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/ccl17 (1) 1
anti-inflammatory agents, non-steroidal - pharmacology (1) 1
atopic-dermatitis (1) 1
biochemistry & molecular biology (1) 1
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chemokine ccl17 - antagonists & inhibitors (1) 1
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proto-oncogene proteins c-raf - metabolism (1) 1
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1. Full Text Suppression of thymus- and activation-regulated chemokine (TARC/CCL17) production by 3-O-β-d-glucopyanosylspinasterol via blocking NF-κB and STAT1 signaling pathways in TNF-α and IFN-γ-induced HaCaT keratinocytes
by Jung, Mi-ra and Lee, Tae Hoon and Bang, Myun-Ho and Kim, Jiyoung and Kim, Hakwon and Son, Youngsook and Chung, Dae Kyun
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 10/2012, Volume 427, Issue 2, pp. 236 - 241
► Spinasterol-Glc suppressed expression of CCL17 induced by TNF-α and IFN-γ in HaCaT cells. ► Activation of c-Raf-1, p38 MAPK, JAK2, and IKK was attenuated by... 
Inflammation | TARC/CCL17 | Spinasterol-glucose | CELLS | CYTOKINES | MDC/CCL22 | MAP KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | (TARC)/CCL17 | ATOPIC-DERMATITIS | BIOPHYSICS | SPINASTEROL | SKIN | KOREANA | Cell Line | NF-kappa B - antagonists & inhibitors | Humans | Stigmasterol - pharmacology | Chemokine CCL17 - antagonists & inhibitors | STAT1 Transcription Factor - antagonists & inhibitors | Proto-Oncogene Proteins c-raf - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Dermatitis, Atopic - drug therapy | Keratinocytes - drug effects | Signal Transduction - drug effects | Chemokine CCL17 - biosynthesis | Keratinocytes - metabolism | Janus Kinase 2 - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Stigmasterol - analogs & derivatives | Dermatitis, Atopic - metabolism | Interferon-gamma - pharmacology | Dermatologic Agents - pharmacology
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