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1. Full Text NCLX Is an Essential Component of Mitochondrial Na⁺ /Ca²⁺ Exchange
by Raz Palty and William F. Silverman and Michal Hershfinkel and Teresa Caporale and Stefano L. Sensi and Julia Parnis and Christiane Nolte and Daniel Fishman and Varda Shoshan-Barmatz and Sharon Herrmann and Daniel Khananshvili and Israel Sekler and Harald Reuter
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2010, Volume 107, Issue 1, pp. 436 - 441
Mitochondrial Ca²⁺ efflux is linked to numerous cellular activities and pathophysiological processes. Although it is established that an Na⁺ -dependent... 
Protein isoforms | Mitochondria | Wavelengths | Calcium | Hepatocytes | Plasmids | Fluorescence | Cell membranes | Mice | CHO cells | CELLS | SODIUM | MULTIDISCIPLINARY SCIENCES | INSULIN-SECRETION | NA+-CA2+ EXCHANGER | CGP-37157 | INHIBITION | CA2 | mitochondrial calcium exchanger | sodium calcium exchanger | mitochondrial calcium homeostasis | CALCIUM-TRANSPORT | ANTIPORTER | HEART-MITOCHONDRIA | Brain - cytology | Thiazepines - metabolism | Calcium - metabolism | Clonazepam - analogs & derivatives | Humans | Clonazepam - metabolism | Homeostasis | Rats | Mitochondria - metabolism | Sodium-Calcium Exchanger - genetics | Sodium - metabolism | Mitochondria - ultrastructure | Mitochondrial Membranes - metabolism | Myocardium - cytology | Brain - metabolism | Animals | Sodium-Calcium Exchanger - metabolism | Myocardium - metabolism | Mitochondrial Membranes - ultrastructure | Sodium-Calcium Exchanger - antagonists & inhibitors | Index Medicus | Biological Sciences
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2. Full Text Molecular Changes in Neurons in Multiple Sclerosis: Altered Axonal Expression of Nav1.2 and Nav1.6 Sodium Channels and Na+/ Ca2+Exchanger
by Matthew J. Craner and Jia Newcombe and Joel A. Black and Caroline Hartle and M. Louise Cuzner and Stephen G. Waxman
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2004, Volume 101, Issue 21, pp. 8168 - 8173
Although voltage-gated sodium channels are known to be deployed along experimentally demyelinated axons, the molecular identities of the sodium channels... 
Axons | Sodium channels | Spinal cord | Neuroscience | Physical Sciences | Sodium | Neurons | Central nervous system | Physical trauma | Lesions | White matter | Demyelinating diseases | Action potential conduction | Axonal degeneration | Neurons - pathology | Acute Disease | Spinal Cord - metabolism | Humans | Middle Aged | Axons - metabolism | Sodium - metabolism | Demyelinating Diseases - metabolism | Nerve Tissue Proteins - metabolism | NAV1.6 Voltage-Gated Sodium Channel | Sodium-Calcium Exchanger - metabolism | Spinal Cord - pathology | Axons - pathology | Amyloid beta-Protein Precursor - metabolism | Sodium Channels - metabolism | Multiple Sclerosis - pathology | Neurons - metabolism | Demyelinating Diseases - pathology | NAV1.2 Voltage-Gated Sodium Channel | Multiple Sclerosis - metabolism | Neurology | Multiple sclerosis | Index Medicus | Biological Sciences | axonal degeneration | demyelinating diseases | action potential conduction
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3. Full Text Structural Insight into the Ion-Exchange Mechanism of the Sodium/Calcium Exchanger
by Jun Liao and Hua Li and Weizhong Zeng and David B. Sauer and Ricardo Belmares and Youxing Jiang and Argonne National Lab. (ANL), Argonne, IL (United States). Advanced Photon Source (APS)
Science, ISSN 0036-8075, 2/2012, Volume 335, Issue 6069, pp. 686 - 690
Sodium/calcium (Na⁺/Ca²⁺) exchangers (NCX) are membrane transporters that play an essential role in maintaining the homeostasis of cytosolic Ca²⁺ for cell... 
Proteins | Colors | Passageways | RESEARCH ARTICLES | Crystals | Crystallization | Ligands | Liposomes | Detergents | Binding sites | Crystal structure | SARCOLEMMAL NA+-CA2+ EXCHANGER | CELLS | NEUROTRANSMITTER TRANSPORTERS | CRYSTAL-STRUCTURE | FUNCTIONAL EXPRESSION | BACTERIAL HOMOLOG | MULTIDISCIPLINARY SCIENCES | SODIUM-CALCIUM EXCHANGE | CARDIAC NA+/CA2+ EXCHANGER | NA-CA EXCHANGE | PROTEINS | Archaeal Proteins - metabolism | Amino Acid Sequence | Protein Structure, Secondary | Calcium - metabolism | Sodium-Calcium Exchanger - chemistry | Archaeal Proteins - chemistry | Models, Molecular | Molecular Sequence Data | Crystallography, X-Ray | Sodium - metabolism | Methanococcales - metabolism | Sodium-Calcium Exchanger - metabolism | Protein Conformation | Ion Transport | Methanococcales - chemistry | Binding Sites | Ion exchange | Research | Signal transduction | Membranes | Sodium | Calcium | Molecular biology | Index Medicus | HOMEOSTASIS | ION EXCHANGE | SYMMETRY | ORIENTATION | MEMBRANES | CRYSTAL STRUCTURE | MATERIALS SCIENCE
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4. Full Text NCLX: The mitochondrial sodium calcium exchanger
by Boyman, Liron and Williams, George S.B and Khananshvili, Daniel and Sekler, Israel and Lederer, W.J
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2013, Volume 59, pp. 205 - 213
Abstract The free Ca 2 + concentration within the mitochondrial matrix ([Ca 2 + ]m ) regulates the rate of ATP production and other [Ca 2 + ]m sensitive... 
Cardiovascular | Sodium/calcium exchange | Mitochondria | Calcium transport | Computational model | NCLX | Cardiac myocytes | OXIDATIVE-PHOSPHORYLATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ESSENTIAL COMPONENT | INNER MEMBRANE | INTRAMITOCHONDRIAL CA2 | NA+-CA2+ EXCHANGER | RABBIT CARDIAC MYOCYTES | PERMEABILITY TRANSITION | CELL BIOLOGY | NA+/CA2+ EXCHANGER | CA2+ UPTAKE | HEART-MITOCHONDRIA | Mitochondria - metabolism | Animals | Calcium - metabolism | Sodium-Calcium Exchanger - metabolism | Humans | Computational Biology | Index Medicus | calcium exchange | Sodium
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5. Full Text Ca²⁺ -dependent structural rearrangements within Na⁺ -Ca²⁺ exchanger dimers
by Scott A. John and Bernard Ribalet and James N. Weiss and Kenneth D. Philipson and Michela Ottolia and Ramón Latorre
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2011, Volume 108, Issue 4, pp. 1699 - 1704
Cytoplasmic Ca²⁺ is known to regulate Na⁺–Ca²⁺ exchanger (NCX) activity by binding to two adjacent Ca²⁺-binding domains (CBD1 and CBD2) located in the large... 
Oligomers | Ratios | Fluorescence | Dimers | Cell membranes | Physiological regulation | Kinetics | Energy transfer | Oocytes | Crystal structure | DYNAMIC PROPERTIES | CONFORMATIONAL-CHANGES | NA+/CA2+ EXCHANGER | MULTIDISCIPLINARY SCIENCES | RESONANCE ENERGY-TRANSFER | CA2+ REGULATION | SODIUM-CALCIUM EXCHANGE | STEADY-STATE | CA2+-BINDING DOMAIN | PLASMA-MEMBRANE | MUTATIONAL ANALYSIS | Calcium - pharmacology | Calcium - metabolism | Oocytes - metabolism | Humans | Protein Conformation - drug effects | Protein Multimerization | Sodium-Calcium Exchanger - chemistry | Xenopus laevis | Cytoplasm - metabolism | Sodium-Calcium Exchanger - genetics | Binding Sites - genetics | Animals | Oocytes - physiology | Membrane Potentials | Sodium-Calcium Exchanger - metabolism | Dogs | HEK293 Cells | Fluorescence Resonance Energy Transfer | Female | Luminescent Proteins - genetics | Cell Membrane - metabolism | Mutation | Luminescent Proteins - metabolism | Index Medicus | Biological Sciences
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6. Full Text Cleavage of the plasma membrane Na+/Ca2+ exchanger in excitotoxicity
by Bano, Daniele and Young, Kenneth W and Guerin, Christopher J and LeFeuvre, Ros and Rothwell, Nancy J and Naldini, Luigi and Rizzuto, Rosario and Carafoli, Ernesto and Nicotera, Pierluigi
Cell, ISSN 0092-8674, 01/2005, Volume 120, Issue 2, pp. 275 - 285
In brain ischemia, gating of postsynaptic glutamate receptors and other membrane channels triggers intracellular Ca overload and cell death. In excitotoxic... 
NEURONAL APOPTOSIS | CARDIAC NA+-CA2+ EXCHANGER | NMDA RECEPTOR | PROTEOLYTIC SYSTEM | BIOCHEMISTRY & MOLECULAR BIOLOGY | ISCHEMIC BRAIN-INJURY | CA2+-BINDING DOMAIN | GLUTAMATE EXCITOTOXICITY | EXCITATORY AMINO-ACIDS | CEREBELLAR GRANULE CELLS | PERMEABILITY TRANSITION | CELL BIOLOGY | Receptors, Glutamate - metabolism | Amino Acid Sequence | Calpain - metabolism | Rats, Wistar | Calcium - metabolism | Cells, Cultured | Molecular Sequence Data | Rats | Brain Ischemia - physiopathology | Cell Membrane - physiology | Ion Channel Gating - physiology | Animals | Cell Death - physiology | Sodium-Calcium Exchanger - metabolism | Cysteine Proteinase Inhibitors - pharmacology | Neurons - enzymology | Neurons - physiology | Membrane Transport Proteins - metabolism | Calcium-Binding Proteins - pharmacology | Cell Death - drug effects | Calpain - antagonists & inhibitors | Cell Membrane - drug effects | Ion Channel Gating - drug effects | Index Medicus
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7. Full Text Mechanism of extracellular ion exchange and binding-site occlusion in a sodium/calcium exchanger
by Liao, Jun and Marinelli, Fabrizio and Lee, Changkeun and Huang, Yihe and Faraldo-Gómez, José D and Jiang, Youxing and UChicago, LLC., Argonne, IL (United States)
Nature Structural and Molecular Biology, ISSN 1545-9993, 06/2016, Volume 23, Issue 6, pp. 590 - 599
Na+/Ca2+ exchangers use the Na+ electrochemical gradient across the plasma membrane to extrude intracellular Ca2+ and play a central role in Ca2+ homeostasis.... 
SARCOLEMMAL NA+-CA2+ EXCHANGER | TRANSPORT | BIOPHYSICS | STRUCTURAL BASIS | CA2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | STOICHIOMETRY | SODIUM-CALCIUM EXCHANGE | CARDIAC NA+/CA2+ EXCHANGER | RESIDUES | SQUID AXONS | CELL BIOLOGY | Archaeal Proteins - metabolism | Calcium - metabolism | Sodium-Calcium Exchanger - chemistry | Archaeal Proteins - chemistry | Crystallography, X-Ray | Sodium - metabolism | Strontium - metabolism | Molecular Dynamics Simulation | Sodium-Calcium Exchanger - metabolism | Ion Exchange | Protein Conformation | Methanocaldococcus - metabolism | Molecular Docking Simulation | Binding Sites | Methanocaldococcus - chemistry | Euryarchaeota | Cell interaction | Genetic aspects | Ion exchange | Observations | Health aspects | Sodium channels | Calcium channels | Homeostasis | Ion transport | Properties | Proteins | Membranes | Binding sites | Crystal structure | Index Medicus | X-ray crystallography | INORGANIC, ORGANIC, PHYSICAL, AND ANALYTICAL CHEMISTRY | membrane proteins | BASIC BIOLOGICAL SCIENCES | computational biology and bioinformatics
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8. Full Text Transient Receptor Potential Channel 6–Mediated, Localized Cytosolic [Na+] Transients Drive Na+/Ca2+ Exchanger–Mediated Ca2+ Entry in Purinergically Stimulated Aorta Smooth Muscle Cells
by Poburko, Damon and Liao, Chiu-Hsiang and Lemos, Virginia S and Lin, Eric and Maruyama, Yoshiaki and Cole, William C and van Breemen, Cornelis
Circulation Research, ISSN 0009-7330, 11/2007, Volume 101, Issue 10, pp. 1030 - 1038
The Na/Ca exchanger (NCX) is increasingly recognized as a physiological mediator of Ca influx and significantly contributes to salt-sensitive hypertension. We... 
Hypertension | elevation | Mitochondria | Calcium | TRPC | Localized [Na | exchanger | CoroNa | ATP | Ca | SODIUM | CARDIAC & CARDIOVASCULAR SYSTEMS | calcium | mitochondria | SARCOPLASMIC-RETICULUM | GUINEA-PIG | VENTRICULAR MYOCYTES | localized [Na+] elevation | CATION CHANNELS | FUZZY SPACE | Na+/Ca2+ exchanger | PERIPHERAL VASCULAR DISEASE | ENDOPLASMIC-RETICULUM | HEMATOLOGY | hypertension | TRPC6 Cation Channel | Calcium - metabolism | Humans | Cells, Cultured | Rats | Mitochondria - metabolism | Sodium - metabolism | Muscle, Smooth, Vascular - cytology | Hypertension - physiopathology | Hypertension - metabolism | Adenosine Triphosphate - pharmacology | Animals | Transfection | Sodium-Calcium Exchanger - metabolism | Adenosine Triphosphate - metabolism | TRPC Cation Channels - metabolism | Cytosol - metabolism | Cell Membrane - metabolism | Sarcoplasmic Reticulum - metabolism | Aorta - cytology | TRPC Cation Channels - genetics | Muscle, Smooth, Vascular - physiology | Index Medicus
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9. Full Text Na+ –Ca2+ exchanger mediates ChR2-induced [Ca2+ ]i elevation in astrocytes
by Yang, Junhua and Yu, Hao and Zhou, Danqing and Zhu, Keqing and Lou, Huifang and Duan, Shumin and Wang, Hao
Cell Calcium, ISSN 0143-4160, 2015, Volume 58, Issue 3, pp. 307 - 316
Graphical abstract 
Advanced Basic Science | Calcium elevation | Channelrhodopsin-2 | Na+–Ca2+ exchanger | Astrocytes | Na | Ca | exchanger | Animals | Sodium-Calcium Exchanger - metabolism | Cells, Cultured | Channelrhodopsins | Mice | Calcium Signaling | Astrocytes - metabolism | Index Medicus
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10. Full Text Redox-mediated reciprocal regulation of SERCA and Na+-Ca2+ exchanger contributes to sarcoplasmic reticulum Ca2+ depletion in cardiac myocytes
by Kuster, Gabriela M and Lancel, Steve and Zhang, Jingmei and Communal, Catherine and Trucillo, Mario P and Lim, Chee C and Pfister, Otmar and Weinberg, Ellen O and Cohen, Richard A and Liao, Ronglih and Siwik, Deborah A and Colucci, Wilson S
Free Radical Biology and Medicine, ISSN 0891-5849, 05/2010, Volume 48, Issue 9, pp. 1182 - 1187
Myocardial failure is associated with increased oxidative stress and abnormal excitation-contraction coupling characterized by depletion of sarcoplasmic... 
Oxidative stress | Free radicals | Sodium-calcium exchanger | Cardiac myocyte | SERCA | Reactive thiols | STIMULATED HYPERTROPHY | NA+/CA2+ EXCHANGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | MUSCLE | S-GLUTATHIOLATION | VENTRICULAR MYOCYTES | FREE-RADICALS | CALCIUM-RELEASE | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | HYDROGEN-PEROXIDE | Oxidants - pharmacology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Oxidation-Reduction | Calcium - metabolism | Sarcoplasmic Reticulum - drug effects | Oxidative Stress - physiology | Cells, Cultured | Hydrogen Peroxide - pharmacology | Rats | Male | Rats, Sprague-Dawley | Animals | Myocytes, Cardiac - drug effects | Sodium-Calcium Exchanger - metabolism | Myocytes, Cardiac - metabolism | Sarcoplasmic Reticulum - metabolism | Index Medicus
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11. Full Text ORM‐10103, a novel specific inhibitor of the Na+/Ca2+ exchanger, decreases early and delayed afterdepolarizations in the canine heart
by Jost, N and Nagy, N and Corici, C and Kohajda, Z and Horváth, A and Acsai, K and Biliczki, P and Levijoki, J and Pollesello, P and Koskelainen, T and Otsomaa, L and Tóth, A and Papp, J Gy and Varró, A and Virág, L
British Journal of Pharmacology, ISSN 0007-1188, 10/2013, Volume 170, Issue 4, pp. 768 - 778
Background and PurposeAt present there are no small molecule inhibitors that show strong selectivity for the Na+/Ca2+ exchanger (NCX). Hence, we studied the... 
EAD | NCX | ORM | sodium/calcium exchanger | 10103 | DAD | ORM-10103 | ARRHYTHMIAS | DE-POINTES | SODIUM-CALCIUM EXCHANGER | KB-R7943 | CARDIAC-MUSCLE | NA/CA EXCHANGE | FAILURE | ATRIAL-FIBRILLATION | sodium | CONTRACTION | PHARMACOLOGY & PHARMACY | calcium exchanger | SEA0400 | Potassium - metabolism | Anti-Arrhythmia Agents - pharmacology | Male | Sodium - metabolism | Action Potentials | Dose-Response Relationship, Drug | Purkinje Fibers - drug effects | Time Factors | Sodium-Calcium Exchanger - metabolism | Female | Papillary Muscles - metabolism | Sodium-Calcium Exchanger - antagonists & inhibitors | Benzopyrans - pharmacology | Guinea Pigs | Purkinje Fibers - metabolism | Animals | Myocytes, Cardiac - drug effects | Calcium Signaling - drug effects | Calcium Channels, L-Type - drug effects | Dogs | Myocytes, Cardiac - metabolism | Heart Ventricles - metabolism | Pyridines - pharmacology | Calcium Channels, L-Type - metabolism | Papillary Muscles - drug effects | Heart Ventricles - drug effects | Index Medicus | Research Paper With
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12. Full Text Na+/H+ exchanger 1, Na+/Ca2+ exchanger 1 and calmodulin complex regulates interleukin 6-mediated cellular behavior of human hepatocellular carcinoma
by Xu, Jingyu and Ji, Bei and Wen, Guorong and Yang, Yuan and Jin, Hai and Liu, Xuemei and Xie, Rui and Song, Wenfeng and Song, Penghong and Dong, Hui and Tuo, Biguang
Carcinogenesis, ISSN 0143-3334, 10/2015, Volume 37, Issue 3, pp. 290 - 300
We demonstrate that IL6 regulates the cellular behavior of human HCC through NHE1, NCX1 and CaM molecular complex-mediated signaling mechanisms, which reveals... 
LIVER-CANCER | CELLS | NHE-1 | ONCOLOGY | CA2 | ROLES | INFLAMMATION | SODIUM-CALCIUM EXCHANGE | PROLIFERATION | EXPRESSION | IL-6 | Immunohistochemistry | Calmodulin - metabolism | Immunoprecipitation | Humans | Male | Blotting, Western | Gene Knockdown Techniques | Sodium-Hydrogen Exchangers - metabolism | Animals | Heterografts | Cation Transport Proteins - metabolism | Mice, Nude | Sodium-Calcium Exchanger - metabolism | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Liver Neoplasms - pathology | Mice | Mice, Inbred BALB C | RNA, Small Interfering | Gene Expression Regulation, Neoplastic - physiology | Interleukin-6 - metabolism | Real-Time Polymerase Chain Reaction | Sodium-Hydrogen Exchanger 1 | Carcinoma, Hepatocellular - metabolism | Index Medicus
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13. Full Text Burst pacemaker activity of the sinoatrial node in sodium–calcium exchanger knockout mice
by Angelo G. Torrente and Rui Zhang and Audrey Zaini and Jorge F. Giani and Jeanney Kang and Scott T. Lamp and Kenneth D. Philipson and Joshua I. Goldhaber
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2015, Volume 112, Issue 31, pp. 9769 - 9774
In sinoatrial node (SAN) cells, electrogenic sodium–calcium exchange (NCX) is the dominant calcium (Ca) efflux mechanism. However, the role of NCX in the... 
Sodium-calcium exchange | Arrhythmia | Intracellular calcium | Pacemaker activity | Sinoatrial node | NONSELECTIVE CATION CHANNEL | MECHANISM | NEURONS IN-VITRO | MULTIDISCIPLINARY SCIENCES | sinoatrial node | NA+-CA2+ EXCHANGER | SICK SINUS SYNDROME | DOPAMINE NEURONS | SPIKE FREQUENCY ADAPTATION | arrhythmia | pacemaker activity | intracellular calcium | HEART-RATE | DYSFUNCTION | sodium-calcium exchange | CARDIAC-SPECIFIC KNOCKOUT | Electric Stimulation | Diastole | Calcium - metabolism | Sinoatrial Node - physiology | Male | Connexins - metabolism | Mice, Knockout | Action Potentials | Animals | Sodium-Calcium Exchanger - metabolism | Intracellular Space - metabolism | Fibrosis | Female | Biological Clocks | Calcium Channels, L-Type - metabolism | Receptors, Adrenergic, beta - metabolism | Usage | Care and treatment | Atrial fibrillation | Pacemaker, Artificial (Heart) | Research | Risk factors | Electrocardiography | Calcium | Rodents | Cells | Index Medicus | Biological Sciences
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14. Full Text Sodium channel TRPM4 and sodium/calcium exchangers (NCX) cooperate in the control of Ca 2 -induced mucin secretion from goblet cells
by Cantero-Recasens, Gerard and Cantero-Recasens, Gerard and Butnaru, Cristian M and Butnaru, Cristian M and Brouwers, Nathalie and Brouwers, Nathalie and Mitrovic, Sandra and Valverde, Miguel A and Valverde, Miguel A and Malhotra, Vivek and Malhotra, Vivek
Journal of Biological Chemistry, ISSN 0021-9258, 01/2019, Volume 294, Issue 3, pp. 816 - 826
Regulated mucin secretion is essential for the formation of the mucus layer that protects the underlying epithelial cells from foreign particles. Alterations... 
transient receptor potential channels (TRP channels) | MUC2 | secretion | cystic fibrosis | mucin |