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Cell Metabolism, ISSN 1550-4131, 2011, Volume 14, Issue 4, pp. 478 - 490
Journal Article
Circulation, ISSN 0009-7322, 12/2017, Volume 136, Issue 25, pp. 2451 - 2467
BACKGROUND: Pulmonary arterial hypertension (PAH) is characterized by abnormal growth and enhanced glycolysis of pulmonary artery endothelial cells. However,... 
microRNAs | hypertension, pulmonary | endothelial cells | metabolism | endothelial progenitor cells | glycolysis | APOPTOSIS | COACTIVATOR | CARDIAC & CARDIOVASCULAR SYSTEMS | hypertension pulmonary | TUMOR | INHIBITION | MIR-124 | GROWTH | PERIPHERAL VASCULAR DISEASE | EXPRESSION | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Humans | MicroRNAs - metabolism | Polypyrimidine Tract-Binding Protein - antagonists & inhibitors | Familial Primary Pulmonary Hypertension - metabolism | Lim Kinases - metabolism | Monocarboxylic Acid Transporters - metabolism | RNA Interference | Smad5 Protein - metabolism | Bone Morphogenetic Protein Receptors, Type II - antagonists & inhibitors | Disease Models, Animal | Familial Primary Pulmonary Hypertension - pathology | Bone Morphogenetic Protein Receptors, Type II - genetics | Pyruvate Kinase - metabolism | Endothelial Cells - metabolism | Cells, Cultured | Heterogeneous-Nuclear Ribonucleoproteins - metabolism | Rats | Familial Primary Pulmonary Hypertension - genetics | Heterogeneous-Nuclear Ribonucleoproteins - antagonists & inhibitors | Polypyrimidine Tract-Binding Protein - metabolism | Bone Morphogenetic Protein Receptors, Type II - metabolism | Symporters - metabolism | Heterogeneous-Nuclear Ribonucleoproteins - genetics | Animals | Endothelial Cells - cytology | Glycolysis | Smad1 Protein - metabolism | MicroRNAs - genetics | Antagomirs - metabolism | Pyruvate Kinase - genetics | Polypyrimidine Tract-Binding Protein - genetics | RNA, Small Interfering - metabolism
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 01/2016, Volume 8, Issue 1, pp. 6 - 24
Cerebral cavernous malformations (CCMs) are vascular malformations located within the central nervous system often resulting in cerebral hemorrhage.... 
endothelial cells | TGFβ‐BMP | CCM | KLF4 | EndMT | Endothelial cells | TGFβ-BMP | MEDICINE, RESEARCH & EXPERIMENTAL | RHO KINASE | BLOOD-BRAIN-BARRIER | KRUPPEL-LIKE FACTORS | ENDOTHELIAL-MESENCHYMAL TRANSITION | TGF beta-BMP | SIGNALING PATHWAY | VASCULAR INTEGRITY | SHEAR-STRESS | VE-CADHERIN | GROWTH-FACTOR | TRANSCRIPTION FACTOR | Cell Proliferation | KRIT1 Protein | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Hemangioma, Cavernous, Central Nervous System - genetics | Hemangioma, Cavernous, Central Nervous System - metabolism | Bone Morphogenetic Protein 6 - genetics | RNA Interference | Kruppel-Like Transcription Factors - metabolism | HEK293 Cells | Disease Models, Animal | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Bone Morphogenetic Protein 6 - antagonists & inhibitors | Proto-Oncogene Proteins - genetics | Microtubule-Associated Proteins - antagonists & inhibitors | Mitogen-Activated Protein Kinase 7 - metabolism | Disease Progression | Mice, Knockout | Animals | Bone Morphogenetic Protein 6 - metabolism | Hemangioma, Cavernous, Central Nervous System - pathology | Endothelial Cells - cytology | Smad1 Protein - metabolism | Mice | Mutation | Kruppel-Like Transcription Factors - antagonists & inhibitors | Kruppel-Like Transcription Factors - genetics | Transforming Growth Factor beta - metabolism | Brain | Bone morphogenetic proteins | Transforming growth factors | Mortality | Stem cells | Vascular Biology & Angiogenesis | Genetics, Gene Therapy & Genetic Disease | Cardiovascular System | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Molecular human reproduction, ISSN 1360-9947, 03/2017, Volume 23, Issue 3, pp. 155 - 165
STUDY QUESTION: Does bone morphogenetic protein 2 (BMP2) regulate connexin43 (Cx43) and modulate cell-cell communication in luteinized human granulosa cells?... 
ALK | luteinized human granulosa cells | BMP2 | SMAD | connexin43 | HUMAN OVARIES | PENTRAXIN 3 | RECEPTOR | DEVELOPMENTAL BIOLOGY | OBSTETRICS & GYNECOLOGY | RECOMBINANT BMP4 | IN-VITRO | REPRODUCTIVE BIOLOGY | GAP-JUNCTIONS | INTERCELLULAR COMMUNICATION | BOVINE CORPUS-LUTEUM | PROGESTERONE PRODUCTION | ESTROUS-CYCLE | Bone Morphogenetic Protein 2 - pharmacology | Gap Junctions - metabolism | RNA, Small Interfering - genetics | Smad8 Protein - genetics | Luteal Cells - metabolism | Smad8 Protein - antagonists & inhibitors | Humans | Quinolines - pharmacology | Protein Isoforms - metabolism | Smad5 Protein - metabolism | Bone Morphogenetic Protein 2 - metabolism | Smad1 Protein - genetics | Female | Phosphorylation - drug effects | Smad5 Protein - genetics | Connexin 43 - genetics | Pyrazoles - pharmacology | Recombinant Proteins - metabolism | Bone Morphogenetic Protein 2 - genetics | Luteal Cells - drug effects | Connexin 43 - metabolism | Signal Transduction | Smad5 Protein - antagonists & inhibitors | Gene Expression Regulation | Activin Receptors, Type I - metabolism | Recombinant Proteins - genetics | Pyrimidines - pharmacology | Smad1 Protein - antagonists & inhibitors | Activin Receptors, Type I - genetics | Luteal Cells - cytology | Cell Communication - drug effects | Smad1 Protein - metabolism | Connexin 43 - antagonists & inhibitors | Smad8 Protein - metabolism | Gap Junctions - drug effects | Cell Line, Transformed | Protein Isoforms - genetics | RNA, Small Interfering - metabolism
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 01/2004, Volume 126, Issue 1, pp. 111 - 121
Background & Aims: The recent findings of bone morphogenetic protein (BMP) receptor la mutations in juvenile polyposis and frequent Smad4 mutations in colon... 
I RECEPTORS | CADHERIN | APOPTOSIS | GROWTH-FACTOR-BETA | SMAD PROTEINS | CANCER CELLS | MOUSE | GERMLINE MUTATIONS | JUVENILE POLYPOSIS | INDUCTION | GASTROENTEROLOGY & HEPATOLOGY | Bone Morphogenetic Proteins - antagonists & inhibitors | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Cytoskeletal Proteins - antagonists & inhibitors | Apoptosis - drug effects | Humans | Colonic Neoplasms - metabolism | Colonic Neoplasms - physiopathology | DNA-Binding Proteins - metabolism | Bone Morphogenetic Proteins - metabolism | Bone Morphogenetic Protein Receptors, Type I | Colon - physiopathology | Bone Morphogenetic Proteins - pharmacology | Protein-Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta | Carrier Proteins | beta Catenin | Bone Morphogenetic Protein 2 | Smad4 Protein | Bone Morphogenetic Protein Receptors, Type II | Cell Division - drug effects | Colon - metabolism | Adenomatous Polyposis Coli - metabolism | Animals | Cell Differentiation - drug effects | Smad1 Protein | Smad Proteins | Bone Morphogenetic Protein Receptors | Cell Line, Tumor | Trans-Activators - metabolism | Mice | Proteins - pharmacology | Trans-Activators - antagonists & inhibitors | Receptors, Growth Factor - metabolism | Immunohistochemistry | Bone morphogenetic proteins | Cellular signal transduction | Colon cancer | Transforming growth factors
Journal Article
Developmental Biology, ISSN 0012-1606, 07/2012, Volume 367, Issue 2, pp. 216 - 227
Bone Morphogenetic Proteins (BMPs) have multiple activities in the developing spinal cord: they specify the identity of the dorsal-most neuronal populations... 
Receptor-activated Smads | Spinal cord | Interneurons | Neurons | BMP signaling | Neural progenitors | Smad1 | Axon outgrowth | BMPs | Smads | Dorsal spinal cord | Cell fate | Bone morphogenetic proteins | Smad5 | Axon guidance | Commissural axons | SIGNALING PATHWAYS | TGF-BETA | NEURAL-TUBE | DEVELOPMENTAL BIOLOGY | ROOF PLATE | SONIC HEDGEHOG | CONDITIONAL KNOCKOUT | SYNAPTIC GROWTH | COMMISSURAL INTERNEURONS | COORDINATE REGULATION | Immunohistochemistry | RNA, Small Interfering - genetics | Spinal Cord - metabolism | Avian Proteins - antagonists & inhibitors | Neurogenesis | In Situ Hybridization | Gene Expression Regulation, Developmental | Mice, Mutant Strains | Smad5 Protein - metabolism | Base Sequence | Smad1 Protein - genetics | Interneurons - cytology | Smad5 Protein - genetics | Spinal Cord - cytology | Avian Proteins - metabolism | Smad Proteins, Receptor-Regulated - genetics | Smad Proteins, Receptor-Regulated - antagonists & inhibitors | Smad5 Protein - antagonists & inhibitors | Axons - metabolism | Bone Morphogenetic Protein Receptors - metabolism | Rats | Smad Proteins, Receptor-Regulated - metabolism | Mice, Transgenic | Smad1 Protein - antagonists & inhibitors | Chick Embryo | Spinal Cord - embryology | Animals | Interneurons - metabolism | Smad1 Protein - metabolism | Mice | Models, Neurological | Avian Proteins - genetics | Proteins | Animal experimentation | Neurosciences
Journal Article
Journal of Dental Research, ISSN 0022-0345, 4/2012, Volume 91, Issue 4, pp. 407 - 412
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 04/2018, Volume 46, Issue 1, pp. 133 - 147
Background/Aims: Bone nonunion remains a challenge for orthopaedists. The technological advancements that have been made in precisely silencing target genes... 
Bone morphogenetic protein-2 | Chordin | 5-imine | Human bone marrow mesenchymal stem cells | Polyspermine imidazole-4 | Osteogenesis | PHYSIOLOGY | OSTEOBLAST DIFFERENTIATION | POLYETHYLENIMINE | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | BMP-INHIBITORS | REPAIR | GENE | IN-VIVO | MORPHOGENETIC PROTEIN ANTAGONISTS | HUMAN FRACTURES | EXPRESSION | Bone Regeneration - physiology | Humans | Glycoproteins - metabolism | Imidazoles - chemistry | Intercellular Signaling Peptides and Proteins - chemistry | Bone Morphogenetic Protein 4 - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | RNA, Small Interfering - chemistry | Intercellular Signaling Peptides and Proteins - metabolism | RNA Interference | Bone Morphogenetic Protein 2 - metabolism | Mesenchymal Stem Cells - cytology | Polyethyleneimine - chemistry | Cell Differentiation | Mesenchymal Stem Cells - metabolism | Spermine - analogs & derivatives | Glycoproteins - genetics | Bone Marrow Cells - cytology | Carrier Proteins - antagonists & inhibitors | Cells, Cultured | Glycoproteins - antagonists & inhibitors | Intercellular Signaling Peptides and Proteins - genetics | Spermine - chemistry | Carrier Proteins - genetics | Carrier Proteins - metabolism | Smad1 Protein - metabolism | Fractures, Bone - pathology | Hydrogen-Ion Concentration | RNA, Small Interfering - metabolism | Nanoparticles | Proteins | Biomedical materials | Fractures | Cytotoxicity | Gene therapy | Gene expression | Patients
Journal Article
Development (Cambridge), ISSN 0950-1991, 12/2017, Volume 144, Issue 23, pp. 4298 - 4312
Journal Article
Journal Article
STEM CELLS, ISSN 1066-5099, 08/2009, Volume 27, Issue 8, pp. 1741 - 1749
Journal Article