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Journal of Cellular Biochemistry, ISSN 0730-2312, 07/2018, Volume 119, Issue 7, pp. 5175 - 5185
miR‐29b was significantly increased during osteoblastic differentiation of hAVICs. The osteoblastic differentiation of hAVICs was significantly inhibited by... 
valvular calcification | miR‐29b | wnt/β‐catenin signaling | calcific aortic valve disease | Runx2/Smad3/TGF‐β3 signaling | miR-29b | Runx2/Smad3/TGF-β3 signaling | wnt/β-catenin signaling | Transforming Growth Factor beta3 - genetics | Humans | Middle Aged | Male | MicroRNAs - metabolism | Smad3 Protein - metabolism | Transforming Growth Factor beta3 - metabolism | Wnt3 Protein - genetics | Aortic Valve - pathology | Cell Differentiation - genetics | Smad3 Protein - genetics | Aged, 80 and over | Female | Wnt3 Protein - metabolism | Cell Differentiation - physiology | Cell Line | Aortic Valve Stenosis | Signal Transduction - genetics | Aortic Valve - cytology | beta Catenin - metabolism | beta Catenin - genetics | Calcinosis | Signal Transduction - physiology | Aged | MicroRNAs - genetics | Calcification (ectopic) | Wnt protein | Calcium | Differentiation (biology) | Interstitial cells | Smad3 protein | mRNA | Kinases | Osteoblasts | Alizarin | Western blotting | Proteins | Biomedical materials | Mineralization | Aorta | Biocompatibility | Inhibition | Bioinformatics | Catenin | Deposition | Cbfa-1 protein | Markers | MiRNA | Molecular chains | Nodules | Signaling | Valve leaflets | Osteoblastogenesis | Calcification | Immunofluorescence | Aortic valve | Index Medicus | TGF‐β3 signaling | Smad3 | β‐catenin signaling | wnt | Runx2
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2017, Volume 127, Issue 10, pp. 3770 - 3783
The master cytokine TGF-beta mediates tissue fibrosis associated with inflammation and tissue injury. TGF-beta induces fibroblast activation and... 
GENETIC MANIPULATION | MEDICINE, RESEARCH & EXPERIMENTAL | HYPERTROPHY | GROWTH-FACTOR-BETA | HEART-DISEASE | TGF-BETA | SMAD3 | MYOCARDIAL-INFARCTION | IN-VIVO | MYOFIBROBLAST DIFFERENTIATION | PROTECTS | Receptors, Transforming Growth Factor beta - genetics | Heart Diseases - metabolism | Male | Smad3 Protein - metabolism | Myofibroblasts - metabolism | Smad3 Protein - genetics | Gene Deletion | Myocardium - metabolism | Smad2 Protein - genetics | Protein-Serine-Threonine Kinases - metabolism | Myofibroblasts - pathology | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Smad2 Protein - metabolism | Mice, Transgenic | Myocardium - pathology | Organ Specificity | Myocytes, Cardiac - pathology | Animals | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Fibrosis | Myocytes, Cardiac - metabolism | Mice | Transforming Growth Factor beta - metabolism | Heart Diseases - genetics | Heart Diseases - pathology | Transcription factors | Phosphorylation | Heart attacks | Disease | Homeostasis | Smad3 protein | Gene deletion | Kinases | Proteins | Clonal deletion | Smad2 protein | Rodents | Fibroblasts | Extracellular matrix | Heart diseases | Growth factors | Heart failure | Cytokines | Cardiomyocytes | Gene expression | Pressure | Latency | Adenoviruses | Genetic engineering | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, pp. e33766 - e33766
MicroRNAs (miRNAs) are non-coding RNAs that regulate gene expression in post-transcriptional fashion, and emerging studies support their importance in... 
TRANSCRIPTION FACTORS | FIBROSIS | TGF-BETA | SMAD3 | RNA-SEQ | MYOGENESIS | BIOLOGY | DUCHENNE MUSCULAR-DYSTROPHY | DIFFERENTIATION | MICRORNAS | INJURED SKELETAL-MUSCLE | RNA, Small Interfering - genetics | MicroRNAs - antagonists & inhibitors | Transcriptome | MicroRNAs - metabolism | Cell Transdifferentiation - genetics | Smad3 Protein - metabolism | YY1 Transcription Factor - metabolism | Myofibroblasts - metabolism | Myoblasts, Skeletal - cytology | Smad3 Protein - genetics | Cell Transdifferentiation - physiology | Base Sequence | Repressor Proteins - metabolism | Myoblasts, Skeletal - metabolism | Cell Line | Promoter Regions, Genetic | Down-Regulation | MyoD Protein - metabolism | Polycomb-Group Proteins | Smad3 Protein - antagonists & inhibitors | Myofibroblasts - cytology | Animals | Models, Biological | Signal Transduction - physiology | Mice | MicroRNAs - genetics | Transforming Growth Factor beta - metabolism | Bone morphogenetic proteins | Genetic aspects | MicroRNA | Genetic transcription | Transforming growth factors | Genes | Post-transcription | Health sciences | Pathogenesis | Transforming growth factor-b | Clinical trials | Smad3 protein | Kinases | Muscular dystrophy | Gene sequencing | Proteins | Cell growth | Rodents | Cell adhesion | Cell cycle | Extracellular matrix | Trends | Inhibition | Hypertension | MyoD protein | Gynecology | MiRNA | Muscles | Gene expression | Ribonucleic acid--RNA | Obstetrics | Biological activity | Myogenesis | Myoblasts | Polycomb group proteins | Pathology | Musculoskeletal system | Signaling | Hypotheses | Differentiation | Index Medicus | RNA | Ribonucleic acid
Journal Article
Molecules, ISSN 1420-3049, 2018, Volume 23, Issue 1, p. 215
Journal Article
细胞研究:英文版, ISSN 1001-0602, 2011, Volume 21, Issue 11, pp. 1591 - 1604
Journal Article
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 06/2017, Volume 42, Issue 1, pp. 357 - 372
Endothelial-to-mesenchymal transition (EndMT) plays significant roles under various pathological conditions including cardiovascular diseases, fibrosis, and... 
Endothelial-to-mesenchymal transition | MALAT1 | SMAD3 | Endothelial progenitor cells | miR-145 | TGFBR2 | Receptors, Transforming Growth Factor beta - genetics | Endothelial Progenitor Cells - cytology | MicroRNAs - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - chemistry | Humans | Transforming Growth Factor beta1 - metabolism | Collagen Type III - metabolism | Epithelial-Mesenchymal Transition - drug effects | MicroRNAs - metabolism | Smad3 Protein - metabolism | Smad3 Protein - genetics | Receptor, Transforming Growth Factor-beta Type II | RNA Interference | Base Sequence | 3' Untranslated Regions | Protein-Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - pharmacology | Endothelial Progenitor Cells - metabolism | Collagen Type I - metabolism | Signal Transduction | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | RNA, Long Noncoding - genetics | Down-Regulation - drug effects | Sequence Alignment | Receptors, Transforming Growth Factor beta - metabolism | Smad3 Protein - chemistry | MicroRNAs - genetics | Protein-Serine-Threonine Kinases - chemistry | Antagomirs - metabolism | RNA, Long Noncoding - antagonists & inhibitors | RNA, Long Noncoding - metabolism | RNA, Small Interfering - metabolism | Studies | Liver cancer | Cell growth | MicroRNAs | Rodents | Smooth muscle | Metastasis | Growth factors | Cell adhesion & migration | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e90807 - e90807
Objective: Increasing evidence shows that TGF-beta 1 is a key mediator in diabetic nephropathy (DN) and induces renal fibrosis positively by Smad3 but... 
RENAL FIBROSIS | GROWTH-FACTOR-BETA | ACTIVATION | TGF-BETA | SMAD3 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DIVERSE ROLES | RECEPTOR | NEPHROPATHY | MICRORNA | Receptors, Transforming Growth Factor beta - genetics | Rats, Wistar | Transforming Growth Factor beta1 - antagonists & inhibitors | Smad7 Protein - agonists | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetic Nephropathies - drug therapy | Drugs, Chinese Herbal - pharmacology | Male | Smad3 Protein - metabolism | Diabetes Mellitus, Experimental - blood | Smad3 Protein - genetics | Smad7 Protein - genetics | Smad7 Protein - metabolism | Diabetic Nephropathies - pathology | Diabetic Nephropathies - metabolism | Gene Expression Regulation | Rats | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Smad3 Protein - antagonists & inhibitors | Hypoglycemic Agents - pharmacology | Animals | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Diabetes Mellitus, Experimental - pathology | Blood Glucose - metabolism | Health sciences | Smad protein | Collagen (type I) | Syngeneic grafts | Streptozocin | Body weight | Smad3 protein | Fibronectin | Receptors | Chinese medicine | Rodents | Extracellular matrix | Inhibition | Drug dosages | Growth factors | Solvents | Diabetes mellitus | MiRNA | Traditional Chinese medicine | Pharmacology | Chemical compounds | Signaling | Hospitals | Nephropathy | Acids | Granular materials | MicroRNAs | Fibrosis | Smad7 protein | Research design | Diabetes | Kidney diseases | Proteinuria | Index Medicus
Journal Article
Journal Article
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