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Nature, ISSN 0028-0836, 02/2010, Volume 463, Issue 7283, pp. 958 - 962
Heterozygous mutations in the gene encoding the CHD (chromodomain helicase DNA-binding domain) member CHD7, an ATP-dependent chromatin remodeller homologous to... 
NETWORK | COMPLEX | PROTEIN | CHROMATIN | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | TRANSCRIPTION | POLYMERASE-II | MUTATIONS | EXPRESSION | CHARGE-SYNDROME | Embryonic Stem Cells - metabolism | Embryo, Nonmammalian - cytology | Embryonic Stem Cells - cytology | Xenopus Proteins - genetics | Humans | Multipotent Stem Cells - metabolism | Embryo, Nonmammalian - metabolism | Embryo, Nonmammalian - embryology | DNA-Binding Proteins - deficiency | Neural Crest - metabolism | Xenopus Proteins - chemistry | DNA-Binding Proteins - metabolism | Gene Expression Regulation, Developmental | Transcription, Genetic | Cell Differentiation | Xenopus laevis - genetics | DNA Helicases - genetics | Snail Family Transcription Factors | SOX9 Transcription Factor - metabolism | Cell Line | DNA Helicases - chemistry | Neural Crest - cytology | Xenopus laevis - embryology | Chromosomal Proteins, Non-Histone - metabolism | DNA Helicases - deficiency | Transcription Factors - genetics | DNA-Binding Proteins - genetics | DNA-Binding Proteins - chemistry | Syndrome | Chromosomal Proteins, Non-Histone - genetics | Transcription Factors - metabolism | Cell Lineage | DNA Helicases - metabolism | Animals | Xenopus Proteins - deficiency | Multipotent Stem Cells - cytology | Neural Crest - embryology | Xenopus laevis - metabolism | Twist-Related Protein 1 - genetics | Protein Binding | Xenopus Proteins - metabolism | Enhancer Elements, Genetic - genetics | Twist-Related Protein 1 - metabolism | SOX9 Transcription Factor - genetics | Cell Movement | Chromatin | Drosophila | Polybrominated biphenyls | Physiological aspects | Genetic aspects | Research | Structure | Embryonic stem cells | Helicases | Proteins | Labeling | Evacuations & rescues | Cell adhesion & migration | Human | Peripheral nervous system | Genes | Charge | Bones | Activation | Numerical control | Gene expression | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0120045 - e0120045
Cervical cancer is the major cause of cancer related deaths in women, especially in developing countries and Human Papilloma Virus infection in conjunction... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | GROWTH-FACTOR-BETA | INVASION | MULTIDISCIPLINARY SCIENCES | TRANSFORMING GROWTH-FACTOR-BETA-1 | PANCREATIC-CANCER | GENE-EXPRESSION | MECHANISMS | HUMAN-PAPILLOMAVIRUS | TRANSCRIPTION FACTOR | Receptors, Transforming Growth Factor beta - genetics | Humans | Collagen - chemistry | Epithelial-Mesenchymal Transition - drug effects | Wnt Proteins - metabolism | Smad4 Protein - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Peptidylprolyl Isomerase - metabolism | Peptidylprolyl Isomerase - genetics | Protein-Serine-Threonine Kinases - metabolism | Emodin - pharmacology | Curcumin - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - antagonists & inhibitors | Smad3 Protein - antagonists & inhibitors | beta Catenin - metabolism | Drug Synergism | Cell Movement - drug effects | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Cyclin D1 - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Laminin - chemistry | HeLa Cells | Cyclin D1 - metabolism | Smad4 Protein - antagonists & inhibitors | Gene Expression Regulation, Neoplastic | Smad3 Protein - metabolism | Proteoglycans - chemistry | Smad3 Protein - genetics | Cyclin D1 - antagonists & inhibitors | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Wnt Proteins - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Transforming Growth Factor beta - antagonists & inhibitors | Female | Snail Family Transcription Factors | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - antagonists & inhibitors | NIMA-Interacting Peptidylprolyl Isomerase | Transcription Factors - genetics | Smad4 Protein - metabolism | beta Catenin - genetics | Transcription Factors - metabolism | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | beta Catenin - antagonists & inhibitors | Cell Proliferation - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Transforming Growth Factor beta - metabolism | Drug Combinations | Biotechnology | Deregulation | Wnt protein | Mesenchyme | Downstream effects | Crosstalk | Developing countries | Viruses | Smad3 protein | Biochemistry | Metastasis | Kinases | Pin1 protein | Developing nations | Cancer therapies | Carcinogenesis | Smad4 protein | Developing countries--LDCs | Cell adhesion & migration | Proteins | β-catenin | Signal transduction | Carcinogens | Pathways | Cell cycle | Curcumin | Tumorigenesis | Inhibition | Downstream | Medical research | Breast cancer | Tumor cell lines | Gene expression | Cervix | Emodin | Signaling | Chemotherapy | Phytochemicals | Cell lines | Ligands | Cell migration | Cervical cancer | Cancer | Apoptosis | Index Medicus | LDCs
Journal Article
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 2, pp. 979 - 994
Epithelial-mesenchymal transition (EMT) plays a critical role in the development of tumor metastases by enhancing migration/invasion. One of the hallmarks of... 
Epithelial-mesenchymal transition (EMT) | Ubiquitin degradation | Fbxo45; miR-27a | EMT-Inducing transcription factors (EMT-TFs) | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, pp. e0177962 - e0177962
Adult neural crest stem-derived cells (NCSC) are of extraordinary high plasticity and promising candidates for use in regenerative medicine. Several locations... 
PROGENITOR CELLS | MESENCHYMAL STROMAL CELLS | STEM-CELLS | TRUNK | MULTIDISCIPLINARY SCIENCES | PERIODONTAL-LIGAMENT | ONTOGENY | GENERATION | SKIN-DERIVED PRECURSORS | DIFFERENTIATION | MELANOCYTES | RNA-Binding Proteins - genetics | Humans | Adipose Tissue - cytology | Melanocytes - metabolism | Neurons - cytology | Receptors, Nerve Growth Factor - metabolism | Neural Stem Cells - cytology | Schwann Cells - cytology | Neural Crest - metabolism | Adipose Tissue - metabolism | Mesenchymal Stromal Cells - cytology | Snail Family Transcription Factors - genetics | Melanocytes - cytology | Nestin - genetics | Adult | Female | Cell Differentiation | Neurons - metabolism | Dermis - cytology | Nuclear Proteins - genetics | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Gene Expression | Neural Crest - cytology | Dermis - metabolism | Snail Family Transcription Factors - metabolism | Microinjections | Bone Marrow Cells - cytology | Neural Crest - growth & development | Mesenchymal Stromal Cells - metabolism | Nuclear Proteins - metabolism | Schwann Cells - metabolism | Transcription Factor Brn-3A - metabolism | Chick Embryo | Nerve Tissue Proteins - genetics | Receptors, Nerve Growth Factor - genetics | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Animals | Transcription Factor Brn-3A - genetics | Twist-Related Protein 1 - genetics | Twist-Related Protein 1 - metabolism | Neural Stem Cells - metabolism | SOX9 Transcription Factor - genetics | Bone Marrow Cells - metabolism | Mesenchymal Stem Cell Transplantation | RNA-Binding Proteins - metabolism | Adipose tissues | Comparative analysis | Skin | Neurosciences | Nestin | Adipose tissue | Leukocyte migration | Laboratories | Sox9 protein | Dermis | Melanocytes | Nervous system | Human tissues | Regeneration (physiology) | Cell adhesion & migration | Pulp | Msi1 protein | Plasticity (neural) | Ethics | Immunology | Pathways | Surgery | Bone marrow | Hair | Hematology | Neurons | Tissue engineering | Schwann cells | Brn-3 protein | Gene expression | Embryos | Neural crest | Medicine | Regeneration | Human performance | Stromal cells | Stem cells | Cell migration | Dental pulp | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2009, Volume 119, Issue 12, pp. 3626 - 3636
The polycomb group protein B lymphoma Mo-MLV insertion region 1 homolog (Bmi-1) is dysregulated in various cancers, and its upregulation strongly correlates... 
MEDICINE, RESEARCH & EXPERIMENTAL | BREAST-CANCER CELLS | DEVELOPMENTAL REGULATORS | CELLULAR MEMORY | E-CADHERIN EXPRESSION | HEMATOPOIETIC STEM-CELLS | PROSTATE-CANCER | MYC TRANSGENIC MICE | SELF-RENEWAL | NF-KAPPA-B | TRANSCRIPTION FACTOR | Nasopharyngeal Neoplasms - genetics | Neoplasm Transplantation | Nasopharyngeal Neoplasms - metabolism | Epithelial Cells - metabolism | Cadherins - metabolism | Humans | Glycogen Synthase Kinase 3 beta | Transplantation, Heterologous | Phosphatidylinositol 3-Kinases - metabolism | Mesoderm - cytology | Repressor Proteins - antagonists & inhibitors | Nasopharyngeal Neoplasms - pathology | Cadherins - genetics | Epithelial Cells - cytology | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Nasopharyngeal Neoplasms - etiology | Snail Family Transcription Factors | Nasopharynx - metabolism | Repressor Proteins - metabolism | Proto-Oncogene Proteins - metabolism | PTEN Phosphohydrolase - genetics | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Neoplasm Invasiveness | Down-Regulation | Gene Silencing | PTEN Phosphohydrolase - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Glycogen Synthase Kinase 3 - metabolism | Transcription Factors - metabolism | Animals | Polycomb Repressive Complex 1 | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Mesoderm - metabolism | Mice | Nasopharynx - cytology | Chromatin | Care and treatment | Lymphomas | Research | Analysis | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2014, Volume 111, Issue 25, pp. 9241 - 9246
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, pp. e73940 - e73940
Stem cell pluripotency, angiogenesis and epithelial-mesenchymal transition (EMT) have been shown to be significantly upregulated in pancreatic ductal... 
EPITHELIAL-MESENCHYMAL TRANSITION | GROWTH-FACTOR RECEPTOR | INTESTINAL EPITHELIUM | REPROGRAMMING FACTOR LIN28 | DUCTAL ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | DOWN-REGULATION | TUMOR-GROWTH | EGF RECEPTOR | FEEDBACK LOOP | Pancreatic Neoplasms - metabolism | RNA Processing, Post-Transcriptional | Humans | Pancreatic Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Gene Silencing | Protein-Serine-Threonine Kinases - genetics | MicroRNAs - metabolism | Pancreatic Neoplasms - genetics | Intracellular Signaling Peptides and Proteins - metabolism | DNA-Binding Proteins - genetics | Epithelial-Mesenchymal Transition - genetics | DNA-Binding Proteins - metabolism | Xenograft Model Antitumor Assays | Animals | Neoplastic Stem Cells - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Tumor Burden - genetics | Mice | MicroRNAs - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | MicroRNA | Analysis | Pancreatic cancer | Stem cells | Development and progression | DNA binding proteins | Metastasis | Vascular endothelial growth factor | Health aspects | Adenocarcinoma | Post-transcription | Health sciences | Animal models | Transcription factors | Mesenchyme | Veterans | Oct-4 protein | Gene regulation | c-Myc protein | Homeostasis | Myc protein | Kinases | K-Ras protein | Metastases | Angiogenesis | Allografts | KLF4 protein | Intestine | Xenografts | MiRNA | siRNA | Gene expression | Ablation | Medicine | Ribonucleic acids | MicroRNAs | Tumor suppressor genes | Poly(lactide-co-glycolide) | Snail protein | Polyps | Pluripotency | Tumors | Cancer | Index Medicus
Journal Article
PLoS Pathogens, ISSN 1553-7366, 05/2017, Volume 13, Issue 5, pp. e1006081 - e1006081
Interactions between early developing Schistosoma mansoni larval stages and the hemolymph of its snail intermediate host represent the first molecular... 
COMPATIBILITY POLYMORPHISM | MICROBIOLOGY | ANOPHELES-GAMBIAE | TANDEM MASS-SPECTROMETRY | RESISTANT | VIROLOGY | INVERTEBRATE | HEMOCYTES | FIBRINOGEN-RELATED PROTEINS | GENE-EXPRESSION | SPOROCYSTS | PARASITOLOGY | SNAIL | Amino Acid Sequence | Schistosoma mansoni - physiology | Sepharose - analogs & derivatives | Biomphalaria - metabolism | Hemolymph - metabolism | Biomphalaria - parasitology | Schistosoma mansoni - immunology | Blood Proteins - metabolism | Schistosoma mansoni - metabolism | Bacterial Proteins | Protein Interaction Mapping | Sequence Alignment | Animals | Larva | Proteomics | Host-Parasite Interactions | Biomphalaria - immunology | Helminth Proteins - metabolism | Mollusks | Research | Hsp60 protein | Enrichment | Plasma | Transformation | LPS-binding protein | Glycoconjugates | Matrices (mathematics) | Funding | Colleges & universities | Genomes | Selectivity | Parasites | Lipopolysaccharides | Proteins | Receptors | Plasma proteins | Placement | Calcium-binding protein | Streptavidin | Fibrinogen | Calmodulin | Immune system | Pathogens | Immunoglobulins | ADAM protein | Nucleotide sequence | Long-term potentiation | Hemolymph | Nanostructure | Pattern recognition | Gene expression | Adhesion | Zinc | Strain | Cytotoxins | Correlation analysis | Lectins | Affinity | Scientific imaging | Stress proteins | Beads | Compatibility | Mass spectrometry | Index Medicus
Journal Article
Molecular Cancer Research, ISSN 1541-7786, 12/2012, Volume 10, Issue 12, pp. 1597 - 1606
To understand the mechanisms leading to trastuzumab resistance in HER2-overexpressing breast tumors, we created trastuzumab-insensitive cell lines (SKBR3/100-8... 
MIGRATION | STEM-CELLS | INVASION | GROWTH-FACTOR-RECEPTOR | MECHANISM | LIGANDS | ONCOLOGY | HERCEPTIN | MESENCHYMAL TRANSITION | BETA-CATENIN | CONTRIBUTES | CELL BIOLOGY | Receptor, Epidermal Growth Factor - genetics | Cadherins - metabolism | Receptor, ErbB-2 - genetics | Humans | Transcriptional Activation | Receptor, ErbB-2 - metabolism | Drug Resistance, Neoplasm | Cadherins - biosynthesis | Wnt3 Protein - genetics | Breast Neoplasms - metabolism | Wnt3 Protein - biosynthesis | beta Catenin - biosynthesis | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Nuclear Proteins - biosynthesis | Antibodies, Monoclonal, Humanized - pharmacology | Female | Wnt3 Protein - metabolism | Twist-Related Protein 1 - biosynthesis | Cadherins - genetics | Nuclear Proteins - genetics | Snail Family Transcription Factors | Wnt Signaling Pathway | Receptor, Epidermal Growth Factor - biosynthesis | Receptor, ErbB-2 - biosynthesis | Organic Cation Transport Proteins - metabolism | Nuclear Proteins - metabolism | Transcription Factors - biosynthesis | Transcription Factors - genetics | Organic Cation Transport Proteins - biosynthesis | beta Catenin - metabolism | beta Catenin - genetics | Transcription Factors - metabolism | Phenotype | Breast Neoplasms - genetics | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Organic Cation Transport Proteins - genetics | Twist-Related Protein 1 - metabolism | Trastuzumab | Index Medicus | trastuzumab | β-catenin | EMT | Breast Cancer | Wnt3
Journal Article