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Alcoholism: Clinical and Experimental Research, ISSN 0145-6008, 10/2017, Volume 41, Issue 10, pp. 1715 - 1724
Background Heavy and chronic ethanol (EtOH) exposure can cause significant structural and functional damage to the adult brain. The most devastating... 
Mcl‐1 | Alternative Pre‐mRNA Splicing | Neurotoxicity | Serine/Arginine‐Rich Splicing Factor 1 | Alcoholism | Alternative Pre-mRNA Splicing | Mcl-1 | Serine/Arginine-Rich Splicing Factor 1 | APOPTOSIS | Serine | SPECTRUM DISORDERS | SUBSTANCE ABUSE | AUTOPHAGY | VIRUS GENE-EXPRESSION | ADULT-RAT BRAIN | ETHANOL | CELL-CULTURE | PROTEINS | RECEPTOR SUBUNIT | SPLICING FACTOR | Arginine-Rich Splicing Factor 1 | Cell Survival - drug effects | Ethanol - toxicity | RNA Precursors - biosynthesis | Humans | Cells, Cultured | Alternative Splicing - physiology | RNA Precursors - genetics | Myeloid Cell Leukemia Sequence 1 Protein - genetics | Dose-Response Relationship, Drug | Alternative Splicing - drug effects | Myeloid Cell Leukemia Sequence 1 Protein - biosynthesis | Neurons - physiology | Cell Line, Tumor | Neurons - drug effects | Cell Survival - physiology | Messenger RNA | Neurons | Analysis | Brain damage | Drinking of alcoholic beverages | Blood proteins | Alternative splicing | Drug abuse | Bcl-2 protein | Transcription | Alcohol | Immunoblotting | Proteins | Reporter gene | Arginine | Dependence | Consumption | Ethanol | Splicing | Exposure | Gene expression | Survival | Mcl-1 protein | Depletion | Structural damage | Drug dependence | Splicing factors | Brain injury | Apoptosis | Structure-function relationships | Index Medicus
Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 1590 - 15
Alternative splicing, a fundamental step in gene expression, is deregulated in many diseases. Splicing factors (SFs), which regulate this process, are up-or... 
APOPTOSIS | DECAY | NONSENSE | ONCOPROTEIN SRSF1 | MULTIDISCIPLINARY SCIENCES | SR PROTEINS | PROLIFERATION | TRACT BINDING-PROTEIN | REGULATOR RBFOX2 | RECOGNITION MOTIFS | CELL | Oligonucleotides - chemistry | Alternative Splicing | Humans | Serine-Arginine Splicing Factors - metabolism | Tandem Repeat Sequences | Serine-Arginine Splicing Factors - genetics | Zebrafish - embryology | Polypyrimidine Tract-Binding Protein - antagonists & inhibitors | RNA Splicing Factors - metabolism | MAP Kinase Signaling System - genetics | Glioblastoma - genetics | Oligonucleotides - pharmacology | HEK293 Cells | Binding Sites | Muscle, Skeletal - growth & development | Animals, Genetically Modified | Heterogeneous-Nuclear Ribonucleoproteins - metabolism | Heterogeneous-Nuclear Ribonucleoproteins - antagonists & inhibitors | Polypyrimidine Tract-Binding Protein - metabolism | RNA Splicing Factors - genetics | Zebrafish - genetics | Heterogeneous-Nuclear Ribonucleoproteins - genetics | Nonsense Mediated mRNA Decay | Serine-Arginine Splicing Factors - antagonists & inhibitors | Xenograft Model Antitumor Assays | Oligonucleotides - metabolism | Animals | Glioblastoma - pathology | RNA Splicing Factors - antagonists & inhibitors | Polypyrimidine Tract-Binding Protein - genetics | Alternative splicing | Deregulation | Splicing | Oligonucleotides | Ribonucleic acid--RNA | Gene expression | Splicing factors | Cancer
Journal Article
Nature (London), ISSN 1476-4687, 2011, Volume 478, Issue 7367, pp. 64 - 69
Journal Article
Journal Article
Genes & development, ISSN 0890-9369, 2013, Volume 27, Issue 2, pp. 129 - 144
Journal Article
Journal Article
Journal of Optical Communications, ISSN 0173-4911, 05/2017, Volume 38, Issue 2, pp. 223 - 229
Journal Article