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1. Full Text Betulinic acid inhibits prostate cancer growth through inhibition of specificity protein transcription factors
by Chintharlapalli, Sudhakar and Papineni, Sabitha and Ramaiah, Shashi K and Safe, Stephen
Cancer research (Chicago, Ill.), ISSN 0008-5472, 03/2007, Volume 67, Issue 6, pp. 2816 - 2823
Life Sciences & Biomedicine | Oncology | Science & Technology | Tumors of the urinary system | Urinary tract. Prostate gland | Gynecology. Andrology. Obstetrics | Pharmacology. Drug treatments | Nephrology. Urinary tract diseases | Biological and medical sciences | Medical sciences | Antineoplastic agents | Male genital diseases | Tumors | Prostatic Neoplasms - metabolism | Sp3 Transcription Factor - antagonists & inhibitors | Triterpenes - pharmacology | Microtubule-Associated Proteins - genetics | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Humans | Male | Sp1 Transcription Factor - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Neoplasm Proteins - metabolism | Promoter Regions, Genetic - drug effects | Sp1 Transcription Factor - antagonists & inhibitors | Liver - drug effects | Neoplasm Proteins - genetics | Prostatic Neoplasms - drug therapy | Sp3 Transcription Factor - metabolism | Prostatic Neoplasms - blood supply | Prostatic Neoplasms - pathology | Inhibitor of Apoptosis Proteins | Cell Growth Processes - drug effects | Liver - metabolism | Sp3 Transcription Factor - genetics | Sp Transcription Factors - antagonists & inhibitors | Angiogenesis Inhibitors - pharmacology | Sp Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Sp4 Transcription Factor - metabolism | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Mice | Proteasome Endopeptidase Complex - metabolism | Antineoplastic Agents, Phytogenic - pharmacology | Sp4 Transcription Factor - antagonists & inhibitors | Sp4 Transcription Factor - genetics | Index Medicus
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2. Full Text The transcription factor Sp1 modulates RNA polymerase III gene transcription by controlling BRF1 and GTF3C2 expression in human cells
by Peng, Feixia and Zhou, Ying and Wang, Juan and Guo, Baoqiang and Wei, Yun and Deng, Huan and Wu, Zihui and Zhang, Cheng and Shi, Kaituo and Li, Yuan and Wang, Xin and Shore, Paul and Zhao, Shasha and Deng, Wensheng and Wek, Ronald C
The Journal of biological chemistry, ISSN 0021-9258, 04/2020, Volume 295, Issue 14, pp. 4617 - 4630
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Transcription Factors, TFIII - genetics | TATA-Binding Protein Associated Factors - metabolism | Up-Regulation | Cell Proliferation | Humans | Sp1 Transcription Factor - metabolism | E1A-Associated p300 Protein - metabolism | TATA-Box Binding Protein - metabolism | Sp1 Transcription Factor - antagonists & inhibitors | RNA Interference | RNA Polymerase III - genetics | TATA-Box Binding Protein - genetics | Transcription Factors, TFIII - metabolism | Transcription, Genetic | Transcription Factors, TFIII - antagonists & inhibitors | Filamins - genetics | Binding Sites | Cell Line | Promoter Regions, Genetic | Mutagenesis, Site-Directed | RNA Polymerase III - metabolism | TATA-Binding Protein Associated Factors - antagonists & inhibitors | Filamins - antagonists & inhibitors | TATA-Binding Protein Associated Factors - genetics | Filamins - metabolism | Sp1 Transcription Factor - genetics | RNA, Small Interfering - metabolism | Index Medicus
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3. Full Text p63α protein up-regulates heat shock protein 70 expression via E2F1 transcription factor 1, promoting Wasf3/Wave3/MMP9 signaling and bladder cancer invasion
by Jin, Honglei and Xie, Qipeng and Guo, Xirui and Xu, Jiheng and Wang, Annette and Li, Jingxia and Zhu, Junlan and Wu, Xue-Ru and Huang, Haishan and Huang, Chuanshu
The Journal of biological chemistry, ISSN 0021-9258, 09/2017, Volume 292, Issue 38, pp. 15952 - 15963
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Up-Regulation | Tumor Suppressor Proteins - metabolism | Cell Proliferation | Signal Transduction | Wiskott-Aldrich Syndrome Protein Family - metabolism | Neoplasm Invasiveness | Humans | HSP70 Heat-Shock Proteins - genetics | Male | Sp1 Transcription Factor - metabolism | E2F1 Transcription Factor - metabolism | HSP70 Heat-Shock Proteins - metabolism | Transcription Factors - metabolism | Matrix Metalloproteinase 9 - metabolism | Animals | Urinary Bladder Neoplasms - genetics | Urinary Bladder Neoplasms - pathology | Cell Line, Tumor | Transcription, Genetic | Mice | E2F1 Transcription Factor - genetics | Urinary Bladder Neoplasms - metabolism | Index Medicus | bladder cancer cell invasion | Wasf3 | Hsp70 | E2F transcription factor | invasion | p63α | E2F1 | 70-kilodalton heat shock protein (Hsp70) | p63 | Wave3 | deleted in bladder cancer 1 (DBC1) | Cell Biology
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4. Full Text Early B-cell factor 1 (EBF1) is critical for transcriptional control of SLAMF1 gene in human B cells
by Schwartz, Anton M and Putlyaeva, Lidia V and Covich, Milica and Klepikova, Anna V and Akulich, Kseniya A and Vorontsov, Ilya E and Korneev, Kirill V and Dmitriev, Sergey E and Polanovsky, Oleg L and Sidorenko, Svetlana P and Kulakovskiy, Ivan V and Kuprash, Dmitry V
Biochimica et biophysica acta. Gene regulatory mechanisms, ISSN 1874-9399, 10/2016, Volume 1859, Issue 10, pp. 1259 - 1268
EBF1 | B cell | RNA isoform | Trascription factor | SLAMF | Transcriptional regulation | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Humans | Interferon Regulatory Factors - metabolism | NF-kappa B - metabolism | Sp1 Transcription Factor - metabolism | STAT6 Transcription Factor - genetics | Luciferases - genetics | Signaling Lymphocytic Activation Molecule Family Member 1 - genetics | Signaling Lymphocytic Activation Molecule Family Member 1 - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | HEK293 Cells | Trans-Activators - genetics | Transcription, Genetic | Nuclear Proteins - genetics | Binding Sites | B-Lymphocytes - metabolism | Genes, Reporter | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | B-Lymphocytes - cytology | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Gene Expression Regulation | Interferon Regulatory Factors - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | STAT6 Transcription Factor - metabolism | Transcription Factors - genetics | Transcription Factors - metabolism | Enhancer Elements, Genetic | NF-kappa B - genetics | Sp1 Transcription Factor - genetics | Cell Line, Tumor | Protein Binding | Trans-Activators - metabolism | Mutation | Primary Cell Culture | Autoimmunity | Promoters (Genetics) | Leukemia | Genes | Genetic aspects | Genetic transcription | B cells | DNA binding proteins | Cell differentiation
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5. Full Text The p65 subunit of NF-κB inhibits COL1A1 gene transcription in human dermal and scleroderma fibroblasts through its recruitment on promoter by protein interaction with transcriptional activators (c-Krox, Sp1, and Sp3)
by Beauchef, Gallic and Bigot, Nicolas and Kypriotou, Magdalini and Renard, Emmanuelle and Porée, Benoît and Widom, Russell and Dompmartin-Blanchere, Anne and Oddos, Thierry and Maquart, François-Xavier and Demoor, Magali and Boumediene, Karim and Galera, Philippe
The Journal of biological chemistry, ISSN 0021-9258, 01/2012, Volume 287, Issue 5, pp. 3462 - 3478
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Scleroderma, Localized - metabolism | Humans | Child, Preschool | Male | Sp1 Transcription Factor - metabolism | Transcription Factor RelA - genetics | DNA-Binding Proteins - metabolism | Collagen Type I - genetics | Adult | Transcription, Genetic | Child | Sp3 Transcription Factor - metabolism | Fibroblasts - metabolism | Dermis - metabolism | Response Elements | Gene Expression Regulation - genetics | Sp3 Transcription Factor - genetics | Scleroderma, Localized - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Fibroblasts - pathology | Transcription Factors - metabolism | Collagen Type I - biosynthesis | Sp1 Transcription Factor - genetics | Transcription Factor RelA - metabolism | Dermis - pathology | Index Medicus | Gene Regulation | Gene Transcription | Transcription Factors, Sp3, c-Krox | Sp1 | Collagen | Fibrosis | Type I Collagen | Skin | Scleroderma | Fibroblast | Nuclear Factor-κB
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6. Full Text Sp1 and krüppel‐like factor family of transcription factors in cell growth regulation and cancer
by Black, Adrian R and Black, Jennifer D and Azizkhan‐Clifford, Jane
Journal of cellular physiology, ISSN 0021-9541, 08/2001, Volume 188, Issue 2, pp. 143 - 160
Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | Humans | Repressor Proteins | Sp1 Transcription Factor - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Cell Division - physiology | DNA-Binding Proteins - metabolism | Kruppel-Like Transcription Factors | Transcription Factors - metabolism | Animals | Sp1 Transcription Factor - genetics | Neoplasms - physiopathology | Gene Expression Regulation, Neoplastic - physiology | Index Medicus
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7. Full Text Sp1 transcription factor: A long-standing target in cancer chemotherapy
by Vizcaíno, Carolina and Mansilla, Sylvia and Portugal, José
Pharmacology & therapeutics (Oxford), ISSN 0163-7258, 06/2015, Volume 152, pp. 111 - 124
Anticancer drugs | Transcription factors | Prognosis | Sp1 | Targeted therapy | Sp3 | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Neoplasms - metabolism | Gene Targeting - methods | Promoter Regions, Genetic | Humans | Gene Expression Regulation, Neoplastic | Sp1 Transcription Factor - metabolism | Antineoplastic Agents - administration & dosage | Neoplasms - therapy | Animals | Neoplasms - genetics | Sp1 Transcription Factor - genetics | Binding Sites - physiology | Drug Delivery Systems - methods | Index Medicus
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