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Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4223 - 4238
A high intake of dietary salt (NaCl) has been implicated in the development of hypertension, chronic inflammation, and autoimmune diseases. We have recently... 
MEDICINE, RESEARCH & EXPERIMENTAL | M2 MACROPHAGES | NA+ STORAGE | TISSUE | GENE-EXPRESSION | URINARY SODIUM | TRANSCRIPTION FACTOR | POTASSIUM EXCRETION | ALTERNATIVE ACTIVATION | BLOOD-PRESSURE | T-CELLS | Sodium Chloride, Dietary - pharmacology | Sodium Chloride - pharmacology | Male | Glycolysis - drug effects | Proto-Oncogene Proteins c-akt - genetics | Oxidative Phosphorylation - drug effects | Interleukin-4 - pharmacology | Histone Code - drug effects | Bone Marrow Cells - drug effects | TOR Serine-Threonine Kinases - physiology | Wound Healing - drug effects | Macrophages - immunology | Macrophages - classification | Immunity, Innate - drug effects | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Inflammation | Proto-Oncogene Proteins c-akt - physiology | Mitochondria - drug effects | Random Allocation | Interleukin-13 - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Sodium Chloride, Dietary - toxicity | Chitin - toxicity | Macrophages - drug effects | Mice | Macrophage Activation - drug effects | Salt | Medical research | Immune response | Medicine, Experimental | Research | Macrophages | Properties | Observations | Biological control systems | Health aspects | Hypertension | Wound healing | Sodium | Cytokines | Kinases | Metabolism | Gene expression | Experiments | Acquisitions & mergers
Journal Article
Infection and Immunity, ISSN 0019-9567, 06/2013, Volume 81, Issue 6, pp. 2258 - 2267
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4281 - 4294
Inhibition of prostaglandin (PG) production with either nonselective or selective inhibitors of cyclooxygenase-2 (COX-2) activity can induce or exacerbate... 
MEDICINE, RESEARCH & EXPERIMENTAL | 11-BETA-HYDROXYSTEROID DEHYDROGENASE | RENIN RELEASE | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | PATHOGENIC T(H)17 CELLS | COLON CARCINOGENESIS | ANGIOTENSIN-II | MACROPHAGE DEPLETION | T-CELL | BLOOD-PRESSURE | MACULA DENSA | Dinoprostone - physiology | Phosphorylation | Kidney - pathology | Intramolecular Oxidoreductases - physiology | Membrane Glycoproteins - biosynthesis | Male | Mice, 129 Strain | Receptors, Prostaglandin E, EP4 Subtype - deficiency | Hypertension - chemically induced | Female | Hypertension - enzymology | Lymphangiogenesis | Cyclooxygenase 2 Inhibitors - toxicity | Macrophages, Peritoneal - drug effects | Kidney - physiopathology | Skin - pathology | Sodium Chloride Symporters - metabolism | Mice, Inbred C57BL | Cyclooxygenase 2 - physiology | Intramolecular Oxidoreductases - deficiency | Radiation Chimera | Vascular Endothelial Growth Factor C - genetics | Membrane Glycoproteins - genetics | Animals | Sodium Chloride, Dietary - toxicity | Prostaglandin-E Synthases | Pyrazoles - toxicity | Myeloid Cells - metabolism | Sulfonamides - toxicity | Mice | Protein Processing, Post-Translational | Cyclooxygenase 2 - deficiency | Vascular Endothelial Growth Factor C - biosynthesis | Hypertension | Salt | Homeostasis | Cyclooxygenases | Properties | Health aspects | Risk factors | Software | Sodium | Rodents
Journal Article
Journal Article
Journal Article
Nephrology, Dialysis, Transplantation, ISSN 0931-0509, 06/2016, Volume 31, Issue 6, pp. 914 - 921
Journal Article
Circulation, ISSN 0009-7322, 04/2016, Volume 133, Issue 14, pp. 1360 - 1370
BACKGROUND—The hypertensive syndrome of Apparent Mineralocorticoid Excess is caused by loss-of-function mutations in the gene encoding 11β-hydroxysteroid... 
pressoreceptors | salt | solitary nucleus | mineralocorticoids | aldosterone | APPARENT MINERALOCORTICOID EXCESS | CARDIAC & CARDIOVASCULAR SYSTEMS | SODIUM APPETITE | BAROREFLEX SENSITIVITY | DISTAL TUBULE | 11-BETA-HYDROXYSTEROID DEHYDROGENASE TYPE-2 | HEART-FAILURE | INTRACEREBROVENTRICULAR INFUSION | BLOOD-PRESSURE | PERIPHERAL VASCULAR DISEASE | MICE | Mineralocorticoid Excess Syndrome, Apparent - drug therapy | Mineralocorticoid Excess Syndrome, Apparent - genetics | Nerve Tissue Proteins - deficiency | Solitary Nucleus - enzymology | Receptors, Mineralocorticoid - physiology | RNA, Messenger - biosynthesis | Nephrons - physiopathology | Dexamethasone - pharmacology | Spironolactone - pharmacology | Reflex, Abnormal | Nestin - genetics | Neurons - physiology | Mineralocorticoid Excess Syndrome, Apparent - physiopathology | Craving - physiology | Corticosterone - blood | Hypertension - genetics | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - genetics | Solitary Nucleus - physiopathology | Nerve Tissue Proteins - physiology | Mice, Inbred C57BL | Genes, Synthetic | Nerve Tissue Proteins - genetics | Hypertension - physiopathology | Drinking Behavior | Mice, Knockout | Mineralocorticoid Receptor Antagonists - therapeutic use | Baroreflex - drug effects | Animals | Sodium Chloride, Dietary - toxicity | Potassium - urine | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - metabolism | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - physiology | Mice | Hypertension | Measurement | Care and treatment | Phenylephrine | Blood pressure | Dosage and administration | Cardiovascular diseases | 10111 | 10014 | 10058 | Original
Journal Article
Free Radical Research, ISSN 1071-5762, 04/2019, Volume 53, Issue 4, pp. 387 - 396
Journal Article