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1. Full Text Sp1 expression regulates lung tumor progression
by Hsu, T.-I and Wang, M.-C and Chen, S.-Y and Yeh, Y.-M and Su, W.-C and Chang, W.-C and Hung, J.-J
Oncogene, ISSN 0950-9232, 08/2012, Volume 31, Issue 35, pp. 3973 - 3988
The role of specificity protein 1 (Sp1) in controlling gene expression in lung tumor development and metastasis is not well understood. In this study, we... 
metastasis | Sp1 | E-cadherin | CANCER-CELLS | CARCINOMA-CELLS | SPECIFICITY PROTEIN-1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION FACTOR SP1 | DOWN-REGULATION | BETA-CATENIN | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | FACTOR-I-RECEPTOR | GENE-EXPRESSION | Lung Neoplasms - genetics | Adenocarcinoma - pathology | Cell Proliferation | Adenocarcinoma of Lung | Humans | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Mice, Transgenic | Sp1 Transcription Factor - metabolism | Cadherins - biosynthesis | beta Catenin - metabolism | Cell Movement - genetics | Disease Progression | Neoplasm Metastasis - genetics | Animals | Sp1 Transcription Factor - genetics | Cell Line, Tumor | Adenocarcinoma - genetics | Mice | Neoplasm Invasiveness - genetics | Care and treatment | Lung cancer | Patient outcomes | Physiological aspects | Development and progression | Genetic aspects | Metastasis | Research | Gene expression | Health aspects | Cancer | Oncology | Tumors | Index Medicus | Adenocarcinoma | Cell proliferation | Invasiveness | catenin | Transgenic mice | Malignancy | Sp1 protein | Nuclear transport | Metastases | Doxycycline | E-Cadherin | Tumorigenesis | Cell migration | Original
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2. Full Text Long non‐coding RNA metastasis‐associated lung adenocarcinoma transcript 1 promotes lung adenocarcinoma by directly interacting with specificity protein 1
by Li, Shufeng and Ma, Fang and Jiang, Kunpeng and Shan, Haitao and Shi, Minke and Chen, Baojun
Cancer Science, ISSN 1347-9032, 05/2018, Volume 109, Issue 5, pp. 1346 - 1356
Metastasis‐associated lung adenocarcinoma transcript 1 (malat1) is an oncogenic long non‐coding RNA (lnc RNA ) which has been proven to be associated with... 
lnc | lung cancer | RNA | transcription | malat1 | lncRNA | SP1 | CELLS | ANGIOGENESIS | TUMOR | SP1 EXPRESSION | GENE | ONCOLOGY | GASTRIC-CANCER | GROWTH-FACTOR | RESISTANT PROSTATE-CANCER | UP-REGULATION | Neoplasm Transplantation | Up-Regulation | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | Sp1 Transcription Factor - metabolism | Aged, 80 and over | Adult | Female | Transcription, Genetic | Adenocarcinoma - genetics | 3' Untranslated Regions | Binding Sites | Lung Neoplasms - genetics | A549 Cells | RNA, Long Noncoding - chemistry | Sp1 Transcription Factor - chemistry | RNA, Long Noncoding - genetics | RNA Stability | Feedback, Physiological | Animals | Sp1 Transcription Factor - genetics | Survival Analysis | Cell Line, Tumor | Aged | Mice | Neoplasm Staging | RNA, Long Noncoding - metabolism | Adenocarcinoma | Metastasis | Lung cancer | Index Medicus | Original
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3. Full Text MicroRNA-22 suppresses the growth, migration and invasion of colorectal cancer cells through a Sp1 negative feedback loop
by Xia, SS and Zhang, GJ and Liu, ZL and Tian, HP and He, Y and Meng, CY and Li, LF and Wang, ZW and Zhou, T
ONCOTARGET, ISSN 1949-2553, 05/2017, Volume 8, Issue 22, pp. 36266 - 36278
MicroRNAs have recently emerged as regulators of many biological processes including cell proliferation, development and differentiation. This study identified... 
colorectal cancer | TARGETING SP1 | METASTASIS | PTEN | TRANSCRIPTION FACTOR SP1 | AKT | POSTTRANSLATIONAL MODIFICATIONS | COLON | PTEN EXPRESSION | CELL BIOLOGY | BREAST-CANCER | Sp1 | GASTRIC-CANCER | miR-22 | CARCINOMA | Recurrence | Cell Proliferation | Prognosis | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Sp1 Transcription Factor - metabolism | Neoplasm Metastasis | RNA Interference | Female | Proto-Oncogene Proteins c-akt - metabolism | Colorectal Neoplasms - metabolism | Disease Models, Animal | Colorectal Neoplasms - mortality | Promoter Regions, Genetic | Signal Transduction | PTEN Phosphohydrolase - metabolism | Disease Progression | Animals | Sp1 Transcription Factor - genetics | Models, Biological | Cell Line, Tumor | Protein Binding | Mice | MicroRNAs - genetics | Colorectal Neoplasms - pathology | Cell Movement | Index Medicus
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4. Full Text Regulation of cytochrome P450 2e1 expression by ethanol: Role of oxidative stress-mediated pkc/jnk/sp1 pathway
by Jin, M and Ande, A and Kumar, A and Kumar, S
Cell Death and Disease, ISSN 2041-4889, 03/2013, Volume 4, Issue 3, pp. e554 - e554
CYP2E1 metabolizes ethanol leading to production of reactive oxygen species (ROS) and acetaldehyde, which are known to cause not only liver damage but also... 
Oxidative stress | Monocytes | CYP2E1 | Ethanol | Astrocytes | PKC/JNK/SP1 | monocytes | PROTEIN-KINASE-C | C/EBP-BETA | INDUCTION | KNOCKOUT MICE | CELL-LINES | ethanol | CELL BIOLOGY | LIVER-INJURY | MESSENGER-RNA | ALCOHOL | astrocytes | oxidative stress | BRAIN | Protein Kinase C - genetics | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Astrocytes - pathology | Monocytes - metabolism | Sp1 Transcription Factor - metabolism | Cytochrome P-450 CYP2E1 - genetics | Cytochrome P-450 CYP2E1 - metabolism | Reactive Oxygen Species - agonists | Sp1 Transcription Factor - antagonists & inhibitors | MAP Kinase Kinase 4 - metabolism | Protein Kinase C - metabolism | Monocytes - pathology | MAP Kinase Kinase 4 - antagonists & inhibitors | Sulfides - pharmacology | Astrocytes - drug effects | Cytochrome P-450 CYP2E1 Inhibitors | Protein Kinase C - antagonists & inhibitors | Allyl Compounds - pharmacology | Plicamycin - pharmacology | Anthracenes - pharmacology | Plicamycin - analogs & derivatives | Gene Expression Regulation - drug effects | Monocytes - drug effects | Ethanol - pharmacology | Reactive Oxygen Species - antagonists & inhibitors | Signal Transduction - drug effects | Sp1 Transcription Factor - genetics | Cell Line, Tumor | MAP Kinase Kinase 4 - genetics | Oxidative Stress - drug effects | Staurosporine - pharmacology | Astrocytes - metabolism | Index Medicus | SP1 | JNK | PKC | Original
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5. Full Text Epigenetic regulation of CD133/PROM1 expression in glioma stem cells by Sp1/myc and promoter methylation
by Gopisetty, G and Xu, J and Sampath, D and Colman, H and Puduvalli, V.K
Oncogene, ISSN 0950-9232, 06/2013, Volume 32, Issue 26, pp. 3119 - 3129
Tumor stem cells, postulated to be the source cells for malignancies, have been identified in several cancers using cell-surface expression of markers... 
glioma Stem Cell | promoter methylation | Sp1 | epigenetic regulation | Myc | CD133 | DNA METHYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | C-MYC | CPG BINDING-PROTEIN | TUMOR-INITIATING CELLS | CELL BIOLOGY | CHROMOSOMAL-PROTEIN | EPITHELIAL-CELLS | ONCOLOGY | GENETICS & HEREDITY | FUNCTIONAL-ANALYSIS | GENE-EXPRESSION | TRANSCRIPTION FACTOR | HEMATOPOIETIC STEM | Nucleic Acid Synthesis Inhibitors - pharmacology | Epigenesis, Genetic | Humans | Glycoproteins - metabolism | Methyl-CpG-Binding Protein 2 - metabolism | Peptides - genetics | Sp1 Transcription Factor - metabolism | Sp1 Transcription Factor - immunology | Antigens, CD - genetics | Methyl-CpG-Binding Protein 2 - genetics | Antigens, CD - metabolism | DNA-Binding Proteins - metabolism | Glioma - genetics | Sp1 Transcription Factor - antagonists & inhibitors | DNA Methylation | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Base Sequence | Transcription, Genetic | Cell Differentiation | Glycoproteins - genetics | Promoter Regions, Genetic | Oncogene Protein p55(v-myc) - metabolism | DNA-Binding Proteins - genetics | Plicamycin - pharmacology | AC133 Antigen | Sequence Analysis, DNA | Transcription Factors - metabolism | Sp1 Transcription Factor - genetics | Cell Line, Tumor | CpG Islands - genetics | Oncogene Protein p55(v-myc) - immunology | Care and treatment | Gliomas | Stem cells | Genetic aspects | Research | Methylation | Health aspects | Risk factors | Epigenetics | DNA methylation | Gene expression | Index Medicus | Chromatin
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6. Full Text Modulation of specificity protein 1 by mithramycin A as a novel therapeutic strategy for cervical cancer
by Choi, Eun-Sun and Choi, Eun-Sun and Nam, Jeong-Seok and Nam, Jeong-Seok and Jung, Ji-Youn and Jung, Ji-Youn and Cho, Nam-Pyo and Cho, Nam-Pyo and Cho, Sung-Dae and Cho, Sung-Dae
Scientific reports, ISSN 2045-2322, 2014, Volume 4, Issue 1, pp. 7162 - 7162
Cervical cancer is the third most common cancer and the third leading cause of death among women. However, the standard treatment for cervical cancer includes... 
TRANSCRIPTION FACTORS | CYTOCHROME-C | DEATH RECEPTORS | CELLS | INHIBITION | SP1 | MULTIDISCIPLINARY SCIENCES | FOLLOW-UP | INDUCE APOPTOSIS | GROWTH-FACTOR | CARCINOMA | Antibiotics, Antineoplastic - pharmacology | Apoptosis - drug effects | Humans | Caspase 8 - metabolism | Sp1 Transcription Factor - metabolism | Transplantation, Heterologous | Uterine Cervical Neoplasms - pathology | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Membrane Potential, Mitochondrial - drug effects | Sp1 Transcription Factor - antagonists & inhibitors | RNA Interference | Female | BH3 Interacting Domain Death Agonist Protein - metabolism | Peptide Hydrolases - metabolism | Plicamycin - therapeutic use | Uterine Cervical Neoplasms - drug therapy | Mitochondria - metabolism | Mitochondria - drug effects | Plicamycin - pharmacology | Plicamycin - analogs & derivatives | Animals | Antibiotics, Antineoplastic - therapeutic use | Signal Transduction - drug effects | Sp1 Transcription Factor - genetics | Mice, Nude | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Kidneys | Toxicity | Body weight | Caspase | Drug delivery | Cervix | Sp1 protein | Cisplatin | Proteins | Caspase-8 | Signal transduction | Side effects | Human papillomavirus | Cervical cancer | Index Medicus
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7. Full Text Inhibition of the transcription factor Sp1 suppresses colon cancer stem cell growth and induces apoptosis in vitro and in nude mouse xenografts
by ZHAO, YINGYING and ZHANG, WENJING and GUO, ZHENG and MA, FENG and WU, YAO and BAI, YANG and GONG, WEI and CHEN, YE and CHENG, TIANMING and ZHI, FACHAO and ZHANG, YALI and WANG, JIDE and JIANG, BO
Oncology Reports, ISSN 1021-335X, 10/2013, Volume 30, Issue 4, pp. 1782 - 1792
The transcription factor specificity protein 1 (Sp1) plays a role in the development and progression of various types of human cancers, while cancer stem cells... 
Sp1 | colon cancer | CD44 | CD166 | CSCs | Colon cancer | DRUG | C-KIT | SELF-RENEWAL | BREAST-CANCER | MELANOMA | ADHESION MOLECULE | ONCOLOGY | COLORECTAL-CANCER | EXPRESSION | CARCINOMA | PROGRESSION | Neoplasm Transplantation | Up-Regulation | Peptides | Antigens, CD - biosynthesis | Apoptosis - drug effects | Colorectal Neoplasms - genetics | Humans | Apoptosis - genetics | Sp1 Transcription Factor - metabolism | Sp1 Transcription Factor - biosynthesis | Sp1 Transcription Factor - antagonists & inhibitors | Heterografts | Neoplastic Stem Cells - metabolism | RNA Interference | Colorectal Neoplasms - drug therapy | Activated-Leukocyte Cell Adhesion Molecule - biosynthesis | Colorectal Neoplasms - metabolism | Down-Regulation | HCT116 Cells | Spheroids, Cellular - metabolism | Hyaluronan Receptors - biosynthesis | Plicamycin - pharmacology | AC133 Antigen | Plicamycin - analogs & derivatives | Glycoproteins - biosynthesis | HT29 Cells | Animals | Sp1 Transcription Factor - genetics | Mice, Nude | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | RNA, Small Interfering | Biomarkers, Tumor - biosynthesis | Cell culture | Immunoglobulins | Transcription factors | Dehydrogenases | Colorectal cancer | Metastasis | Gene expression | Drug resistance | Polymerase chain reaction | Proteins | Cell growth | Medical prognosis | Stem cells | Tumorigenesis | Growth factors | Apoptosis | Index Medicus
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8. Full Text Transcription Factor Sp1 Regulates the Expression of Calcium Channel α2δ-1 Subunit in Neuropathic Pain
by Gómez, Kimberly and Sandoval, Alejandro and Barragán-Iglesias, Paulino and Granados-Soto, Vinicio and Delgado-Lezama, Rodolfo and Felix, Ricardo and González-Ramírez, Ricardo
Neuroscience, ISSN 0306-4522, 08/2019, Volume 412, pp. 207 - 215
High voltage-activated (HVA) Ca (Ca ) channels are oligomeric complexes formed by an ion-conducting main subunit (Cavα ) and at least two auxiliary subunits... 
Sp1 | neuropathic pain | calcium channels
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9. Full Text Mithramycin depletes specificity protein 1 and activates p53 to mediate senescence and apoptosis of malignant pleural mesothelioma cells
by Rao, Mahadev and Atay, Scott M and Shukla, Vivek and Hong, Young and Upham, Trevor and Ripley, R. Taylor and Hong, Julie A and Zhang, Mary and Reardon, Emily and Fetsch, Patricia and Miettinen, Markku and Li, Xinmin and Peer, Cody J and Sissung, Tristan and Figg, William D and De Rienzo, Assunta and Bueno, Raphael and Schrump, David S
Clinical Cancer Research, ISSN 1078-0432, 03/2016, Volume 22, Issue 5, pp. 1197 - 1210
Purpose: Specificity protein 1 (SP1) is an oncogenic transcription factor overexpressed in various human malignancies. This study sought to examine SP1... 
CANCER-CELLS | LUNG-CANCER | IN-VITRO | THERAPY | GENE | ONCOLOGY | SIGNALING PATHWAY | GROWTH | TRANSCRIPTION FACTOR SP1 | MUTATIONS | EXPRESSION | Lung Neoplasms - drug therapy | Pleural Neoplasms - genetics | Tumor Suppressor Protein p53 - biosynthesis | Mesothelioma - pathology | Apoptosis - drug effects | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Cellular Senescence - drug effects | Lung Neoplasms - pathology | Sp1 Transcription Factor - biosynthesis | Tumor Suppressor Protein p53 - genetics | Pleural Neoplasms - drug therapy | Sp1 Transcription Factor - antagonists & inhibitors | RNA, Messenger - biosynthesis | Plicamycin - administration & dosage | Female | Pleural Neoplasms - pathology | Lung Neoplasms - genetics | Mesothelioma - genetics | Mesothelioma - drug therapy | Xenograft Model Antitumor Assays | Animals | Sp1 Transcription Factor - genetics | Biomarkers, Tumor - genetics | Cell Proliferation - drug effects | Mice | Biomarkers, Tumor - biosynthesis | Index Medicus | senescence | malignant pleural mesothelioma | apoptosis | SP1 | epigenetics | p53
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10. Full Text Sp1 is upregulated in human glioma, promotes MMP‐2‐mediated cell invasion and predicts poor clinical outcome
by Guan, Hongyu and Cai, Junchao and Zhang, Nu and Wu, Jueheng and Yuan, Jie and Li, Jun and Li, Mengfeng
International Journal of Cancer, ISSN 0020-7136, 02/2012, Volume 130, Issue 3, pp. 593 - 601
Sp1, the first identified transcription factor, has been reported to be associated with the development and progression of various human cancer types. However,... 
glioma | invasion | Sp1 | prognosis | MMP‐2 | MMP-2 | FACTOR FAMILY | ACTIVATION | MATRIX-METALLOPROTEINASE | TRANSCRIPTION FACTOR SP1 | DNA-BINDING ACTIVITY | ONCOLOGY | GASTRIC-CANCER | MALIGNANT GLIOMAS | EXPRESSION | CARCINOMA | ENDOTHELIAL GROWTH-FACTOR | Gene Expression | Glioma - mortality | Prognosis | Glioma - diagnosis | Matrix Metalloproteinase 2 - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Sp1 Transcription Factor - metabolism | Up-Regulation - genetics | Glioma - metabolism | Glioma - genetics | Matrix Metalloproteinase 2 - genetics | Sp1 Transcription Factor - genetics | HEK293 Cells | Aged, 80 and over | Cell Line, Tumor | Adult | Female | Aged | Neoplasm Invasiveness - genetics | Neoplasm Staging | Multivariate analysis | Medical research | Index Medicus
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11. Full Text SP1 mediates the link between methylation of the tumour suppressor miR‐149 and outcome in colorectal cancer
by Wang, Feng and Ma, Yan‐Lei and Zhang, Peng and Shen, Tong‐Yi and Shi, Chen‐Zhang and Yang, Yong‐Zhi and Moyer, Mary‐Pat and Zhang, Hui‐Zhen and Chen, Hong‐Qi and Liang, Yong and Qin, Huan‐Long
The Journal of Pathology, ISSN 0022-3417, 01/2013, Volume 229, Issue 1, pp. 12 - 24
Although recent studies indicate that DNA methylation contributes to the down‐regulation of microRNAs (miRNAs) in colorectal cancer (CRC), this field remains... 
overall survival | epigenetic silencing | promoter methylation | Sp1 | CRC | miR‐149 | miR-149 | SURVIVAL | DNA METHYLATION | TRANSCRIPTION FACTOR SP1 | GENETIC-VARIANTS | PATHOLOGY | MICRORNA EXPRESSION PROFILES | HUMAN GASTRIC-CANCER | COLON-CANCER | ONCOLOGY | SQUAMOUS-CELL CARCINOMA | PROGRESSION | ENDOTHELIAL GROWTH-FACTOR | Multivariate Analysis | Cell Proliferation | Prognosis | Oligonucleotide Array Sequence Analysis | CpG Islands - drug effects | DNA Modification Methylases - antagonists & inhibitors | Colorectal Neoplasms - genetics | Humans | Middle Aged | Molecular Sequence Data | Male | MicroRNAs - metabolism | Sp1 Transcription Factor - metabolism | Gene Knockdown Techniques | Transfection | Time Factors | Base Sequence | Polymerase Chain Reaction | Female | Colorectal Neoplasms - metabolism | Colorectal Neoplasms - mortality | DNA Modification Methylases - metabolism | Risk Assessment | Signal Transduction | Neoplasm Invasiveness | Down-Regulation | HCT116 Cells | Risk Factors | Enzyme Inhibitors - pharmacology | Gene Silencing | Kaplan-Meier Estimate | Proportional Hazards Models | Azacitidine - analogs & derivatives | Gene Expression Profiling - methods | Chi-Square Distribution | HT29 Cells | Azacitidine - pharmacology | DNA Modification Methylases - genetics | Sp1 Transcription Factor - genetics | Colorectal Neoplasms - pathology | DNA Methylation - drug effects | Index Medicus
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12. Full Text miR‐145 sensitizes ovarian cancer cells to paclitaxel by targeting Sp1 and Cdk6
by Zhu, Xiaolan and Li, Yuefeng and Xie, Chanjuan and Yin, Xinming and Liu, Yueqin and Cao, Yuan and Fang, Yue and Lin, Xin and Xu, Yao and Xu, Wenlin and Shen, Huiling and Wen, Jian
International Journal of Cancer, ISSN 0020-7136, 09/2014, Volume 135, Issue 6, pp. 1286 - 1296
Multidrug resistance (MDR) remains a major obstacle to effective chemotherapy treatment in ovarian cancer. In our study, paclitaxel‐resistant ovarian cancer... 
pRb | miR‐145 | Cdk6 | Sp1 | P‐gp | P-gp | miR-145 | ACTIVATION | DOWN-REGULATION | GENE PROMOTER | INHIBITION | ONCOLOGY | RESISTANCE | TRANSCRIPTION FACTOR INTERACTIONS | AGENTS | TUMOR-SUPPRESSOR MIR-145 | EXPRESSION | MICRORNA SIGNATURES | RNA, Small Interfering - genetics | Paclitaxel - pharmacology | Cyclin-Dependent Kinase 6 - biosynthesis | Humans | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Drug Resistance, Neoplasm | MicroRNAs - metabolism | Sp1 Transcription Factor - metabolism | Paclitaxel - pharmacokinetics | Sp1 Transcription Factor - biosynthesis | Ovarian Neoplasms - genetics | Retinoblastoma Protein - biosynthesis | Transfection | MCF-7 Cells | Cell Cycle Checkpoints - genetics | Female | Ovarian Neoplasms - metabolism | Antineoplastic Agents, Phytogenic - pharmacokinetics | Ovarian Neoplasms - drug therapy | MicroRNAs - administration & dosage | Retinoblastoma Protein - metabolism | Cyclin-Dependent Kinase 6 - genetics | Cyclin-Dependent Kinase 6 - metabolism | ATP-Binding Cassette, Sub-Family B, Member 1 - biosynthesis | Xenograft Model Antitumor Assays | Animals | Sp1 Transcription Factor - genetics | ATP-Binding Cassette, Sub-Family B, Member 1 - genetics | Cell Cycle Checkpoints - drug effects | Retinoblastoma Protein - genetics | Cell Line, Tumor | Ovarian Neoplasms - therapy | Mice | Mice, Inbred BALB C | MicroRNAs - genetics | Antineoplastic Agents, Phytogenic - pharmacology | RNA, Small Interfering - administration & dosage | Antimitotic agents | Yuan (China) | Chemotherapy | Cancer cells | Drug resistance | Antineoplastic agents | Cancer | Ovarian cancer | Medical research | Cancer therapies | Index Medicus
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13. Full Text SP1-induced upregulation of the long noncoding RNA TINCR regulates cell proliferation and apoptosis by affecting KLF2 mRNA stability in gastric cancer
by Xu, T.-P and Liu, X.-X and Xia, R and Yin, L and Kong, R and Chen, W.-M and Huang, M.-D and Shu, Y.-Q
Oncogene, ISSN 0950-9232, 03/2015, Volume 34, Issue 45, pp. 5648 - 5661
The long noncoding RNA TINCR shows aberrant expression in human squamous carcinomas. However, its expression and function in gastric cancer remain unclear. We... 
SURVIVAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION FACTOR SP1 | CELL BIOLOGY | NCRNAS | REPRESSION | ONCOLOGY | POOR-PROGNOSIS | GENETICS & HEREDITY | CARCINOMAS | KRUPPEL-LIKE FACTOR-2 | INHIBITORS | HALLMARKS | DECREASED EXPRESSION | RNA-Binding Proteins - genetics | Cell Proliferation | Cytoskeletal Proteins - genetics | Humans | Stomach Neoplasms - metabolism | Sp1 Transcription Factor - metabolism | Stomach Neoplasms - pathology | RNA, Messenger - metabolism | RNA, Neoplasm - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclin-Dependent Kinase Inhibitor p15 - genetics | RNA, Long Noncoding - bios