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Journal Article
Human molecular genetics, ISSN 0964-6906, 12/2015, Volume 24, Issue 23, pp. 6614 - 6623
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2015, Volume 290, Issue 20, pp. 12558 - 12571
Resistance toward imatinib (IM) and other BCR/ABL tyrosine kinase inhibitors remains troublesome in the treatment of advanced stage chronic myeloid leukemia... 
TYROSINE KINASE INHIBITORS | SURVIVAL | APOPTOSIS | ACTIVATION | THERAPY | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | BCR-ABL | FREQUENCY | MUTATIONS | EXPRESSION | Apoptosis - drug effects | Humans | Middle Aged | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Protein Kinase C beta - genetics | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Apoptosis - genetics | Male | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Sodium-Hydrogen Exchangers - metabolism | Heme Oxygenase-1 - genetics | MAP Kinase Signaling System - genetics | Cation Transport Proteins - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Enzyme Induction - drug effects | Adult | Cation Transport Proteins - genetics | Female | NF-E2-Related Factor 2 - genetics | Sodium-Hydrogen Exchangers - genetics | Benzamides - pharmacology | Neoplasm Proteins - genetics | Heme Oxygenase-1 - biosynthesis | Oxidative Stress - genetics | Gene Expression Regulation, Leukemic | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Drug Resistance, Neoplasm - genetics | Enzyme Induction - genetics | MAP Kinase Signaling System - drug effects | NF-E2-Related Factor 2 - metabolism | K562 Cells | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Protein Kinase C beta - metabolism | Aged | Protein Kinase Inhibitors - pharmacology | Oxidative Stress - drug effects | Protein Kinase C beta - antagonists & inhibitors | Sodium-Hydrogen Exchanger 1 | Drug Resistance, Neoplasm - drug effects | Index Medicus | heme oxygenase | H+ ion exchanger 1 | mitogen-activated protein kinase | p38 | protein kinase C (PKC) | chronic myelogenous leukemia (CML) | imatinib resistance | Na+ | drug resistance | p38 MAPK | Cell Biology
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 3050 - 3059
Our study described that DJ‐1 deficient CD4 + T cells leads to upregulation of NHE1 and enhanced ROS production, therefore results in increase in NHE1... 
HEART | IMMUNITY | PHYSIOLOGY | PROTEIN | DENDRITIC CELLS | GENE | ROS | DJ-1 | EXPRESSION | PARKINSONS-DISEASE | PLASTICITY | CELL BIOLOGY | Up-Regulation | Cation Transport Proteins - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Protein-Tyrosine Kinases - metabolism | Protein Deglycase DJ-1 - genetics | RNA, Messenger - metabolism | Sodium-Hydrogen Exchangers - metabolism | Time Factors | Cation Transport Proteins - metabolism | Cation Transport Proteins - genetics | Sodium-Hydrogen Exchangers - genetics | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Protein Deglycase DJ-1 - deficiency | Protein Deglycase DJ-1 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Genotype | Antioxidants - pharmacology | Mice, Knockout | Phenotype | Animals | Protein Kinase Inhibitors - pharmacology | Sodium-Hydrogen Exchanger 1 | CD4-Positive T-Lymphocytes - drug effects | Hydrogen-Ion Concentration | Protein-Tyrosine Kinases - antagonists & inhibitors | T cells | Cysteine | Oxidative stress | Ammonium | Parkinson's disease | Staurosporine | Acetylcysteine | Fluorescence | Lymphocytes T | Gene deletion | Kinases | Western blotting | Proteins | Clonal deletion | Lymphocytes | Rodents | Deletion | Oxidation | Protein-tyrosine kinase | Movement disorders | Tyrosine | Neurodegenerative diseases | Na+/H+-exchanging ATPase | Inflammation | CD4 antigen | Polymerase chain reaction | PARK7 protein | Mice | Index Medicus
Journal Article
Nature Methods, ISSN 1548-7091, 10/2010, Volume 7, Issue 10, pp. 801 - 805
Mitogen-activated protein kinase (MAPK) pathways form the backbone of signal transduction in the mammalian cell. Here we applied a systematic experimental and... 
INTERACTION NETWORK | YEAST | PATHWAYS | ORGANIZATION | ACTIVATED PROTEIN-KINASE | H EXCHANGER NHE1 | PHOSPHORYLATION | BIOCHEMICAL RESEARCH METHODS | GLOBAL ANALYSIS | SACCHAROMYCES-CEREVISIAE | SYSTEMS BIOLOGY | RNA, Small Interfering - genetics | MAP Kinase Signaling System - physiology | Saccharomyces cerevisiae - genetics | Transcription Factor AP-1 - genetics | Humans | DNA, Complementary - genetics | Molecular Sequence Data | NF-kappa B - metabolism | Transcription Factor AP-1 - metabolism | Luciferases - genetics | Sodium-Hydrogen Exchangers - metabolism | Saccharomyces cerevisiae - metabolism | MAP Kinase Signaling System - genetics | Cation Transport Proteins - metabolism | Base Sequence | Cloning, Molecular | Mucins - metabolism | Cation Transport Proteins - genetics | Sodium-Hydrogen Exchangers - genetics | Cell Membrane - metabolism | HSP90 Heat-Shock Proteins - genetics | Proteomics - methods | Intracellular Signaling Peptides and Proteins - genetics | Cell Line | Saccharomyces cerevisiae Proteins - genetics | Two-Hybrid System Techniques | Mitogen-Activated Protein Kinases - physiology | NF-kappa B - genetics | HSP90 Heat-Shock Proteins - metabolism | Mitogen-Activated Protein Kinases - genetics | Sodium-Hydrogen Exchanger 1 | Mucins - genetics | Mitogen-Activated Protein Kinases - metabolism | Chromosome mapping | Cellular signal transduction | Genetic aspects | Properties | Methods | Mitogen-activated protein kinases | Signal transduction | Computational mathematics | Kinases | Ribonucleic acid--RNA | Research methodology | Index Medicus | conservation | NHE1 | interactome | scaffold | Two-hybrid
Journal Article
Journal Article
Nature, ISSN 0028-0836, 04/2017, Volume 544, Issue 7649, pp. 235 - 239
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 07/2018, Volume 115, Issue 28, pp. E6640 - E6649
Endosomes have emerged as a central hub and pathogenic driver of Alzheimer's disease (AD). The earliest brain cytopathology in neurodegeneration, occurring... 
Histone deacetylase | Trichostatin A | Amyloid beta | ApoE4 | exchanger | Na+/H+ exchanger | CHRISTIANSON SYNDROME | HUMAN BRAIN | trichostatin A | MULTIDISCIPLINARY SCIENCES | SPORADIC ALZHEIMERS-DISEASE | histone deacetylase | SYNAPTIC PLASTICITY | amyloid beta | APOLIPOPROTEIN-E | LYSOSOMAL DYSFUNCTION | DOWN-SYNDROME | A-BETA | GENE-EXPRESSION | PRECURSOR PROTEIN | Microcephaly - genetics | Epigenesis, Genetic | Humans | Astrocytes - pathology | Epilepsy - metabolism | Endosomes - metabolism | Alzheimer Disease - pathology | Intellectual Disability - metabolism | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | Ataxia - drug therapy | Genetic Diseases, X-Linked - genetics | Ataxia - genetics | Ataxia - metabolism | Microcephaly - drug therapy | Ataxia - pathology | Ocular Motility Disorders - pathology | Tumor Suppressor Proteins - metabolism | Apolipoprotein E4 - metabolism | Endosomes - genetics | Histone Deacetylases - genetics | Ocular Motility Disorders - metabolism | Intellectual Disability - pathology | Alzheimer Disease - drug therapy | Receptors, LDL - metabolism | Mice, Knockout | Genetic Diseases, X-Linked - pathology | Epilepsy - drug therapy | Histone Deacetylase Inhibitors - pharmacology | Mice | Astrocytes - metabolism | Hydrogen-Ion Concentration | Apolipoprotein E4 - genetics | Ocular Motility Disorders - genetics | Intellectual Disability - genetics | Sodium-Hydrogen Exchangers - metabolism | Genetic Diseases, X-Linked - drug therapy | Microcephaly - pathology | Tumor Suppressor Proteins - genetics | Epilepsy - genetics | Sodium-Hydrogen Exchangers - genetics | Receptors, LDL - genetics | Microcephaly - metabolism | Histone Deacetylases - metabolism | Endosomes - pathology | Genetic Diseases, X-Linked - metabolism | Intellectual Disability - drug therapy | Ocular Motility Disorders - drug therapy | Animals | Alzheimer Disease - metabolism | Alzheimer Disease - genetics | Epilepsy - pathology | Physiological aspects | Epigenetic inheritance | Glycoproteins | Genetic aspects | Observations | Astrocytes | Protons | Brain | Membranes | Transcription | Gene regulation | Trafficking | Cognitive ability | Acidification | pH effects | Neuronal-glial interactions | Risk factors | Proteins | Apolipoprotein E | Neurodegeneration | Compartments | Down-regulation | Alzheimer's disease | Plaques | Translocation | Pathogens | Apolipoprotein E4 | Therapeutic applications | Na+/H+-exchanging ATPase | Risk analysis | Apolipoproteins | Nuclear transport | Alleles | Epigenetics | Cytopathology | Receptor density | Mutation | Alzheimers disease |