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Development, ISSN 0950-1991, 01/2010, Volume 137, Issue 2, pp. 203 - 212
The transcription factor neurogenin 3 (Neurog3 or Ngn3) controls islet cell fate specification in multipotent pancreatic progenitor cells in the mouse embryo.... 
Mouse | Rfx | Endocrine | Zebrafish | Cell differentiation | Pancreas | Transcription factor | Neurogenin 3 | PROGENITOR CELLS | NEUROGENIN3 | BETA-CELLS | DEVELOPMENTAL BIOLOGY | HORMONE-EXPRESSING CELLS | PROTEIN ISL-1 | ENDOCRINE PANCREAS | GENE | EMBRYONIC STEM-CELLS | DIFFERENTIATION | Immunohistochemistry | Paired Box Transcription Factors - genetics | Homeodomain Proteins - metabolism | Pancreas - cytology | Winged-Helix Transcription Factors - metabolism | Embryo, Nonmammalian - metabolism | Stem Cells - cytology | Stem Cells - metabolism | Embryo, Mammalian - metabolism | Endocrine Cells - metabolism | In Situ Hybridization | Basic Helix-Loop-Helix Transcription Factors - metabolism | Gene Expression Regulation, Developmental | Islets of Langerhans - metabolism | Islets of Langerhans - cytology | Somatostatin - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Zebrafish Proteins - metabolism | Winged-Helix Transcription Factors - genetics | Cells, Cultured | Pancreas - metabolism | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Nerve Tissue Proteins - genetics | Pancreas - embryology | Homeodomain Proteins - genetics | Endoderm - metabolism | Ghrelin - metabolism | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Blotting, Northern | Animals | Endocrine Cells - cytology | Glucagon - metabolism | Mice | Zebrafish Proteins - genetics | In Vitro Techniques | Paired Box Transcription Factors - metabolism | Development and Stem Cells
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2052 - 2062
Background & Aims The winged helix transcription factors Foxa1 and Foxa2 are expressed in all epithelia of the gastrointestinal tract from its embryonic origin... 
Gastroenterology and Hepatology | RESPONSE ELEMENT | GLUCOSE-HOMEOSTASIS | GLUCAGON-LIKE PEPTIDE-1 | PROGLUCAGON GENE-TRANSCRIPTION | IN-VIVO | INSULIN-SECRETION | PANCREATIC BETA-CELLS | GASTROENTEROLOGY & HEPATOLOGY | ISLET CELLS | EXPRESSION | ENDOCRINE-CELLS | Immunohistochemistry | Hepatocyte Nuclear Factor 3-beta - genetics | Intestine, Small - pathology | Peptide YY - metabolism | Homeodomain Proteins - metabolism | Male | RNA, Messenger - metabolism | Cell Differentiation | Enteroendocrine Cells - metabolism | Proglucagon - metabolism | Somatostatin - metabolism | Repressor Proteins - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Mucin-2 - metabolism | Enteroendocrine Cells - pathology | Somatostatin-Secreting Cells - pathology | Glucagon-Like Peptide 1 - metabolism | Glucagon-Like Peptide 2 - metabolism | Hepatocyte Nuclear Factor 3-alpha - deficiency | Hepatocyte Nuclear Factor 3-alpha - genetics | PAX6 Transcription Factor | Goblet Cells - metabolism | Hepatocyte Nuclear Factor 3-alpha - metabolism | Mice, Knockout | Mucin-5B - metabolism | Animals | Eye Proteins - metabolism | Mucin 5AC - metabolism | LIM-Homeodomain Proteins | Mice | Transcription Factors | Goblet Cells - pathology | Intestine, Small - metabolism | Somatostatin-Secreting Cells - metabolism | Hepatocyte Nuclear Factor 3-beta - deficiency | Paired Box Transcription Factors - metabolism | Chromatin | Messenger RNA | DNA binding proteins | Cholecystokinin | Peptides | Mucins
Journal Article
Journal Article
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2008, Volume 134, Issue 7, pp. 1842 - 1860
Recent milestones in the understanding of gastric acid secretion and treatment of acid-peptic disorders include the (1) discovery of histamine H2 -receptors... 
Gastroenterology and Hepatology | RABBIT PARIETAL-CELLS | PERFUSED RAT STOMACH | COMMUNITY-ACQUIRED PNEUMONIA | ATRIAL-NATRIURETIC-PEPTIDE | HELICOBACTER-PYLORI INFECTION | GASTROESOPHAGEAL-REFLUX DISEASE | PROTON PUMP INHIBITORS | ENTEROCHROMAFFIN-LIKE CELLS | SOMATOSTATIN RECEPTOR SCINTIGRAPHY | GASTROENTEROLOGY & HEPATOLOGY | GLYCINE-EXTENDED GASTRINS | Gastrointestinal Diseases - physiopathology | Gastroesophageal Reflux - physiopathology | Humans | Acetylcholine - metabolism | Stomach - metabolism | Gastric Mucosa - metabolism | Gastrointestinal Diseases - microbiology | Gastric Acid - secretion | Histamine - metabolism | Helicobacter pylori | Gastrointestinal Diseases - drug therapy | Helicobacter Infections - metabolism | Duodenal Ulcer - metabolism | Stomach - innervation | Stomach - drug effects | Somatostatin - metabolism | Helicobacter Infections - physiopathology | Anti-Ulcer Agents - therapeutic use | Eating | Proton Pump Inhibitors - adverse effects | Gastroesophageal Reflux - metabolism | Stomach Ulcer - metabolism | H(+)-K(+)-Exchanging ATPase - metabolism | Paracrine Communication | Anti-Ulcer Agents - adverse effects | Histamine H2 Antagonists - adverse effects | Digestion | Duodenal Ulcer - physiopathology | Gastrins - metabolism | Proton Pump Inhibitors - therapeutic use | Histamine H2 Antagonists - therapeutic use | Stomach Ulcer - physiopathology | Animals | Gastrointestinal Diseases - metabolism | Ion Channels - metabolism | Gastric Mucosa - secretion | Stomach - enzymology | Stomach - microbiology | Gastroesophageal reflux | Polypeptides | Gastrin
Journal Article
Journal Article
Diabetes (New York, N.Y.), ISSN 0012-1797, 11/2011, Volume 60, Issue 11, pp. 2861 - 2871
OBJECTIVE-Class Ha histone deacetylases (HDACs) belong to a large family of enzymes involved in protein deacetylation and play a role in regulating gene... 
PLAY | PROGENITORS | RESPONSIVENESS | CONTROLS CHONDROCYTE HYPERTROPHY | SMALL-MOLECULE | GROWTH | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | MEF2 TRANSCRIPTION FACTOR | DIFFERENTIATION | INHIBITORS | Embryo, Mammalian - drug effects | Homeodomain Proteins - metabolism | Somatostatin-Secreting Cells - cytology | RNA, Messenger - metabolism | Embryo, Mammalian - metabolism | Repressor Proteins - antagonists & inhibitors | Insulin-Secreting Cells - metabolism | Isoenzymes - metabolism | Mice, Mutant Strains | Somatostatin-Secreting Cells - drug effects | Insulin-Secreting Cells - cytology | Somatostatin - metabolism | Repressor Proteins - metabolism | Animals, Newborn | Histone Deacetylases - genetics | Isoenzymes - genetics | Tissue Culture Techniques | Enzyme Inhibitors - pharmacology | Gene Expression Regulation, Developmental - drug effects | Histone Deacetylases - chemistry | Organ Size | Pancreas - drug effects | Repressor Proteins - genetics | Histone Deacetylases - metabolism | Pancreas - metabolism | Organ Specificity | Insulin - metabolism | Algorithms | Animals | Insulin-Secreting Cells - drug effects | Cell Differentiation - drug effects | Embryo, Mammalian - cytology | Mice | Pancreas - growth & development | Somatostatin-Secreting Cells - metabolism | Isoenzymes - antagonists & inhibitors | Paired Box Transcription Factors - metabolism | Islet Studies
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2017, Volume 127, Issue 7, pp. 2631 - 2646
Somatostatin secreted by pancreatic delta cells mediates important paracrine interactions in Langerhans islets, including maintenance of glucose metabolism through the control of reciprocal insulin... 
SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | CALCIUM-CHANNELS | UBIQUITIN LIGASE | INSULIN-SECRETION | BETA-CELLS | ALPHA | DIFFERENTIATION | SOMATOSTATIN RECEPTOR | ISLET CELLS | CUL4B | Calcium - metabolism | Epigenesis, Genetic | Homeodomain Proteins - metabolism | Somatostatin-Secreting Cells - cytology | Multienzyme Complexes - metabolism | Cullin Proteins - metabolism | Insulin - genetics | Cyclic AMP - metabolism | Insulin Secretion | Somatostatin - metabolism | Somatostatin - genetics | Paracrine Communication | Multienzyme Complexes - genetics | Adenylyl Cyclases - metabolism | Transcription Factors - genetics | Homeodomain Proteins - genetics | Mice, Knockout | Cullin Proteins - genetics | Transcription Factors - metabolism | Insulin - metabolism | Animals | Calcium Channels, L-Type - genetics | Mice | Calcium Channels, L-Type - metabolism | Adenylyl Cyclases - genetics | Somatostatin-Secreting Cells - metabolism | Histone-lysine N-methyltransferase | Glucagon | Methyltransferase | Homeostasis | Paracrine signalling | Bipolar disorder | Glucose | Cullin | Homeobox | Proteins | Glucose metabolism | Transgenic animals | Rodents | N-Methyltransferase | Somatostatin | Pancreas | Ubiquitin-protein ligase | Obesity | Calcium (intracellular) | Secretion | Diabetes mellitus | Calcium channels (voltage-gated) | Cyclic AMP | Insulin | Studies | Glucose tolerance | Polycomb group proteins | Urocortin | Lysine | Epigenetics | Diabetes | Mutation | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Endocrinology, ISSN 0013-7227, 11/2015, Volume 156, Issue 11, pp. 3924 - 3936
Journal Article