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Journal of Bone and Mineral Research, ISSN 0884-0431, 04/2011, Volume 26, Issue 4, pp. 777 - 791
Mesenchymal stem cell commitment to an osteoprogenitor lineage requires the activity of Runx2, a molecule implicated in the etiopathology of multiple... 
CLEIDOCRANIAL DYSPLASIA | CRANIOFACIAL DEVELOPMENT | TRANSGENIC ANIMAL | NELL‐1 | RUNX2 | NELL-1 | craniofacial development | transgenic animal | cleidocranial dysplasia | CELLS | OSTEOBLAST DIFFERENTIATION | BONE MORPHOGENETIC PROTEIN-2 | CRANIOSYNOSTOSIS-ASSOCIATED GENE | CBFA1-DEFICIENT MICE | BIOLOGY | ENDOCRINOLOGY & METABOLISM | CBFA1 | TRANSCRIPTION FACTOR | EXPRESSION | RNA, Small Interfering - genetics | Osteogenesis - physiology | Core Binding Factor Alpha 1 Subunit - deficiency | Gene Expression - genetics | Alkaline Phosphatase - metabolism | Osteocalcin - genetics | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Glycoproteins - pharmacology | Core Binding Factor Alpha 1 Subunit - metabolism | Skull - pathology | Skull - abnormalities | Craniofacial Abnormalities - pathology | p38 Mitogen-Activated Protein Kinases - metabolism | Cleidocranial Dysplasia - metabolism | Phosphorylation - drug effects | Craniofacial Abnormalities - genetics | Animals, Newborn | Glycoproteins - genetics | Skull - metabolism | Alkaline Phosphatase - genetics | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Calcification, Physiologic - drug effects | Femur - metabolism | Calcification, Physiologic - physiology | Mice, Transgenic | Mice, Knockout | Cleidocranial Dysplasia - genetics | Skull - cytology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Sp7 Transcription Factor | Dura Mater - metabolism | Mice | Calcium-Binding Proteins - pharmacology | Calcium-Binding Proteins - genetics | Osteopontin - genetics | Glycoproteins - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Osteopontin - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Tibia - metabolism | Osteoblasts - cytology | Cell Differentiation - physiology | Craniofacial Abnormalities - metabolism | Calcium-Binding Proteins - metabolism | Haploinsufficiency - genetics | Osteocalcin - metabolism | Tissue Culture Techniques | Osteogenesis - drug effects | Cells, Cultured | Transcription Factors - genetics | Mice, Inbred Strains | Cleidocranial Dysplasia - pathology | Skull - embryology | Animals | Heterozygote | Protein Kinase Inhibitors - pharmacology | Osteoblasts - metabolism | Core Binding Factor Alpha 1 Subunit - genetics | Original
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 05/2011, Volume 26, Issue 5, pp. 1035 - 1046
Journal Article
Stem Cell Research and Therapy, ISSN 1757-6512, 08/2018, Volume 9, Issue 1, pp. 232 - 14
Background: Periodontal ligament stem cells (PDLSCs) are considered as candidate cells for the regeneration of periodontal and alveolar bone tissues. Antisense... 
Periodontal ligament | RNA, circular | MicroRNAs | Osteogenesis | Stem cells | MEDICINE, RESEARCH & EXPERIMENTAL | RNA | REGENERATION | OSTEOGENIC DIFFERENTIATION | circular | CELL & TISSUE ENGINEERING | CELL BIOLOGY | CRITICAL-SIZE | BINDING | MIR-7 | Autoantigens - metabolism | MicroRNAs - antagonists & inhibitors | Bone Regeneration - genetics | Humans | Alkaline Phosphatase - metabolism | RNA - agonists | Male | MicroRNAs - metabolism | Stem Cells - cytology | Periodontal Ligament - metabolism | Stem Cells - metabolism | Autoantigens - genetics | Skull Fractures - metabolism | RNA - genetics | Stem Cell Transplantation | Periodontal Ligament - cytology | Growth Differentiation Factor 5 - genetics | Oligoribonucleotides - metabolism | Skull - injuries | Skull Fractures - pathology | Cell Differentiation | MicroRNAs - agonists | p38 Mitogen-Activated Protein Kinases - metabolism | Osteoblasts - cytology | Skull Fractures - genetics | Osteogenesis - genetics | RNA - metabolism | Skull - metabolism | Alkaline Phosphatase - genetics | RNA - antagonists & inhibitors | Signal Transduction | Gene Expression Regulation | p38 Mitogen-Activated Protein Kinases - genetics | Smad Proteins - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Animals | Mice, Nude | Growth Differentiation Factor 5 - metabolism | Mice | MicroRNAs - genetics | Primary Cell Culture | Osteoblasts - metabolism | Smad Proteins - metabolism | Skull Fractures - therapy | Oligoribonucleotides - genetics | Cellular signal transduction | Research | Cell differentiation | Gene expression | Osteoblasts | Cerebellum | Alkaline phosphatase | Phosphorylation | Smad protein | Stem cell transplantation | Antisense | Kinases | Proteins | Signal transduction | Bone growth | Computed tomography | Degeneration | Alveolar bone | MAP kinase | Ribonucleic acid--RNA | Orthodontics | Polymerase chain reaction | Regeneration | Osteoblastogenesis | Circular RNA | Growth differentiation factor 5 | Immunofluorescence | Ligaments
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2017, Volume 12, Issue 5, p. e0177557
Highly coordinated regulation of cell proliferation and differentiation contributes to the formation of functionally shaped and sized teeth; however, the... 
DENTINOGENESIS IMPERFECTA | METABOLIC CHECKPOINT | MUTATIONS CAUSE | OSTEOBLAST DIFFERENTIATION | MULTIDISCIPLINARY SCIENCES | TEETH | PULP CELLS | AMPK | CELL-PROLIFERATION | GAP-JUNCTION PROTEINS | EXPRESSION | AMP-Activated Protein Kinases - metabolism | Sialoglycoproteins - metabolism | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Aminoimidazole Carboxamide - pharmacology | Ribonucleotides - pharmacology | Cell Differentiation - genetics | Bone Morphogenetic Proteins - metabolism | Transfection | Adenosine Triphosphate - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Phosphorylation - drug effects | Odontoblasts - cytology | Extracellular Matrix Proteins - metabolism | Odontoblasts - metabolism | Tooth Germ - metabolism | RNA, Messenger - genetics | Connexins - genetics | Enzyme Activation - drug effects | Mice, Inbred ICR | Connexins - metabolism | Gene Expression Regulation - drug effects | Dental Papilla - cytology | Animals | Aminoimidazole Carboxamide - analogs & derivatives | Signal Transduction - drug effects | Cell Differentiation - drug effects | Models, Biological | Intracellular Space - metabolism | Odontoblasts - drug effects | Cell Proliferation - drug effects | Smad Proteins - metabolism | RNA, Small Interfering - metabolism | Cellular signal transduction | Genetic aspects | Gene expression | Health aspects | Heart | Pediatrics | Regulations | Transcription factors | Differentiation (biology) | Central nervous system | Nervous system | AKT protein | Lethality | Glucose | Proteins | Cartilage | Bone growth | Cell growth | Cellular communication | Mineralization | Neurodegeneration | Biocompatibility | Tension | Hair | Deafness | AMP | Developmental biology | Teeth | Cell division | Ions | Anatomy | Metabolism | Epithelium | Root development | Embryonic growth stage | Osteoblastogenesis | Stem cells | Mice | Mutation | Endoplasmic reticulum | Hippocampus | Tracers | Dentin | Phosphorylation | Laboratories | Homology | Biology | Kinases | AMP-activated protein kinase | Tissues | Autophagy | Defects | Morphogenesis | Biomedical materials | Metabolites | Rodents | Cell cycle | Heart diseases | Communication | University graduates | Bone morphogenetic protein 2 | Dentistry | Cell differentiation | Ribonucleic acid--RNA | Dental enamel | Skull | Bone | ATP | Apoptosis | RNA | Ribonucleic acid
Journal Article
Cell Metabolism, ISSN 1550-4131, 03/2017, Volume 25, Issue 3, pp. 661 - 672
Journal Article
STEM CELLS, ISSN 1066-5099, 11/2016, Volume 34, Issue 11, pp. 2721 - 2732
Prenatal folic acid (FA) supplementation prevents neural tube defects. Folate receptor alpha (FRα) is critical for embryonic development, including neural... 
Oct4 | Sox2 | Enhancer/promoters | Folate receptor alpha | miR‐138 | Multipotency | Klf4 | miR‐let‐7 | Cranial neural crest cells | miR-138 | miR-let-7 | SURVIVAL | TUBE | TRANSCRIPTIONAL NETWORK | MOUSE | PAX3 | PLURIPOTENCY | MICRORNAS | FOLIC-ACID | CELL & TISSUE ENGINEERING | CELL BIOLOGY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | EMBRYONIC STEM-CELLS | TARGET GENE | HEMATOLOGY | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | Folic Acid - pharmacology | MicroRNAs - antagonists & inhibitors | Transcriptional Activation | MicroRNAs - metabolism | Neural Stem Cells - cytology | SOXB1 Transcription Factors - agonists | Neural Crest - metabolism | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | SOXB1 Transcription Factors - genetics | p300-CBP Transcription Factors - genetics | Gene Expression Regulation, Developmental | Kruppel-Like Transcription Factors - agonists | Kruppel-Like Transcription Factors - metabolism | PAX3 Transcription Factor - genetics | Female | Promoter Regions, Genetic | Neural Crest - cytology | Ribonuclease III - genetics | Ribonuclease III - metabolism | Signal Transduction | Neural Stem Cells - drug effects | PAX3 Transcription Factor - deficiency | Folate Receptor 1 - antagonists & inhibitors | Transcription Factors - genetics | Mice, Knockout | Neural Crest - drug effects | Octamer Transcription Factor-3 - agonists | Transcription Factors - metabolism | p300-CBP Transcription Factors - metabolism | Animals | Histones - genetics | Folate Receptor 1 - genetics | Folate Receptor 1 - metabolism | Octamer Transcription Factor-3 - metabolism | Folic Acid - metabolism | Protein Binding | Mice | MicroRNAs - genetics | Histones - metabolism | Antagomirs - metabolism | Antagomirs - genetics | Kruppel-Like Transcription Factors - genetics | Neural Stem Cells - metabolism | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Embryonic development | Chromatin | Biosynthesis | MicroRNA | Folic acid | Stem cells | Genes | Cell proliferation | Immunoprecipitation | Oct-4 protein | Effector cells | Event-related potentials | Nuclei | Defects | Embryogenesis | KLF4 protein | Coding | Supplementation | Prenatal experience | Neural tube defects | MiRNA | Neural crest | Promoters | Embryonic growth stage | Polymerase chain reaction | Gene silencing | Neural stem cells | Skull
Journal Article
Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7690, pp. 112 - 117
Many craniofacial disorders are caused by heterozygous mutations in general regulators of housekeeping cellular functions such as transcription or ribosome... 
PATHOGENESIS | CELLS | PROTEIN | ENDONUCLEASE | RNA HELICASE DDX21 | DNA | MULTIDISCIPLINARY SCIENCES | PREVENTION | TREACHER-COLLINS-SYNDROME | NEURAL CREST | P53 | Xenopus | Chromatin - metabolism | Cell Nucleolus - pathology | Embryonic Stem Cells - cytology | Mandibulofacial Dysostosis - embryology | Humans | Protein Transport - drug effects | DNA-Directed RNA Polymerases - deficiency | RNA, Ribosomal - genetics | RNA Polymerase I - antagonists & inhibitors | Mandibulofacial Dysostosis - genetics | Cell Nucleus - metabolism | Skull - pathology | Nuclear Proteins - deficiency | Cell Nucleolus - drug effects | Stress, Physiological - drug effects | Neural Crest - enzymology | Benzothiazoles - pharmacology | Phenotype | Zebrafish Proteins - deficiency | Ribosomes - genetics | Mice | DNA Damage | HeLa Cells | Embryonic Stem Cells - metabolism | RNA Helicases - metabolism | Mandibulofacial Dysostosis - pathology | Neural Crest - pathology | Ribosomal Proteins - biosynthesis | Ribosomes - metabolism | DNA, Ribosomal - metabolism | Phosphoproteins - metabolism | Zebrafish - embryology | Cell Nucleus - pathology | Cell Nucleolus - metabolism | DEAD-box RNA Helicases - deficiency | DEAD-box RNA Helicases - metabolism | Nuclear Proteins - genetics | DNA, Ribosomal - genetics | Cell Nucleolus - genetics | Ribosomal Proteins - genetics | Tumor Suppressor Protein p53 - metabolism | RNA, Ribosomal - metabolism | Nuclear Proteins - metabolism | Phosphoproteins - genetics | Organ Specificity | DEAD-box RNA Helicases - genetics | Animals | Phosphoproteins - deficiency | RNA, Ribosomal - biosynthesis | Cell Nucleus - drug effects | Naphthyridines - pharmacology | Apoptosis | Nucleolus organizer region | Genetic aspects | Gene mutations | Observations | Neural crest | Craniofacial dysostosis | Chromatin | Transcription | Genomics | p53 Protein | Genes | DNA damage | Ribosomal DNA | Disorders | Homeostasis | Biosynthesis | Genomes | DNA-directed RNA polymerase | Proteins | DNA helicase | Nucleoli | Embryogenesis | Cell activation | RNA processing | Chemical synthesis | Deoxyribonucleic acid--DNA | Anemia | rRNA | RNA polymerase | Embryonic growth stage | Polymerase | DNA biosynthesis | Craniofacial growth | Ribonucleic acids | Stem cells | Mutation | Developmental disabilities | Tumors | RNA helicase
Journal Article