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PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 5, p. e36800
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 6, p. e21549
Background: Acidification of the cytoplasm and the extracellular environment is associated with many physiological and pathological conditions, such as intense... 
TUMOR PH | ACTIVATED PROTEIN-KINASE | CARDIAC-ARREST | CYTOPLASMIC PH | MULTIDISCIPLINARY SCIENCES | INTRACELLULAR PH | PHOSPHATIDIC-ACID | TUBEROUS SCLEROSIS | CELL-GROWTH | EXTRACELLULAR PH | HUMAN CANCER | Phosphorylation - physiology | Cell Line | Tumor Suppressor Proteins - metabolism | Humans | Multiprotein Complexes | Signal Transduction - genetics | Mechanistic Target of Rapamycin Complex 1 | Proteins - genetics | Phosphorylation - genetics | Animals | Proteins - metabolism | Tumor Suppressor Proteins - genetics | Cell Line, Tumor | Signal Transduction - physiology | TOR Serine-Threonine Kinases | Hydrogen-Ion Concentration | Tuberous sclerosis | Protein biosynthesis | Hydrogen-ion concentration | Protein kinases | Drugs | Cell culture | Phosphorylation | Tuberous Sclerosis Complex 2 | Brain cancer | Acidification | Raf protein | AKT protein | Cytotoxicity | Biosynthesis | Biochemistry | Kinases | pH effects | Autophagy | Tuberous Sclerosis Complex 1 | Proteins | Breast carcinoma | Signal transduction | Pathways | Hydrogen ions | Immunotherapy | Fibroblasts | Physiology | Vascular endothelial growth factor | Starvation | Extracellular signal-regulated kinase | Energy expenditure | Substrate inhibition | Embryo fibroblasts | Metabolism | Signaling | Acids | Perfusion | Protein synthesis | Protein kinase | Signal processing | Hypoxia | Molecular biology | Biosensors | Cytoplasm | Tumors
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 4, p. e0154638
[This corrects the article DOI: 10.1371/journal.pone.0147830.]. 
Signaling
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 3, p. e16781
Icaritin, a compound from Epimedium Genus, has selective estrogen receptor (ER) modulating activities, and posses antitumor activity. Here, we examined... 
CYCLIN-DEPENDENT KINASES | SIGNALING PATHWAYS | INHIBITION | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | MAP KINASES | DEATH | STRESS | TRANSIENT | ESTROGEN-RECEPTOR | Endometrial Neoplasms - enzymology | Apoptosis - drug effects | Humans | Enzyme Activation - drug effects | Flavonoids - therapeutic use | Proto-Oncogene Proteins c-bcl-2 - metabolism | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Caspases - biosynthesis | Cell Line, Tumor | Enzyme Induction - drug effects | Endometrial Neoplasms - drug therapy | Endometrial Neoplasms - pathology | Female | Cell Proliferation - drug effects | Flavonoids - pharmacology | Cell Cycle - drug effects | Flavonoids - chemistry | Mitogen-Activated Protein Kinase 1 - metabolism | Cytochrome c | Endometrial cancer | Health aspects | Estrogen | Cancer | Apoptosis | Cell proliferation | Cytochrome | Phosphorylation | Bax protein | Bcl-2 protein | Estrogens | Estrogen receptors | Activation | Kinases | MAP kinase kinase | Anticancer properties | Cell activation | Cell growth | Antitumor agents | Ribose | Inhibition | Pretreatment | Endometrium | Cyclin-dependent kinases | Extracellular signal-regulated kinase | Caspase | Poly(ADP-ribose) polymerase | Adenosine diphosphate | MAP kinase | Inhibitors | Cyclin-dependent kinase inhibitor p27
Journal Article
PloS one, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92917
It has been proposed that differential activation kinetics allows cells to use a common set of signaling pathways to specify distinct cellular outcomes. For... 
PHEOCHROMOCYTOMA CELLS | SIGNALING PATHWAYS | ACTIVATED PROTEIN-KINASE | MAP KINASE | MULTIDISCIPLINARY SCIENCES | NEURONAL DIFFERENTIATION | MAMMALIAN-CELLS | PLASMA-MEMBRANE | NUCLEAR TRANSLOCATION | NERVE GROWTH-FACTOR | LIVING CELLS | NIH 3T3 Cells | Protein Transport - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | raf Kinases - metabolism | PC12 Cells | Light | Cell Membrane - metabolism | Cell Membrane - drug effects | Neurites - drug effects | Cell Membrane - radiation effects | Cryptochromes - metabolism | Cell Differentiation - radiation effects | Protein Structure, Tertiary | MAP Kinase Signaling System - radiation effects | Proto-Oncogene Proteins c-raf | Nerve Growth Factor - pharmacology | Rats | MAP Kinase Kinase Kinases - metabolism | Neurites - metabolism | Enzyme Activation - drug effects | Enzyme Activation - radiation effects | Animals | MAP Kinase Signaling System - drug effects | Cell Differentiation - drug effects | Protein Transport - radiation effects | Cryptochromes - chemistry | Mice | Kinetics | Dose-Response Relationship, Radiation | Neurites - radiation effects | Nerve growth factor | Cellular signal transduction | Epidermal growth factor | Protein-protein interactions | Phosphorylation | Raf protein | Stimulation | Activation | Kinases | Proteins | Signal transduction | Cell growth | Cell cycle | Localization | Growth factors | Axonogenesis | Extracellular signal-regulated kinase | Gene expression | Studies | Signaling | Cell lines | Pheochromocytoma cells | Transduction | Genetic engineering | Protein interaction
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35826
Receptor tyrosine kinase signaling cooperates with WNT/beta-catenin signaling in regulating many biological processes, but the mechanisms of their interaction... 
INSULIN | MUTATIONS | MECHANISM | WNT | FGFR3 | MULTIDISCIPLINARY SCIENCES | Phosphorylation | Humans | Gene Expression Regulation | Phosphatidylinositol 3-Kinases - metabolism | Glycogen Synthase Kinase 3 - metabolism | Wnt Proteins - metabolism | Receptor Protein-Tyrosine Kinases | beta Catenin - metabolism | Proto-Oncogene Proteins c-akt - genetics | beta Catenin - genetics | Phosphatidylinositol 3-Kinases - genetics | Low Density Lipoprotein Receptor-Related Protein-6 - genetics | Low Density Lipoprotein Receptor-Related Protein-6 - metabolism | MAP Kinase Signaling System - genetics | Wnt Proteins - genetics | Glycogen Synthase Kinase 3 - genetics | Wnt Signaling Pathway - genetics | HEK293 Cells | Mitogen-Activated Protein Kinases - genetics | Proto-Oncogene Proteins c-akt - metabolism | Mitogen-Activated Protein Kinases - metabolism | Wnt protein | AKT protein | Biology | Kinases | Throat cancer | Proteins | Signal transduction | β-catenin | Immunology | TrkA protein | Protein-tyrosine kinase receptors | Physiology | Standard deviation | Protein-tyrosine kinase | Fibroblast growth factor receptor 2 | Tyrosine | Epidermal growth factor receptors | Extracellular signal-regulated kinase | MAP kinase | Biophysics | Cadherin | Biological activity | 1-Phosphatidylinositol 3-kinase | Golgi apparatus | Medicine | Signaling | Low density lipoprotein receptors | Mutation | Prostate cancer | Fibroblast growth factor receptors | Tumors
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 2017, Volume 37, Issue 7, pp. 1772 - 1784
Triggering Receptor Expressed on Myeloid cells 2 (TREM2), which is expressed on myeloid cells including microglia in the CNS, has recently been identified as a... 
Akt/GSK3β signaling pathway | TREM2 | Cell survival | Alzheimer’s disease | Wnt/β-catenin signaling pathway | Microglia | NEURODEGENERATIVE DISEASE | NERVOUS-SYSTEM | CELLS | cell survival | ALZHEIMERS-DISEASE | AMYLOID PLAQUES | PROLIFERATION | BETA-CATENIN | NEUROSCIENCES | Wnt/beta-catenin signaling pathway | SIGNALING PATHWAY | PSEUDOMONAS-AERUGINOSA | Akt/GSK3 beta signaling pathway | IN-VIVO | Alzheimer's disease | microglia | Cell Cycle - genetics | Cyclin D1 - metabolism | Microglia - metabolism | Adjuvants, Immunologic - pharmacology | Membrane Glycoproteins - metabolism | Caspase 3 - metabolism | Cell Survival - genetics | Wnt Signaling Pathway - physiology | Male | Proteolysis - drug effects | Lithium Chloride - pharmacology | Thiadiazoles - pharmacology | Animals, Newborn | Cell Survival - drug effects | Brain - cytology | Microglia - drug effects | Gene Expression Regulation - genetics | Kainic Acid - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Excitatory Amino Acid Agonists - pharmacology | beta Catenin - metabolism | Membrane Glycoproteins - genetics | Gene Expression Regulation - drug effects | Animals | Wnt Signaling Pathway - drug effects | Cyclin D1 - genetics | Mice | Cell Cycle - drug effects | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | β-catenin signaling pathway | GSK3β signaling pathway | Akt | Wnt
Journal Article
10.