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Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2012, Volume 143, Issue 3, pp. 646 - 654
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 1, p. e0141159
Background Spleen enlargement is often detected in patients with liver cirrhosis, but the precise pathogenetic mechanisms behind the phenomenon have not been... 
MAMMALIAN TARGET | FIBROSIS | LIVER-CIRRHOSIS | SPLEEN | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | HYPERSPLENISM | MECHANISMS | HYPERDYNAMIC SPLANCHNIC CIRCULATION | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Splenomegaly - drug therapy | TOR Serine-Threonine Kinases - metabolism | Hypertension, Portal - complications | Humans | Middle Aged | Male | Spleen - drug effects | Splenomegaly - pathology | Neovascularization, Pathologic - pathology | Portal Pressure - drug effects | TOR Serine-Threonine Kinases - antagonists & inhibitors | Splenomegaly - complications | Female | Neovascularization, Pathologic - prevention & control | Spleen - pathology | Sirolimus - therapeutic use | Hypertension, Portal - pathology | Anti-Infective Agents - therapeutic use | Rats | Splenomegaly - metabolism | Rats, Sprague-Dawley | Hypertension, Portal - drug therapy | Animals | Signal Transduction - drug effects | Spleen - metabolism | Hypertension, Portal - metabolism | Fibrosis | Spleen - blood supply | Hypertension | Complications and side effects | Splenomegaly | Care and treatment | Patient outcomes | Development and progression | Rapamycin | Research | Liver cirrhosis | Risk factors | TOR protein | Animal models | Identification methods | Pathogenesis | Liver | Mitochondrial DNA | Activation | Kinases | Proteins | Splenectomy | Angiogenesis | Signal transduction | Lymphocytes | Rodents | Hepatology | Surgery | Tumor necrosis factor-TNF | Vascular endothelial growth factor | Enlargement | Spleen | Cytokines | Lymphoid tissue | Patients | Bile duct | Medicine | Cirrhosis | Signaling | Hospitals | Portal vein | Laboratory animals | Apoptosis
Journal Article
EMBO molecular medicine, ISSN 1757-4684, 2011, Volume 3, Issue 10, pp. 605 - 615
MicroRNAs (miRNAs) have been implicated in B cell lineage commitment, regulation of T cell differentiation, TCR signalling, regulation of IFN signalling, and... 
autoimmunity | miR‐21/PDCD4 | SLE | splenomegaly | TRANSFORMATION | MEDICINE, RESEARCH & EXPERIMENTAL | miR-21/PDCD4 | SIGNALING PROTEINS | TUMOR-SUPPRESSOR PDCD4 | PROGRAMMED CELL-DEATH-4 | PATHOGENESIS | GENETIC DISSECTION | THERAPEUTIC TARGETS | MIR-21 | EXPRESSION | T-CELLS | Transcription, Genetic - drug effects | Lupus Erythematosus, Systemic - complications | Gene Silencing - drug effects | Aging - drug effects | MicroRNAs - metabolism | Gene Expression Profiling | Splenomegaly - pathology | fas Receptor - metabolism | Lymphocyte Subsets - immunology | Autoimmune Diseases - genetics | Splenomegaly - complications | CD4-CD8 Ratio | Oligonucleotides - pharmacology | Lupus Erythematosus, Systemic - immunology | Lymphocytes - metabolism | Splenomegaly - genetics | Lymphocyte Subsets - drug effects | Mice, Inbred C57BL | Splenomegaly - immunology | Autoimmune Diseases - immunology | Aging - pathology | Apoptosis Regulatory Proteins - metabolism | Gene Expression Regulation - drug effects | B-Lymphocytes - drug effects | Animals | B-Lymphocytes - immunology | Lupus Erythematosus, Systemic - genetics | Lymphocytes - drug effects | Mice | MicroRNAs - genetics | RNA-Binding Proteins - metabolism | Glomerulonephritis | Splenomegaly | Flow cytometry | Pathogenesis | CD8 antigen | Population studies | Lymphocytes T | T-cell receptor | Cell growth | Systemic lupus erythematosus | Lymphocytes | miRNA | Age | Lupus | Autoantibodies | Statistical analysis | Cell lineage | T cell receptors | Cell differentiation | Gene expression | CD4 antigen | Studies | Lymphocytes B | MicroRNAs | Interferon | Kidney diseases | Autoimmune diseases | Apoptosis | PDCD4 | miR-21
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