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Nature Genetics, ISSN 1061-4036, 11/2013, Volume 45, Issue 11, pp. 1309 - 1321
Journal Article
Journal Article
Blood, ISSN 0006-4971, 08/2008, Volume 112, Issue 4, pp. 1503 - 1509
Journal Article
Biochemical Journal, ISSN 0264-6021, 02/2015, Volume 466, Issue 1, pp. 55 - 68
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 07/2012, Volume 32, Issue 14, pp. 2904 - 2916
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2009, Volume 11, Issue 5, pp. 637 - 643
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166382
There is confusion about the role that IFN-alpha plays in the pathogenesis of pulmonary arterial hypertension (PAH) with different investigators reporting a... 
ENDOPLASMIC-RETICULUM STRUCTURE | CONTACT SITES | CELLS | INTERFERON | PULMONARY ARTERIAL-HYPERTENSION | MULTIDISCIPLINARY SCIENCES | GOLGI-APPARATUS | NITRIC-OXIDE | ANTIVIRAL ACTIVITY | RECEPTOR | ENDOCYTOSIS | Smad8 Protein - genetics | rab5 GTP-Binding Proteins - genetics | Bone Morphogenetic Protein 4 - genetics | Humans | Bone Morphogenetic Protein 4 - metabolism | rab5 GTP-Binding Proteins - metabolism | Endosomes - metabolism | Pulmonary Artery - metabolism | Interferon-alpha - genetics | Smad5 Protein - metabolism | HEK293 Cells | Smad1 Protein - genetics | Growth Differentiation Factors - genetics | Transcription, Genetic | Endocytosis - genetics | Smad5 Protein - genetics | Vesicular Transport Proteins | Interferon-alpha - metabolism | Signal Transduction | Endothelial Cells - metabolism | Hypertension, Pulmonary - genetics | Clathrin - genetics | Hypertension, Pulmonary - metabolism | Clathrin - metabolism | Growth Differentiation Factors - metabolism | Smad1 Protein - metabolism | Smad8 Protein - metabolism | Endothelial Cells - pathology | Myxovirus Resistance Proteins - genetics | Myxovirus Resistance Proteins - metabolism | Pulmonary Artery - pathology | Bone morphogenetic proteins | Clathrin | Genetic aspects | Genetic transcription | Pulmonary hypertension | Transcription | Pathogenesis | Proteins | α-Interferon | Nocodazole | Stimulators | Endocytosis | Vascular diseases | Rodents | Paclitaxel | Cell cycle | Immunoelectron microscopy | Growth factors | Hypertension | Bone morphogenetic protein 4 | Bone morphogenetic protein receptor type II | Caveolin | Tubules | Endothelial cells | Mutants | Signaling | Hypotheses | Inhibitors | Microscopy | Nitric oxide | Microtubules | Dynamin | Hypoxia | Mutation | Immunofluorescence | Endoplasmic reticulum | Prostate | Bone morphogenetic protein 9 | Endosomes | Guanosinetriphosphatase
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166984
Birth defects are among the leading causes of infant mortality and contribute substantially to illness and long-term disability. Defects in Bone Morphogenetic... 
BRANCHIAL ARCH | DLX GENES | HEAD SKELETON | ALAGILLE-SYNDROME | DOWNS-SYNDROME | MULTIDISCIPLINARY SCIENCES | AURICULOCONDYLAR SYNDROME | LEFT-RIGHT ASYMMETRY | REPEAT PROTEIN | HUMAN JAGGED1 | NEURAL CREST CELLS | Jagged-1 Protein - metabolism | Zebrafish - embryology | Intercellular Signaling Peptides and Proteins - metabolism | Somites - embryology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Nuclear Proteins - biosynthesis | Smad5 Protein - metabolism | Smad1 Protein - genetics | Smad5 Protein - genetics | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | Zebrafish Proteins - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - genetics | Endothelin-1 - genetics | Endothelin-1 - metabolism | Zebrafish Proteins - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Repressor Proteins - genetics | Jagged-1 Protein - genetics | Zebrafish - genetics | Body Patterning - physiology | Animals | Transforming Growth Factor beta - genetics | Facial Bones - enzymology | Smad1 Protein - metabolism | Signal Transduction - physiology | Zebrafish Proteins - genetics | Transforming Growth Factor beta - metabolism | Bone morphogenetic proteins | Genetic aspects | Research | Craniofacial abnormalities | Neural crest | Pattern formation | Jaw | Transcription factors | Endothelin | Arches | Congenital defects | Alagille syndrome | Genomics | Transforming growth factor-b | Genomes | Somites | Kinases | Defects | Proteins | Cartilage | Signal transduction | Pathways | Growth factors | Territory | Patterning | Mandible | Interference | Craniofacial syndromes | Zebrafish | Infant mortality | Birth defects | Endothelin 1 | Gene expression | Embryos | Mutants | Cleft lip/palate | Craniofacial growth | Signaling | Scaffolding | In vivo methods and tests | Notch protein
Journal Article
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, ISSN 0892-6638, 11/2017, Volume 31, Issue 11, pp. 4720 - 4733
Before the onset of sprouting angiogenesis, the endothelium is prepatterned for the positioning of tip and stalk cells. Both cell identities are not static, as... 
cell migration | ALK2 | ALK3 | p38 MAPK | SMAD1/5 | MIGRATION | ACTIVATION | TGF-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | NOTCH | CANCER | ALK1 | CELL BIOLOGY | ENDOTHELIAL-CELLS | BIOLOGY | GROWTH-FACTOR | EXPRESSION | MAP Kinase Signaling System - physiology | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor Receptor-2 - genetics | HSP27 Heat-Shock Proteins - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Bone Morphogenetic Protein 6 - genetics | Smad5 Protein - metabolism | Bone Morphogenetic Protein 2 - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Smad1 Protein - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Smad5 Protein - genetics | Bone Morphogenetic Protein 2 - genetics | Intercellular Signaling Peptides and Proteins - genetics | Bone Morphogenetic Protein Receptors, Type I - genetics | Vascular Endothelial Growth Factor Receptor-2 - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | Activin Receptors, Type I - metabolism | Bone Morphogenetic Protein Receptors, Type I - metabolism | Activin Receptors, Type I - genetics | Neovascularization, Physiologic - physiology | Bone Morphogenetic Protein 6 - metabolism | HSP27 Heat-Shock Proteins - metabolism | Smad1 Protein - metabolism | Complex formation | Genes | Clinical trials | Cell adhesion & migration | Proteins | Angiogenesis | Signal transduction | Receptors | Pathways | Bone morphogenetic proteins | Vascular endothelial growth factor | Protein-tyrosine kinase | Enhancer-of-split protein | Bone morphogenetic protein 6 | Tyrosine | Medical research | Bone morphogenetic protein 2 | Heat shock proteins | Gene expression | Spheroids | Endothelial cells | Bone morphogenetic protein receptor type I | Endothelium | Signaling | Antiangiogenics | Hsp27 protein | Bone | Activin | Cell migration | Heat shock | SMAD1 | Research
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 01/2008, Volume 28, Issue 1, pp. 248 - 257
Journal Article
Oncogene, ISSN 0950-9232, 2013, Volume 32, Issue 47, pp. 5409 - 5420
Glioblastoma multiforme (GBM) is an aggressive brain malignancy characterized by high heterogeneity and invasiveness. It is increasingly accepted that the... 
BMP | Snail | Invasiveness | Glioblastoma multiforme | Glioma-initiating cells | INITIATING CELLS | glioblastoma multiforme | snail | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | MESENCHYMAL TRANSITION | CELL BIOLOGY | invasiveness | NEURAL STEM-CELLS | GROWTH-FACTOR-BETA | glioma-initiating cells | ONCOLOGY | GENETICS & HEREDITY | SUPPRESSES | DIFFERENTIATION | HIGH-GRADE GLIOMA | BRAIN | PROGRESSION | Cell Proliferation | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Brain Neoplasms - metabolism | Cell Movement - genetics | Cell Differentiation - genetics | Smad3 Protein - genetics | Bone Morphogenetic Proteins - metabolism | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | RNA Interference | Smad4 Protein - genetics | Smad2 Protein - genetics | Smad1 Protein - genetics | Glioblastoma - metabolism | Smad5 Protein - genetics | Snail Family Transcription Factors | Brain Neoplasms - genetics | Signal Transduction - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Glioblastoma - pathology | Cell Line, Tumor | Mice | RNA, Small Interfering | Bone morphogenetic proteins | Genetic aspects | Cellular signal transduction | Research | Gene expression | Identification and classification | Proteins | Signal transduction | Brain cancer | Cell adhesion & migration | Original | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Cell and Molecular Biology | Cell- och molekylärbiologi | glioma initiating cells
Journal Article
Developmental Cell, ISSN 1534-5807, 03/2012, Volume 22, Issue 3, pp. 501 - 514
Gradients of vascular endothelial growth factor (VEGF) induce single endothelial cells to become leading tip cells of emerging angiogenic sprouts. Tip cells... 
DEFECTS | TIP CELLS | ANGIOGENESIS | VEGF | ENDOTHELIAL-CELLS | ID PROTEINS | HES1 | DEVELOPMENTAL BIOLOGY | DIFFERENTIATION | EXPRESSION | INHIBITS TUMOR-GROWTH | CELL BIOLOGY | Transcription Factor HES-1 | Humans | Intercellular Signaling Peptides and Proteins - biosynthesis | Intracellular Signaling Peptides and Proteins - metabolism | Inhibitor of Differentiation Proteins - biosynthesis | Inhibitor of Differentiation Protein 1 - biosynthesis | Serrate-Jagged Proteins | Smad5 Protein - metabolism | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | Smad1 Protein - genetics | Membrane Proteins - metabolism | Smad5 Protein - genetics | Intracellular Signaling Peptides and Proteins - genetics | Jagged-1 Protein | Calcium-Binding Proteins - biosynthesis | Homeodomain Proteins - biosynthesis | Signal Transduction | Inhibitor of Differentiation Protein 2 - biosynthesis | Membrane Proteins - genetics | Down-Regulation | Cells, Cultured | Mice, Transgenic | Cell Cycle Proteins - biosynthesis | Vascular Endothelial Growth Factor Receptor-1 - biosynthesis | Mice, Knockout | Membrane Proteins - biosynthesis | Phenotype | Animals | Smad1 Protein - metabolism | Mice | Neovascularization, Physiologic | Proteins | Endothelial growth factors | Genes | Genetic aspects | Transforming growth factors | Vascular endothelial growth factor | Endothelium | Dll4 | tip cell | lateral inhibition | sprouting angiogenesis | BMP | directed migration | Notch | stalk cell | mouse embryo | Smad | polarity
Journal Article