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1. Full Text L-DOPA Treatment Selectively Restores Spine Density in Dopamine Receptor D2–Expressing Projection Neurons in Dyskinetic Mice
by Suárez, Luz M and Solís, Oscar and Caramés, Jose M and Taravini, Irene R and Solís, Jose M and Murer, Mario G and Moratalla, Rosario
Biological psychiatry (1969), ISSN 0006-3223, 2014, Volume 75, Issue 9, pp. 711 - 722
Psychiatry | three-dimensional neuronal reconstruction | medium spiny neuron | striatum | Behavioral sensitization | L-DOPA | dyskinesia | Parkinson’s disease | Parkinson's disease | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurology | Organic mental disorders. Neuropsychology | Psychology. Psychoanalysis. Psychiatry | Adult and adolescent clinical studies | Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases | Psychopathology. Psychiatry | Biological and medical sciences | Nervous system as a whole | Medical sciences | Nervous system (semeiology, syndromes) | Neurons - pathology | Dyskinesia, Drug-Induced - physiopathology | Cerebral Cortex - pathology | Male | Oxidopamine | Receptors, Dopamine D2 - metabolism | Cerebral Cortex - physiopathology | Receptors, Dopamine D1 - metabolism | Antiparkinson Agents - therapeutic use | Dendritic Spines - drug effects | Parkinsonian Disorders - drug therapy | Neurons - physiology | Receptors, Dopamine D1 - agonists | Cerebral Cortex - drug effects | Neurons - drug effects | Corpus Striatum - pathology | Membrane Potentials - drug effects | Dopamine Agonists - pharmacology | Parkinsonian Disorders - physiopathology | Corpus Striatum - physiopathology | Levodopa - therapeutic use | Mice, Inbred C57BL | Dendritic Spines - physiology | Proto-Oncogene Proteins c-fos - metabolism | Mice, Transgenic | Dyskinesia, Drug-Induced - pathology | Membrane Potentials - physiology | Antiparkinson Agents - adverse effects | Animals | Parkinsonian Disorders - pathology | Dendritic Spines - pathology | Levodopa - adverse effects | 2,3,4,5-Tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine - pharmacology | Corpus Striatum - drug effects | Phenols | Dopamine | Dopa | Neurons | Movement disorders | Resveratrol | Index Medicus
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2. Full Text Physiology and pharmacology of striatal neurons
by Kreitzer, Anatol C
Annual Review of Neuroscience, ISSN 0147-006X, 06/2009, Volume 32, Issue 1, pp. 127 - 147
Medium spiny neuron | Basal ganglia | Acetylcholine | Dopamine | Interneuron | Fundamental and applied biological sciences. Psychology | Vertebrates: nervous system and sense organs | Biological and medical sciences | General aspects. Models. Methods | Neuropil - physiology | Humans | Neurotransmitter Agents - metabolism | Neurons - cytology | Neuropeptides - metabolism | Corpus Striatum - cytology | Neural Pathways - physiology | Action Potentials - physiology | Neuropil - ultrastructure | Basal Ganglia Diseases - pathology | Animals | Interneurons - metabolism | Basal Ganglia Diseases - physiopathology | Neural Pathways - cytology | Corpus Striatum - physiology | Interneurons - cytology | Neurons - metabolism | Physiological aspects | Motor neurons | Basal forebrain | Research | Health aspects | Learning | Brain | Pharmacology | Motivation | Neurons | Motor ability | Index Medicus
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3. Full Text Striatal neurons directly converted from Huntington's disease patient fibroblasts recapitulate age-associated disease phenotypes
by Victor, Matheus B and Richner, Michelle and Olsen, Hannah E and Lee, Seong Won and Monteys, Alejandro M and Ma, Chunyu and Huh, Christine J and Zhang, Bo and Davidson, Beverly L and Yang, X. William and Yoo, Andrew S
Nature neuroscience, ISSN 1097-6256, 03/2018, Volume 21, Issue 3, pp. 341 - 352
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Mitochondrial Diseases - pathology | Neostriatum - pathology | Neurons - pathology | Neurodegenerative Diseases - pathology | Oxidative Stress | Humans | Huntington Disease - pathology | Gene Expression Profiling | Fibroblasts - pathology | Aging - pathology | Phenotype | Pluripotent Stem Cells | MicroRNAs - genetics | DNA Damage | Huntingtin Protein - genetics | Huntingtin Protein - biosynthesis | Excitatory Postsynaptic Potentials - genetics | Cell culture | Brain research | Huntington's chorea | Age factors in disease | Neurons | Fibroblasts | Development and progression | Genetic aspects | Research | Huntingtons disease | Huntingtin | DNA damage | Mitochondrial DNA | Neostriatum | miRNA | Modelling | Degeneration | Age | Deoxyribonucleic acid--DNA | Phenotypes | Polyglutamine | Trinucleotide repeats | Ribonucleic acid--RNA | Huntington's disease | Spiny neurons | Conversion | Neurological diseases | Aggregates | Cell death | Stem cells | Codons | Pluripotency | Index Medicus
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4. Full Text Manifestation of Huntington's disease pathology in human induced pluripotent stem cell-derived neurons
by Nekrasov, Evgeny D and Vigont, Vladimir A and Klyushnikov, Sergey A and Lebedeva, Olga S and Vassina, Ekaterina M and Bogomazova, Alexandra N and Chestkov, Ilya V and Semashko, Tatiana A and Kiseleva, Elena and Suldina, Lyubov A and Bobrovsky, Pavel A and Zimina, Olga A and Ryazantseva, Maria A and Skopin, Anton Yu and Illarioshkin, Sergey N and Kaznacheyeva, Elena V and Lagarkova, Maria A and Kiselev, Sergey L
Molecular neurodegeneration, ISSN 1750-1326, 04/2016, Volume 11, Issue 1, pp. 27 - 27
Neuroprotection | Nuclear indentations | Neurodegeneration | Store-operated calcium entry | Aging | Human induced pluripotent stem cells | GABAergic medium spiny neurons | Differentiation | Huntington's disease | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cell Line | Lysosomes - metabolism | Calcium - metabolism | Humans | Huntington Disease - pathology | Mutant Proteins - metabolism | Neurons - cytology | Nuclear Proteins - metabolism | Cell Differentiation | Induced Pluripotent Stem Cells - cytology | Corpus Striatum - metabolism | Huntington Disease - metabolism | Complications and side effects | Stem cell research | Care and treatment | Huntington's chorea | GABA | Influence | Research | Index Medicus
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5. GABAergic Neurons from Mouse Embryonic Stem Cells Possess Functional Properties of Striatal Neurons In Vitro, and Develop into Striatal Neurons In Vivo in a Mouse Model of Huntington's Disease
by Shin, Eunju and Palmer, Mary J and Li, Meng and Fricker, Rosemary A
Stem cell reviews, ISSN 1550-8943, 06/2012, Volume 8, Issue 2, pp. 513 - 531
Glutamate receptors | Mouse | Medium spiny neurons | Electrophysiology | Embryonic stem cell | Neuronal differentiation | Action potential | DARPP-32 | Transplantation | Quinolinic acid lesion | Cell & Tissue Engineering | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | GABAergic Neurons - transplantation | Immunohistochemistry | Receptors, Glutamate - metabolism | Synaptic Potentials | Embryonic Stem Cells - metabolism | Embryonic Stem Cells - cytology | Neostriatum - cytology | Huntington Disease - pathology | Neurogenesis | Huntington Disease - therapy | Stem Cell Transplantation | Patch-Clamp Techniques | Animals | GABAergic Neurons - cytology | Neostriatum - metabolism | Cell Differentiation | Mice | Electrophysiological Phenomena | Huntington Disease - physiopathology | Disease Models, Animal | gamma -Aminobutyric acid | Animal models | Neurodegenerative diseases | Embryo cells | Glutamate decarboxylase | Huntington's disease | Grafting | Phosphoproteins | Neostriatum | Stem cells | puromycin | Neural stem cells | Sodium channels (voltage-gated) | Potassium channels (voltage-gated) | Receptor mechanisms | Differentiation
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6. Full Text Differential vulnerability of neurons in Huntington’s disease: the role of cell type‐specific features
by Han, Ina and You, YiMei and Kordower, Jeffrey H and Brady, Scott T and Morfini, Gerardo A
Journal of neurochemistry, ISSN 0022-3042, 06/2010, Volume 113, Issue 5, pp. 1073 - 1091
axonal transport | dying back degeneration | Huntington’s disease | medium‐sized spiny neurons | huntingtin | Medium-sized spiny neurons | Dying back degeneration | Huntingtin | Axonal transport | Huntington's disease | Biochemistry & Molecular Biology | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurology | Organic mental disorders. Neuropsychology | Psychology. Psychoanalysis. Psychiatry | Adult and adolescent clinical studies | Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases | Psychopathology. Psychiatry | Biological and medical sciences | Medical sciences | Brain-Derived Neurotrophic Factor - genetics | Neurons - pathology | Signal Transduction | Gene Expression - genetics | Humans | Huntington Disease - pathology | Axonal Transport - physiology | Mitochondria - pathology | Nerve Tissue Proteins - genetics | Nerve Degeneration - pathology | Nuclear Proteins - toxicity | Brain Chemistry - physiology | Huntington Disease - etiology | Huntingtin Protein | Mutation - physiology | Nerve Tissue Proteins - toxicity | Neurons - classification | Brain-Derived Neurotrophic Factor - physiology | Gene Expression - physiology | Brain - pathology | Huntington Disease - genetics | Nuclear Proteins - genetics | Nervous system diseases | Disease susceptibility | Universities and colleges | Huntington's chorea | Neurons | Huntingtons disease | Cells | Index Medicus | medium-sized spiny neurons
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