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Cell Metabolism, ISSN 1550-4131, 01/2015, Volume 21, Issue 1, pp. 39 - 50
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 02/2016, Volume 310, Issue 4, pp. F300 - F310
Journal Article
Kidney International, ISSN 0085-2538, 01/2017, Volume 91, Issue 1, pp. 24 - 33
Gitelman syndrome (GS) is a rare, salt-losing tubulopathy characterized by hypokalemic metabolic alkalosis with hypomagnesemia and hypocalciuria. The disease... 
hypokalemic metabolic alkalosis | hypomagnesemia | SLC12A3 | thiazide-sensitive sodium-chloride cotransporter | salt-losing tubulopathy | SERUM POTASSIUM | FOLLOW-UP | HYPOKALEMIC ALKALOSIS | THERAPEUTIC APPROACH | BARTTERS-SYNDROME | BLOOD-PRESSURE | NCC GENE | GLUCOSE-METABOLISM | UROLOGY & NEPHROLOGY | CHANNEL GENE | SODIUM-CHLORIDE COTRANSPORTER | Rare Diseases - genetics | Gitelman Syndrome - drug therapy | Genetic Testing | Hypokalemia - blood | Magnesium - administration & dosage | Humans | Bartter Syndrome - diagnosis | Chloride Channels - genetics | Bartter Syndrome - urine | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Bartter Syndrome - genetics | Solute Carrier Family 12, Member 3 - genetics | Ultrasonography | Bartter Syndrome - blood | Diagnosis, Differential | Chondrocalcinosis - prevention & control | Sodium Chloride, Dietary - therapeutic use | Calcium - urine | Consensus Development Conferences as Topic | Magnesium - therapeutic use | Phenotype | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Potassium - administration & dosage | Chondrocalcinosis - etiology | Potassium - therapeutic use | Gitelman Syndrome - complications | Gitelman Syndrome - diagnosis | Quality of Life | Potassium - blood | Gitelman Syndrome - genetics | Mutation | Angiotensin Receptor Antagonists - therapeutic use | Dietary Supplements | Hypokalemia - genetics | Magnesium - blood | Practice Guidelines as Topic
Journal Article
by Zhang, Y and Zhang, F and Chen, D and Lü, Q and Tang, L and Yang, C and Lei, M and Tong, N
Brazilian Journal of Medical and Biological Research, ISSN 0100-879X, 2016, Volume 49, Issue 11, p. e5261
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 06/2014, Volume 6, Issue 6, pp. 744 - 759
Fibroblast growth factor‐23 (FGF23) is a bone‐derived hormone regulating renal phosphate reabsorption and vitamin D synthesis in renal proximal tubules. Here,... 
heart hypertrophy | blood pressure | aldosterone | sodium homeostasis | fibroblast growth factor‐23 | Sodium homeostasis | Blood pressure | Fibroblast growth factor-23 | Aldosterone | Heart hypertrophy | KIDNEY-DISEASE | MEDICINE, RESEARCH & EXPERIMENTAL | X-LINKED HYPOPHOSPHATEMIA | D METABOLISM | VITAMIN-D-RECEPTOR | ANGIOTENSIN-II | DEFICIENT MICE | fibroblast growth factor-23 | GLUCOSE-HOMEOSTASIS | NA+-CL-COTRANSPORTER | SIGNALING PATHWAY | CHLORIDE COTRANSPORTER | Blood Pressure | Up-Regulation | Humans | Fibroblast Growth Factors - genetics | Hypertension - drug therapy | Male | Sodium - metabolism | Sodium Chloride Symporter Inhibitors - therapeutic use | Fibroblast Growth Factors - metabolism | Kidney - metabolism | Chlorothiazide - therapeutic use | Fibroblast Growth Factors - administration & dosage | Gene Deletion | Solute Carrier Family 12, Member 3 - genetics | Hypertension - genetics | Recombinant Proteins - metabolism | Signal Transduction | Mice, Inbred C57BL | Recombinant Proteins - genetics | Antihypertensive Agents - therapeutic use | Recombinant Proteins - administration & dosage | Hypertension - metabolism | Animals | Glucuronidase - genetics | Mice | Solute Carrier Family 12, Member 3 - metabolism | Cardiomegaly - metabolism | Hypertension | Heart | Phosphates | Corticosteroids | Vitamin D | Chronic kidney failure | Analysis | Calcifediol | Fibroblast growth factors | Alfacalcidol
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 08/2017, Volume 313, Issue 2, pp. F326 - F338
Kidney transplantation (KTX) is a life-saving procedure for patients with end-stage renal disease. Expression levels of many genes in the kidney vary between... 
Receptors and sodium transporter | Mouse kidney transplantation | Sex difference | receptors and sodium transporter | PHYSIOLOGY | sex difference | SPONTANEOUSLY HYPERTENSIVE-RATS | LONG-TERM OUTCOMES | REPERFUSION INJURY | ANGIOTENSIN-II | mouse kidney transplantation | EPITHELIAL SODIUM-CHANNEL | II-INDUCED HYPERTENSION | MINOR HISTOCOMPATIBILITY ANTIGENS | NITRIC-OXIDE | UROLOGY & NEPHROLOGY | GENDER-DIFFERENCES | AMBULATORY BLOOD-PRESSURE | Receptors, Angiotensin - metabolism | Kidney - blood supply | Receptors, Endothelin - genetics | Kidney - pathology | Nephritis - genetics | Male | Nephritis - pathology | Epithelial Sodium Channels - metabolism | Sodium - metabolism | RNA, Messenger - metabolism | Gene-Environment Interaction | Sodium-Hydrogen Exchangers - metabolism | Kidney - metabolism | Epithelial Sodium Channels - genetics | Time Factors | Solute Carrier Family 12, Member 3 - genetics | Inflammation Mediators - metabolism | Female | Models, Animal | Sodium-Hydrogen Exchangers - genetics | Kidney - surgery | Solute Carrier Family 12, Member 1 - metabolism | Nephritis - physiopathology | Endothelin-1 - genetics | Endothelin-1 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | Receptors, Endothelin - metabolism | Genotype | Receptors, Angiotensin - genetics | Renal Elimination | Solute Carrier Family 12, Member 1 - genetics | Animals | Renal Circulation | Sex Factors | Nephritis - metabolism | Hemodynamics | Solute Carrier Family 12, Member 3 - metabolism | Kidney Transplantation - adverse effects | Sodium-Hydrogen Exchanger 3
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 03/2017, Volume 312, Issue 3, pp. F489 - F501
Calcineurin dephosphorylates nuclear factor of activated T cells transcription factors, thereby facilitating T cell-mediated immune responses. Calcineurin... 
Hypertension | Salt transport | Vasopressin | Sodium-chloride cotransporter | Sodium-potassium-chlo-ride cotransporter | sodium-potassium-chloride cotransporter | PHYSIOLOGY | CYCLOOXYGENASE-2 | RAT | PHOSPHORYLATION | vasopressin | TAMM-HORSFALL PROTEIN | SENSITIVE NA+ | TRANSPORT | SHORT-TERM STIMULATION | salt transport | UROLOGY & NEPHROLOGY | sodium-chloride cotransporter | hypertension | EXPRESSION | THICK ASCENDING LIMB | Rats, Brattleboro | Renin - metabolism | Epithelial Cells - metabolism | Rats, Wistar | Epithelial Cells - drug effects | Loop of Henle - metabolism | Male | Water-Electrolyte Balance - drug effects | Time Factors | Hypertension - chemically induced | Solute Carrier Family 12, Member 3 - genetics | Loop of Henle - drug effects | Solute Carrier Family 12, Member 1 - agonists | Solute Carrier Family 12, Member 1 - metabolism | Cells, Cultured | Loop of Henle - physiopathology | Arginine Vasopressin - pharmacology | Immunosuppressive Agents - toxicity | Hypertension - physiopathology | Hypertension - metabolism | Solute Carrier Family 12, Member 1 - genetics | Cyclosporine - toxicity | Animals | Calcineurin Inhibitors - toxicity | Kidney Tubules, Distal - drug effects | Cyclooxygenase 2 - metabolism | Kidney Tubules, Distal - physiopathology | Solute Carrier Family 12, Member 3 - agonists | Solute Carrier Family 12, Member 3 - metabolism | Kidney Tubules, Distal - metabolism | Carrier proteins | Health aspects | Calcineurin
Journal Article
Journal Article
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 09/2014, Volume 307, Issue 6, pp. R653 - R663
Guanylin (GN) inhibited water absorption and short-circuit current (Isc) in seawater eel intestine. Similar inhibition was observed after bumetanide, and the... 
Ion transport | Guanylin | Water absorption | Seawater adaptation | Na + -K + -Cl - transport system | eel intestine | PHYSIOLOGY | ATRIAL-NATRIURETIC-PEPTIDE | ANGUILLA-ANGUILLA | water absorption | ion transport | ANION-EXCHANGE | guanylin | seawater adaptation | Na+-K+-Cl- transport system | TRANSMEMBRANE CONDUCTANCE REGULATOR | HCO3-SECRETION | TELEOST FISH | CYSTIC-FIBROSIS | JAPANESE EEL | EUROPEAN EEL | MARINE FISH OSMOREGULATION | Adaptation, Physiological | Intestines - drug effects | Sodium Potassium Chloride Symporter Inhibitors - pharmacology | Solute Carrier Family 12, Member 1 - antagonists & inhibitors | Gastrointestinal Hormones - pharmacology | Intestines - metabolism | Aquaporin 1 - metabolism | Fish Proteins - genetics | Solute Carrier Family 12, Member 3 - genetics | Ion Transport | Fish Proteins - antagonists & inhibitors | Seawater | Solute Carrier Family 12, Member 1 - metabolism | Water - metabolism | Eels - genetics | Fish Proteins - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Natriuretic Peptides - pharmacology | Bumetanide - pharmacology | Aquaporin 1 - genetics | Solute Carrier Family 12, Member 1 - genetics | Animals | Membrane Potentials | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Eels - metabolism | Solute Carrier Family 12, Member 3 - drug effects | Kinetics | Solute Carrier Family 12, Member 3 - metabolism
Journal Article
PLOS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, p. e0147831
The (Pro) renin receptor (P)RR/Atp6ap2 is a cell surface protein capable of binding and non-proteolytically activate prorenin. Additionally, (P)RR is... 
V-ATPASE | LUMINAL MEMBRANE | METABOLIC-ACIDOSIS | RAT-KIDNEY | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | MOLECULAR-CLONING | RENAL TUBULAR-ACIDOSIS | (PRO)RENIN RECEPTOR | UP-REGULATION | TURTLE-BLADDER | Kidney Cortex - drug effects | Kidney Medulla - cytology | Membrane Glycoproteins - metabolism | Kidney Cortex - metabolism | Sodium Chloride - pharmacology | Male | Kidney Tubules, Collecting - cytology | Sodium-Phosphate Cotransporter Proteins, Type IIa - metabolism | Sulfate Transporters | Proton-Translocating ATPases - metabolism | Ammonium Chloride - pharmacology | Proton-Translocating ATPases - genetics | Solute Carrier Family 12, Member 3 - genetics | Madin Darby Canine Kidney Cells | Cell Membrane - metabolism | Renin - pharmacology | Cell Membrane - drug effects | Kidney Tubules, Collecting - drug effects | Kidney Cortex - cytology | Kidney Tubules, Proximal - cytology | Solute Carrier Family 12, Member 1 - metabolism | Signal Transduction | Kidney Medulla - drug effects | Mice, Inbred C57BL | Gene Expression Regulation | Sodium Bicarbonate - pharmacology | Receptors, Cell Surface - metabolism | Anion Transport Proteins - metabolism | Membrane Glycoproteins - genetics | Aquaporin 2 - metabolism | Sodium-Phosphate Cotransporter Proteins, Type IIa - genetics | Solute Carrier Family 12, Member 1 - genetics | Kidney Tubules, Collecting - metabolism | Animals | Dogs | Kidney Medulla - metabolism | Kidney Tubules, Proximal - metabolism | Renin-Angiotensin System - drug effects | Mice | Aquaporin 2 - genetics | Anion Transport Proteins - genetics | Solute Carrier Family 12, Member 3 - metabolism | Kidney Tubules, Proximal - drug effects | Receptors, Cell Surface - genetics | Immunohistochemistry | Plasma | Drinking water | Salts | Membranes | Phosphorylation | Wnt protein | Syngeneic grafts | Hydrogen | Acidification | mRNA | Proximal tubules | Kinases | Experiments | Cell surface | Protein turnover | Proteins | Bicarbonate | Endocytosis | Renin | Rodents | Physiology | Carbonates | Localization | H+-transporting ATPase | Hypertension | Kidneys | Incubation | Internal medicine | Ducts | Extracellular signal-regulated kinase | Tubules | Reabsorption | Pharmacology | Metabolism | Sodium chloride | Membrane proteins | Medicine | Signaling | Adenosine triphosphatase
Journal Article
Scientific Reports, ISSN 2045-2322, 07/2015, Volume 5, Issue 1, p. 12480
Ureteral obstruction with subsequent hydronephrosis is a common clinical complication. Downregulation of renal sodium transporters in obstructed kidneys could... 
DUCT-SPECIFIC KNOCKOUT | CELLS | RENAL SODIUM TRANSPORTERS | CAUSES HYPERTENSION | MULTIDISCIPLINARY SCIENCES | INJURY | NATRIURETIC PEPTIDE | P2Y RECEPTOR | STRESS | PROGRESSION | WATER | Sodium-Potassium-Exchanging ATPase - genetics | Kidney - pathology | Solute Carrier Family 12, Member 1 - antagonists & inhibitors | Natriuretic Peptide, Brain - metabolism | Male | Ureteral Obstruction - metabolism | Epithelial Sodium Channels - metabolism | Electron Transport Complex I - metabolism | Kidney Diseases - genetics | Sodium-Hydrogen Exchangers - metabolism | Kidney - metabolism | Epithelial Sodium Channels - genetics | Electron Transport Complex I - genetics | Solute Carrier Family 12, Member 3 - genetics | Rotenone - pharmacology | Ureteral Obstruction - genetics | Sodium-Hydrogen Exchangers - genetics | Kidney Diseases - metabolism | Solute Carrier Family 12, Member 1 - agonists | Solute Carrier Family 12, Member 1 - metabolism | Electron Transport Complex I - antagonists & inhibitors | Kidney - drug effects | Angiotensin II - metabolism | Signal Transduction | Kidney Diseases - pathology | Mice, Inbred C57BL | Gene Expression Regulation | Kidney Diseases - drug therapy | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Dinoprostone - metabolism | Solute Carrier Family 12, Member 1 - genetics | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Nitric Oxide Synthase Type II - genetics | Ureteral Obstruction - pathology | Mice | Nitric Oxide - metabolism | Solute Carrier Family 12, Member 3 - metabolism | Nitric Oxide Synthase Type II - metabolism | Sodium-Hydrogen Exchanger 3
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 11/2016, Volume 480, Issue 4, pp. 745 - 751
Kelch-like 3 (KLHL3) is a component of an E3 ubiquitin ligase complex that regulates blood pressure by targeting With-No-Lysine (WNK) kinases for degradation.... 
Post-translational modification | Hypokalemia | Distal nephron | Ubiquitin proteasome pathway | SPAK | PROTEIN-KINASE-C | BIOCHEMISTRY & MOLECULAR BIOLOGY | WNK4 | KLHL3 | BLOOD-PRESSURE | TRANSPORT | BIOPHYSICS | DEGRADATION | MUTATIONS | HYPERKALEMIC HYPERTENSION | CHLORIDE | Potassium Deficiency - genetics | Protein Kinase C - genetics | Phosphorylation | Kidney - pathology | Humans | Male | Potassium, Dietary - administration & dosage | Sodium - metabolism | Kidney - metabolism | WNK Lysine-Deficient Protein Kinase 1 | Protein Kinase C - metabolism | Potassium Deficiency - pathology | Potassium, Dietary - metabolism | Solute Carrier Family 12, Member 3 - genetics | HEK293 Cells | Hypokalemia - metabolism | Minor Histocompatibility Antigens - genetics | Microfilament Proteins - metabolism | Hypertension - genetics | Hypokalemia - pathology | Microfilament Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Hypertension - pathology | Hypertension - metabolism | Minor Histocompatibility Antigens - metabolism | Animals | Diet | Potassium Deficiency - metabolism | Mice | Hypokalemia - genetics | Solute Carrier Family 12, Member 3 - metabolism | Hypertension | Ubiquitin | Medical colleges | Lysine | Ligases | Protein kinases | hypokalemia | distal nephron | post-translational modification | ubiquitin proteasome pathway
Journal Article