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1. Full Text Hemorrhagic shock activation of NLRP3 inflammasome in lung endothelial cells
by Xiang, Meng and Shi, Xiaolian and Li, Yuehua and Xu, Jia and Yin, Lianhua and Xiao, Guozhi and Scott, Melanie J and Billiar, Timothy R and Wilson, Mark A and Fan, Jie
The Journal of immunology (1950), ISSN 0022-1767, 11/2011, Volume 187, Issue 9, pp. 4809 - 4817
Life Sciences & Biomedicine | Immunology | Science & Technology | Inflammasomes - metabolism | Shock, Hemorrhagic - metabolism | Carrier Proteins - secretion | NLR Family, Pyrin Domain-Containing 3 Protein | Male | Respiratory Mucosa - pathology | Respiratory Mucosa - immunology | HMGB1 Protein - physiology | Respiratory Mucosa - secretion | Disease Models, Animal | Endothelium, Vascular - immunology | Interleukin-1beta - secretion | Shock, Hemorrhagic - pathology | Lung - pathology | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Mice, Knockout | Animals | Endothelium, Vascular - secretion | Inflammasomes - immunology | Endothelium, Vascular - pathology | Mice | Shock, Hemorrhagic - immunology | Lung - secretion | Inflammasomes - secretion | Lung - immunology | Index Medicus | Abridged Index Medicus
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2. Full Text Frontline Science: Macrophage‐derived exosomes promote neutrophil necroptosis following hemorrhagic shock
by Jiao, Yang and Li, Zhigang and Loughran, Patricia A and Fan, Erica K and Scott, Melanie J and Li, Yuehua and Billiar, Timothy R and Wilson, Mark A and Shi, Xueyin and Fan, Jie
Journal of leukocyte biology, ISSN 0741-5400, 02/2018, Volume 103, Issue 2, pp. 175 - 183
cell death | cell‐cell interaction | extracellular vesicles | cell-cell interaction | Immunology | Life Sciences & Biomedicine | Hematology | Science & Technology | Cell Biology | Inflammation - pathology | Shock, Hemorrhagic - pathology | Macrophages, Alveolar - pathology | Reactive Oxygen Species - metabolism | Coculture Techniques | Humans | Mice, Inbred C57BL | NADPH Oxidases - metabolism | Exosomes - pathology | Male | Necrosis | Animals | Mice | Primary Cell Culture | Disease Models, Animal | Neutrophils - pathology | Index Medicus
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3. Full Text Poor microcirculatory flow dynamics are associated with endothelial cell damage and glycocalyx shedding after traumatic hemorrhagic shock
by Naumann, David N and Hazeldine, Jon and Midwinter, Mark J and Hutchings, Sam D and Harrison, Paul
The journal of trauma and acute care surgery, ISSN 2163-0755, 01/2018, Volume 84, Issue 1, pp. 81 - 88
glycocalyx | trauma | microcirculation | Endothelium | hemorrhage | Life Sciences & Biomedicine | Critical Care Medicine | General & Internal Medicine | Surgery | Science & Technology | Biomarkers - metabolism | Shock, Hemorrhagic - pathology | Glycocalyx - pathology | Prospective Studies | Syndecan-1 - metabolism | Shock, Hemorrhagic - metabolism | Endothelial Cells - metabolism | Humans | Middle Aged | Shock, Traumatic - pathology | Male | Microscopy, Video | Microcirculation - physiology | Thrombomodulin - metabolism | Shock, Traumatic - physiopathology | Shock, Hemorrhagic - physiopathology | Adult | Female | Endothelial Cells - pathology | Shock, Traumatic - metabolism | Longitudinal Studies | Complications and side effects | Hemorrhagic shock | Research | Biological markers | Health aspects | Index Medicus | Abridged Index Medicus
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4. Full Text Pathogenesis of filoviral haemorrhagic fevers
by Mahanty, Siddhartha and Bray, Mike
The Lancet infectious diseases, ISSN 1473-3099, 08/2004, Volume 4, Issue 8, pp. 487 - 498
Life Sciences & Biomedicine | Infectious Diseases | Science & Technology | Africa - epidemiology | Marburg Virus Disease - immunology | Marburg Virus Disease - epidemiology | Marburg Virus Disease - pathology | Humans | Ebolavirus - pathogenicity | Marburgvirus - pathogenicity | Hemorrhagic Fever, Ebola - pathology | Ebolavirus - genetics | Hemorrhagic Fever, Ebola - epidemiology | Ebolavirus - immunology | Hemorrhagic Fever, Ebola - etiology | Marburg Virus Disease - etiology | Marburgvirus - genetics | Animals | Marburgvirus - immunology | Hemorrhagic Fever, Ebola - immunology | Disease Reservoirs | Primates | Development and progression | Hemorrhagic fever | Health aspects | Epidemics | Immune response | Laboratories | Pathogenesis | Viral diseases | Coagulation | Septic shock | Viruses | Disseminated intravascular coagulation | Inflammation | Fever | Fatalities | Index Medicus
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5. Full Text Polydatin - a new mitochondria protector for acute severe hemorrhagic shock treatment
by Wang, Xingmin and Song, Rui and Chen, Yunyan and Zhao, Ming and Zhao, Ke-seng
Expert opinion on investigational drugs, ISSN 1354-3784, 02/2013, Volume 22, Issue 2, pp. 169 - 179
mitochondrial injury | hemorrhagic shock | mitochondrial protector | mitochondrial permeability transition pores | polydatin | brain neuron | Brain neuron | Hemorrhagic shock | Mitochondrial protector | Mitochondrial injury | Mitochondrial permeability transition pores | Polydatin | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Stilbenes - administration & dosage | Rats, Wistar | Shock, Hemorrhagic - metabolism | Stilbenes - therapeutic use | Lipid Peroxides - metabolism | Protective Agents - chemistry | Stilbenes - chemistry | Parietal Lobe - metabolism | Protective Agents - therapeutic use | Shock, Hemorrhagic - drug therapy | Membrane Potential, Mitochondrial - drug effects | Mitochondria - ultrastructure | Dose-Response Relationship, Drug | Neurons - ultrastructure | Parietal Lobe - drug effects | Lipid Peroxidation - drug effects | Glucosides - chemistry | Glucosides - therapeutic use | Neurons - metabolism | Neurons - drug effects | Disease Models, Animal | Severity of Illness Index | Shock, Hemorrhagic - pathology | Acute Disease | Drugs, Chinese Herbal | Mitochondrial Membrane Transport Proteins - metabolism | Rats | Mitochondria - metabolism | Mitochondria - drug effects | Parietal Lobe - pathology | Animals | Glucosides - administration & dosage | Survival Analysis | Protective Agents - administration & dosage | Index Medicus
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6. Full Text Blockade of Stellate Ganglion Remediates Hemorrhagic Shock–Induced Intestinal Barrier Dysfunction
by Zhang, Jing and Lin, Xue-Rong and Zhang, Yu-Ping and Zhang, Li-Min and Du, Hui-Bo and Jiang, Li-Na and Zhao, Zi-Gang and Niu, Chun-Yu
The Journal of surgical research, ISSN 0022-4804, 12/2019, Volume 244, pp. 69 - 76
Stellate ganglion blockade | Hemorrhagic shock | Intestinal barrier function | Life Sciences & Biomedicine | Surgery | Science & Technology | Intestinal Diseases - prevention & control | Permeability - drug effects | Shock, Hemorrhagic - complications | Specific Pathogen-Free Organisms | Intestinal Mucosa - innervation | Ropivacaine - administration & dosage | Rats | Male | Survival Rate | Treatment Outcome | Shock, Hemorrhagic - therapy | Nerve Block - methods | Rats, Sprague-Dawley | Stellate Ganglion - drug effects | Animals | Shock, Hemorrhagic - mortality | Anesthetics, Local - administration & dosage | Intestinal Diseases - etiology | Resuscitation | Disease Models, Animal | Intestinal Diseases - pathology | Intestinal Mucosa - pathology | Oxidases | Dextran | Actin | Fluorescein | Lactic acid | Permeability | Muscle proteins | Ganglion | Index Medicus
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7. Full Text Remote ischemic conditioning prevents lung and liver injury after hemorrhagic shock/resuscitation: Potential role of a humoral plasma factor
by Leung, Chung Ho and Caldarone, Christopher A and Wang, Feng and Venkateswaran, Seetha and Ailenberg, Menachem and Vadasz, Brian and Wen, Xiao-Yan and Rotstein, Ori D
Annals of surgery, ISSN 0003-4932, 06/2015, Volume 261, Issue 6, pp. 1215 - 1225
Remote ischemic conditioning | Hemorrhagic shock | Ischemia/reperfusion | Trauma | Resuscitation | Life Sciences & Biomedicine | Surgery | Science & Technology | Chemotaxis, Leukocyte | Reperfusion Injury - etiology | Lung Injury - pathology | Shock, Hemorrhagic - complications | Tumor Necrosis Factor-alpha - blood | Liver Diseases - pathology | Male | Alanine Transaminase - blood | Shock, Hemorrhagic - therapy | Lung Injury - etiology | Interleukin-1beta - blood | Reperfusion Injury - blood | Time Factors | Liver Diseases - etiology | Systemic Inflammatory Response Syndrome - immunology | Systemic Inflammatory Response Syndrome - prevention & control | Disease Models, Animal | Liver Diseases - blood | Animals, Genetically Modified | Lung Injury - blood | Mice, Inbred C57BL | Neutrophils - immunology | Plasma - immunology | Ischemic Preconditioning | Treatment Outcome | Zebrafish | Biomarkers - blood | Shock, Hemorrhagic - blood | Animals | Reperfusion Injury - prevention & control | Reperfusion Injury - immunology | Mice | Shock, Hemorrhagic - immunology | Index Medicus | Abridged Index Medicus
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8. Full Text Hemorrhagic Cholecystitis: An Uncommon Disease Resulting in Hemorrhagic Shock
by Lauria, Alexis L and Bradley, Matthew J and Rodriguez, Carlos J and Franklin, Brenton R
The American surgeon, ISSN 0003-1348, 6/2019, Volume 85, Issue 6, pp. 279 - 281
Life Sciences & Biomedicine | Surgery | Science & Technology | Cholecystitis - diagnosis | Shock, Hemorrhagic - complications | Humans | Cholecystitis - etiology | Male | Shock, Hemorrhagic - diagnosis | Cholecystitis - surgery | Computed Tomography Angiography | Hemorrhage - diagnosis | Laparotomy - methods | Hemorrhage - etiology | Ultrasonography | Aged | Hemorrhage - surgery | Hemostasis, Surgical - methods | Heart failure | Hemorrhagic shock | Complications | Diabetes mellitus | Case reports | Gallbladder diseases | Neuropathy | Patients | Hemorrhage | Disease control | Bleeding | Blood clots | Abdomen | Pathology | Ultrasonic imaging | Hospitals | Pain | Gallbladder | Diabetes | Cholecystitis | Index Medicus
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9. Full Text Protective effects of plasma products on the endothelial-glycocalyx barrier following trauma-hemorrhagic shock: Is sphingosine-1 phosphate responsible?
by Diebel, Mark E and Diebel, Lawrence N and Liberati, David M
The journal of trauma and acute care surgery, ISSN 2163-0755, 11/2019, Volume 87, Issue 5, pp. 1061 - 1069
Blood Component Transfusion - methods | Endothelium - pathology | Human Umbilical Vein Endothelial Cells | Microfluidic Analytical Techniques | Cell Line | Lysophospholipids - metabolism | Shock, Hemorrhagic - pathology | Glycocalyx - pathology | Resuscitation - methods | Shock, Hemorrhagic - etiology | Humans | Wounds and Injuries - complications | Shock, Hemorrhagic - therapy | Endothelium - cytology | Plasma - metabolism | Sphingosine - analogs & derivatives | Wounds and Injuries - therapy | Endothelium - metabolism | Sphingosine - metabolism | Glycocalyx - metabolism | Plasma products | Usage | Glycocalyx | Care and treatment | Hemorrhagic shock | Physiological aspects | Sphingolipids | Health aspects | Index Medicus | Abridged Index Medicus
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10. Full Text Histone deacetylase inhibitors: Isoform selectivity improves survival in a hemorrhagic shock model
by Chang, Panpan and Weykamp, Michael and Dennahy, Isabel S and Williams, Aaron M and Bhatti, Umar F and Liu, Baoling and Nikolian, Vahagn C and Li, Yongqing and Alam, Hasan B
The journal of trauma and acute care surgery, ISSN 2163-0755, 05/2018, Volume 84, Issue 5, pp. 795 - 801
hemorrhagic shock | isoform-selective | prosurvival pathways | valproic acid | Histone deacetylase inhibitors | Life Sciences & Biomedicine | Critical Care Medicine | General & Internal Medicine | Surgery | Science & Technology | Shock, Hemorrhagic - pathology | Phosphorylation | Survival Rate - trends | Apoptosis - drug effects | Rats | Male | Myocardium - pathology | Random Allocation | Shock, Hemorrhagic - drug therapy | Rats, Sprague-Dawley | Animals | Myocardium - metabolism | Shock, Hemorrhagic - mortality | Valproic Acid - therapeutic use | Histone Deacetylase Inhibitors - therapeutic use | Disease Models, Animal | Divalproex | Care and treatment | Usage | Rattus | Research | Valproic acid | Hemorrhage | Index Medicus | Abridged Index Medicus | pro-survival pathways | histone deacetylase inhibitors
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