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Nature, ISSN 0028-0836, 2016, Volume 539, Issue 7629, pp. 378 - 383
Sleep is conserved from invertebrates to vertebrates, and is tightly regulated in a homeostatic manner. The molecular and cellular mechanisms that determine... 
CLOCK GENE | MUTANTS | AROUSAL | RHYTHMS | SALT-INDUCIBLE KINASE | MULTIDISCIPLINARY SCIENCES | CIRCADIAN PACEMAKER | NEURONAL EXCITABILITY | GABAERGIC NEURONS | REM-SLEEP | CIRCUIT | Sleep Deprivation | Phosphorylation | Sleep, REM - physiology | Caenorhabditis elegans Proteins - chemistry | Caenorhabditis elegans Proteins - metabolism | Ion Channels - genetics | Electroencephalography | Drosophila Proteins - metabolism | Drosophila melanogaster - genetics | Nerve Tissue Proteins - chemistry | Time Factors | RNA Splicing - genetics | Conserved Sequence | Electromyography | Neurons - metabolism | Sleep - physiology | Ion Channels - chemistry | Protein-Serine-Threonine Kinases - metabolism | Amino Acid Sequence | Caenorhabditis elegans - genetics | Wakefulness - genetics | Protein-Serine-Threonine Kinases - genetics | Sleep - genetics | Drosophila Proteins - chemistry | Random Allocation | Sleep, REM - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Animals | Ion Channels - metabolism | Mutagenesis | Wakefulness - physiology | Mice | Protein-Serine-Threonine Kinases - chemistry | Drosophila Proteins - genetics | Mutation | Caenorhabditis elegans Proteins - genetics | Homeostasis - genetics | Usage | Sleep | Physiological research | Genetic aspects | Genetic screening | Methods | Proteins | Genotype & phenotype | Rodents | Genetics | Kinases | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, p. e9259
Journal Article
Science Signaling, ISSN 1945-0877, 01/2017, Volume 10, Issue 463, p. eaai9219
Journal Article
PLoS Genetics, ISSN 1553-7390, 07/2011, Volume 7, Issue 7, p. e1002171
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2012, Volume 7, Issue 12, p. e51954
Genetic variants responsible for susceptibility to obesity and its comorbidities among Hispanic children have not been identified. The VIVA LA FAMILIA Study... 
ENERGY-BALANCE | FASTING GLUCOSE-LEVELS | XERODERMA-PIGMENTOSUM | EUROPEAN POPULATIONS | MULTIDISCIPLINARY SCIENCES | QUANTITATIVE TRAIT LOCUS | BODY-MASS INDEX | PHYSICAL-ACTIVITY | VIVA LA FAMILIA | GENOME-WIDE ASSOCIATION | CHILDREN | Anthropometry | Humans | Child, Preschool | Genetic Loci | Obesity - genetics | Hormones - genetics | Young Adult | Inflammation - metabolism | Body Composition - genetics | Fasting - metabolism | Child | Genetic Predisposition to Disease | Hispanic Americans - genetics | Obesity - ethnology | Genotype | Obesity - metabolism | Triglycerides - metabolism | Diet | Energy Metabolism | Hormones - metabolism | Genetic Markers - genetics | Adolescent | Glucose - metabolism | Inflammation - genetics | Polymorphism, Single Nucleotide | Genome-Wide Association Study - methods | Genes | Body weight | Genomics | Hispanic Americans | Disease susceptibility | Genomes | Inflammation | Obesity in children | Genetic markers | Exercise | Metabolites | Analysis | Genetic aspects | Single nucleotide polymorphisms | Energy use | Physical activity | Pathogenesis | Population studies | Single-nucleotide polymorphism | Hormones | Population genetics | Body composition | Antioxidants | Body mass index | Body composition (biology) | Children | Localization | Obesity | Energy expenditure | Triglycerides | ABO system | Loci | Biological activity | Substrates | Genetic variance | Nutrition research | Sleep | Genotyping | Phenotyping | Gene loci | Monocyte chemoattractant protein 1
Journal Article
Genes, Brain and Behavior, ISSN 1601-1848, 03/2006, Volume 5, Issue 2, pp. 150 - 157
We hypothesize that circadian dysfunction could underlie, at least partially, the liability for bipolar 1 disorder (BD1). Our hypothesis motivated tests for... 
genetic | circadian | bipolar I disorder | PER3 | TIMELESS | association | linkage | schizophrenia | ARNTL | schizoaffective disorder | Association | Linkage | Bipolar I disorder | Genetic | Schizophrenia | Schizoaffective disorder | Circadian | SLEEP PHASE SYNDROME | GENOMIC CONTROL | POLYMORPHISM | MORNINGNESS-EVENINGNESS | NEUROSCIENCES | TRANSMISSION | LINKAGE DISEQUILIBRIUM | INDIVIDUAL-DIFFERENCES | RHYTHM DISORDERS | BEHAVIORAL SCIENCES | DIURNAL PREFERENCE | CLOCK | Genetic Testing | Humans | Bipolar Disorder - physiopathology | Male | Chronobiology Disorders - complications | Case-Control Studies | Psychotic Disorders - physiopathology | Schizophrenia - genetics | DNA Mutational Analysis | Schizophrenia - physiopathology | Female | Nuclear Proteins - genetics | Infant, Newborn | Brain Chemistry - genetics | ARNTL Transcription Factors | Chronobiology Disorders - genetics | Genetic Predisposition to Disease - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Gene Expression Regulation - genetics | Circadian Rhythm - genetics | Chronobiology Disorders - physiopathology | Intracellular Signaling Peptides and Proteins | Transcription Factors - genetics | Bipolar Disorder - genetics | Nerve Tissue Proteins - genetics | Period Circadian Proteins | Genome, Human - genetics | Psychotic Disorders - genetics | Biological Clocks - genetics | Polymorphism, Single Nucleotide - genetics | Cell Cycle Proteins | Genetic research | Medicine, Experimental | Medical research | Bipolar disorder | Clinical psychiatry
Journal Article
Forensic Science International, ISSN 0379-0738, 12/2018, Volume 293, pp. 70 - 76
Cases of sudden cardiac death (SCD) in young and apparently healthy individuals represent a devastating event in affected families. Hereditary arrhythmia... 
Genetics | Massive parallel sequencing | Sudden cardiac death | Arrhythmia syndromes | Sudden death | BRUGADA SYNDROME | VARIANTS | MEDICINE, LEGAL | UNEXPLAINED DEATH | EUROPEAN-SOCIETY | HYPERTROPHIC CARDIOMYOPATHY | RECOMMENDATIONS | MOLECULAR AUTOPSY | CARDIAC DEATH | EPIDEMIOLOGY | CHANNELOPATHIES | Cardiac Myosins - genetics | Humans | Connectin - genetics | Myosin Heavy Chains - genetics | Infant | Male | Death, Sudden, Cardiac - etiology | Sequence Analysis, DNA | Genetic Variation | Young Adult | Cardiomyopathies - genetics | Channelopathies - genetics | Calcium Channels, L-Type - genetics | Dystrophin - genetics | Hypertrophy, Left Ventricular - pathology | Ankyrins - genetics | Forensic Genetics | Adult | Female | High-Throughput Nucleotide Sequencing | alpha Catenin - genetics | Microfilament Proteins - genetics | Arrhythmia | Demographic aspects | Cardiac arrest | Causes of | Youth | Genetic aspects | Teenagers | Health aspects | Death of | Heart | Cardiac arrhythmia | Cardiomyopathy | Epilepsy | Genes | Cardiovascular disease | Family medical history | Genetic screening | Gene sequencing | Analgesics | Autopsy | Genetic analysis | Deoxyribonucleic acid--DNA | Genetic disorders | Forensic science | Legal medicine | Mortality | Preventive medicine | Disease prevention | Studies | Pathology | Sleep | Genetic counseling | Death
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2009, Volume 4, Issue 4, pp. e5371 - e5371
Background: Constant hypoxia (CH) and intermittent hypoxia (IH) occur during several pathological conditions such as asthma and obstructive sleep apnea. Our... 
MULTIDISCIPLINARY SCIENCES | Multigene Family | Drosophila melanogaster - physiology | Male | DNA Primers - genetics | Gene Expression Profiling | Drosophila Proteins - metabolism | RNA, Messenger - metabolism | Drosophila melanogaster - genetics | Heat-Shock Proteins - genetics | Time Factors | Base Sequence | Female | Genome, Insect | Disease Models, Animal | Genome-Wide Association Study | Genes, MDR | Oxidoreductases - metabolism | Animals, Genetically Modified | Oxidoreductases - genetics | Heat-Shock Proteins - metabolism | RNA, Messenger - genetics | Gene Expression Regulation | HSP70 Heat-Shock Proteins - genetics | Symporters - metabolism | HSP70 Heat-Shock Proteins - metabolism | Hypoxia - genetics | Animals | Symporters - genetics | ATP Binding Cassette Transporter, Sub-Family B - metabolism | Hypoxia - physiopathology | Drosophila Proteins - genetics | ATP Binding Cassette Transporter, Sub-Family B - genetics | Proteins | DNA microarrays | Analysis | Drosophila | Genes | Genomics | Heat shock proteins | Sleep apnea syndromes | Genomes | Gene expression | Brain | Pediatrics | Transcription | Disease | P elements | Nervous system | Tissues | Defects | Sleep disorders | Apnea | Neurodegeneration | Trends | Bioinformatics | Adaptation | Adenosine | Cell survival | Gene families | Hsp70 protein | Metabolism | Survival | Morbidity | Asthma | Medicine | Studies | Sleep | Cell injury | Molecular modelling | Insects | Hypoxia | Heat shock
Journal Article