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1. Full Text Reactive Hyperemia Occurs Via Activation of Inwardly Rectifying Potassium Channels and Na+/K+-ATPase in Humans
by Crecelius, Anne R and Richards, Jennifer C and Luckasen, Gary J and Larson, Dennis G and Dinenno, Frank A
Circulation research, ISSN 0009-7330, 09/2013, Volume 113, Issue 8, pp. 1023 - 1032
Ischemia | Blood flow regulation | Hyperpolarization | Vasodilation | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Humans | Nitric Oxide Synthase - antagonists & inhibitors | Male | Endothelium, Vascular - enzymology | Case-Control Studies | Brachial Artery - enzymology | Young Adult | Microcirculation | Time Factors | Forearm - blood supply | Adult | Brachial Artery - physiopathology | Female | Potassium Channels, Inwardly Rectifying - antagonists & inhibitors | Brachial Artery - drug effects | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Potassium Channel Blockers - administration & dosage | Cyclooxygenase Inhibitors - administration & dosage | Endothelium, Vascular - physiopathology | Regional Blood Flow | Sodium-Potassium-Exchanging ATPase - metabolism | Analysis of Variance | Hyperemia - enzymology | Hyperemia - physiopathology | Adolescent | Blood Flow Velocity | Hemodynamics - drug effects | Nitric Oxide Synthase - metabolism | Nitric Oxide - metabolism | Potassium Channels, Inwardly Rectifying - metabolism | Vasodilator Agents - administration & dosage | Plethysmography | Infusions, Intra-Arterial | Prostaglandins - metabolism | Index Medicus | hyperpolarization | vasodilation | ischemia
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2. Full Text Na+/K+‐ATPase coupled to endothelin receptor type B stimulates peripheral nerve regeneration via lactate signalling
by Tu, Nguyen H and Katano, Tayo and Matsumura, Shinji and Funatsu, Nobuo and Pham, Vuong Minh and Fujisawa, Jun‐ichi and Ito, Seiji
The European journal of neuroscience, ISSN 0953-816X, 09/2017, Volume 46, Issue 5, pp. 2096 - 2107
monocarboxylate transporter | in vivo mouse model | dorsal root ganglion | Schwann cells | sciatic nerve | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Sciatic Nerve - injuries | Nerve Regeneration - physiology | Male | Endothelin B Receptor Antagonists - pharmacology | RNA, Messenger - metabolism | Monocarboxylic Acid Transporters - antagonists & inhibitors | Monocarboxylic Acid Transporters - metabolism | Sciatic Nerve - metabolism | Protein Kinase C - metabolism | Sciatic Nerve - drug effects | Endothelin-3 - metabolism | Schwann Cells - drug effects | Neuronal Outgrowth - physiology | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Nerve Growth Factor - metabolism | Endothelin-1 - metabolism | Mice, Inbred C57BL - metabolism | Sciatic Nerve - pathology | Lactic Acid - metabolism | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mice, Transgenic | Schwann Cells - pathology | Schwann Cells - metabolism | Neuronal Outgrowth - drug effects | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Ganglia, Spinal - pathology | Nerve Regeneration - drug effects | Receptor, Endothelin B - metabolism | Ganglia, Spinal - drug effects | Ganglia, Spinal - metabolism | Lactates | Endothelin | Neurons | Analysis | Ouabain | Nitriles | Protein kinases | Adenosine triphosphatase | Cell culture | Brain | Protein kinase C | Cinnamic acid | Recovery of function | Kinases | Axotomy | Dorsal root ganglia | Rodents | Axonogenesis | Endothelin 3 | Endothelin 1 | Toe | Na+/K+-exchanging ATPase | Ganglia | Regeneration | Signaling | Inhibitors | Reinnervation | Lactic acid | Transporter | Sciatic nerve | Index Medicus
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3. Full Text Selective Assembly of Na,K-ATPase α2β2 Heterodimers in the Heart: DISTINCT FUNCTIONAL PROPERTIES AND ISOFORM-SELECTIVE INHIBITORS
by Habeck, Michael and Tokhtaeva, Elmira and Nadav, Yotam and Ben Zeev, Efrat and Ferris, Sean P and Kaufman, Randal J and Bab-Dinitz, Elizabeta and Kaplan, Jack H and Dada, Laura A and Farfel, Zvi and Tal, Daniel M and Katz, Adriana and Sachs, George and Vagin, Olga and Karlish, Steven J D
The Journal of biological chemistry, ISSN 0021-9258, 10/2016, Volume 291, Issue 44, pp. 23159 - 23174
Potassium - metabolism | Sodium-Potassium-Exchanging ATPase - genetics | Cation Transport Proteins - antagonists & inhibitors | Myocardium - chemistry | Sodium - chemistry | Sodium-Potassium-Exchanging ATPase - chemistry | Humans | Isoenzymes - chemistry | Sodium - metabolism | Cation Transport Proteins - metabolism | Isoenzymes - metabolism | Enzyme Inhibitors - chemistry | Cell Adhesion Molecules, Neuronal - metabolism | Dimerization | Cell Adhesion Molecules, Neuronal - antagonists & inhibitors | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Cell Adhesion Molecules, Neuronal - chemistry | Enzyme Inhibitors - metabolism | Isoenzymes - genetics | Adenosine Triphosphatases - metabolism | Potassium - chemistry | Adenosine Triphosphatases - antagonists & inhibitors | Sodium-Potassium-Exchanging ATPase - metabolism | Myocardium - enzymology | Animals | Adenosine Triphosphatases - chemistry | Mice | Kinetics | Cation Transport Proteins - chemistry | Isoenzymes - antagonists & inhibitors | Index Medicus | molecular modeling | Membrane Biology | cardiomyocyte | excitation-contraction coupling (E-C coupling) | isoform-selective inhibitors | Na+ | K+-ATPase | kinetics | medicinal chemistry | isoform assembly | alpha2beta2
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4. Full Text Functional consequences of the CAPOS mutation E818K of Na,K-ATPase
by Roenn, Christian P and Li, Melody and Schack, Vivien R and Forster, Ian C and Holm, Rikke and Toustrup-Jensen, Mads S and Andersen, Jens P and Petrou, Steven and Vilsen, Bente
The Journal of biological chemistry, ISSN 0021-9258, 2019, Volume 294, Issue 1, pp. 269 - 280
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Sodium-Potassium-Exchanging ATPase - genetics | Hearing Loss, Sensorineural - metabolism | Cerebellar Ataxia - metabolism | Humans | Xenopus laevis | Adenosine Triphosphate - genetics | Foot Deformities, Congenital - genetics | Foot Deformities, Congenital - metabolism | Hearing Loss, Sensorineural - genetics | Mutation, Missense | Reflex, Abnormal - genetics | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Cerebellar Ataxia - genetics | Membrane Potentials | Optic Atrophy - metabolism | Adenosine Triphosphate - metabolism | Vanadates - pharmacology | Protein Domains | Amino Acid Substitution | Optic Atrophy - genetics | Index Medicus | site-directed mutagenesis | membrane enzyme | potassium transport | Membrane Biology | neurological disease | sodium transport | ATP1A3 | K+-ATPase | electrophysiology | Na+,K+-pump | membrane transport | Na+ | enzyme mechanism | mutant | P-type ATPase | Na+ site
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5. Full Text Cationic nanocarriers induce cell necrosis through impairment of Na^＋/K^＋-ATPase and cause subsequent inflammatory response
by Xiawei Wei Bin Shao Zhiyao He Tinghong Ye Min Luo Yaxiong Sang Xiao Liang Wei Wang Shuntao Luo Shengyong Yang Shuang Zhang Changyang Gong Maling Gou Hongxing Deng Yinglan Zhao Hanshuo Yang Senyi Deng Chengjian Zhao Li Yang Zhiyong Qian Jiong Li Xun Sun Jiahuai Han Chengyu Jiang Min Wu Zhirong Zhang
Cell research, ISSN 1001-0602, 2015, Volume 25, Issue 2, pp. 237 - 253
线粒体DNA | 阳离子脂质体 | ATP酶 | ATPase活性 | 纳米载体 | 细胞坏死 | 炎症反应 | damage-associated molecular patterns | inflammation | necrosis | ATPase | cationic nanocarriers | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Inflammation - pathology | Sodium-Potassium-Exchanging ATPase - genetics | Apoptosis - drug effects | Humans | Drug Carriers - toxicity | Drug Carriers - chemistry | Necrosis | Polyethyleneimine - toxicity | Polyethyleneimine - chemistry | Ouabain - pharmacology | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Cations - chemistry | Liposomes - toxicity | DNA, Mitochondrial - metabolism | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Chitosan - toxicity | Mice, Knockout | Sodium-Potassium-Exchanging ATPase - metabolism | Liposomes - chemistry | Animals | Chitosan - chemistry | Signal Transduction - drug effects | Cell Line, Tumor | Nanostructures - chemistry | Mice | Nanostructures - toxicity | Toll-Like Receptor 9 - metabolism | Myeloid Differentiation Factor 88 - metabolism | Index Medicus | Na+ | K+-ATPase | Original
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6. Full Text Creatine increases hippocampal Na+ ,K+ -ATPase activity via NMDA–calcineurin pathway
by Rambo, Leonardo Magno and Ribeiro, Leandro Rodrigo and Schramm, Vanessa Grigoletto and Berch, Andriely Moreira and Stamm, Daniel Neis and Della-Pace, Iuri Domingues and Silva, Luiz Fernando Almeida and Furian, Ana Flávia and Oliveira, Mauro Schneider and Fighera, Michele Rechia and Royes, Luiz Fernando Freire
Brain research bulletin, ISSN 0361-9230, 2012, Volume 88, Issue 6, pp. 553 - 559
Neurology | Sodium pump | Creatine | NMDA receptors | Calcineurin | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Creatine - pharmacology | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Rats, Wistar | Nitric Oxide Synthase - antagonists & inhibitors | Male | Hippocampus - drug effects | N-Methylaspartate - pharmacology | Calcineurin Inhibitors | Piperidines - pharmacology | Isoenzymes - metabolism | Hippocampus - enzymology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cell Membrane - drug effects | Nerve Tissue Proteins - antagonists & inhibitors | Rats | Excitatory Amino Acid Antagonists - pharmacology | Protein Kinase C - antagonists & inhibitors | Receptors, N-Methyl-D-Aspartate - physiology | Calcineurin - physiology | Nerve Tissue Proteins - metabolism | Cell Membrane - enzymology | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Signal Transduction - drug effects | Signal Transduction - physiology | Isoenzymes - drug effects | Sodium-Potassium-Exchanging ATPase - drug effects | Dizocilpine Maleate - pharmacology | Index Medicus
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7. Full Text The interactive contributions of Na+/K+‐ATPase and nitric oxide synthase to sweating and cutaneous vasodilatation during exercise in the heat
by Louie, Jeffrey C and Fujii, Naoto and Meade, Robert D and Kenny, Glen P
The Journal of physiology, ISSN 0022-3751, 06/2016, Volume 594, Issue 12, pp. 3453 - 3462
Neurosciences | Physiology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | NG-Nitroarginine Methyl Ester - pharmacology | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Sodium-Potassium-Exchanging ATPase - physiology | Humans | Nitric Oxide Synthase - antagonists & inhibitors | Nitric Oxide Synthase - physiology | Male | Sweating - physiology | Hot Temperature | Skin - blood supply | Young Adult | Adult | Exercise - physiology | Vasodilation - physiology | Ouabain - pharmacology | Evaluation | Cardiotonic agents | Dilatation | Nitric oxide | Ouabain | Blood vessels | Skin | Adenosine triphosphatase | Cardiac glycosides | Metabolism | Fixed rates | Index Medicus | Integrative | Research Paper | Exercise Physiology | Vasculature
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8. Full Text Rostafuroxin: an ouabain antagonist that corrects renal and vascular Na+-K+- ATPase alterations in ouabain and adducin-dependent hypertension
by Patrizia Ferrari and Mara Ferrandi and Giovanni Valentini and Giuseppe Bianchi
American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 03/2006, Volume 290, Issue 3, pp. 529 - 535
Antihypertensive therapy | pump | Adducin | Endogenous ouabain | Life Sciences & Biomedicine | Physiology | Science & Technology | Kidney - drug effects | Blood Vessels - metabolism | Humans | Antihypertensive Agents - administration & dosage | Hypertension - drug therapy | Treatment Outcome | Calmodulin-Binding Proteins - metabolism | Androstanols - administration & dosage | Hypertension - metabolism | Ouabain - antagonists & inhibitors | Ouabain - metabolism | Kidney - metabolism | Animals | Blood Vessels - drug effects | Sodium-Potassium-Exchanging ATPase | Index Medicus
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9. Full Text Na+‐K+‐ATPase, a potent neuroprotective modulator against Alzheimer disease
by Zhang, Li‐Nan and Sun, Yong‐Jun and Pan, Shuo and Li, Jun‐Xia and Qu, Yin‐E and Li, Yao and Wang, Yong‐Li and Gao, Zi‐Bin
Fundamental & clinical pharmacology, ISSN 0767-3981, 02/2013, Volume 27, Issue 1, pp. 96 - 103
pathogenesis | Alzheimer disease | Na+‐K+‐ATPase | Pathogenesis | ATPase | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Neurology | Organic mental disorders. Neuropsychology | Psychology. Psychoanalysis. Psychiatry | Adult and adolescent clinical studies | Pharmacology. Drug treatments | Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases | Psychopathology. Psychiatry | Biological and medical sciences | Medical sciences | Alzheimer Disease - etiology | Neuroprotective Agents - therapeutic use | Sodium-Potassium-Exchanging ATPase - chemistry | Humans | Brain - enzymology | Isoenzymes - chemistry | Protein Subunits - metabolism | Brain - metabolism | Alzheimer Disease - prevention & control | Isoenzymes - metabolism | Enzyme Induction - drug effects | Neurons - metabolism | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Protein Subunits - agonists | Alzheimer Disease - therapy | Nootropic Agents - therapeutic use | Isoenzymes - deficiency | Enzyme Activation - drug effects | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Neurons - enzymology | Alzheimer Disease - metabolism | Protein Subunits - deficiency | Protein Subunits - antagonists & inhibitors | Oxidative Stress - drug effects | Dietary Supplements | Sodium-Potassium-Exchanging ATPase - deficiency | Alzheimer's disease | Cysteine | Adenosine triphosphatase | Index Medicus
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