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Seminars in immunopathology, ISSN 1863-2300, 10/2011, Volume 34, Issue 1, pp. 43 - 62
... of inflammatory human diseases. In particular, a proinflammatory role for fibrinogen has been reported in vascular wall disease, stroke, spinal cord injury, brain trauma... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | Pathology | Life Sciences & Biomedicine | Science & Technology | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Index Medicus | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 09/2014, Volume 83, Issue 5, pp. 1098 - 1116
Journal Article
American journal of physiology: endocrinology and metabolism, ISSN 1522-1555, 05/2016, Volume 310, Issue 9, pp. E754 - E761
Individuals with long-standing spinal cord injury (SCI) often present with extreme muscle atrophy and impaired glucose metabolism at both the skeletal muscle and whole body level... 
Nuclear factor κ-light-chainenhancer of activated B cells | TNF-like weak inducer of apoptosis | Endocrinology & Metabolism | Physiology | Life Sciences & Biomedicine | Science & Technology | Tumor Necrosis Factors - genetics | SKP Cullin F-Box Protein Ligases - genetics | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Transcriptome | Immunoblotting | Male | Muscle, Skeletal - metabolism | NF-kappa B - metabolism | TWEAK Receptor | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Spinal Cord Injuries - pathology | Adult | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Interleukin-6 - genetics | Muscular Atrophy - metabolism | NF-kappa B p50 Subunit - metabolism | Signal Transduction | Spinal Cord Injuries - metabolism | Receptors, Interleukin-6 - genetics | Muscular Atrophy - pathology | Tumor Necrosis Factors - metabolism | Organ Size | Tripartite Motif Proteins - genetics | Inflammation | Reverse Transcriptase Polymerase Chain Reaction | Muscle Proteins - genetics | Cytokine TWEAK | Transcription Factor RelA - metabolism | Fibrosis | Muscle Fibers, Skeletal - pathology | Muscle, Skeletal - pathology | Ubiquitin-Protein Ligases - genetics | Chronic Disease | Index Medicus | nuclear factor κ-light-chain-enhancer of activated B cells
Journal Article
PloS one, ISSN 1932-6203, 12/2017, Volume 12, Issue 12, pp. e0189151 - e0189151
Sensorimotor dysfunction following incomplete spinal cord injury (SCI) is often characterized by paralysis, spasticity and pain... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Albumins - chemistry | Tenascin - agonists | Recovery of Function - drug effects | Male | Pain - metabolism | Tenascin - metabolism | Spinal Cord Injuries - pathology | Pain - genetics | Prostaglandin-E Synthases - antagonists & inhibitors | Prostaglandin-E Synthases - genetics | Nociception - drug effects | Recovery of Function - physiology | Growth Differentiation Factor 10 - agonists | Locomotion - drug effects | Prostaglandin-E Synthases - metabolism | Extracellular Matrix Proteins - genetics | Nerve Tissue Proteins - agonists | Rats | Pain - pathology | Extracellular Matrix Proteins - agonists | Locomotion - physiology | Spinal Cord - drug effects | Spinal Cord - metabolism | Spinal Cord Injuries - drug therapy | Tenascin - genetics | Paralysis - drug therapy | Rats, Wistar | Spinal Cord Injuries - genetics | Paralysis - pathology | Paralysis - metabolism | Phospholipases - genetics | Growth Differentiation Factor 10 - metabolism | Spinal Cord - pathology | Oleic Acids - pharmacology | Extracellular Matrix Proteins - metabolism | Injections, Spinal | Drug Administration Schedule | Pain - prevention & control | Spinal Cord Injuries - metabolism | Gene Expression Regulation | Treatment Outcome | Paralysis - genetics | Nerve Tissue Proteins - genetics | Phospholipases - antagonists & inhibitors | Nerve Tissue Proteins - metabolism | Rotarod Performance Test | Albumins - pharmacology | Animals | Phospholipases - metabolism | Growth Differentiation Factor 10 - genetics | Oleic Acids - chemistry | Monounsaturated fatty acids | Care and treatment | DNA microarrays | Spinal cord injuries | Research | Diagnosis | Gene expression | Tenascin | Spinal cord | Laboratories | Genes | Recovery of function | Lipids | Spinal cord injury | Morphogenesis | Pain | Oleic acid | Rodents | Sensorimotor system | Anxiety | Paralysis | Prostaglandin E | Injuries | Hypertension | Aspirin | Pain perception | Cell survival | Tenascin C | Albumin | Spasticity | Fatty acids | Polymerase chain reaction | Acids | Index Medicus
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 10/2011, Volume 31, Issue 42, pp. 15173 - 15187