordon, Rita and Graus, Francesc and Rudy, Bernardo and Dalmau, Josep
Annals of Neurology, ISSN 0364-5134, 01/2013, Volume 73, Issue 1, pp. 120 - 128
Objective: To report a novel cell surface autoantigen of encephalitis that is a critical regulatory subunit of the Kv4.2 potassium channels. Methods: Four... 
LIMBIC ENCEPHALITIS | NMDA-RECEPTOR ENCEPHALITIS | DPPX | NEURONS | PATIENT | NEUROSCIENCES | CLINICAL NEUROLOGY | Encephalitis - enzymology | Nerve Tissue Proteins - immunology | Dipeptidyl-Peptidases and Tripeptidyl-Peptidases - immunology | Shal Potassium Channels - immunology | Autoantibodies - chemistry | Humans | Middle Aged | Potassium Channels - immunology | Encephalitis - pathology | Encephalitis - immunology | Male | Autoantibodies - biosynthesis | Mice, Knockout | Shal Potassium Channels - metabolism | Animals | HEK293 Cells | Female | Shal Potassium Channels - chemistry | Aged | Mice | Antigen-Antibody Reactions - immunology | Proteins | Diarrhea | Encephalitis | Potassium | Index Medicus
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3. Full Text Tetrameric Assembly of K+ Channels Requires ER-Located Chaperone Proteins
by Li, Kai and Jiang, Qiang and Bai, Xue and Yang, Yi-Feng and Ruan, Mei-Yu and Cai, Shi-Qing
Molecular Cell, ISSN 1097-2765, 01/2017, Volume 65, Issue 1, pp. 52 - 65
Tetrameric assembly of channel subunits in the endoplasmic reticulum (ER) is essential for surface expression and function of K channels, but the molecular... 
J-protein | hERG | protein trafficking | long QT syndrome | chaperone protein | C. elegans | protein oligomerization | potassium channel | protein biogenesis | subunit assembly | PAS DOMAIN | CAENORHABDITIS-ELEGANS | HERG | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARDIAC-ARRHYTHMIA | LONG QT SYNDROME | SHAKER POTASSIUM CHANNEL | I-KR | CONFORMATIONAL DEFECT | MEMBRANE-PROTEINS | MOLECULAR-BASIS | CELL BIOLOGY | HSP47 Heat-Shock Proteins - chemistry | Caenorhabditis elegans Proteins - chemistry | Humans | Protein Multimerization | Caenorhabditis elegans Proteins - metabolism | Endoplasmic Reticulum - metabolism | Potassium Channels - metabolism | Shal Potassium Channels - genetics | Transfection | RNA Interference | Time Factors | HEK293 Cells | Protein Structure, Quaternary | ERG1 Potassium Channel - genetics | Protein Stability | HSP40 Heat-Shock Proteins - chemistry | Induced Pluripotent Stem Cells - metabolism | HSP40 Heat-Shock Proteins - metabolism | Caenorhabditis elegans - metabolism | ERG1 Potassium Channel - chemistry | HSP40 Heat-Shock Proteins - genetics | Animals, Genetically Modified | Caenorhabditis elegans - genetics | ERG1 Potassium Channel - metabolism | HSP47 Heat-Shock Proteins - genetics | HSP70 Heat-Shock Proteins - metabolism | Potassium Channels - genetics | Long QT Syndrome - metabolism | Shal Potassium Channels - metabolism | Animals | Membrane Potentials | Cell Line, Tumor | Long QT Syndrome - genetics | Myocytes, Cardiac - metabolism | HSP47 Heat-Shock Proteins - metabolism | Potassium Channels - chemistry | Mutation | Caenorhabditis elegans Proteins - genetics | Index Medicus
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4. Full Text Divergent roles of ALS-linked proteins FUS/TLS and TDP-43 intersect in processing long pre-mRNAs
by Lagier-Tourenne, Clotilde and Polymenidou, Magdalini and Hutt, Kasey R and Vu, Anthony Q and Baughn, Michael and Huelga, Stephanie C and Clutario, Kevin M and Ling, Shuo-Chien and Liang, Tiffany Y and Mazur, Curt and Wancewicz, Edward and Kim, Aneeza S and Watt, Andy and Freier, Sue and Hicks, Geoffrey G and Donohue, John Paul and Shiue, Lily and Bennett, C Frank and Ravits, John and Cleveland, Don W and Yeo, Gene W
Nature Neuroscience, ISSN 1097-6256, 11/2012, Volume 15, Issue 11, pp. 1488 - 1497
FUS/TLS (fused in sarcoma/translocated in liposarcoma) and TDP-43 are integrally involved in amyotrophic lateral sclerosis (ALS) and frontotemporal dementia.... 
NEURODEGENERATIVE DISEASE | GENE | AMYOTROPHIC-LATERAL-SCLEROSIS | FAMILY PROTEINS | FUS PATHOLOGY | MUTATIONS | FRONTOTEMPORAL LOBAR DEGENERATION | BINDING | NEUROSCIENCES | BRAIN | NASCENT TRANSCRIPTION | RNA, Small Interfering - genetics | Protein Binding - genetics | Oligonucleotide Array Sequence Analysis | Humans | tau Proteins - metabolism | Gene Expression Profiling | RNA, Messenger - metabolism | Kv Channel-Interacting Proteins - metabolism | Brain - metabolism | Frontotemporal Dementia - metabolism | RNA Splicing - genetics | Frontotemporal Dementia - genetics | RNA-Binding Protein FUS - deficiency | Amyotrophic Lateral Sclerosis - genetics | Cell Cycle Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | RNA-Binding Protein FUS - genetics | Mice, Knockout | Motor Neurons - metabolism | Amyotrophic Lateral Sclerosis - pathology | Shal Potassium Channels - metabolism | Brain - pathology | Mice | Neurofilament Proteins - metabolism | RNA, Small Interfering - metabolism | Immunoprecipitation | Spinal Cord - metabolism | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | tau Proteins - genetics | Cell Cycle Proteins - genetics | Female | RNA Precursors - metabolism | Excitatory Amino Acid Transporter 2 - genetics | Membrane Proteins - metabolism | Frontotemporal Dementia - pathology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | RNA Precursors - genetics | Protein Structure, Tertiary - genetics | RNA-Binding Protein FUS - metabolism | DNA-Binding Proteins - genetics | Excitatory Amino Acid Transporter 2 - metabolism | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Histone-Lysine N-Methyltransferase - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Neural Cell Adhesion Molecules - metabolism | Neural Stem Cells - metabolism | Cell Line, Transformed | Amyotrophic lateral sclerosis | Development and progression | Genetic aspects | Messenger RNA | Health aspects | Index Medicus
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5. Full Text Electrophysiological consequences of dyssynchronous heart failure and its restoration by resynchronization therapy
by Aiba, Takeshi and Hesketh, Geoffrey G and Barth, Andreas S and Liu, Ting and Daya, Samantapudi and Chakir, Khalid and Dimaano, Veronica Lea and Abraham, Theodore P and O'Rourke, Brian and Akar, Fadi G and Kass, David A and Tomaselli, Gordon F
Circulation, ISSN 0009-7322, 03/2009, Volume 119, Issue 9, pp. 1220 - 1230
Background-Cardiac resynchronization therapy (CRT) is widely applied in patients with heart failure and dyssynchronous contraction (DHF), but the... 
Heart failure | Ion channels | Resynchronization, electrophysiology | Remodeling | heart failure | remodeling | CARDIAC & CARDIOVASCULAR SYSTEMS | SUDDEN-DEATH | resynchronization | IONIC MECHANISM | VENTRICULAR MYOCYTES | QT INTERVAL | ion channels | OUTWARD POTASSIUM CURRENT | BUNDLE-BRANCH-BLOCK | electrophysiology | CARDIAC-RESYNCHRONIZATION | CALCIUM-CHANNELS | FAILING HEART | TRANSMURAL DISPERSION | PERIPHERAL VASCULAR DISEASE | Calcium Channels - metabolism | Calcium - metabolism | Heart Failure - physiopathology | Bundle-Branch Block - physiopathology | Male | Bundle-Branch Block - diagnosis | RNA, Messenger - metabolism | Kv Channel-Interacting Proteins - genetics | Kv Channel-Interacting Proteins - metabolism | Pacemaker, Artificial | Bundle-Branch Block - therapy | Heart Failure - therapy | Shal Potassium Channels - genetics | Electrocardiography | Heart Failure - diagnostic imaging | Calcium Channels - genetics | Coronary Circulation | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Echocardiography | Potassium Channels, Inwardly Rectifying - genetics | Action Potentials - physiology | Patch-Clamp Techniques | Shal Potassium Channels - metabolism | Animals | Myocytes, Cardiac - physiology | Homeostasis - physiology | Dogs | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Potassium Channels, Inwardly Rectifying - metabolism | Care and treatment | Usage | Animal models in research | Cardiac pacing | Electrophysiology | Physiological aspects | Research | Index Medicus | Abridged Index Medicus
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6. Full Text High temperature sensitivity is intrinsic to voltage-gated potassium channels
by Yang, Fan and Zheng, Jie
eLife, ISSN 2050-084X, 2014, Volume 3, pp. e03255 - e03255
Temperature-sensitive transient receptor potential (TRP) ion channels are members of the large tetrameric cation channels superfamily but are considered to be... 
gating | TRP channel | allosteric coupling | potassium channel | conformational changes | temperature sensing | Potassium - metabolism | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Allosteric Regulation | Humans | Shaker Superfamily of Potassium Channels - metabolism | Large-Conductance Calcium-Activated Potassium Channels - chemistry | TRPV Cation Channels - metabolism | Shal Potassium Channels - genetics | Shaker Superfamily of Potassium Channels - chemistry | HEK293 Cells | Large-Conductance Calcium-Activated Potassium Channels - genetics | Shal Potassium Channels - chemistry | Ion Transport | Transgenes | Protein Structure, Tertiary | Shab Potassium Channels - genetics | Gene Expression | Shaker Superfamily of Potassium Channels - genetics | Shab Potassium Channels - chemistry | TRPV Cation Channels - chemistry | Hot Temperature | Membrane Potentials - physiology | TRPV Cation Channels - genetics | Patch-Clamp Techniques | Shab Potassium Channels - metabolism | Shal Potassium Channels - metabolism | Animals | Mice | Ion Channel Gating | GATING CHARGE | DEPENDENT POTASSIUM | ACTIVATION | TRP channels | CAPSAICIN RECEPTOR | TRPV1 ION-CHANNEL | potassium channels | BIOLOGY | CLOSED-STATE INACTIVATION | SHAKER | K+ CHANNEL | PORE DOMAIN | KV4.2 CHANNELS | Data analysis | Pain perception | Channel gating | Capsaicin receptors | Equilibrium | Thermal energy | Transient receptor potential proteins | Heat | Allosteric properties | Temperature effects | Nociceptors | Physiology | Potassium channels (voltage-gated) | Ion channels | Sensors | Potassium | Index Medicus
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7. Full Text Specific sorting and post-golgi trafficking of dendritic potassium channels in living neurons
by Jensen, Camilla Stampe and Watanabe, Shoji and Rasmussen, Hanne Borger and Schmitt, Nicole and Olesen, Søren-Peter and Frost, Nicholas A and Blanpied, Thomas A and Misonou, Hiroaki
Journal of Biological Chemistry, ISSN 0021-9258, 04/2014, Volume 289, Issue 15, pp. 10566 - 10581
Background: Voltage-gated K+ (Kv) channels are localized in specific dendritic subcompartments of neurons to regulate dendritic excitability. Results:... 
LOCALIZATION | Ion Channels | MICROTUBULES | BIOCHEMISTRY & MOLECULAR BIOLOGY | Intracellular Trafficking | IDENTIFICATION | Protein Sorting | TRANSPORT | Actin | Myosin | Golgi | PROTEINS | RECEPTORS | KV2.1 K+ CHANNEL | EXPRESSION | MODULATION | Dendrites - metabolism | Green Fluorescent Proteins - metabolism | Temperature | Rats, Wistar | Signal Transduction | Humans | Rats | Amino Acid Motifs | Protein Transport | Hippocampus - metabolism | Shab Potassium Channels - metabolism | Shal Potassium Channels - metabolism | Animals | HEK293 Cells | Cytoskeleton - metabolism | Golgi Apparatus - metabolism | Myosins - metabolism | Neurons - metabolism | Index Medicus | Neurobiology
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8. Full Text Compartmentalized dendritic plasticity and input feature storage in neurons
by Losonczy, Attila and Makara, Judit K and Magee, Jeffrey C
Nature, ISSN 0028-0836, 03/2008, Volume 452, Issue 7186, pp. 436 - 441
Although information storage in the central nervous system is thought to be primarily mediated by various forms of synaptic plasticity, other mechanisms, such... 
ACTION-POTENTIALS | HIPPOCAMPUS | CELLS | EXCITABILITY | MEMORY | RAT | KV4.2 | SPIKES | MULTIDISCIPLINARY SCIENCES | PATTERNS | CA1 PYRAMIDAL NEURONS | Pyramidal Cells - cytology | Pyramidal Cells - metabolism | Receptors, N-Methyl-D-Aspartate - metabolism | Rats | Male | Rats, Sprague-Dawley | Action Potentials - physiology | Shal Potassium Channels - genetics | Shal Potassium Channels - metabolism | Animals | Neuronal Plasticity - physiology | Cell Shape | Dendrites - physiology | Mice | Models, Neurological | Ion Channel Gating | Shal Potassium Channels - deficiency | Brain | Neurosciences | Membranes | Cellular biology | Electricity | Rodents | Index Medicus
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9. Full Text Exome sequencing identifies de novo gain of function missense mutation in KCND2 in identical twins with autism and seizures that slows potassium channel inactivation
by Lee, Hane and Lin, Meng-chin A and Kornblum, Harley I and Papazian, Diane M and Nelson, Stanley F
Human Molecular Genetics, ISSN 0964-6906, 2014, Volume 23, Issue 13, pp. 3481 - 3489
Numerous studies and case reports show comorbidity of autism and epilepsy, suggesting some common molecular underpinnings of the two phenotypes. However, the... 
YOUNG-ADULTS | INDIVIDUALS | KV4.2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | CLOSED-STATE INACTIVATION | SPECTRUM DISORDERS | EPILEPSY | ASSOCIATION | K+ CHANNELS | MODULATION | CHILDREN | Autistic Disorder - genetics | Seizures - genetics | Humans | Electrophysiology | Male | Mutation, Missense - genetics | Potassium Channels - genetics | Potassium Channels - metabolism | Exome - genetics | Shal Potassium Channels - genetics | Shal Potassium Channels - metabolism | Pedigree | Female | Mutation | Index Medicus
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10. Full Text Regulation of Kv4.3 and hERG potassium channels by KChIP2 isoforms and DPP6 and response to the dual K+ channel activator NS3623
by Lainez, Sergio and Doray, Adélaïde and Hancox, Jules C and Cannell, Mark B
Biochemical Pharmacology, ISSN 0006-2952, 04/2018, Volume 150, pp. 120 - 130
Transient outward potassium current (I ) contributes to early repolarization of many mammalian cardiac action potentials, including human, whilst the rapid... 
Heart | KChIP2 | hERG | DPP6 | Transient outward current | Action potential | Potassium ion channels | NS3623 | PIG VENTRICULAR MYOCYTES | CARDIAC-ARRHYTHMIA | BETA-SUBUNITS | GUINEA-PIG | SPLICE VARIANTS | INTERACTING PROTEIN-2 | REPOLARIZATION | PHARMACOLOGY & PHARMACY | ACTION-POTENTIAL CLAMP | EXPRESSION | I-TO | Kv Channel-Interacting Proteins - agonists | Tetrazoles - pharmacology | Humans | Nerve Tissue Proteins - agonists | ERG1 Potassium Channel - agonists | ERG1 Potassium Channel - metabolism | Kv Channel-Interacting Proteins - metabolism | Nerve Tissue Proteins - metabolism | Potassium Channels - metabolism | Dipeptidyl-Peptidases and Tripeptidyl-Peptidases - metabolism | Shal Potassium Channels - agonists | Shal Potassium Channels - metabolism | Protein Isoforms - metabolism | Protein Isoforms - agonists | HEK293 Cells | Potassium Channels - agonists | Phenylurea Compounds - pharmacology | Index Medicus
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11. Full Text Evaluation of batch variations in induced pluripotent stem cell-derived human cardiomyocytes from 2 major suppliers
by Huo, Jianhua and Kamalakar, Archana and Yang, Xi and Word, Beverly and Stockbridge, Norman and Lyn-Cook, Beverly and Pang, Li
Toxicological Sciences, ISSN 1096-6080, 03/2017, Volume 156, Issue 1, pp. 25 - 38
Drug-induced proarrhythmia is a major safety issue in drug development. Developing sensitive in vitro assays that can predict drug-induced cardiotoxicity in... 
Drug-induced proarrhythmia | iPSC-CMs | Batch variations | ACTION-POTENTIALS | TORSADES-DE-POINTES | DOFETILIDE | CONTRACTILITY | drug-induced proarrhythmia | PROLONGATION | LONG-QT SYNDROME | VENTRICULAR MYOCYTES | MATURATION | IMPEDANCE | TOXICOLOGY | batch variations | RISK-ASSESSMENT | Arrhythmias, Cardiac - chemically induced | Drug Evaluation, Preclinical - economics | Calcium Channel Blockers - adverse effects | Anti-Arrhythmia Agents - pharmacology | Humans | Potassium Channel Blockers - antagonists & inhibitors | Toxicity Tests, Acute - methods | Potassium Channel Blockers - adverse effects | Arrhythmias, Cardiac - pathology | Shal Potassium Channels - genetics | Kv1.5 Potassium Channel - genetics | Toxicity Tests, Acute - economics | Induced Pluripotent Stem Cells - cytology | Voltage-Gated Sodium Channel Blockers - adverse effects | Potassium Channel Blockers - pharmacology | Induced Pluripotent Stem Cells - metabolism | Arrhythmias, Cardiac - metabolism | Induced Pluripotent Stem Cells - pathology | Cell Line | Drug Evaluation, Preclinical - methods | Reproducibility of Results | Induced Pluripotent Stem Cells - drug effects | Myocytes, Cardiac - cytology | Antioxidants - pharmacology | Calcium Channel Blockers - pharmacology | Gene Expression Regulation - drug effects | Myocytes, Cardiac - pathology | Shal Potassium Channels - metabolism | Calcium Channels, L-Type - genetics | Myocytes, Cardiac - drug effects | Cell Differentiation - drug effects | Kv1.5 Potassium Channel - metabolism | Calcium Channel Blockers - chemistry | Myocytes, Cardiac - metabolism | Voltage-Gated Sodium Channel Blockers - pharmacology | Calcium Channels, L-Type - metabolism | Kinetics | Electrophysiological Phenomena - drug effects | Index Medicus
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12. Full Text Cold-Inducible RNA-Binding Protein Regulates Cardiac Repolarization by Targeting Transient Outward Potassium Channels
by Li, Jun and Xie, Duanyang and Huang, Jian and Lv, Fei and Shi, Dan and Liu, Yi and Lin, Li and Geng, Li and Wu, Yufei and Liang, Dandan and Chen, Yi-Han
Circulation Research, ISSN 0009-7330, 05/2015, Volume 116, Issue 10, pp. 1655 - 1659
RATIONALE:Cold-inducible RNA-binding protein (CIRP) is constitutively expressed at low levels across various tissues. It is rapidly upregulated by multiple... 
Repolarization | Action potential | Ion channels | Myocytes | Cardiac | Cold shock proteins | myocytes, cardiac | APOPTOSIS | CELLS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | repolarization | action potential | ion channels | TEMPERATURE | PERIPHERAL VASCULAR DISEASE | cold shock proteins | CIRP | HEMATOLOGY | EXPRESSION | Heart Rate | RNA-Binding Proteins - genetics | Rats, Transgenic | Cold Shock Proteins and Peptides - metabolism | Cells, Cultured | Cold Shock Proteins and Peptides - genetics | Genotype | RNA, Messenger - metabolism | Kv Channel-Interacting Proteins - genetics | Kv Channel-Interacting Proteins - metabolism | Action Potentials | Phenotype | Shal Potassium Channels - genetics | Shal Potassium Channels - metabolism | Animals | Transfection | Time Factors | Myocytes, Cardiac - metabolism | Protein Binding | Transcription, Genetic | Cold Shock Proteins and Peptides - deficiency | Binding Sites | Ion Channel Gating | RNA-Binding Proteins - metabolism | Index Medicus
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13. Full Text Acquired Dendritic Channelopathy in Temporal Lobe Epilepsy
by Christophe Bernard and Anne Anderson and Albert Becker and Nicholas P. Poolos and Heinz Beck and Daniel Johnston
Science, ISSN 0036-8075, 7/2004, Volume 305, Issue 5683, pp. 532 - 535
Inherited channelopathies are at the origin of many neurological disorders. Here we report a form of channelopathy that is acquired in experimental temporal... 
Phosphorylation | Neuroscience | Channelopathies | Neurons | Pyramidal cells | Epilepsy | Temporal lobe epilepsy | Reports | Dendrites | Membrane potential | Seizures | FIRING MODE | ACTIVATED PROTEIN-KINASE | GABAERGIC INHIBITION | POTASSIUM CHANNELS | MULTIDISCIPLINARY SCIENCES | STATUS EPILEPTICUS | H-CHANNELS | HIPPOCAMPAL-NEURONS | UP-REGULATION | K+ CHANNELS | CA1 PYRAMIDAL NEURONS | Shal Potassium Channels | Potassium Channels, Voltage-Gated | Nitriles - pharmacology | Potassium Channels - physiology | Epilepsy, Temporal Lobe - physiopathology | Male | Potassium Channels - metabolism | Protein Kinase C - metabolism | Dendrites - physiology | Pyramidal Cells - physiology | Pilocarpine - administration & dosage | 4-Aminopyridine - pharmacology | Action Potentials - drug effects | Potassium Channel Blockers - pharmacology | Butadienes - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Protein Kinase C - antagonists & inhibitors | Hippocampus - cytology | Potassium Channels - drug effects | Rats, Sprague-Dawley | Animals | Membrane Potentials | Mitogen-Activated Protein Kinases - antagonists & inhibitors | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Hippocampus - physiopathology | Mitogen-Activated Protein Kinases - metabolism |