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1996, Current topics in microbiology and immunology, ISBN 354061026X, Volume 216., viii, 321
Book
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 539 - 550
In contrast to microbially triggered inflammation, mechanisms promoting sterile inflammation remain poorly understood. Damage-associated molecular patterns... 
MEDICINE, RESEARCH & EXPERIMENTAL | DANGER SIGNALS | STERILE INFLAMMATION | CHROMATIN PROTEIN HMGB1 | IN-VIVO | SEPTIC SHOCK | MOBILITY GROUP BOX-1 | RECEPTOR | DEFICIENT MICE | CELL-DEATH | TRANSGENIC MICE | Acetaminophen - adverse effects | Inflammation - chemically induced | Leukocyte Elastase - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Tumor Necrosis Factor-alpha - genetics | Analgesics, Non-Narcotic - pharmacology | Hepatocytes - pathology | Hepatocytes - metabolism | Neutrophil Infiltration | fas Receptor - metabolism | Necrosis - pathology | HMGB1 Protein - genetics | Shock, Septic - metabolism | Inflammation - metabolism | Acetaminophen - pharmacology | Analgesics, Non-Narcotic - adverse effects | HMGB1 Protein - metabolism | fas Receptor - genetics | Chemical and Drug Induced Liver Injury - pathology | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Neutrophils - pathology | Shock, Septic - genetics | Shock, Septic - pathology | Lipopolysaccharides - toxicity | Macrophages - pathology | Shock, Septic - chemically induced | Leukocyte Elastase - genetics | Necrosis - metabolism | Chemical and Drug Induced Liver Injury - genetics | Mice, Knockout | Necrosis - chemically induced | Macrophages - metabolism | Animals | Chemical and Drug Induced Liver Injury - metabolism | Inflammation - genetics | Mice | Necrosis - genetics | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Physiological aspects | Causes of | Inflammation | Genetic aspects | Cell death | Necrosis | Chromosomal proteins | Mass spectrometry | Analysis | Proteins | Studies | Rodents | Mortality | Gangrene | Homeostasis | Mitochondrial DNA | Laboratory animals | Deoxyribonucleic acid--DNA | Apoptosis
Journal Article
Nature Communications, ISSN 2041-1723, 10/2013, Volume 4, Issue 1, pp. 2657 - 2657
Although it has long been known that patients with sepsis often have thrombocytopenia and that septic patients with severe thrombocytopenia have a poor... 
IMMUNE-RESPONSE | TUMOR-NECROSIS-FACTOR | NEUTROPHIL EXTRACELLULAR TRAPS | TOLL-LIKE RECEPTOR-4 | MULTIDISCIPLINARY SCIENCES | AUTOLOGOUS PLATELETS | LIPOPOLYSACCHARIDE | SEPSIS | NF-KAPPA-B | CRITICALLY-ILL PATIENTS | FACTOR-ALPHA | Inflammation - chemically induced | Inflammation - pathology | Blood Platelets - immunology | Receptors, Prostaglandin E, EP4 Subtype - immunology | Antibodies, Anti-Idiotypic - pharmacology | Humans | Macrophages, Peritoneal - pathology | Male | Dinoprostone - immunology | Shock, Septic - therapy | Thrombocytopenia - genetics | Blood Platelets - cytology | Tumor Necrosis Factor-alpha - immunology | Macrophages, Peritoneal - drug effects | Cyclooxygenase 1 - immunology | Disease Models, Animal | Shock, Septic - genetics | Receptors, Prostaglandin E, EP4 Subtype - genetics | Shock, Septic - pathology | Cyclooxygenase 1 - genetics | Shock, Septic - chemically induced | Signal Transduction | Dinoprostone - genetics | Membrane Proteins - genetics | Platelet Transfusion | Mice, Inbred C57BL | Gene Expression Regulation | Macrophages, Peritoneal - immunology | Membrane Proteins - immunology | Thrombocytopenia - chemically induced | Thrombocytopenia - pathology | Animals | Interleukin-6 - immunology | Lipopolysaccharides - pharmacology | Inflammation - genetics | Interleukin-6 - biosynthesis | Thrombocytopenia - prevention & control | Inflammation - prevention & control | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Platelet Glycoprotein GPIb-IX Complex - immunology
Journal Article
Journal Article
Journal Article
SHOCK, ISSN 1073-2322, 05/2016, Volume 45, Issue 5, pp. 518 - 524
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, p. e0148142
Journal Article