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Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 06/2002, Volume 195, Issue 11, pp. 1433 - 1444
Interleukin (IL)-5 and IL-13 are thought to play key roles in the pathogenesis of asthma. Although both cytokines use eotaxin to regulate eosinophilia, IL-13... 
Allergy | Inflammation | Cytokines | Lung | Eosinophils | MEDICINE, RESEARCH & EXPERIMENTAL | TISSUE EOSINOPHILIA | BRONCHIAL BIOPSIES | MESSENGER-RNA EXPRESSION | BONE-MARROW | allergy | eosinophils | IMMUNOLOGY | BRONCHOALVEOLAR LAVAGE | lung | LUNG DAMAGE | MURINE MODEL | inflammation | ALLERGEN INHALATION CHALLENGE | cytokines | GUINEA-PIG MODEL | ATOPIC ASTHMA | Eosinophilia - metabolism | Asthma - metabolism | Eosinophils - metabolism | Eosinophils - transplantation | Bronchial Hyperreactivity - immunology | Chemokines, CC - blood | Humans | Th2 Cells - immunology | Adoptive Transfer | Interleukin-5 - genetics | RNA, Messenger - metabolism | CD4-Positive T-Lymphocytes - immunology | Eosinophilia - pathology | Interleukin-13 - biosynthesis | Gene Deletion | Asthma - immunology | Bronchial Hyperreactivity - metabolism | Disease Models, Animal | Signal Transduction | Interleukin-5 - metabolism | RNA, Messenger - genetics | CD4-Positive T-Lymphocytes - metabolism | Chemokine CCL11 | Interleukin-13 - metabolism | Bronchial Hyperreactivity - complications | Th2 Cells - metabolism | Interleukin-5 - blood | Sputum - metabolism | Bronchial Hyperreactivity - pathology | Animals | Eosinophilia - immunology | Asthma - complications | Chemokines, CC - genetics | Eosinophilia - complications | Mice | Mice, Inbred BALB C | Interleukin-18 - metabolism | Chemokines, CC - metabolism | Index Medicus
Journal Article
Thorax, ISSN 0040-6376, 12/2017, Volume 72, Issue 12, pp. 1074 - 1083
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2016, Volume 113, Issue 31, pp. 8765 - 8770
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 06/2013, Volume 48, Issue 6, pp. 694 - 702
Up-regulation of arginase contributes to airways hyperresponsiveness (AHR) in asthma by reducing L-arginine bioavailability for the nitric oxide (NO) synthase... 
Arginase | α- difluoromethylornithine | Spermine | Polyamines | Airways hyperresponsiveness | CARCINOMA CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | spermine | GUINEA-PIG | arginase | airways hyperresponsiveness | CELL BIOLOGY | INCREASED ARGINASE ACTIVITY | SMOOTH-MUSCLE | INHIBITION | METABOLISM | RESPIRATORY SYSTEM | INFLAMMATION | alpha-difluoromethylornithine | ALLERGIC-ASTHMA | CYSTIC-FIBROSIS | ENDOGENOUS NITRIC-OXIDE | polyamines | Inflammation - pathology | Asthma - metabolism | Nitric Oxide Synthase - immunology | Ornithine Decarboxylase - metabolism | Ovalbumin - immunology | Spermine - adverse effects | Humans | Middle Aged | Polyamines - antagonists & inhibitors | Hypersensitivity - immunology | Methacholine Chloride - metabolism | Eflornithine - pharmacology | Male | Hypersensitivity - drug therapy | Young Adult | Inflammation - metabolism | Hypersensitivity - metabolism | Methacholine Chloride - pharmacology | Ovalbumin - adverse effects | Asthma - immunology | Adult | Female | Lung - metabolism | Disease Models, Animal | Lung - pathology | Spermine - administration & dosage | Ornithine - metabolism | Inflammation - immunology | Asthma - drug therapy | Sputum - metabolism | Polyamines - metabolism | Nitrates - metabolism | Ornithine Decarboxylase Inhibitors | Animals | Spermine - pharmacology | Hypersensitivity - pathology | Lung - drug effects | Adolescent | Mice | Nitric Oxide Synthase - metabolism | Nitric Oxide - metabolism | Asthma - pathology | Lung - immunology | Index Medicus
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 09/2011, Volume 184, Issue 6, pp. 662 - 671
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 05/2015, Volume 52, Issue 5, pp. 554 - 562
Cigarette smoking, the major causative factor for the development of chronic obstructive pulmonary disease, is associated with neutrophilic airway... 
Chronic obstructive pulmonary disease | Inflammation | Damage-associated molecular patterns | Cigarette smoke | Neutrophils | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | MITOCHONDRIA | cigarette smoke | CELL BIOLOGY | RESPONSES | NECROSIS | damage-associated molecular patterns | EPITHELIAL-CELLS | inflammation | RESPIRATORY SYSTEM | APOPTOTIC CELLS | LUNG-DISEASE | MICE | neutrophils | chronic obstructive pulmonary disease | COPD | Smoking - adverse effects | Smoking - immunology | Humans | Pneumonia - pathology | Bronchoalveolar Lavage Fluid - immunology | Necrosis | Sputum - immunology | Time Factors | HMGB1 Protein - metabolism | Respiratory Mucosa - immunology | Inflammation Mediators - metabolism | Membrane Potential, Mitochondrial | Inhalation Exposure - adverse effects | Pneumonia - immunology | Female | Lung - metabolism | Interleukin-8 - metabolism | Neutrophils - metabolism | Inflammation Mediators - immunology | Lung - pathology | Pneumonia - etiology | Signal Transduction | Smoke - adverse effects | Mice, Inbred C57BL | Cells, Cultured | Neutrophils - immunology | Immunity, Innate | Sputum - metabolism | Cross-Over Studies | Smoking - metabolism | Phenotype | Animals | Respiratory Mucosa - metabolism | Pneumonia - metabolism | Lung - immunology | Apoptosis | Peroxidase - metabolism | Mitochondrial DNA | Kinases | Immune system | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e87400 - e87400
We have previously reported that the lungs of patients with very severe chronic obstructive pulmonary disease (COPD) contain significantly higher numbers of... 
SPUTUM | SMOKERS | MULTIDISCIPLINARY SCIENCES | PHENOTYPE | MONOCYTES | RECEPTOR | SMOKING | ALTERNATIVE ACTIVATION | T-CELLS | COPD | Mannose-Binding Lectins - metabolism | Macrophages, Alveolar - pathology | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Lectins, C-Type - genetics | Antigens, CD - genetics | Antigens, CD - metabolism | Lectins, C-Type - metabolism | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 9 - genetics | Pulmonary Alveoli - metabolism | Female | Scavenger Receptors, Class A - genetics | Pulmonary Disease, Chronic Obstructive - metabolism | Pulmonary Disease, Chronic Obstructive - genetics | Severity of Illness Index | Lung - pathology | Pulmonary Alveoli - pathology | Receptors, Cell Surface - metabolism | Smoking - genetics | Antigens, Differentiation, Myelomonocytic - genetics | Smoking - metabolism | Mannose-Binding Lectins - genetics | Antigens, Differentiation, Myelomonocytic - metabolism | Smoking - pathology | Aged | Macrophages, Alveolar - metabolism | Scavenger Receptors, Class A - metabolism | Receptors, Cell Surface - genetics | Development and progression | Lung diseases, Obstructive | Smokers | Macrophages | Analysis | Pathogenesis | Lung | Genotype & phenotype | Tumor necrosis factor-TNF | Chronic obstructive pulmonary disease | Alveoli | Gold | Antigens | Cytokines | Lung diseases | Neutrophils | Rheumatology | Inflammation | Patients | Medicine | CD163 antigen | Pathology | Neurology | Hospitals | Lungs | Obstructive lung disease | Cancer | Smoking | Tumors | Index Medicus
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 2012, Volume 47, Issue 6, pp. 738 - 745
Journal Article