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Journal Article
Surgery: Official Journal of the Society of University Surgeons, Central Surgical Association, and the American Association of Endocrine Surgeons, ISSN 0039-6060, 2013, Volume 153, Issue 6, pp. 848 - 856
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2018, Volume 200, Issue 5, pp. 1817 - 1828
Journal Article
Expert Opinion on Investigational Drugs, ISSN 1354-3784, 02/2013, Volume 22, Issue 2, pp. 169 - 179
Objective: The aim of the study was find out whether neuronal mitochondrial injury does take place in severe shock and to explore effective therapy for severe... 
mitochondrial injury | hemorrhagic shock | mitochondrial protector | mitochondrial permeability transition pores | polydatin | brain neuron | Brain neuron | Hemorrhagic shock | Mitochondrial protector | Mitochondrial injury | Mitochondrial permeability transition pores | Polydatin | TARGET | PERMEABILITY TRANSITION PORE | RESVERATROL | OXIDATIVE STRESS | REPERFUSION | CYCLOSPORINE-A | ARTERIAL SMOOTH-MUSCLE | PHARMACOLOGY & PHARMACY | UNDERLIES HYPOREACTIVITY | DYSFUNCTION | BRAIN | Stilbenes - administration & dosage | Rats, Wistar | Shock, Hemorrhagic - metabolism | Stilbenes - therapeutic use | Lipid Peroxides - metabolism | Protective Agents - chemistry | Stilbenes - chemistry | Parietal Lobe - metabolism | Protective Agents - therapeutic use | Shock, Hemorrhagic - drug therapy | Membrane Potential, Mitochondrial - drug effects | Mitochondria - ultrastructure | Dose-Response Relationship, Drug | Neurons - ultrastructure | Parietal Lobe - drug effects | Lipid Peroxidation - drug effects | Glucosides - chemistry | Glucosides - therapeutic use | Neurons - metabolism | Neurons - drug effects | Disease Models, Animal | Severity of Illness Index | Shock, Hemorrhagic - pathology | Acute Disease | Drugs, Chinese Herbal | Mitochondrial Membrane Transport Proteins - metabolism | Rats | Mitochondria - metabolism | Mitochondria - drug effects | Parietal Lobe - pathology | Animals | Glucosides - administration & dosage | Survival Analysis | Protective Agents - administration & dosage
Journal Article
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 07/2017, Volume 102, Issue 1, pp. 127 - 134
NK1.1+ cell activation contributes to the persistence of inflammation leading to end organ damage in the liver and gut after HS/T. Various cell populations... 
leukocytes | cytokines | natural killer cell | T cell | Natural killer cell | Leukocytes | Cytokines | NATURAL-KILLER | MACROPHAGES | MEDIATOR | DEPLETION | IMMUNOLOGY | HEPATIC-INJURY | CELL BIOLOGY | SYSTEMIC INFLAMMATION | ORGAN INJURY | NK CELLS | LIVER-INJURY | INFECTION | HEMATOLOGY | Shock, Hemorrhagic - pathology | Wounds and Injuries - pathology | Wounds and Injuries - blood | Alanine Transaminase - blood | Aspartate Aminotransferases - immunology | Wounds and Injuries - immunology | NK Cell Lectin-Like Receptor Subfamily B - immunology | Shock, Hemorrhagic - blood | NK Cell Lectin-Like Receptor Subfamily B - blood | Animals | Antigens, Ly - immunology | Aspartate Aminotransferases - blood | Killer Cells, Natural - immunology | Mice | Killer Cells, Natural - metabolism | Shock, Hemorrhagic - immunology | Alanine Transaminase - immunology | Antigens, Ly - blood | Cytokines - blood | Cytokines - immunology | Flow cytometry | Populations | Liver | Transaminase | Lymphocytes T | Macrophages | Hemorrhage | Interleukin 6 | Cell activation | Rodents | Natural killer cells | Damage | Injuries | Resuscitation | Alanine | Leukocytes (neutrophilic) | Histology | Inflammation | Tumor necrosis factor-α | Trauma | Cytometry | Depletion | Hepatocytes | γ-Interferon | Alanine transaminase | Plasma levels | Interferon | Aspartate aminotransferase | Monocyte chemoattractant protein 1 | Inflammation, Extracellular Mediators, & Effector Molecules
Journal Article