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Molecular Neurobiology, ISSN 0893-7648, 3/2018, Volume 55, Issue 3, pp. 2577 - 2590
After spinal cord injury (SCI), tight junction (TJ) protein degradation increases permeability and disrupts the blood... 
Neurology | Neurosciences | Biomedicine | Tight junctions | Blood–spinal cord barrier | Neurobiology | Neuroinflammation | MMP-8 | Spinal cord injury | Cell Biology | Occludin | FUNCTIONAL RECOVERY | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | NEUTROPHIL INFILTRATION | VASCULAR EVENTS | NEUROSCIENCES | BRAIN-BARRIER | CELL JUNCTIONS | MESSENGER-RNA | Blood-spinal cord barrier | NITRIC-OXIDE | HEME OXYGENASE-1 | CEREBRAL-ISCHEMIA | Inflammation - pathology | Spinal Cord - drug effects | Spinal Cord - enzymology | Rats | Tight Junctions - enzymology | Blood-Brain Barrier - drug effects | Rats, Sprague-Dawley | Matrix Metalloproteinase Inhibitors - therapeutic use | Spinal Cord Injuries - pathology | Dose-Response Relationship, Drug | Spinal Cord Injuries - prevention & control | Blood-Brain Barrier - pathology | Matrix Metalloproteinase Inhibitors - pharmacology | Animals | Blood-Brain Barrier - enzymology | Spinal Cord Injuries - enzymology | Inflammation - drug therapy | Matrix Metalloproteinase 8 - metabolism | Spinal Cord - pathology | Female | Inflammation - enzymology | Tight Junctions - pathology | Disease Models, Animal | Tight Junctions - drug effects | Prevention | Immunohistochemistry | Cysteine | Proteolysis | Surgery | Nervous system | Spinal cord injuries | Inflammation | Permeability | Recombinant proteins | Endothelium | Compression | Matrix metalloproteinase | Blood | Western blotting | Degradation | Interleukin 6 | Proteins | Antioxidants | Cell activation | Extravasation | Rodents | Metalloproteinase | Neutrophils | Leukocytes (neutrophilic) | Tumor necrosis factor-α | Zonula occludens-1 protein | Nitric-oxide synthase | Endothelial cells | Cell injury | Sodium | Neutrophil collagenase | Protein expression | Infiltration | N-Acetyl-L-cysteine
Journal Article
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 2017, Volume 216, Issue 8, pp. 2533 - 2550
Understanding how the transcription factor signal transducer and activator of transcription-3 (STAT3) controls glial scar formation may have important clinical... 
PROTEIN | MEDIATED RECOMBINATION | PTEN | CANCER-CELL INVASION | REACTIVE ASTROCYTES | SPINAL-CORD-INJURY | CENTRAL-NERVOUS-SYSTEM | MATRIX METALLOPROTEINASE-2 | FOCAL ADHESION KINASE | TRANSGENIC MICE | CELL BIOLOGY | Phosphorylation | Cytoskeletal Proteins - genetics | Neuroglia - pathology | Coculture Techniques | Astrocytes - pathology | MicroRNAs - metabolism | Spinal Cord Injuries - genetics | Astrocytes - enzymology | Wounds, Stab - pathology | Spinal Cord Injuries - pathology | Transfection | Spinal Cord Injuries - enzymology | rho-Associated Kinases - metabolism | Proteolysis | Spinal Cord - pathology | Wounds, Stab - enzymology | Nestin - genetics | STAT3 Transcription Factor - deficiency | Cytoskeletal Proteins - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Animals, Newborn | PTEN Phosphohydrolase - genetics | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | rho GTP-Binding Proteins - genetics | Mice, Inbred C57BL | Neuroglia - enzymology | Cells, Cultured | Spinal Cord - enzymology | PTEN Phosphohydrolase - metabolism | Genotype | Actomyosin - metabolism | Cell Adhesion | Mice, Knockout | Cicatrix - genetics | Matrix Metalloproteinase 2 - genetics | Macrophages - metabolism | Phenotype | Animals | rho GTP-Binding Proteins - metabolism | Cicatrix - enzymology | Cicatrix - pathology | MicroRNAs - genetics | Wounds, Stab - genetics | Integrases - genetics | Cell Movement | Development and progression | Genetic aspects | Gene expression | Health aspects | Neuroglia | Cicatrices
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 12/2013, Volume 127, Issue 6, pp. 880 - 890
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2003, Volume 23, Issue 10, pp. 4017 - 4022
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 04/2006, Volume 26, Issue 16, pp. 4406 - 4414
Journal Article
Nature Neuroscience, ISSN 1097-6256, 03/2008, Volume 11, Issue 3, pp. 251 - 253
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0152750
...). Inhibition of matrix metalloproteinases (MMPs) and maintaining a neurotrophic support could represent two approaches to prevent or reduce the maladaptive plastic changes in the ventral horn of spinal cord following PNI... 
NEUROPATHIC PAIN | ASTROGLIOSIS | REGENERATION | MULTIDISCIPLINARY SCIENCES | DISEASE | EXTRACELLULAR-MATRIX | AMYOTROPHIC-LATERAL-SCLEROSIS | GROWTH-FACTOR | MODEL | EXPRESSION | MALADAPTIVE PLASTICITY | Glutamate Plasma Membrane Transport Proteins - metabolism | Glycine Plasma Membrane Transport Proteins - metabolism | Sciatic Nerve - injuries | gamma-Aminobutyric Acid - metabolism | Astrocytes - pathology | Male | Astrocytes - enzymology | Synapses - pathology | Anterior Horn Cells - enzymology | Spinal Cord - pathology | Gelatinases - metabolism | Microglia - pathology | Gelatinases - antagonists & inhibitors | Sciatic Nerve - pathology | Dipeptides - pharmacology | Anterior Horn Cells - pathology | Peripheral Nerve Injuries - pathology | Spinal Cord - enzymology | Microglia - enzymology | Rats | Synapses - enzymology | Rats, Sprague-Dawley | Animals | Peripheral Nerve Injuries - enzymology | Glutamic Acid - metabolism | Sciatic Nerve - enzymology | Care and treatment | Proteases | Physiological aspects | Extracellular matrix | Genetic aspects | Research | Peripheral nerve diseases | Ventral horn | Spinal cord | Matrix metalloproteinases | Central nervous system | Homeostasis | Nerve growth factor | Spinal cord injury | Glycine | Neuronal-glial interactions | Infusion | Pain | Motor neuron diseases | Rodents | Modulation | Inhibition | Paralysis | Enzymes | Cytokines | Neurons | Glial fibrillary acidic protein | Amyotrophic lateral sclerosis | Liquid chromatography | High-performance liquid chromatography | Microglia | Injury prevention | Gliosis | Plasticity (synaptic) | Glycine transporter | Correlation analysis | Spinal cord injuries | Glutamic acid transporter | Transporter | Sodium-glutamic acid cotransporter | Sciatic nerve
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2008, Volume 107, Issue 1, pp. 50 - 60
.... We investigated the possible activation of this major IL‐6 signaling system in the spinal cord after peripheral nerve injury and its role in neuropathic pain. Ligation of L5–L6 spinal nerves (SNL... 
spinal cord microglia | neuropathic pain | peripheral nerve injury | Janus kinase/signal transducer and activator of transcription 3 | Peripheral nerve injury | Spinal cord microglia | Neuropathic pain | MECHANICAL ALLODYNIA | BIOCHEMISTRY & MOLECULAR BIOLOGY | LEUKEMIA INHIBITORY FACTOR | INTERLEUKIN-6 | NEUROSCIENCES | IL-6 | FAMILY | STAT3 PHOSPHORYLATION | TRANSCRIPTION-3 | GLIA | POTENTIAL ROLE | SIGNAL TRANSDUCER | Microglia - metabolism | Phosphorylation | Spinal Cord - metabolism | Spinal Nerves - surgery | Male | Peripheral Nervous System Diseases - physiopathology | Janus Kinase 1 - metabolism | Ligation | Janus Kinase 2 - metabolism | Up-Regulation - physiology | Interleukin-6 - metabolism | Janus Kinase 2 - antagonists & inhibitors | STAT3 Transcription Factor - metabolism | Spinal Nerves - injuries | Disease Models, Animal | Spinal Nerves - physiopathology | Interleukin-6 - genetics | Spinal Cord - enzymology | Microglia - enzymology | Peripheral Nervous System Diseases - enzymology | Rats | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Peripheral Nerves - physiopathology | Antibodies - pharmacology | Peripheral Nervous System Diseases - metabolism | Peripheral Nerves - enzymology | Animals | Interleukin-6 - immunology | Signal Transduction - physiology | Spinal Cord - physiopathology | Enzyme Activation - physiology | Hyperalgesia - enzymology | Neurology | Signal transduction | Biochemistry | Cytokines | Ribonucleic acid--RNA
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 03/2008, Volume 25, Issue 3, pp. 184 - 195
...) dysfunction after central nervous system injury. Oxidative stress is also implicated in the pathogenesis after cerebral ischemia and spinal cord injury (SCI... 
Copper/zinc-superoxide dismutase | Endothelial | Bloodbrain barrier | Spinal cord injury | MMP-9 | blood-brain barrier | copper/zinc-superoxide dismutase | spinal cord injury | FUNCTIONAL RECOVERY | SUPEROXIDE | ACTIVATION | REPERFUSION | PROTEOLYSIS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | MATRIX METALLOPROTEINASES | OVEREXPRESSION | endothelial | MATRIX-METALLOPROTEINASE-9 | NEURONS | CRITICAL CARE MEDICINE | Superoxide Dismutase - genetics | Apoptosis - drug effects | Humans | Blood-Brain Barrier - physiopathology | Nerve Degeneration - physiopathology | Nerve Degeneration - genetics | Spinal Cord Injuries - genetics | Evans Blue - pharmacokinetics | Microcirculation - physiopathology | Matrix Metalloproteinase 9 - metabolism | Spinal Cord Injuries - enzymology | Matrix Metalloproteinase 9 - genetics | Female | Superoxide Dismutase - metabolism | Disease Models, Animal | Nerve Degeneration - enzymology | Animals, Genetically Modified | Enzyme Inhibitors - pharmacology | Oxidative Stress - genetics | Spinal Cord - enzymology | Rats | Up-Regulation - genetics | Spinal Cord - blood supply | Rats, Sprague-Dawley | Animals | Blood-Brain Barrier - enzymology | Microcirculation - pathology | Gene Expression Regulation, Enzymologic - genetics | Microcirculation - enzymology | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Apoptosis - physiology | Superoxide Dismutase-1 | Endothelial Cells - pathology | Endothelial Cells - enzymology | Oxidative stress | Care and treatment | Spinal cord injuries | Genetic aspects | Research | Gene expression | Health aspects | Methods | Risk factors | Neurosciences | Rodents | Brain damage | Oxidation | Stress | copper | zinc-superoxide dismutase
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, p. e38381
Background: Spinal cord injury (SCI), including immediate mechanical injury and secondary injury, is associated with the inflammatory response, apoptosis and oxidative stress in response to traumatic injury. Tanshinone IIA (TIIA... 
GLIAL APOPTOSIS | TUMOR-NECROSIS-FACTOR | CYTOKINES | INHIBITION | MULTIDISCIPLINARY SCIENCES | FACTOR-KAPPA-B | TRANSCRIPTION FACTOR | EXPRESSION | ASTROCYTES | FACTOR-ALPHA | CELL-DEATH | Inflammation - pathology | Spinal Cord - drug effects | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | Apoptosis - drug effects | Spinal Cord Injuries - complications | Aging - drug effects | Astrocytes - pathology | Male | NF-kappa B - metabolism | Spinal Cord Injuries - pathology | Inflammation - complications | Inflammation - drug therapy | Spinal Cord - pathology | Inflammation Mediators - metabolism | Neutrophil Infiltration - drug effects | Biomarkers - metabolism | Astrocytes - drug effects | Cytokines - metabolism | Wounds and Injuries - pathology | Diterpenes, Abietane - therapeutic use | Spinal Cord - enzymology | Wounds and Injuries - complications | Rats | Wounds and Injuries - physiopathology | Rats, Sprague-Dawley | Aging - pathology | Diterpenes, Abietane - pharmacology | Animals | MAP Kinase Signaling System - drug effects | Wounds and Injuries - drug therapy | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Oxidative Stress - drug effects | Inflammation - physiopathology | Nitric Oxide Synthase Type II - metabolism | Spinal cord injuries | Apoptosis | Oxidative stress | Spinal cord | Bcl-2 protein | Inflammatory response | Spinal cord injury | Kinases | Caspase-3 | Interleukin 6 | Pathways | Rodents | Tumor necrosis factor-TNF | Peroxidase | Inhibition | Redox properties | Edema | NF-κB protein | Damage assessment | Cytokines | Astrocytes | Caspase | MAP kinase | Pharmacology | Inflammation | Tumor necrosis factor-α | Trauma | Nitric-oxide synthase | Signaling | Injury prevention | Nitric oxide | Infiltration
Journal Article