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Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 3050 - 3059
Our study described that DJ‐1 deficient CD4 + T cells leads to upregulation of NHE1 and enhanced ROS production, therefore results in increase in NHE1... 
HEART | IMMUNITY | PHYSIOLOGY | PROTEIN | DENDRITIC CELLS | GENE | ROS | DJ-1 | EXPRESSION | PARKINSONS-DISEASE | PLASTICITY | CELL BIOLOGY | Up-Regulation | Cation Transport Proteins - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Protein-Tyrosine Kinases - metabolism | Protein Deglycase DJ-1 - genetics | RNA, Messenger - metabolism | Sodium-Hydrogen Exchangers - metabolism | Time Factors | Cation Transport Proteins - metabolism | Cation Transport Proteins - genetics | Sodium-Hydrogen Exchangers - genetics | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Protein Deglycase DJ-1 - deficiency | Protein Deglycase DJ-1 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Genotype | Antioxidants - pharmacology | Mice, Knockout | Phenotype | Animals | Protein Kinase Inhibitors - pharmacology | Sodium-Hydrogen Exchanger 1 | CD4-Positive T-Lymphocytes - drug effects | Hydrogen-Ion Concentration | Protein-Tyrosine Kinases - antagonists & inhibitors | T cells | Cysteine | Oxidative stress | Ammonium | Parkinson's disease | Staurosporine | Acetylcysteine | Fluorescence | Lymphocytes T | Gene deletion | Kinases | Western blotting | Proteins | Clonal deletion | Lymphocytes | Rodents | Deletion | Oxidation | Protein-tyrosine kinase | Movement disorders | Tyrosine | Neurodegenerative diseases | Na+/H+-exchanging ATPase | Inflammation | CD4 antigen | Polymerase chain reaction | PARK7 protein | Mice | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2015, Volume 290, Issue 20, pp. 12558 - 12571
Resistance toward imatinib (IM) and other BCR/ABL tyrosine kinase inhibitors remains troublesome in the treatment of advanced stage chronic myeloid leukemia... 
TYROSINE KINASE INHIBITORS | SURVIVAL | APOPTOSIS | ACTIVATION | THERAPY | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | BCR-ABL | FREQUENCY | MUTATIONS | EXPRESSION | Apoptosis - drug effects | Humans | Middle Aged | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Protein Kinase C beta - genetics | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Apoptosis - genetics | Male | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Sodium-Hydrogen Exchangers - metabolism | Heme Oxygenase-1 - genetics | MAP Kinase Signaling System - genetics | Cation Transport Proteins - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Enzyme Induction - drug effects | Adult | Cation Transport Proteins - genetics | Female | NF-E2-Related Factor 2 - genetics | Sodium-Hydrogen Exchangers - genetics | Benzamides - pharmacology | Neoplasm Proteins - genetics | Heme Oxygenase-1 - biosynthesis | Oxidative Stress - genetics | Gene Expression Regulation, Leukemic | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Drug Resistance, Neoplasm - genetics | Enzyme Induction - genetics | MAP Kinase Signaling System - drug effects | NF-E2-Related Factor 2 - metabolism | K562 Cells | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Protein Kinase C beta - metabolism | Aged | Protein Kinase Inhibitors - pharmacology | Oxidative Stress - drug effects | Protein Kinase C beta - antagonists & inhibitors | Sodium-Hydrogen Exchanger 1 | Drug Resistance, Neoplasm - drug effects | Index Medicus | heme oxygenase | H+ ion exchanger 1 | mitogen-activated protein kinase | p38 | protein kinase C (PKC) | chronic myelogenous leukemia (CML) | imatinib resistance | Na+ | drug resistance | p38 MAPK | Cell Biology
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 10/2008, Volume 103, Issue 8, pp. 891 - 899
Activation of the sarcolemmal Na/H exchanger (NHE)1 is increasingly documented as a process involved in cardiac hypertrophy and heart failure. However, whether... 
Na+and Ca2+overload | CaMKII-HDAC pathway | Na+/H+ exchanger | Cardiac remodeling | Calcineurin-NFAT pathway | MYOCARDIUM | NHE1 | CARDIAC & CARDIOVASCULAR SYSTEMS | RAT | PHOSPHORYLATION | CARDIOMYOPATHY | ENHANCED ACTIVITY | VENTRICULAR MYOCYTES | Na+ and Ca2+ overload | INHIBITION | HOMOLOGOUS PROTEIN | H+ EXCHANGE | calcineurin-NFAT pathway | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Phosphorylation | Cation Transport Proteins - antagonists & inhibitors | Humans | Heart Failure - physiopathology | Myocardial Contraction - drug effects | Cytoplasm - metabolism | Guanidines - pharmacology | Sodium - metabolism | Sulfones - pharmacology | Sodium-Hydrogen Exchangers - metabolism | Myocytes, Cardiac - enzymology | Time Factors | Cation Transport Proteins - metabolism | Cell Shape | Cation Transport Proteins - genetics | Sodium-Hydrogen Exchangers - genetics | Sarcoplasmic Reticulum - metabolism | Active Transport, Cell Nucleus | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Calcium-Binding Proteins - metabolism | Signal Transduction | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Cardiomegaly - physiopathology | Heart Failure - genetics | Histone Deacetylases - metabolism | Mice, Transgenic | Heart Failure - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Calcium Signaling - drug effects | Myocytes, Cardiac - metabolism | Mice | Mutation | Cardiomegaly - genetics | Sodium-Hydrogen Exchanger 1 | Calcineurin - metabolism | Cardiomegaly - metabolism | Hydrogen-Ion Concentration | Index Medicus
Journal Article
Journal Article
by Chang, G and Wang, J and Zhang, H and Zhang, Y and Wang, C and Xu, H and Zhang, H and Lin, Y and Ma, L and Li, Q and Pang, T
British Journal of Cancer, ISSN 0007-0920, 02/2014, Volume 110, Issue 4, pp. 916 - 927
Journal Article
BMC Biology, ISSN 1741-7007, 04/2016, Volume 14, Issue 1, pp. 31 - 31
Journal Article
Cancer Letters, ISSN 0304-3835, 2010, Volume 295, Issue 2, pp. 198 - 204
Journal Article