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Breast Cancer Research, ISSN 1465-5411, 09/2011, Volume 13, Issue 5, pp. R87 - R87
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2009, Volume 119, Issue 12, pp. 3626 - 3636
The polycomb group protein B lymphoma Mo-MLV insertion region 1 homolog (Bmi-1) is dysregulated in various cancers, and its upregulation strongly correlates... 
MEDICINE, RESEARCH & EXPERIMENTAL | BREAST-CANCER CELLS | DEVELOPMENTAL REGULATORS | CELLULAR MEMORY | E-CADHERIN EXPRESSION | HEMATOPOIETIC STEM-CELLS | PROSTATE-CANCER | MYC TRANSGENIC MICE | SELF-RENEWAL | NF-KAPPA-B | TRANSCRIPTION FACTOR | Nasopharyngeal Neoplasms - genetics | Neoplasm Transplantation | Nasopharyngeal Neoplasms - metabolism | Epithelial Cells - metabolism | Cadherins - metabolism | Humans | Glycogen Synthase Kinase 3 beta | Transplantation, Heterologous | Phosphatidylinositol 3-Kinases - metabolism | Mesoderm - cytology | Repressor Proteins - antagonists & inhibitors | Nasopharyngeal Neoplasms - pathology | Cadherins - genetics | Epithelial Cells - cytology | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Nasopharyngeal Neoplasms - etiology | Snail Family Transcription Factors | Nasopharynx - metabolism | Repressor Proteins - metabolism | Proto-Oncogene Proteins - metabolism | PTEN Phosphohydrolase - genetics | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Neoplasm Invasiveness | Down-Regulation | Gene Silencing | PTEN Phosphohydrolase - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Glycogen Synthase Kinase 3 - metabolism | Transcription Factors - metabolism | Animals | Polycomb Repressive Complex 1 | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Mesoderm - metabolism | Mice | Nasopharynx - cytology | Chromatin | Care and treatment | Lymphomas | Research | Analysis | Cancer
Journal Article
Cancer Letters, ISSN 0304-3835, 2014, Volume 356, Issue 2, pp. 156 - 164
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, p. e0177962
Adult neural crest stem-derived cells (NCSC) are of extraordinary high plasticity and promising candidates for use in regenerative medicine. Several locations... 
PROGENITOR CELLS | MESENCHYMAL STROMAL CELLS | STEM-CELLS | TRUNK | MULTIDISCIPLINARY SCIENCES | PERIODONTAL-LIGAMENT | ONTOGENY | GENERATION | SKIN-DERIVED PRECURSORS | DIFFERENTIATION | MELANOCYTES | RNA-Binding Proteins - genetics | Humans | Adipose Tissue - cytology | Melanocytes - metabolism | Neurons - cytology | Receptors, Nerve Growth Factor - metabolism | Neural Stem Cells - cytology | Schwann Cells - cytology | Neural Crest - metabolism | Adipose Tissue - metabolism | Mesenchymal Stromal Cells - cytology | Snail Family Transcription Factors - genetics | Melanocytes - cytology | Nestin - genetics | Adult | Female | Cell Differentiation | Neurons - metabolism | Dermis - cytology | Nuclear Proteins - genetics | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Gene Expression | Neural Crest - cytology | Dermis - metabolism | Snail Family Transcription Factors - metabolism | Microinjections | Bone Marrow Cells - cytology | Neural Crest - growth & development | Mesenchymal Stromal Cells - metabolism | Nuclear Proteins - metabolism | Schwann Cells - metabolism | Transcription Factor Brn-3A - metabolism | Chick Embryo | Nerve Tissue Proteins - genetics | Receptors, Nerve Growth Factor - genetics | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Animals | Transcription Factor Brn-3A - genetics | Twist-Related Protein 1 - genetics | Twist-Related Protein 1 - metabolism | Neural Stem Cells - metabolism | SOX9 Transcription Factor - genetics | Bone Marrow Cells - metabolism | Mesenchymal Stem Cell Transplantation | RNA-Binding Proteins - metabolism | Adipose tissues | Comparative analysis | Skin | Neurosciences | Nestin | Adipose tissue | Leukocyte migration | Laboratories | Sox9 protein | Dermis | Melanocytes | Nervous system | Human tissues | Regeneration (physiology) | Cell adhesion & migration | Msi1 protein | Plasticity (neural) | Ethics | Immunology | Pathways | Surgery | Bone marrow | Hair | Hematology | Neurons | Tissue engineering | Schwann cells | Brn-3 protein | Gene expression | Embryos | Neural crest | Medicine | Regeneration | Human performance | Stromal cells | Stem cells | Cell migration | Dental pulp
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 03/2014, Volume 306, Issue 6, pp. L534 - L542
MicroRNAs are small noncoding RNAs that inhibit protein expression. We have previously shown that the inhibition of the microRNA let-7d in epithelial cells... 
High-mobility group-A2 protein | Epithelial-to-mesenchymal transition | Idiopathic pulmonary fibrosis | Transforming growth factor-β | Fibrosis | microRNA | Slug | PHYSIOLOGY | HMGA2 | idiopathic pulmonary fibrosis | INDUCTION | EMT | high-mobility group-A2 protein | epithelial-to-mesenchymal transition | transforming growth factor-beta | LOOP-HELIX PROTEINS | REGULATOR | MIR-29 | RESPIRATORY SYSTEM | fibrosis | GROWTH-FACTOR | PULMONARY-FIBROSIS | SNAIL | Cell Proliferation | HMGB2 Protein - metabolism | Cadherins - metabolism | Humans | Actins - metabolism | Pulmonary Fibrosis - genetics | Lung - cytology | Cell Movement - genetics | Idiopathic Pulmonary Fibrosis - metabolism | Myofibroblasts - metabolism | Transfection | HMGA2 Protein - metabolism | Pulmonary Alveoli - metabolism | Epithelial-Mesenchymal Transition | Lung - metabolism | Pulmonary Fibrosis - metabolism | Wound Healing - genetics | Zonula Occludens-1 Protein - metabolism | Snail Family Transcription Factors | Fibroblasts - metabolism | Calcium-Binding Proteins - metabolism | Cells, Cultured | Transcription Factors - genetics | Fibronectins - metabolism | Transcription Factors - metabolism | Idiopathic Pulmonary Fibrosis - pathology | Fibroblasts - cytology | MicroRNAs - genetics | Keratin-19 - metabolism | Transforming Growth Factor beta - metabolism | MicroRNA | Physiological aspects | Fibroblasts | Phenotypic plasticity | Genetic research | Research | Transforming growth factors | transforming growth factor-β
Journal Article
Journal Article
Cancer, ISSN 0008-543X, 04/2005, Volume 103, Issue 8, pp. 1631 - 1643
BACKGROUND It was demonstrated previously that the Snail family of transcription factors and Smad‐interacting protein 1 (Sip1) regulate E‐cadherin and matrix... 
breast carcinoma | survival | ovarian carcinoma | cadherins | matrix metalloproteinases | serous effusions | chemotherapy | Snail transcription factors | Breast carcinoma | Serous effusions | Chemotherapy | Matrix metalloproteinases | Ovarian carcinoma | Cadherins | Survival | FINGER TRANSCRIPTION FACTORS | CANCER PATIENTS | E-CADHERIN EXPRESSION | REPRESSES E-CADHERIN | MALIGNANT PLEURAL EFFUSION | ONCOLOGY | GASTRIC-CANCER | EPITHELIAL TUMOR-CELLS | CELL-ADHESION MOLECULES | ALTERED EXPRESSION | Cadherins - metabolism | Homeodomain Proteins - metabolism | Humans | Middle Aged | Ovarian Neoplasms - pathology | Cystadenocarcinoma, Serous - genetics | Drosophila Proteins - metabolism | RNA, Messenger - metabolism | Zinc Finger E-box Binding Homeobox 2 | RNA, Neoplasm - metabolism | Immunoenzyme Techniques | Ovarian Neoplasms - genetics | Carcinoma, Lobular - genetics | Carcinoma, Ductal - metabolism | Ovarian Neoplasms - metabolism | Cadherins - genetics | Repressor Proteins - metabolism | Carcinoma, Lobular - secondary | Cystadenocarcinoma, Serous - secondary | Matrix Metalloproteinase 2 - metabolism | Repressor Proteins - genetics | Survival Rate | Adenocarcinoma, Mucinous - secondary | Reverse Transcriptase Polymerase Chain Reaction | Matrix Metalloproteinase 2 - genetics | Breast Neoplasms - genetics | Adenocarcinoma, Clear Cell - secondary | RNA, Neoplasm - genetics | Carcinoma, Lobular - metabolism | Adenocarcinoma, Mucinous - genetics | Prognosis | Carcinoma, Ductal - genetics | Cystadenocarcinoma, Serous - metabolism | Gene Expression Regulation, Neoplastic | Breast Neoplasms - metabolism | DNA-Binding Proteins - metabolism | In Situ Hybridization | Adult | Female | Snail Family Transcription Factors | Adenocarcinoma, Clear Cell - metabolism | RNA, Messenger - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Carcinoma, Ductal - secondary | Breast Neoplasms - pathology | Adenocarcinoma, Mucinous - metabolism | Aged | Drosophila Proteins - genetics | Adenocarcinoma, Clear Cell - genetics | Proteins | Care and treatment | Usage | Innovations | Breast cancer | Research | Metastasis | Ovarian cancer
Journal Article
Microvascular Research, ISSN 0026-2862, 2008, Volume 75, Issue 2, pp. 144 - 154
The Notch ligand, Dll4, is essential for angiogenesis during embryonic vascular development and is involved in tumour angiogenesis. Several recent publications... 
Dll4 | Angiogenesis | cDNA microarray | HUVEC | Sprout formation | Notch | Transcription Factor HES-1 | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Receptors, Notch - metabolism | Homeodomain Proteins - metabolism | Humans | Pregnancy Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Receptors, Growth Factor - genetics | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | STAT1 Transcription Factor - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Serrate-Jagged Proteins | Membrane Proteins - metabolism | Snail Family Transcription Factors | Vascular Endothelial Growth Factor Receptor-1 - genetics | Calcium-Binding Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Reproducibility of Results | Basic Helix-Loop-Helix Transcription Factors - genetics | Transduction, Genetic | Endothelial Cells - metabolism | Membrane Proteins - genetics | Neovascularization, Physiologic - genetics | Proto-Oncogene Proteins c-met | Cells, Cultured | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Gene Expression Profiling - methods | Proto-Oncogene Proteins - genetics | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Signal Transduction - genetics | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | STAT1 Transcription Factor - genetics | Homeodomain Proteins - genetics | Hepatocyte Growth Factor - metabolism | Transcription Factors - metabolism | Placenta Growth Factor | Enzyme Activation | Endothelial Cells - enzymology | Calcium-Binding Proteins - genetics | Cluster Analysis | Receptors, Growth Factor - metabolism | Genetic research | Fibroblast growth factors | Neovascularization | Vascular endothelial growth factor | Analysis | Endothelium
Journal Article
by Liu, Z and Li, Q and Li, K and Chen, L and Li, W and Hou, M and Liu, T and Yang, J and Lindvall, C and Björkholm, M and Jia, J and Xu, D
Oncogene, ISSN 0950-9232, 09/2013, Volume 32, Issue 36, pp. 4203 - 4213
Telomerase activation through induction of telomerase reverse transcriptase (hTERT) contributes to malignant transformation by stabilizing telomeres. Clinical... 
hTERT | gastric cancer | EMT | CSCs | SURVIVAL | METASTASIS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BETA-CATENIN | TUMOR-INITIATING CELLS | CELL BIOLOGY | ONCOLOGY | GASTRIC-CANCER | GROWTH | GENETICS & HEREDITY | EXPRESSION | CARCINOMA | SNAIL | Neoplasms - metabolism | Vimentin - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Stomach Neoplasms - pathology | Epithelial-Mesenchymal Transition - genetics | Cell Nucleus - metabolism | Telomerase - genetics | Neoplasms - genetics | Neoplastic Stem Cells - metabolism | RNA Interference | Cell Transformation, Neoplastic - genetics | Vimentin - genetics | Hyaluronan Receptors - metabolism | Telomerase - metabolism | Transcription, Genetic | Catalysis | Snail Family Transcription Factors | Biomarkers - metabolism | Stomach Neoplasms - genetics | Promoter Regions, Genetic | Gene Expression | Signal Transduction | Neoplasm Invasiveness | Transcription Factors - genetics | Cell Transformation, Neoplastic - metabolism | beta Catenin - metabolism | Protein Transport | Transcription Factors - metabolism | Animals | Cell Line, Tumor | Protein Binding | Mice | Neoplasms - pathology | Quantitative Trait Loci | Neoplastic processes | Cellular control mechanisms | Cancer cells | Research | Properties | Observations | Telomerase | Proteins | Signal transduction | Gene expression | Stomach cancer | Stem cells | Index Medicus
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6028, pp. 458 - 461
Journal Article