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PLoS pathogens, ISSN 1553-7374, 2018, Volume 14, Issue 8, p. e1007264
Herpes Simplex Virus 1 (HSV1) is amongst the most clinically advanced oncolytic virus platforms. However, efficient and sustained viral replication within... 
RAPAMYCIN | IMMUNE-RESPONSE | MESSENGER-RNA TRANSLATION | MYXOMA-VIRUS | ACTIVATION | INITIATION | ONCOLYTIC VIROTHERAPY | MICROBIOLOGY | RESISTANT | HERPES-SIMPLEX-VIRUS | INTERFERON | VIROLOGY | PARASITOLOGY | Catalytic Domain - drug effects | Humans | Cercopithecus aethiops | Eukaryotic Initiation Factor-4E - metabolism | Phosphoproteins - metabolism | Herpes Simplex - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | Neoplasms - complications | Neoplasms - virology | Neoplasms - genetics | HEK293 Cells | Immediate-Early Proteins - deficiency | Gene Expression Regulation, Neoplastic - drug effects | Organisms, Genetically Modified | Eukaryotic Initiation Factor-4E - genetics | Vero Cells | Cells, Cultured | Signal Transduction - genetics | Phosphoproteins - genetics | Herpesvirus 1, Human - drug effects | Animals | Immediate-Early Proteins - genetics | TOR Serine-Threonine Kinases - chemistry | Herpes Simplex - pathology | Adaptor Proteins, Signal Transducing - genetics | Ubiquitin-Protein Ligases - deficiency | Herpes Simplex - complications | Herpesvirus 1, Human - genetics | Mice | Protein Kinase Inhibitors - pharmacology | Adaptor Proteins, Signal Transducing - metabolism | Neoplasms - pathology | Ubiquitin-Protein Ligases - genetics | Physiological aspects | Genetic aspects | Research | Genetic regulation | Herpes simplex virus | Cancer cells | TOR protein | Transplants & implants | Funding | Xenotransplantation | Herpes simplex | Viruses | Genomes | mRNA | Infections | Biochemistry | Kinases | Proteins | Immunology | Repressors | Transformed cells | Immunotherapy | Xenografts | Oncolysis | Inhibition | Supervision | Medical research | Protein biosynthesis | Rapamycin | Pharmacology | Children & youth | Infectious diseases | Inhibitors | Protein synthesis | Herpes viruses | Replication | Initiation factor eIF-4E | Tumors | Cancer | TOR Serine-Threonine Kinases/antagonists & inhibitiors | Herpes Simplex/genetics | Herpes Simplex/pathology | Neoplasms/pathology | Eukaryotic Initiation Factor-4E/genetics | Ubiquitin-Protein Ligases/deficiency | Immediate-Early Proteins/genetics | Neoplasms/genetics | Herpes Simplex/complications | Life Sciences | Eukaryotic Initiation Factor-4E/metabolism | Neoplasms/complications | Herpesvirus 1, Human/genetics | Catalytic Domain/drug effects | Signal Transduction/genetics | Adaptator Proteins, Signal Transducing/genetics | Gene Expression Regulation, Neoplastic/drug effects | Phosphoproteins/genetics | Adaptator Proteins, Signal Transducing/metabolism | Immediate-Early Proteins/deficiency | Phosphoproteins/metabolims | TOR Serine-Threonine Kinases/chemistry | Protein Kinase Inhibitors/pharmacology | Herpesvirus 1, Human/drug effects | Ubiquitin-Protein Ligases/genetics | Neoplasms/virology
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e109890
Herpes Simplex Virus type-1 (HSV-1) and type-2 (HSV-2) establish life-long infections and cause significant orofacial and genital infections in humans. HSV-1... 
AMINO-TERMINUS | HERPES-SIMPLEX-VIRUS | TYPE-2 INFECTION | IMMUNODEFICIENCY-VIRUS | MULTIDISCIPLINARY SCIENCES | GENITAL HERPES | OCULARLY-INFECTED MICE | HERPES-SIMPLEX-VIRUS-2 INFECTION | T-CELL EPITOPE | GUINEA-PIGS | REPLICATION-COMPETENT | Herpes Simplex - mortality | Humans | Vaccination | Viral Plaque Assay | Viral Proteins - genetics | Injections, Intramuscular | Herpes Simplex Virus Vaccines - immunology | Animals | Herpes Simplex Virus Vaccines - administration & dosage | Herpesvirus 2, Human - immunology | Immunity, Humoral | Virus Replication | Female | Herpes Simplex - prevention & control | Herpesvirus 1, Human - genetics | Herpesvirus 1, Human - immunology | Mice | Mutation | Herpes Simplex Virus Vaccines - genetics | Immunity, Cellular | Disease Models, Animal | Cell culture | Veterinary colleges | Biotechnology | Herpes simplex | Viruses | Infections | Vaccines | Veterinary medicine | Immunity | Immunity (cell-mediated) | Proteins | Lymphocytes | Animal tissues | Human immunodeficiency virus--HIV | Immunity (humoral) | Deoxyribonucleic acid--DNA | Pathogens | Antigens | Immunization | Neurons | Glycoproteins | FDA approval | Membrane proteins | Virology | Axons | Herpes viruses | Blindness | Replication | Laboratory animals | Deoxyribonucleic acid | Recombinant | HIV | DNA | Human immunodeficiency virus
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 491, Issue 7426, pp. 769 - 773
In the course of primary infection with herpes simplex virus 1 (HSV-1), children with inborn errors of toll-like receptor 3 (TLR3) immunity are prone to HSV-1... 
TOLL-LIKE RECEPTOR-3 | TLR3 DEFICIENCY | DOUBLE-STRANDED-RNA | HUMAN ES | PROTECTIVE IMMUNITY | VIRUS ENCEPHALITIS | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | CENTRAL-NERVOUS-SYSTEM | HERPES-SIMPLEX ENCEPHALITIS | RETICULAR THALAMIC NEURONS | Neurons - pathology | Encephalitis, Herpes Simplex - immunology | Humans | Central Nervous System - pathology | Interferons - immunology | Central Nervous System - immunology | Membrane Transport Proteins - deficiency | Neural Stem Cells - immunology | Oligodendroglia - immunology | Astrocytes - immunology | Membrane Transport Proteins - genetics | Toll-Like Receptor 3 - deficiency | Herpesvirus 1, Human - immunology | Cell Differentiation | Toll-Like Receptor 3 - genetics | Induced Pluripotent Stem Cells - cytology | Child | Astrocytes - virology | Central Nervous System - virology | Neurons - virology | Disease Susceptibility | Cell Separation | Induced Pluripotent Stem Cells - virology | Neural Stem Cells - virology | Cells, Cultured | Neurons - immunology | Oligodendroglia - virology | Immunity, Innate | Encephalitis, Herpes Simplex - pathology | Oligodendroglia - pathology | Cell Lineage | Central Nervous System - cytology | Encephalitis, Herpes Simplex - virology | Encephalitis, Herpes Simplex - metabolism | Biomarkers | Herpesvirus 1, Human - pathogenicity | Neurons | Stem cells | Physiological aspects | Toll-like receptors | Virulence (Microbiology) | Research | Health aspects | Herpes simplex virus | Proteins | Epidermal growth factor | Patients
Journal Article
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2018, Volume 99, Issue 1, pp. 56 - 63.e3
Amyloid-β peptide (Aβ) fibrilization and deposition as β-amyloid are hallmarks of Alzheimer’s disease (AD) pathology. We recently reported Aβ is an innate... 
human herpesvirus 6 | herpesviridae | infection | Alzheimer’s disease | oligomers | amyloid-β | herpes simplex virus 1 | β-amyloid peptide | antimicrobial peptide | herpesvirus glycoprotein | Alzheimer's disease | CELL-CULTURE MODEL | SIMPLEX-VIRUS TYPE-1 | SEMENOGELIN-I | ANTIMICROBIAL PEPTIDE | RISK | OLIGOMERIZATION | EXPRESSION | NEUROSCIENCES | CATHELICIDIN LL-37 | HUMAN ALPHA-DEFENSIN | INNATE IMMUNITY | Encephalitis, Viral - virology | Humans | Brain - virology | Herpesvirus 6, Human | Brain - metabolism | Neurofibrillary Tangles - metabolism | Amyloidosis - virology | Amyloid beta-Peptides - metabolism | Encephalitis, Viral - metabolism | Disease Models, Animal | Neurons | Herpesviridae | Cells, Cultured | Mice, Transgenic | Roseolovirus Infections - metabolism | Herpesvirus 1, Human | Alzheimer Disease - virology | Animals | Encephalitis, Herpes Simplex - virology | Encephalitis, Herpes Simplex - metabolism | Alzheimer Disease - metabolism | Plaque, Amyloid - metabolism | Roseolovirus Infections - virology | Mice | Amyloidosis - metabolism | Pathogens | Encephalitis | Cell culture | Brain | Animal models | Peptides | Central nervous system | Herpes simplex | Innate immunity | Infections | Glycoproteins | Investigations | Fibrillogenesis | Microscopy | Transgenic animals | Rodents | β-Amyloid | Amyloidosis
Journal Article