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Cell reports (Cambridge), ISSN 2211-1247, 2016, Volume 16, Issue 10, pp. 2576 - 2592
.... Here, we describe the derivation and characterization, including single-cell RNA-seq, of neocortical and spinal cord neuroepithelial stem (NES... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
2016, Fourth edition., ISBN 9780781778176, xlii, 975 pages
"Neuroscience: Exploring the Brain surveys the organization and function of the human nervous system. We present material at the cutting edge of neuroscience... 
Spinal Cord | Brain | Neurosciences
Book
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 6, p. e38381
Background: Spinal cord injury (SCI), including immediate mechanical injury and secondary injury, is associated with the inflammatory response, apoptosis and oxidative stress in response to traumatic injury. Tanshinone IIA (TIIA... 
GLIAL APOPTOSIS | TUMOR-NECROSIS-FACTOR | CYTOKINES | INHIBITION | MULTIDISCIPLINARY SCIENCES | FACTOR-KAPPA-B | TRANSCRIPTION FACTOR | EXPRESSION | ASTROCYTES | FACTOR-ALPHA | CELL-DEATH | Inflammation - pathology | Spinal Cord - drug effects | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | Apoptosis - drug effects | Spinal Cord Injuries - complications | Aging - drug effects | Astrocytes - pathology | Male | NF-kappa B - metabolism | Spinal Cord Injuries - pathology | Inflammation - complications | Inflammation - drug therapy | Spinal Cord - pathology | Inflammation Mediators - metabolism | Neutrophil Infiltration - drug effects | Biomarkers - metabolism | Astrocytes - drug effects | Cytokines - metabolism | Wounds and Injuries - pathology | Diterpenes, Abietane - therapeutic use | Spinal Cord - enzymology | Wounds and Injuries - complications | Rats | Wounds and Injuries - physiopathology | Rats, Sprague-Dawley | Aging - pathology | Diterpenes, Abietane - pharmacology | Animals | MAP Kinase Signaling System - drug effects | Wounds and Injuries - drug therapy | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Oxidative Stress - drug effects | Inflammation - physiopathology | Nitric Oxide Synthase Type II - metabolism | Spinal cord injuries | Apoptosis | Oxidative stress | Spinal cord | Bcl-2 protein | Inflammatory response | Spinal cord injury | Kinases | Caspase-3 | Interleukin 6 | Pathways | Rodents | Tumor necrosis factor-TNF | Peroxidase | Inhibition | Redox properties | Edema | NF-κB protein | Damage assessment | Cytokines | Astrocytes | Caspase | MAP kinase | Pharmacology | Inflammation | Tumor necrosis factor-α | Trauma | Nitric-oxide synthase | Signaling | Injury prevention | Nitric oxide | Infiltration
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 1, pp. e30012 - 326
... from bisperoxovanadium treatment following spinal trauma. The goal of this study was to assess acute bisperoxovanadium treatment effects on neuroprotection and functional recovery following cervical unilateral contusive spinal cord injury, and investigate... 
SURVIVAL | PROTEIN-KINASE-B | TYROSINE-PHOSPHATASE INHIBITION | MULTIDISCIPLINARY SCIENCES | CONTUSION INJURY | PTEN | AKT | UP-REGULATION | EXPRESSION | BRAIN | CELL-DEATH | Neuroprotective Agents - therapeutic use | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | TOR Serine-Threonine Kinases - metabolism | Microtubule-Associated Proteins - metabolism | Cervical Vertebrae - blood supply | Protein Transport - drug effects | Autophagy - drug effects | Motor Neurons - pathology | Spinal Cord Injuries - pathology | Forelimb - physiopathology | Neuroprotective Agents - pharmacology | Spinal Cord Injuries - enzymology | Vanadium Compounds - pharmacology | Female | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Forelimb - drug effects | Motor Neurons - drug effects | Contusions - complications | Phagosomes - drug effects | Cervical Vertebrae - drug effects | Phagosomes - metabolism | Contusions - physiopathology | PTEN Phosphohydrolase - metabolism | Rats | Vanadium Compounds - therapeutic use | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Contusions - drug therapy | Animals | Signal Transduction - drug effects | Models, Biological | Cervical Vertebrae - pathology | Spinal Cord Injuries - physiopathology | Spinal cord injuries | Phosphatases | TOR protein | Neuroprotection | Neurosciences | Spinal cord | Vanadium compounds | Traumatic brain injury | Central nervous system | Vanadium | Recovery of function | Homology | AKT protein | Spinal cord injury | Kinases | Phosphatase | Autophagy | Western blotting | Proteins | Rodents | Cell cycle | Bisperoxovanadium | Tumorigenesis | Tensin | Hematology | Neurons | Rapamycin | Pharmacology | Functional analysis | Trauma | Medicine | Signaling | Injury prevention | Brain research | Insects | Immunofluorescence | Potassium | Histochemistry | Phagocytosis | Apoptosis
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2016, Volume 213, Issue 12, pp. 2603 - 2620
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2015, Volume 35, Issue 37, pp. 12733 - 12752
.... Acute DHA administration is neuroprotective and promotes functional recovery in animal models of adult spinal cord injury (SCI... 
Neuroplasticity | Docosahexaenoic acid | Spinal cord injury | Omega-3 polyunsaturated fatty acids | spinal cord injury | NERVOUS-SYSTEM | PTEN INHIBITION | SYNAPTIC PLASTICITY | NEUROSCIENCES | omega-3 polyunsaturated fatty acids | IN-VITRO | TRAUMATIC BRAIN-INJURY | ADULT-RAT | docosahexaenoic acid | CORTICOSPINAL TRACT | POLYUNSATURATED FATTY-ACIDS | PTEN/MTOR PATHWAY | EXPRESSION | neuroplasticity | Pyramidal Tracts - drug effects | Nerve Regeneration - physiology | Neuronal Plasticity - drug effects | Serotonergic Neurons - ultrastructure | Neuronal Plasticity - physiology | Neuroprotective Agents - pharmacology | Neurites - ultrastructure | Pyramidal Tracts - injuries | Exploratory Behavior - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Pyramidal Tracts - pathology | Neurites - drug effects | Docosahexaenoic Acids - therapeutic use | PTEN Phosphohydrolase - genetics | Cervical Vertebrae | Motor Neurons - physiology | Serotonergic Neurons - physiology | PTEN Phosphohydrolase - biosynthesis | Rats | MicroRNAs - biosynthesis | Rats, Sprague-Dawley | Spinal Cord - physiology | Nerve Regeneration - drug effects | Mice | MicroRNAs - genetics | Gait Disorders, Neurologic - etiology | Neuroprotective Agents - therapeutic use | Interneurons - physiology | Injections, Intravenous | Spinal Cord - drug effects | Spinal Cord Injuries - drug therapy | Spinal Cord Injuries - complications | Protein Processing, Post-Translational - drug effects | Female | Drug Evaluation, Preclinical | Neuroprotective Agents - administration & dosage | Pyramidal Tracts - physiology | Motor Neurons - drug effects | Cells, Cultured | Interneurons - drug effects | Docosahexaenoic Acids - administration & dosage | Nerve Tissue Proteins - genetics | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Docosahexaenoic Acids - pharmacology | Animals | Gait Disorders, Neurologic - drug therapy | Spinal Cord Injuries - physiopathology
Journal Article
Acta biomaterialia, ISSN 1742-7061, 2016, Volume 42, pp. 77 - 89
[Display omitted] The hostile environment after spinal cord injury (SCI) can compromise effects of regenerative therapies... 
Functional recovery | Self-assembling peptides | Tissue-preservation | Neural precursor cells | Cervical spinal cord injury | SCAR FORMATION | MATERIALS SCIENCE, BIOMATERIALS | NEUROLOGICAL RECOVERY | ENGINEERING, BIOMEDICAL | REGENERATION | TRANSPLANTATION | REPAIR | ADULT-RAT | GROWTH | STEM/PROGENITOR CELLS | PRECURSOR CELLS | Spinal Cord Injuries - drug therapy | Rats, Wistar | Pyramidal Tracts - drug effects | Cervical Cord - drug effects | Synapses - pathology | Neural Stem Cells - cytology | Choline O-Acetyltransferase - metabolism | Cervical Cord - physiopathology | Spinal Cord Injuries - pathology | Gliosis - pathology | Stem Cell Transplantation | Neural Stem Cells - transplantation | Behavior, Animal - drug effects | Biotin - metabolism | Female | Spinal Cord Injuries - therapy | Cicatrix - complications | Pyramidal Tracts - pathology | Motor Neurons - drug effects | Dextrans - metabolism | Cell Survival - drug effects | Synapses - drug effects | Cervical Cord - pathology | Hyperalgesia - complications | Neural Stem Cells - drug effects | Wounds and Injuries - complications | Wounds and Injuries - physiopathology | Gliosis - complications | Hyperalgesia - physiopathology | Peptides - pharmacology | Animals | Hyperalgesia - therapy | Cell Differentiation - drug effects | Wounds and Injuries - drug therapy | Cicatrix - pathology | Spinal Cord Injuries - physiopathology | Microscopy, Fluorescence | Peptides - therapeutic use | Biotin - analogs & derivatives | Transplantation | Spinal cord injuries | Peptides | Stem cells | Regenerative | Therapy | Spinal cord | Cysts | Differentiation | Injuries
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 2016, Volume 20, Issue 6, pp. 1062 - 1075
After spinal cord injury (SCI), disruption of blood–spinal cord barrier (BSCB) elicits blood cell infiltration such as neutrophils and macrophages, contributing to permanent neurological disability... 
spinal cord injury | Rac1 | blood–spinal cord barrier | epidermal growth factor | PI3K/Akt | Blood-spinal cord barrier | Epidermal growth factor | Spinal cord injury | MEDICINE, RESEARCH & EXPERIMENTAL | FUNCTIONAL RECOVERY | blood-spinal cord barrier | ACTIVATION | MODEL | RHOA | CELL BIOLOGY | BRAIN-BARRIER | ENDOTHELIAL-CELLS | MEK/ERK | ISCHEMIA-REPERFUSION | EXPRESSION | Epidermal Growth Factor - administration & dosage | Spinal Cord - drug effects | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Epidermal Growth Factor - therapeutic use | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Adherens Junctions - metabolism | Proteolysis - drug effects | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Female | Spinal Cord Injuries - blood | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Tight Junctions - drug effects | Tight Junctions - metabolism | Oxygen | Endothelial Cells - metabolism | Morpholines - pharmacology | Rats, Sprague-Dawley | Glucose - deficiency | Animals | Signal Transduction - drug effects | Adherens Junctions - drug effects | Epidermal Growth Factor - pharmacology | rac1 GTP-Binding Protein - metabolism | Endothelial Cells - drug effects | Spinal cord injuries | Permeability | Analysis | PI3K | Akt | Original
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 02/2018, Volume 22, Issue 2, pp. 1148 - 1166
Spinal cord injury (SCI) is a severe neurological disease; however, few drugs have been proved to treat SCI effectively... 
spinal cord injury | autophagic flux | salidroside | AMPK | M2 polarization | microglia | M1 polarization | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | OXIDATIVE STRESS | M2 MACROPHAGES | ACTIVATION | PROMOTES LOCOMOTOR RECOVERY | CELL BIOLOGY | RESPONSES | ADOPTIVE TRANSFER | INDUCED AUTOPHAGY | MACROPHAGE POLARIZATION | MEDIATED NEUROTOXICITY | Neurons - pathology | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Spinal Cord Injuries - complications | Motor Activity - drug effects | Autophagy - drug effects | Lipopolysaccharides | Spinal Cord Injuries - pathology | Inflammation - complications | Inflammation - drug therapy | Adenylate Kinase - metabolism | Inflammation Mediators - metabolism | Microglia - pathology | Female | Glucosides - therapeutic use | Cell Polarity - drug effects | Neurons - metabolism | Neurons - drug effects | Phenols - pharmacology | Cell Line | Glucosides - pharmacology | Macrophages - pathology | Microglia - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Rats, Sprague-Dawley | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Models, Biological | Phenols - therapeutic use | Macrophages - drug effects | Mice | Spinal Cord Injuries - physiopathology | Nervous system diseases | Neurons | Analysis | Therapeutics | Spinal cord injuries | Homeopathy | Materia medica and therapeutics | TOR protein | Drugs | Polarization | Spinal cord | Pathogenesis | Recovery of function | Rats | Inflammation | Spinal cord injury | Drug development | Autophagy | Polarization modulation | Microglia | Respiratory tract | Salidroside | Blood-brain barrier | Rodents | Pheochromocytoma cells | Immunofluorescence | Injuries | Apoptosis | Original
Journal Article