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1. Full Text Genetics of ankylosing spondylitis
by Robinson, Philip C and Brown, Matthew A
Molecular Immunology, ISSN 0161-5890, 01/2014, Volume 57, Issue 1, pp. 2 - 11
Ankylosing spondylitis (AS) is a chronic inflammatory arthritis that affects the spine and sacroiliac joints. It causes significant disability and is... 
Single nucleotide polymorphism association | Spondyloarthritis | Ankylosing spondylitis | BIOCHEMISTRY & MOLECULAR BIOLOGY | SINGLE-NUCLEOTIDE POLYMORPHISM | IMMUNOLOGY | RETICULUM AMINOPEPTIDASE 1 | ACUTE ANTERIOR UVEITIS | PSORIASIS SUSCEPTIBILITY LOCI | HLA-B27 POLYMORPHISM | ENDOPLASMIC-RETICULUM | AXIAL SPONDYLOARTHRITIS | ER AMINOPEPTIDASE | GENOME-WIDE ASSOCIATION | DISEASE SUSCEPTIBILITY | Aminopeptidases - genetics | Genetic Predisposition to Disease | Major Histocompatibility Complex - genetics | Spondylitis, Ankylosing - genetics | Minor Histocompatibility Antigens | Humans | Psoriasis - complications | Interleukin-23 - genetics | Sacroiliac Joint - pathology | HLA-B27 Antigen - immunology | Uveitis, Anterior - complications | Inflammatory Bowel Diseases - complications | Spine - pathology | Spondylitis, Ankylosing - pathology | HLA-B27 Antigen - genetics | Genetic aspects | Psoriasis | Gastrointestinal diseases
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2. Full Text Genetics of ankylosing spondylitis
by Brown, Matthew A
Current Opinion in Rheumatology, ISSN 1040-8711, 03/2010, Volume 22, Issue 2, pp. 126 - 132
PURPOSE OF REVIEWOver the past 3 years, several new genes and gene deserts have been identified that are associated with ankylosing spondylitis (AS). The... 
Genetics | Major histocompatibility complex | KIR | IL23R | ERAP1 | POPULATION | NECROSIS-FACTOR RECEPTOR | CROHNS-DISEASE | VARIANTS | SUSCEPTIBILITY LOCI | major histocompatibility complex | RHEUMATOLOGY | HYPER-IGE SYNDROME | genetics | GENES | HLA-B27 POLYMORPHISM | GENOME-WIDE ASSOCIATION | Chromosomes, Human, Pair 2 - genetics | Genetic Predisposition to Disease - genetics | Genetic Association Studies | Major Histocompatibility Complex - genetics | Spondylitis, Ankylosing - genetics | Humans | Alleles | Chromosomes, Human, Pair 21 - genetics
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3. Full Text Genetics of ankylosing spondylitis - Insights into pathogenesis
by Brown, Matthew A and Kenna, Tony and Wordsworth, B. Paul
Nature Reviews Rheumatology, ISSN 1759-4790, 02/2016, Volume 12, Issue 2, pp. 81 - 91
Ankylosing spondylitis (AS), an immune-mediated arthritis, is the prototypic member of a group of conditions known as spondyloarthropathies that also includes... 
RHEUMATOID-ARTHRITIS | INFLAMMATORY-BOWEL-DISEASE | HL-A-ANTIGEN | CROHNS-DISEASE | VARIANTS | PSORIASIS SUSCEPTIBILITY LOCI | ERAP1 | RHEUMATOLOGY | RETICULUM AMINOPEPTIDASE 1 | HLA-B27 | GENOME-WIDE ASSOCIATION | Predictive Value of Tests | Genetic Predisposition to Disease | Spondylitis, Ankylosing - genetics | Interleukin-12 - genetics | Humans | Risk Factors | Biomarkers - blood | Evidence-Based Medicine | Interleukin-23 - genetics | Sensitivity and Specificity | Alleles | Polymorphism, Single Nucleotide - genetics | Spondylitis, Ankylosing - pathology | HLA-B27 Antigen - genetics | Phenotype | Development and progression | Genetic aspects | Gene expression | Identification and classification | Health aspects | Ankylosing spondylitis
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4. Full Text Global prevalence of ankylosing spondylitis
by Dean, Linda E and Jones, Gareth T and Macdonald, Alan G and Downham, Christina and Sturrock, Roger D and Macfarlane, Gary J
Rheumatology (United Kingdom), ISSN 1462-0324, 04/2014, Volume 53, Issue 4, pp. 650 - 657
Methods. A systematic literature search was conducted. Prevalence data were extracted and used to calculate the mean prevalence by continent and the expected... 
Spondyloarthropathies | Systematic review | Prevalence | Epidemiology | Ankylosing spondylitis | MUSCULOSKELETAL PAIN | URBAN | ankylosing spondylitis | RHEUMATOLOGY | systematic review | ARTHRITIS | prevalence | epidemiology | BACK-PAIN | NORTHERN NORWAY | WHO-ILAR COPCORD | RURAL POPULATIONS | RHEUMATIC-DISEASES | AXIAL SPONDYLOARTHRITIS | spondyloarthropathies | Latin America - epidemiology | Africa - epidemiology | Asia - epidemiology | Global Health | Spondylitis, Ankylosing - genetics | Humans | Spondylitis, Ankylosing - epidemiology | Europe - epidemiology | North America - epidemiology | HLA-B27 Antigen - genetics
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5. Full Text Genome-wide association study of ankylosing spondylitis identifies non-MHC susceptibility loci
by Reveille, John D and Sims, Anne-Marie and Danoy, Patrick and Evans, David M and Leo, Paul and Pointon, Jennifer J and Jin, Rui and Zhou, Xiaodong and Bradbury, Linda A and Appleton, Louise H and Davis, John C and Diekman, Laura and Doan, Tracey and Dowling, Alison and Duan, Ran and Duncan, Emma L and Farrar, Claire and Hadler, Johanna and Harvey, David and Karaderi, Tugce and Mogg, Rebecca and Pomeroy, Emma and Pryce, Karena and Taylor, Jacqueline and Savage, Laurie and Deloukas, Panos and Kumanduri, Vasudev and Peltonen, Leena and Ring, Sue M and Whittaker, Pamela and Glazov, Evgeny and Thomas, Gethin P and Maksymowych, Walter P and Inman, Robert D and Ward, Michael M and Stone, Millicent A and Weisman, Michael H and Wordsworth, B Paul and Brown, Matthew A and Australo-Anglo-Amer Spondyloarthri and Australo-Anglo-American Spondyloarthritis Consortium (TASC) and The Australo-Anglo-American Spondyloarthritis Consortium (TASC)
Nature Genetics, ISSN 1061-4036, 02/2010, Volume 42, Issue 2, pp. 123 - 127
To identify susceptibility loci for ankylosing spondylitis, we undertook a genome-wide association study in 2,053 unrelated ankylosing spondylitis cases among... 
POPULATION | INFLAMMATORY-BOWEL-DISEASE | VARIANTS | GENETICS & HEREDITY | GENETIC-SUSCEPTIBILITY | RISK | PHENOTYPES | TNF | Genetic Predisposition to Disease | Genome-Wide Association Study | Reproducibility of Results | Major Histocompatibility Complex - genetics | Spondylitis, Ankylosing - genetics | Humans | Genetic Loci - genetics | Polymorphism, Single Nucleotide - genetics | Cohort Studies | Medical research | Inflation | Biomedical research | Disease | Colleges & universities | Genetics | Grants | Regression analysis | Gene expression | Chromosomes
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6. Full Text Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci
by Cortes, Adrian and Hadler, Johanna and Pointon, Jenny P and Robinson, Philip C and Karaderi, Tugce and Leo, Paul and Cremin, Katie and Pryce, Karena and Harris, Jessica and Lee, Seunghun and Joo, Kyung Bin and Shim, Seung-Cheol and Weisman, Michael and Ward, Michael and Zhou, Xiaodong and Garchon, Henri-Jean and Chiocchia, Gilles and Nossent, Johannes and Lie, Benedicte A and Førre, Øystein and Tuomilehto, Jaakko and Laiho, Kari and Jiang, Lei and Liu, Yu and Wu, Xin and Bradbury, Linda A and Elewaut, Dirk and Burgos-Vargas, Ruben and Stebbings, Simon and Appleton, Louise and Farrah, Claire and Lau, Jonathan and Kenna, Tony J and Haroon, Nigil and Ferreira, Manuel A and Yang, Jian and Mulero, Juan and Fernandez-Sueiro, Jose Luis and Gonzalez-Gay, Miguel A and Lopez-Larrea, Carlos and Deloukas, Panos and Donnelly, Peter and Bowness, Paul and Gafney, Karl and Gaston, Hill and Gladman, Dafna D and Rahman, Proton and Maksymowych, Walter P and Xu, Huji and Crusius, J Bart A and Van Der Horst-Bruinsma, Irene E and Chou, Chung-Tei and Valle-Oñate, Raphael and Romero-Sánchez, Consuelo and Hansen, Inger Myrnes and Pimentel-Santos, Fernando M and Inman, Robert D and Videm, Vibeke and Martin, Javier and Breban, Maxime and Reveille, John D and Evans, David M and Kim, Tae-Hwan and Wordsworth, Bryan Paul and Brown, Matthew A and Australo-Anglo-Amer and GFEGS and Spondyloarthrit Res Consortium and Int Genetics Ankylosing and WTCCC2 and Nord-Trondelag Hlth Study HUNT and Groupe Française d'Etude Génétique des Spondylarthrites (GFEGS) and Nord-Trøndelag Health Study (HUNT) and Spondyloarthritis Research Consortium of Canada (SPARCC) and Australo-Anglo-American Spondyloarthritis Consortium (TASC) and Wellcome Trust Case Control Consortium 2 (WTCCC2) and International Genetics of Ankylosing Spondylitis Consortium (IGAS)
Nature Genetics, ISSN 1061-4036, 07/2013, Volume 45, Issue 7, pp. 730 - 738
Ankylosing spondylitis is a common, highly heritable inflammatory arthritis affecting primarily the spine and pelvis. In addition to HLA-B*27 alleles, 12 loci... 
GENETIC SUSCEPTIBILITY | ACTIVATION | HLA CLASS-I | ERAP1 | GENETICS & HEREDITY | COMMON | ENDOPLASMIC-RETICULUM | T-CELL | BEHCETS-DISEASE | DISEASE SUSCEPTIBILITY LOCI | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease - genetics | Spondylitis, Ankylosing - genetics | Humans | Immune System Phenomena - genetics | Risk Factors | Genotype | DNA Mutational Analysis - methods | Case-Control Studies | Genetic Loci - immunology | Spondylitis, Ankylosing - immunology | Alleles | Genetic Predisposition to Disease - ethnology | High-Throughput Nucleotide Sequencing | Polymorphism, Single Nucleotide | Spondylitis, Ankylosing - ethnology | Genome-Wide Association Study - methods | Genotyping Techniques - methods | HLA-B27 Antigen - genetics | Care and treatment | Histocompatibility antigens | Aminopeptidases | HLA histocompatibility antigens | Physiological aspects | Genetic aspects | Research | Health aspects | Ankylosing spondylitis | Studies | Medical research | Arthritis | Autoimmune diseases | Pharmaceutical industry | Market shares
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7. Full Text Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility
by Evans, David M and Spencer, Chris C. A and Pointon, Jennifer J and Su, Zhan and Harvey, David and Kochan, Grazyna and Opperman, Udo and Dilthey, Alexander and Pirinen, Matti and Stone, Millicent A and Appleton, Louise and Moutsianis, Loukas and Leslie, Stephen and Wordsworth, Tom and Kenna, Tony J and Karaderi, Tugce and Thomas, Gethin P and Ward, Michael M and Weisman, Michael H and Farrar, Claire and Bradbury, Linda A and Danoy, Patrick and Inman, Robert D and Maksymowych, Walter and Gladman, Dafna and Rahman, Proton and Morgan, Ann and Marzo-Ortega, Helena and Bowness, Paul and Gaffney, Karl and Gaston, J. S. Hill and Smith, Malcolm and Bruges-Armas, Jacome and Couto, Ana-Rita and Sorrentino, Rosa and Paladini, Fabiana and Ferreira, Manuel A and Xu, Huji and Liu, Yu and Jiang, Lei and Lopez-Larrea, Carlos and Díaz-Peña, Roberto and Lóepez-Vázquez, Antonio and Zayats, Tetyana and Band, Gavin and Bellenguez, Céline and Blackburn, Hannah and Blackwell, Jenefer M and Bramon, Elvira and Bumpstead, Suzannah J and Casas, Juan P and Corvin, Aiden and Craddock, Nicholas and Deloukas, Panos and Dronov, Serge and Duncanson, Audrey and Edkins, Sarah and Freeman, Colin and Gillman, Matthew and Gray, Emma and Gwilliam, Rhian and Hammond, Naomi and Hunt, Sarah E and Jankowski, Janusz and Jayakumar, Alagurevathi and Langford, Cordelia and Liddle, Jennifer and Markus, Hugh S and Mathew, Christopher G and McCann, Owen T and McCarthy, Mark I and Palmer, Colin N. A and Peltonen, Leena and Plomin, Robert and Potter, Simon C and Rautanen, Anna and Ravindrarajah, Radhi and Ricketts, Michelle and Samani, Nilesh and Sawcer, Stephen J and Strange, Amy and Trembath, Richard C and Viswanathan, Ananth C and Waller, Matthew and Weston, Paul and Whittaker, Pamela and Widaa, Sara and Wood, Nicholas W and McVean, Gilean and Reveille, John D and Wordsworth, B. Paul and Brown, Matthew A and Donnelly, Peter and Australo-Anglo-Amer Spondyloarthri and Wellcome Trust Case Control Consor and Spondyloarthrit Res Consortium Can and Wellcome Trust Case Control Consortium 2 (WTCCC2) and Spondyloarthritis Research Consortium of Canada (SPARCC) and Australo-Anglo-American Spondyloarthritis Consortium (TASC) and The Australo-Anglo-American Spondyloarthritis Consortium (TASC) and the Wellcome Trust Case Control Consortium 2 (WTCCC2)
Nature Genetics, ISSN 1061-4036, 08/2011, Volume 43, Issue 8, pp. 761 - 767
Ankylosing spondylitis is a common form of inflammatory arthritis predominantly affecting the spine and pelvis that occurs in approximately 5 out of 1,000... 
POPULATION | CELLS | INFLAMMATORY-BOWEL-DISEASE | GENETIC ASSOCIATION | GENETICS & HEREDITY | RISK | TRIMS PRECURSORS | AMINOPEPTIDASE | HLA | PROMOTES | GENOME-WIDE ASSOCIATION | Receptors, Prostaglandin E, EP4 Subtype - genetics | Interleukin-12 Subunit p40 - genetics | Aminopeptidases - genetics | Genome-Wide Association Study | Meta-Analysis as Topic | Peptide Fragments - metabolism | Disease Susceptibility | Spondylitis, Ankylosing - genetics | Membrane Proteins - genetics | Minor Histocompatibility Antigens | Humans | European Continental Ancestry Group | Receptors, Tumor Necrosis Factor, Type I - genetics | Spondylitis, Ankylosing - metabolism | Case-Control Studies | CARD Signaling Adaptor Proteins - genetics | Latent TGF-beta Binding Proteins - genetics | Receptors, Peptide | CD8-Positive T-Lymphocytes - metabolism | Core Binding Factor Alpha 3 Subunit - genetics | Polymorphism, Genetic - genetics | Aminopeptidases - metabolism | HLA-B27 Antigen - genetics | Histocompatibility antigens | HLA histocompatibility antigens | Physiological aspects | Disease susceptibility | Genetic aspects | Research | Single nucleotide polymorphisms | Ankylosing spondylitis | Risk factors | Inflammatory bowel disease | Medical research | Biomedical research | Lymphocytes | Immunotherapy | Quality control | Genomes | Gene expression | Deoxyribonucleic acid--DNA
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8. Full Text Genetics of ankylosing spondylitis
by Castro-Santos, P and Gutierrez, MA and Diaz-Pena, R
REVISTA MEDICA DE CHILE, ISSN 0034-9887, 09/2014, Volume 142, Issue 9, pp. 1165 - 1173
Ankylosing spondylitis (AS) is a prototypical inflammatory disease of the.. locomotor system affecting axial skeleton. It is part of the general group of... 
ankylosing | HLA-B27 HOMODIMERS | SUBTYPES | CHLAMYDIA-TRACHOMATIS | SUSCEPTIBILITY | RECEPTOR | IDENTIFICATION | RETICULUM AMINOPEPTIDASE 1 | Spondylitis | MEDICINE, GENERAL & INTERNAL | Genetic predisposition to disease | ENDOPLASMIC-RETICULUM | HLA-B27 antigen | ASSOCIATION | PRESENTED PEPTIDES | Female | Male | Genetic Predisposition to Disease | Spondylitis, Ankylosing - genetics | Humans | HLA-B27 Antigen - genetics
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9. Full Text Pathogenesis of ankylosing spondylitis-recent advances and future directions
by Ranganathan, Vidya and Gracey, Eric and Brown, Matthew A and Inman, Robert D and Haroon, Nigil
Nature Reviews Rheumatology, ISSN 1759-4790, 06/2017, Volume 13, Issue 6, pp. 359 - 367
Over the past 5 years, advances in high-throughput techniques and studies involving large cohorts of patients have led to considerable advances in the... 
UNFOLDED PROTEIN RESPONSE | FUNCTIONAL INTERACTION | T(H)17 CELLS | INFLAMMATORY DISEASE | PERIPHERAL-BLOOD | TRIMS PRECURSORS | RHEUMATOLOGY | T-CELLS | GENOME-WIDE ASSOCIATION | PROOF-OF-CONCEPT | TH17 CELLS | Spondylitis, Ankylosing - immunology | Spondylitis, Ankylosing - etiology | Spondylitis, Ankylosing - genetics | Humans | Antigen Presentation - physiology | Interleukin-17 - immunology | Development and progression | Genotype | Care and treatment | Genetic aspects | Health aspects | Ankylosing spondylitis | Antigen presentation | Spondylitis | Signal transduction | Transcription factors | Immune response | Pathogenesis | Cytokine receptors | Histocompatibility antigen HLA
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