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Blood, ISSN 0006-4971, 08/2008, Volume 112, Issue 4, pp. 1503 - 1509
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 07/2012, Volume 32, Issue 14, pp. 2904 - 2916
Journal Article
Nucleic Acids Research, ISSN 0305-1048, 09/2018, Volume 46, Issue 17, pp. 9220 - 9235
Journal Article
Nature Genetics, ISSN 1061-4036, 11/2013, Volume 45, Issue 11, pp. 1309 - 1321
Journal Article
Journal Article
Endocrinology, ISSN 0013-7227, 10/2017, Volume 158, Issue 10, pp. 3501 - 3511
Bone morphogenetic protein 2 (BMP2) belongs to the transforming growth factor-beta superfamily and plays a critical role in regulating ovarian follicle... 
OVARY | STEROIDOGENESIS | ACTIVIN-A | FERTILIZATION | BONE MORPHOGENETIC PROTEIN-2 | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | TGF-BETA SUPERFAMILY | PROGESTERONE PRODUCTION | GROWTH-DIFFERENTIATION FACTOR-8 | ESTRADIOL PRODUCTION | Bone Morphogenetic Protein 2 - pharmacology | Luteal Cells - metabolism | Ovulation Induction | Humans | Smad8 Protein - drug effects | Serum Amyloid P-Component - drug effects | Gene Knockdown Techniques | C-Reactive Protein - metabolism | Smad4 Protein - genetics | Smad5 Protein - metabolism | Dioxoles - pharmacology | Female | Benzamides - pharmacology | C-Reactive Protein - drug effects | Phosphorylation - drug effects | Pyrazoles - pharmacology | Luteal Cells - drug effects | Bone Morphogenetic Protein Receptors, Type II - genetics | Enzyme-Linked Immunosorbent Assay | Down-Regulation | Bone Morphogenetic Protein Receptors, Type I - genetics | C-Reactive Protein - genetics | Pyrimidines - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Activin Receptors, Type I - genetics | Blotting, Western | Serum Amyloid P-Component - genetics | Smad5 Protein - drug effects | Activin Receptors, Type II - genetics | Smad1 Protein - metabolism | Protein Kinase Inhibitors - pharmacology | RNA, Small Interfering | Serum Amyloid P-Component - metabolism | Smad1 Protein - drug effects | Smad8 Protein - metabolism | Phosphorylation | Bone morphogenetic protein 2 | Bone morphogenetic protein receptor type II | Lutein | Homology | Ovulation | Smad4 protein | Signaling | Receptors | Molecular modelling | Luteinizing hormone | Activin | Growth factors | Expansion | Pentraxins
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0120045 - e0120045
Cervical cancer is the major cause of cancer related deaths in women, especially in developing countries and Human Papilloma Virus infection in conjunction... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | GROWTH-FACTOR-BETA | INVASION | MULTIDISCIPLINARY SCIENCES | TRANSFORMING GROWTH-FACTOR-BETA-1 | PANCREATIC-CANCER | GENE-EXPRESSION | MECHANISMS | HUMAN-PAPILLOMAVIRUS | TRANSCRIPTION FACTOR | Receptors, Transforming Growth Factor beta - genetics | Humans | Collagen - chemistry | Epithelial-Mesenchymal Transition - drug effects | Wnt Proteins - metabolism | Smad4 Protein - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Peptidylprolyl Isomerase - metabolism | Peptidylprolyl Isomerase - genetics | Protein-Serine-Threonine Kinases - metabolism | Emodin - pharmacology | Curcumin - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - antagonists & inhibitors | Smad3 Protein - antagonists & inhibitors | beta Catenin - metabolism | Drug Synergism | Cell Movement - drug effects | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Cyclin D1 - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Laminin - chemistry | HeLa Cells | Cyclin D1 - metabolism | Smad4 Protein - antagonists & inhibitors | Gene Expression Regulation, Neoplastic | Smad3 Protein - metabolism | Proteoglycans - chemistry | Smad3 Protein - genetics | Cyclin D1 - antagonists & inhibitors | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Wnt Proteins - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Transforming Growth Factor beta - antagonists & inhibitors | Female | Snail Family Transcription Factors | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - antagonists & inhibitors | NIMA-Interacting Peptidylprolyl Isomerase | Transcription Factors - genetics | Smad4 Protein - metabolism | beta Catenin - genetics | Transcription Factors - metabolism | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | beta Catenin - antagonists & inhibitors | Cell Proliferation - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Transforming Growth Factor beta - metabolism | Drug Combinations | Biotechnology | Deregulation | Wnt protein | Mesenchyme | Downstream effects | Crosstalk | Viruses | Smad3 protein | Biochemistry | Metastasis | Kinases | Pin1 protein | Cancer therapies | Carcinogenesis | Smad4 protein | Developing countries--LDCs | Cell adhesion & migration | Proteins | β-catenin | Signal transduction | Carcinogens | Pathways | Cell cycle | Curcumin | Tumorigenesis | Inhibition | Downstream | Medical research | Breast cancer | Tumor cell lines | Gene expression | Cervix | Emodin | Signaling | Chemotherapy | Phytochemicals | Cell lines | Ligands | Cell migration | Cervical cancer | Cancer | Apoptosis | Index Medicus | Developing countries | Developing nations | LDCs
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e15151
Background: Development of molecules chemically modifying the expression of crucial orchestrator(s) of stem cell commitment may have significant biomedical... 
MIXED ESTERS | HEART | IN-VITRO | TGF-BETA | MESODERM | HIGH-THROUGHPUT | TRANSCRIPTION | BIOLOGY | DIFFERENTIATION | NKX2-5 | DROSOPHILA | Embryonic Stem Cells - metabolism | Embryonic Stem Cells - cytology | Humans | Esters | Hyaluronic Acid - pharmacology | Smad3 Protein - metabolism | Smad3 Protein - genetics | Mesenchymal Stromal Cells - cytology | RNA Interference | Smad4 Protein - genetics | HEK293 Cells | Smad1 Protein - genetics | Butyric Acid - pharmacology | Smad7 Protein - genetics | Smad7 Protein - metabolism | Tretinoin - pharmacology | Myocytes, Cardiac - cytology | Cells, Cultured | Hyaluronic Acid - chemistry | Mesenchymal Stromal Cells - metabolism | Smad Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Smad4 Protein - metabolism | Blotting, Western | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Myocytes, Cardiac - metabolism | Smad1 Protein - metabolism | Mice | Smad Proteins - metabolism | Analysis | Genes | Stem cells | Genetic research | Transplantation | Genetic transcription | Precipitation (Meteorology) | Hyaluronic acid | Saturated fatty acids | Heart | Pattern formation | Actinin | Embryo cells | Nuclei | Smad4 protein | Proteins | Hyaluronan | Growth factors | Nkx2.5 protein | Medical research | Fetuses | Conductors | Gene expression | Embryos | Studies | Polymerase chain reaction | Placenta | Insects | Runoff | Biotechnology | Chromatin | Membranes | Smad protein | Transcription | Mesenchyme | Laboratories | Fluorescence | Stem cell transplantation | Kinases | Engineering | Embryogenesis | Allografts | Cell fate | Transgenic animals | Rodents | Mathematical models | Gene transfer | Cardiomyocytes | Medicine | Signaling | Acids | Smad7 protein | Molecular biology | Immunofluorescence | Combinatorial analysis | Cancer
Journal Article
Trends in Immunology, ISSN 1471-4906, 2004, Volume 25, Issue 10, pp. 513 - 517
Journal Article
Developmental Dynamics, ISSN 1058-8388, 01/2008, Volume 237, Issue 1, pp. 259 - 269
textabstractAlthough transforming growth factor-beta (TGF-β) signaling negatively regulates branching morphogenesis in early lung development, few studies to... 
mouse | development | septation | rat | TGF‐β, transforming growth factor | human | alveolarization | Human | Mouse | Rat | Transforming growth factor | Development | TGF-β | Alveolarization | Septation | BRONCHIAL EPITHELIAL-CELLS | ANATOMY & MORPHOLOGY | BRANCHING MORPHOGENESIS | BRONCHOPULMONARY-DYSPLASIA | EMBRYONIC LUNG | DEVELOPMENTAL BIOLOGY | INDUCTION | II-RECEPTOR | TGF-beta | TRANSFORMING-GROWTH-FACTOR | DIFFERENTIATION | transforming growth factor | EXPRESSION | MOUSE LUNG | Immunohistochemistry | Receptors, Transforming Growth Factor beta - genetics | Humans | Immunoblotting | Intracellular Signaling Peptides and Proteins - metabolism | Smad4 Protein - genetics | Smad2 Protein - genetics | Activin Receptors, Type I - physiology | Smad6 Protein - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Smad7 Protein - metabolism | Smad2 Protein - metabolism | Proteoglycans - metabolism | Smad6 Protein - physiology | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Proteoglycans - physiology | Activin Receptors, Type II - genetics | Lung - embryology | Mice | Smad6 Protein - metabolism | Intracellular Signaling Peptides and Proteins - physiology | Smad3 Protein - physiology | Proteoglycans - genetics | Activin Receptors, Type II - metabolism | Smad3 Protein - metabolism | Receptors, Transforming Growth Factor beta - physiology | Endoglin | Smad3 Protein - genetics | Smad7 Protein - physiology | Gene Expression Regulation, Developmental | Lung - metabolism | Smad7 Protein - genetics | Smad2 Protein - physiology | Protein-Serine-Threonine Kinases - physiology | Mice, Inbred C57BL | Smad4 Protein - physiology | Protein-Serine-Threonine Kinases - genetics | Transforming Growth Factor beta - physiology | Activin Receptors, Type I - metabolism | Activin Receptors, Type I - genetics | Smad4 Protein - metabolism | Animals | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - physiology | Activin Receptors, Type II - physiology | Transforming Growth Factor beta - metabolism
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 358 - 361
Transforming growth factor—β (TGFβ) signaling drives aneurysm progression in multiple disorders, including Marfan syndrome (MFS), and therapies that inhibit... 
Transcriptional regulatory elements | Echocardiography | Aortic diseases | Root growth | REPORTS | Placebos | Aneurysms | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | CELLS | ACTIVATION | VALSALVA | MULTIDISCIPLINARY SCIENCES | N-TERMINAL KINASE | MOUSE MODEL | NOONANS-SYNDROME | RECEPTOR | MUTATIONS | INHIBITOR | Diphenylamine - pharmacology | Aortic Aneurysm - metabolism | Mitogen-Activated Protein Kinase 8 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Anthracenes - therapeutic use | MAP Kinase Signaling System | Diphenylamine - analogs & derivatives | Marfan Syndrome - drug therapy | Smad4 Protein - genetics | Transforming Growth Factor beta - antagonists & inhibitors | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Transforming Growth Factor beta - immunology | Diphenylamine - therapeutic use | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 8 - metabolism | Aortic Aneurysm - physiopathology | Losartan - pharmacology | Smad2 Protein - metabolism | Sulfonamides - pharmacology | Anthracenes - pharmacology | Disease Progression | Aorta - pathology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Sulfonamides - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Protein Kinase Inhibitors - pharmacology | Smad4 Protein - deficiency | Enzyme Activation | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article